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1 nal cortisol infusion (1.2 mg kg(-1) day(-1) i.v. for 5 days, n = 20) on fetal glucose, lactate and o
2 ty was rejected as CL316,243 (0.15 mg kg(-1) i.v.) evoked similar rises in oxygen consumption (VO2) i
3 rial lipopolysaccharide (LPS, 500 mug kg(-1) i.v.) in rats at an ambient temperature of 22 degrees C.
4 hase 2 clinical trials of i.v. hu14.18-IL-2 (i.v.-IC) in neuroblastoma and melanoma are underway and
5         The aim of the AIDA STEMI (Abciximab i.v. Versus i.c. in ST-elevation Myocardial Infarction)
6 lectrolyte homeostasis in response to acute [i.v. volume expansion (VE)] or chronic stressful stimuli
7 re likely to personally offer and administer i.v. sedation services for their patients.
8 ium-based contrast agent can be administered i.v. to the animal to detect mural inflammation or tumor
9 .c. HT-1080 tumors in nude mice administered i.v. docetaxel-containing nanoparticles was more effecti
10   However, the majority of MSCs administered i.v. encounter a pulmonary impasse and soon disappear fr
11  at a dose of 9.0 x 10(5) PfSPZ administered i.v. three times at 8-wk intervals to 15 malaria-naive a
12                  Paclitaxel was administered i.v. at 50 mg/m and i.p. at 20 mg/m on days 1 and 8.
13                  Paclitaxel was administered i.v. at 50 mg/m and i.p. at 20 mg/m on days 1 and 8.
14                        ConA was administered i.v. to control or HSC-depleted mice; hepatic histopatho
15 h a mean diameter of 83 nm were administered i.v. in hamsters.
16 s were experimentally seeded via an adoptive i.v. transfer of luciferase-expressing CT26 CRC cells th
17                                        After i.v. administration in C57BL/6 mice, we observed marked
18                                        After i.v. or s.c. priming, partial proliferation and activati
19 nism, but the antinociceptive activity after i.v. administration could not be directly correlated to
20 ycosylation was essential for activity after i.v. administration.
21 s computed tomography (CT), before and after i.v. contrast media enhancement.
22 acquisitions were performed before and after i.v. or intrapulmonary administration of the nanoparticl
23 on, the amount of accumulated antibody after i.v. application was calculated relative to its apoptosi
24 um concentration obtained in the blood after i.v. administration correlates with body weight across a
25 layed glucose clearance from the blood after i.v. loading, and significantly decreased glucose uptake
26   The mean t1/2 of KRN23 was 8-12 days after i.v. administration and 13-19 days after s.c. administra
27 late in the wound margins several days after i.v. administration.
28 1, ELDP and CT-proET-1 were determined after i.v. injection in anaesthetised rats.
29 anti-ICAM/NCs by pulmonary endothelium after i.v. injection in mice, similar to that of flow-adapted
30  cell line showed transgene expression after i.v. CPS injection in an animal model of metastasis.
31  levels of effector cytokines in hosts after i.v. AML induction, consistent with abortive T cell acti
32  4.7 T before, during and up to 60 min after i.v. injection of EP-3533, or of its non-binding isomer
33 ssue rapidly and homogeneously; 30 min after i.v. injection, siRNA-L2 achieved uptake in 99% of tumor
34  Bacteria were captured within minutes after i.v. injection and were associated with Mphis in both li
35 MRI and optical imaging were performed after i.v. injection of the liposomal nanoprobes into mice bea
36 s spectrometry (ICP-MS), respectively, after i.v. injection of the payload loaded HA NPs in tumor bea
37 nal centers (GCs), and the Ab response after i.v. administration of IgG3 anti-trinitrophenyl (TNP) in
38 illary endothelial cells in the retina after i.v. application.
39 e antigen to erythrocyte cell surfaces after i.v. injection, one using a conjugate with an erythrocyt
40 nized mice were completely protected against i.v. Stx2 challenge, and weaned mice receiving an oral c
41 netics and immune responses induced after an i.v. infection with a Brazilian ZIKV clinical isolate (H
42 hol cue-exposure session (day 6; AM), and an i.v. alcohol administration session (day 6; PM).
43 n the legs of mice followed 3 wk later by an i.v. exposure to Staphylococcus aureus This procedure re
44 n and puncture, followed 6 hours later by an i.v. injection of either MSCs or saline.
45     The dendribodies were administered in an i.v. bolus to male Sprague Dawley rats after starting a
46  time each active lever press resulted in an i.v. cocaine infusion paired with one of two cues that a
47 ich time active lever presses resulted in an i.v. infusion of cocaine that was paired with a light/to
48 unized and five control subjects received an i.v. challenge with P. falciparum-infected erythrocytes.
49 w paradigm in the treatment of PAH, using an i.v./sublingual tissue-penetrating homing peptide to sel
50 ective role, we studied both aerosolized and i.v. models of mycobacterial infection and observed MIF-
51 everal volatile (isoflurane, desflurane) and i.v. (propofol) general anesthetics excite peripheral se
52 of immune suppression to more frequently and i.v. administered licensed therapies.
53 ortant finding is that both intratumoral and i.v. administration demonstrated a significant enhanceme
54 mmed death ligand 1 binding site just before i.v. injection of IgM/LPS-pretreated BMDC or using IL-10
55 cer cell lines with anti-CD73 AD2 mAb before i.v. injection into mice inhibited extravasation/coloniz
56                                         Both i.v. hexamethonium and locally applied prazosin abolishe
57 erum, and up to 70% were protected from both i.v. and mosquito bite challenge with transgenic Plasmod
58 e, results in significant protection in both i.v. and airway-induced models of IA.
59 ntation of in vivo-formed Ag-Ab complexes by i.v. injecting mice with Ag-specific Abs followed by the
60 llers with low viremia) were CD8 depleted by i.v. administration of the antibody M-T807R1.
61  the common marmoset (Callithrix jacchus) by i.v., subcutaneous (s.c.), and intranasal exposure route
62             Retinal vessels were labelled by i.v. injection of a fluorescent dye and imaged with scan
63  levels of corticosterone were normalized by i.v. leptin infusion at a dose that raises low plasma le
64  The second hypoxic exposure was preceded by i.v. infusion of iron.
65 mportantly, this loss is swiftly restored by i.v. administration of human recombinant ANXA1.
66  exposure was evaluated in a monkey study by i.v. dosing a mixture of the SIL and the unlabeled drugs
67 us aureus implant infections were treated by i.v. administration of activated or non-activated MSC, w
68 NV infection, we administered exogenous CCL7 i.v. to WNV-infected Ccl7(-/-) mice and observed a signi
69                            After challenging i.v. with 1.5 x 10(4) cell/g, 60% of control C57BL/6 mic
70                   In this study, we compared i.v. and foodborne transmission of L. monocytogenes in m
71 fter the SIL drug (100 mug) was concurrently i.v. dosed to humans, at T(max) of an oral therapeutic d
72 ced septic acute kidney injury by continuous i.v. infusion of Escherichia coli.
73   Macrophage depletion was achieved by daily i.v. clodronate liposomes (-1 day to +3 days) during Ang
74 t up to 0.20% to 0.97% of antibody delivered i.v. reached the brain tumor, but that apoptosis inducti
75 ffected lung tumor growth in three different i.v. metastasis models.
76                                    High-dose i.v. Ig (IVIg) is a prominent immunomodulatory therapy f
77                                    High-dose i.v. Ig (IVIG) is used to treat various autoimmune and i
78  the anti-inflammatory effects of large-dose i.v. Ig (IVIg) in autoimmunity.
79 MyTrCa(-/-)) mice that succumbed to low-dose i.v. VSV infection with similar kinetics as IFN-I recept
80                            In the three-dose i.v. arm, 246/333 (74%) children had >/= 99% reduction i
81 ared both a three-dose i.m. and a three-dose i.v. parenteral ARS regimen with the standard five-dose
82  = 331; three-dose i.m.: n = 338; three-dose i.v.: n = 333); 139 participants were lost to follow-up.
83                     Adoptive transfer (i.e., i.v. injection) of glycolipid (Ag)-loaded WT but not S1P
84  with ischemic AKI, administration of ECFCs (i.v.) at the time of reperfusion significantly attenuate
85           Garcinol administered after either i.v. or i.p. cocaine reactivation significantly decrease
86 rug memory was later reactivated with either i.v. or i.p. cocaine presentation in the absence of any
87 subjects underwent experimental endotoxemia (i.v. administration of 2 ng/kg Escherichia coli endotoxi
88 e HPAIV background were lethal and exhibited i.v. pathogenicity indices of 2.79, 2.37, 2.85, and 2.61
89                  Administration of exosomes (i.v.) directly to mice with ischemic AKI attenuated rena
90                                     In fact, i.v. AML cell inoculation prevented functional T cell ac
91                                    Following i.v. peptide injection, in vivo Helios expression in ado
92 ylation (FosPEG 2% and FosPEG 8%), following i.v. administration to normal and tumour bearing rats.
93 y superior compared with free drug following i.v. delivery, exploiting the "enhanced permeability and
94 ns, and promoted lung tumor growth following i.v. injection of MADB106 tumor cells.
95 d to 1.7% of injected dose at 24 h following i.v. delivery.
96  a fluorescent dye, illuminated MB following i.v. injection and was able to distribute throughout the
97 t to the spleen, were all observed following i.v. but not foodborne transmission of L. monocytogenes.
98 ntrauterine infection was observed following i.v. rhCMV inoculation during the early second trimester
99  half-life was 46.1 h in mini-pigs following i.v. administration, and semaglutide has an MRT of 63.6
100 ecific regulatory function in vivo following i.v. injection of non-T cells derived from the spleen of
101 , and significant triglyceride reduction for i.v.-injected TPP-HDL-apoA-I-QD NPs in rats.
102 available in vivo gene delivery formulation, i.v. delivery of the core/PEGylated shell (CPS) nanopart
103 lated with their in vivo Ag acquisition from i.v. injected Ag-loaded splenic non-T cells, and in vivo
104 ly protected mice with humanized livers from i.v.- and mosquito bite-delivered P. falciparum sporozoi
105 -/-) mice had impaired response to CpG given i.v. or s.c.
106 n with Abraxane(R) (12mg paclitaxel/kg given i.v.), ACT induced a strong increase in the therapeutic
107 when submitted to hyperglycemic (40% glucose i.v.) and hypoglycemic (5 U/kg insulin i.v.) challenges.
108 (10 and 20 mg/kg, 10 and 20 mg kg/h for 1 h, i.v.), pazopanib (30 and100 mg/kg, i.p.), or vandetanib
109 nib (3 and 6 mg/kg, 3 and 6 mg/kg/h for 1 h, i.v.), sorafenib (10 and 20 mg/kg, 10 and 20 mg kg/h for
110 f blast trauma with multiorgan hemorrhaging, i.v. administration of the hemostatic nanoparticles led
111             In subordinate monkeys, however, i.v. buspirone was ineffective on average, and oral busp
112 e in the anti-inflammatory activity of human i.v. immunoglobulin therapy.
113                                      Indeed, i.v. administration of DCs differentiated in the presenc
114 n the VMS and DLS of rats bearing indwelling i.v. catheters over the course of 3 wk of cocaine self-a
115     Splenocytes from wild-type mice infected i.v. produced significantly more IFN-beta than did those
116 CR7) in mice that were subsequently infected i.v. with Listeria monocytogenes.
117                           Mice were infected i.v. using 8 different S. aureus strains, and developmen
118 ter 5 days of recovery, mothers were infused i.v. for 30 min with either vehicle (n = 11), low dose (
119                  Animals received an initial i.v. bolus of placebo, natalizumab (30 mg/kg), or vedoli
120 y charged microdroplet clusters are injected i.v., activated within the target pathology by diagnosti
121 on biased data collected with cells injected i.v., a route in which most transferred cells enter via
122               The radiotracers were injected i.v. into antigen-positive, antigen-negative, immunodefi
123        When breast tumor cells were injected i.v. into IL-15(-/-), C57BL/6, IL-15 transgenic (TG) and
124 s with OSA or control subjects were injected i.v. into mice.
125 e radiolabelled imaging probes were injected i.v. into wild-type, Sglt1(-/-) , Sglt2(-/-) and Glut2(-
126 scapular and suprarenal region were injected i.v. with 1 mg of TF12 and, after 16 hours of tumor accu
127                                When injected i.v. in mice bearing CT26 colon carcinoma or B16 melanom
128                   Furthermore, when injected i.v., these HSCs migrated to BM, self-replicated, provid
129 tered methamphetamine (0.03 mg/kg/injection, i.v.) under a fixed-ratio 20 schedule of reinforcement (
130  cell responses against AML cells inoculated i.v. versus s.c.
131 ucose i.v.) and hypoglycemic (5 U/kg insulin i.v.) challenges.
132 himeric H77/JFH1 virus (HJ3-5), intravenous (i.v.) challenge with 10(6) FFU H77S.2 virus resulted in
133  body localization of NPs after intravenous (i.v.) injection into live mice bearing human lung tumors
134 (NHP) model for RVF utilizes an intravenous (i.v.) exposure route in rhesus macaques (Macaca mulatta)
135 n CSF following intrathecal and intravenous (i.v.) administration of ziconotide was investigated.
136 transmission efficiency between intravenous (i.v.) and i.c. routes to estimate dose-dependent TTvCJD
137 bioavailability with concurrent intravenous (i.v.) microdosing a stable isotopically labeled (SIL) dr
138 WHO-recommended regimen of five intravenous (i.v.) or i.m. injections over 4 d.
139 ry parent formulation following intravenous (i.v.) delivery.
140 ent (STING(-/-)) mice following intravenous (i.v.) infection.
141 ycoside moiety was required for intravenous (i.v.) but not for intracerebroventricular (i.c.v.) activ
142 n of an SIL analogue for use in intravenous (i.v.) microdosing for the determination of absolute bioa
143 al efficacy and tolerability of intravenous (i.v.) and intraperitoneal (i.p.) paclitaxel combined wit
144 a progressive-ratio schedule of intravenous (i.v.) drug injection.
145 igate trends in the training of intravenous (i.v.) sedation in residency and its use in periodontal p
146 d investigate the efficiency of intravenous (i.v.), intraperitoneal (i.p.), and intramuscular (i.m.)
147 1:1 to treatment with serelaxin intravenous (i.v.) infusion (for 60 min at 80 mug/kg/d and then 60 mi
148 ion (MCAO) model after a single intravenous (i.v.) injection.
149 es following inoculation by the intravenous (i.v.) or intrarectal (i.r.) route.
150 ied Carba NP test that utilized intravenous (i.v.) imipenem-cilastatin, which is less expensive than
151 ing elevated drug levels versus intravenous (i.v.) injection.
152 cked C. albicans mutant given intravenously (i.v.) and S. aureus given intraperitoneally (i.p.) faile
153 hesus macaques (RMs) infected intravenously (i.v.) with SIVmac239.
154  into two groups and injected intravenously (i.v.) 6 hours post-MCAO with either 1 mg/kg PNU-120596 (
155              The animals were intravenously (i.v.) administered PGT121 or 3BNC117 at 10 and 2 mg/kg o
156 a clinically desirable therapy would involve i.v. infusion of a recombinant enzyme.
157  Current treatment of these attacks involves i.v. administration of hemin, but a faster-acting, more
158 ., NIR light exposure; group 4=cet-tra-IR700 i.v. and additional cet-tra-IR700 i.v. at 24h but no lig
159 -tra-IR700 i.v. and additional cet-tra-IR700 i.v. at 24h but no light exposure; group 5=cet-tra-IR700
160  light exposure and additional cet-tra-IR700 i.v. immediately after NIR but no additional NIR light e
161 but no light exposure; group 5=cet-tra-IR700 i.v., NIR light exposure and additional cet-tra-IR700 i.
162 v., no light exposure; group 3=cet-tra-IR700 i.v., NIR light exposure; group 4=cet-tra-IR700 i.v. and
163  group 1=no treatment; group 2=cet-tra-IR700 i.v., no light exposure; group 3=cet-tra-IR700 i.v., NIR
164 tion of an injection of placebo or 0.5 mg/kg i.v. methylphenidate.
165 eive a single dose of KRN23 (0.003-0.3 mg/kg i.v. or 0.1-1 mg/kg s.c.) or placebo.
166 ed T1/2 values of 1.1-1.2 h (dose of 5 mg/kg i.v.) and MTDs of 60-80 mg/kg (i.p.).
167 ly treated with artesunate (2.4 or 4.8 mg/kg i.v.) or vehicle upon resuscitation.
168 ly treated with artesunate (2.4 or 4.8 mg/kg i.v.) or vehicle upon resuscitation.
169 sepsis, the administration of LXA4 (7 mug/kg i.v.) 1 h after surgery increased neutrophil phagocytic
170        Pretreatment with leptin (0.12 mug/kg i.v.) 24 h before ischemia/reperfusion abolished cardiop
171        Pretreatment with leptin (0.12 mug/kg i.v.) abolished cardioprotection produced by Goodbelly.
172 days, received an insulin bolus (10 units/kg i.v.) plus insulin infusion (50 mU/kg/min i.v.) until hy
173  (KOR) agonist salvinorin A (0.01-1.8 mg/kg, i.v.) before administration of the KOR selective radiotr
174 i.m.) attenuated cocaine-induced (1.0 mg/kg, i.v.) dopamine overflow in the caudate nucleus but not i
175 istering subanesthetic ketamine (1-30 mg/kg, i.v.) or vehicle to conscious rats (n=12) and testing th
176 at brain demonstrated that 4-MEC (1-3 mg/kg, i.v.) produced large increases in extracellular 5-HT, sm
177             In contrast, 4-MePPP (1-3 mg/kg, i.v.) produced selective increases in dopamine and robus
178  single daily cocaine-reinforced (1.0 mg/kg, i.v.) runway trials 10 min after intracranial injection
179 cotinic receptors by hexamethonium (3 mg/kg, i.v.).
180 on for earning cocaine infusions (0.3 mg/kg, i.v.).
181 24 h after amphetamine infusion (0.56 mg/kg, i.v.).
182 pha2-AR agonists dexmedetomidine (25 mug/kg, i.v.) and clonidine (100 mug/kg, i.v.) inhibited shiveri
183     The effects of CP 55 940 (1.0-10 mug/kg, i.v.) and THC (3.0-300 mug/kg, i.v.) on food-maintained
184 (25 mug/kg, i.v.) and clonidine (100 mug/kg, i.v.) inhibited shivering EMGs, BAT SNA, and BAT thermog
185 .0-10 mug/kg, i.v.) and THC (3.0-300 mug/kg, i.v.) on food-maintained responding and body temperature
186 olidine methiodide (nicotine(PM), 30 mug/kg, i.v.) resulted in habituation (tolerance) of the same ph
187  human annexin A5 treatment (5 or 10 mug/kg, i.v.).
188  to a low, pyrogenic dose of LPS (10 mug/kg, i.v.).
189 gh, shock-inducing dose of LPS (5000 mug/kg, i.v.); this attenuation was due to a blockade of cold-se
190 uated ACTH responses to IL-1beta (500 ng/kg, i.v.) in PNS females, but not in PNS males.
191                              LPS (1.0 ng/kg, i.v.) was administered 180 min before the second [11C]PB
192                           Aspirin (10 mg/kg; i.v.) inhibited COX-1, measured as blood thromboxane and
193 ully applied to the selection of the labeled i.v.-dosed drugs for use in two microdose absolute bioav
194 at 30 mug/kg/d) or terlipressin (single 2-mg i.v. bolus), and the regional hemodynamic effects were q
195 fter fetal treatment with pravastatin (25 mg i.v.).
196 del plasma and in plasma from wild-type mice i.v. injected with Abeta42.
197 kg i.v.) plus insulin infusion (50 mU/kg/min i.v.) until hypoglycemia ensued (</=3.9 mmol/L) (experim
198 n = 7) or SSTR2a infusion (3,000 nmol/kg/min i.v., n = 12) 60 min prior to the same insulin regimen.
199                                    Moreover, i.v. injection of FB28.4.2 in rats blocked complement ac
200 erated against AML cells after s.c., but not i.v., inoculation.
201 f the profound tolerance-inducing ability of i.v. administered Ag-coupled splenocytes (Ag-SP) in mice
202 associate odor cues with the availability of i.v. cocaine or oral sucrose, respectively.
203 ly enhance the pulmonary vascular effects of i.v. vasodilators in Sugen5416/hypoxia/normoxia-exposed
204     At day 7, another injection of 10 mug of i.v. TDM in oil/water emulsion was given (TDM-IV).
205                                The number of i.v. sedations performed in residency was moderately cor
206 was moderately correlated with the number of i.v. sedations personally performed in periodontal pract
207 cokinetics, pharmacodynamics, and potency of i.v. administrations in healthy subjects.
208      This pattern was observed regardless of i.v. contrast medium type, diagnostic criteria for AKI,
209 te an increasing use of high-dose therapy of i.v. gammaglobulin (IVIg) in the treatment of various T
210 differences exist in the use and training of i.v. sedation.
211                   Phase 2 clinical trials of i.v. hu14.18-IL-2 (i.v.-IC) in neuroblastoma and melanom
212 half (49.8%) of the survey respondents offer i.v. sedation in their practices.
213 at 0, 24, and 48 h) either i.m. (n = 348) or i.v. (n = 351), both of which were the intervention arms
214 ated efficient thymic entry of endogenous or i.v. transfused pDCs.
215                         After orotracheal or i.v. administration of USRPs, an excellent colocalizatio
216                                       In ovo i.v. injection of PE resulted in deletion of VLR(PE)Tmu-
217                                     The i.p./i.v. TDM (TDM-IVIP) group was compared with mice injecte
218 y assigned to receive either 50 mg pethidine i.v. (n = 48), or an equal volume of 0.9% normal saline
219 in the liver and 42% in the kidneys 16h post i.v. injection.
220 roximately half of all periodontists provide i.v. sedation, with more recent periodontal graduates mo
221                                     In rats, i.v. ANG2002 induced a dose-dependent analgesia in the f
222                    A subset of mice received i.v. BrdU injection 20 h after challenge.
223 ter i.p. injection of alcohol, mice received i.v. challenge with 5 x 10(7) Escherichia coli for 8 or
224 ial adverse event in NPC1 patients receiving i.v. cyclodextrin therapy.
225 a phase II study, with 22 patients receiving i.v. mangafodipir following oxaliplatin.
226 acteria and wound healing following repeated i.v. administration of activated allogeneic canine MSC.
227 nt OVA TCR transgenic (OT-II) mice or saline i.v. 48 h before challenge with aerosolized OVA.
228 in data obtained during a frequently sampled i.v. glucose tolerance test showed that the antidiabetic
229  relapsed HL were evaluated with up to seven i.v. infusions of the radiolabeled anti-CD25 antibody (9
230                                     A single i.v. dose of Alas1-siRNA prevented the phenobarbital-ind
231 ee groups of mice were treated with a single i.v. injection of rAvPAL-RBCs at three different doses t
232 re examined a model without injury; a single i.v. injection of unfractionated BMCs in healthy mice.
233 d immunogenicity of KRN23 following a single i.v. or s.c. dose of KRN23 in adults with XLH.
234 ynamic stress via sodium nitroprusside (SNP) i.v.] on stimulus evoked responses of sensory processing
235 urnover under post-absorptive and fed state (i.v. Glamin to double amino acids, dextrose to sustain g
236                     In microdialysis studies i.v. PRE-084 did not significantly stimulate dopamine at
237  detailed account is provided for successful i.v. administration of SERRS nanoparticles such that del
238 o experiments were carried out upon systemic i.v. injections with anti-CD137 mAbs and showed CD137 in
239 munized human MUC1 transgenic mice (MUC1.Tg) i.v. with a MUC1 peptide vaccine against which they gene
240 amma, and monokine induced by IFN-gamma than i.v. challenged controls.
241             In addition, we demonstrate that i.v. infusion of the isolated EVs shortly after inductio
242                                We found that i.v. administration of the anti-CD40Ab induced rapid and
243                                 We show that i.v. administration of these NPs can target WAT vasculat
244 ialysis in rat nucleus accumbens showed that i.v. administration of 0.3 and 1.0 mg/kg of mephedrone o
245 the tumor Ag MUC1, we previously showed that i.v. immunization of MUC1 transgenic mice, but not wild-
246                 We report in this study that i.v. delivery of rhabdoviral vectors leads to direct inf
247                                          The i.v. administration of HBD3/CpG complexes induced proinf
248                                          The i.v. injection of oxygen bubbles has recently emerged as
249                                          The i.v. injection of thrombin-activated platelets into CD40
250 consequence of infection of marmosets by the i.v. and s.c. exposure routes.
251 table role in mediating host survival by the i.v. route was not recapitulated following a mucosal inf
252 s routinely lethal to STING(-/-) mice by the i.v. route.
253 o L. monocytogenes may be an artifact of the i.v. infection model.
254 ecting a labeling scheme that overlabels the i.v.-dosed drug or leads to incorrect conclusions on the
255    Administration of T4(+) T cells using the i.v. or intratumoral routes achieves partial tumor regre
256 s a higher bioavailability compared with the i.v. administration of the commercial docetaxel solution
257    We added stable isotope-labeled threonine i.v. to mice and combined fluorescence in situ hybridiza
258 ronegative despite repeated exposure through i.v. injection drug use (IDU-HESN individuals) as witnes
259 with the addition of aerosolized colistin to i.v. treatment, whereas the addition of aerosolized coli
260 ntranasal administration (15min) compared to i.v. administration (120min).
261  the incidence of AKI in patients exposed to i.v. contrast medium was directly compared with the inci
262 -equivalent sheep fetuses were randomized to i.v. bolus infusion of either saline-vehicle or LPS.
263 y cortex of anesthetized mice in response to i.v. NH4(+).
264 ia (10% O(2)) or cardiovascular responses to i.v. injection of sodium cyanide.
265 or rapid TNF-alpha and IFN-beta responses to i.v.-injected sialylated C. jejuni.
266 erosolized colistin as adjunctive therapy to i.v. antimicrobials or as monotherapy in the treatment o
267 ne (4get) or Cd45.1 allele was used to track i.v. transferred eosinophils into the airway following a
268 Accordingly, when infected mice were treated i.v. with an MHC-II-restricted M. tuberculosis epitope p
269 icant tumor gene silencing for 7 d after two i.v. doses.
270 capillaris and intraretinal capillaries upon i.v. injection and 1-h circulation time.
271         In this article, we report that upon i.v. transfer (to avoid major iatrogenic interference),
272                      Propofol, a widely used i.v. anesthetic, has been shown to modulate immunoinflam
273             Propofol is the most widely used i.v. general anesthetic to induce and maintain anesthesi
274          Contrary to what was observed using i.v. infection, IFNAR1(-/-) and wild-type mice had simil
275 he largest percentage of periodontists using i.v. sedation (74.0%) was reported from American Academy
276 were comparing adjunctive aerosolized versus i.v. colistin (seven observational cohort or case-contro
277 ncing in xenograft and orthotopic tumors via i.v. injection or aerosol inhalation, respectively.
278 inally enhanced susceptibility to lethal VSV i.v. infection.
279 tions, we compared the efficacy of 12 weekly i.v. infusions of PerT-GUS versus native GUS on (i) deli
280                                 Animals were i.v. administered 5 mg/kg, 1 mg/kg, or 0.2 mg/kg PGT121
281                                  Horses were i.v. injected with NPHV containing plasma.
282                                    Mice were i.v. infected with a recombinant adenovirus, and within
283                            C57BL/6 mice were i.v. injected with adeno-associated viral vectors encodi
284 ve rats received three weekly sessions where i.v. injected Lipo-DOX was combined with FUS-BBBD; an ad
285 Blue FCF increased bladder capacity, whereas i.v. administration did not.
286           In this study, we assessed whether i.v. administration of oxLDL-induced apoptotic DCs (apop
287 y almost an order of magnitude compared with i.v. delivery with substantially lower plasma concentrat
288 ntrations of drug to the tumor compared with i.v. delivery.
289 reater tumor growth inhibition compared with i.v. FOLFIRINOX.
290 e improved anticancer activity compared with i.v. Taxotere, observed in the metastatic lung cancer mo
291 tumor site was seen with IT-IC compared with i.v.-IC.
292  growth, and improved survival compared with i.v.-IC.
293 tumor activity in mouse models compared with i.v.-IC.
294 sculature, achieved by pretreating mice with i.v. liposome-encapsulated clodronate, significantly att
295  imaged with [(18)F]-FAC PET/CT and MRI with i.v. contrast.
296 up was compared with mice injected once with i.v. or i.p. TDM.
297 ing (8-15 days) compared with that seen with i.v. dosing (0.5-4 days).
298  with IT-IC compared with those treated with i.v.-IC or control mice.
299 pared with control mice or mice treated with i.v.-IC.
300                               Treatment with i.v. bscEGFRvIIIxCD3 yielded extended survival in mice w

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