ライフサイエンスコーパス: ibuprofen

1 ith the non-steroidal anti-inflammatory drug ibuprofen.

2 igher CPP score in response to morphine than ibuprofen.

3 ly well studied, bisphenol A, triclosan, and ibuprofen.

4 nal antiinflammatory therapy with aspirin or ibuprofen.

5 easing use of aspirin, nonaspirin NSAIDs, or ibuprofen.

6 e in mediating the RhoA-inhibiting effect of ibuprofen.

7 C, LIN28A, ALPP and KIT were also reduced by ibuprofen.

8 induction of p75(NTR) by R-flurbiprofen and ibuprofen.

9 induction of p75(NTR) by R-flurbiprofen and ibuprofen.

10 cam, and indometacin but not when given with ibuprofen.

11 ymes, possesses similar potency as the NSAID ibuprofen.

12 ation ratio was 1.9 for naproxen and 7.2 for ibuprofen.

13 solution of 0.2 mM (approximately 4 microg) ibuprofen.

14 cy and profile strikingly similar to that of ibuprofen.

15 nticancer drug toremifene and the painkiller ibuprofen.

16 ssed in some patients with a positive DPT to ibuprofen.

17 different) in those patients that tolerated ibuprofen.

18 rse asthma control than was as-needed use of ibuprofen.

19 harmaceutically relevant solids Ambroxol and Ibuprofen.

20 ) than non-steroidal anti-inflammatory agent ibuprofen (0.44) described the safety profile of the tit

21 0.71 to 1.15; hazard ratio for celecoxib vs. ibuprofen, 0.81; 95% CI, 0.65 to 1.02; P<0.001 for nonin

22 0.76 to 1.13; hazard ratio for celecoxib vs. ibuprofen, 0.85; 95% CI, 0.70 to 1.04; P<0.001 for nonin

23 one of three different protocols: 1) 600 mg ibuprofen 1 hour before surgery and another 600 mg 6 hou

24 loids could also facilitate the transport of ibuprofen (~10% sorbed on colloids) by ~50% due likely t

25 In contrast, depletion of endogenous PGE2 by ibuprofen (100 microM) and celecoxib (5 microM) enhanced

26 high accuracy (90.0% for warfarin, 83.3% for ibuprofen, 70.0% for furosemide and 100% for sertraline

27 once daily, naproxen 500 mg twice daily, or ibuprofen 800 mg 3 times daily for 12 months.

28 crine [a phospholipase A2 (PLA2) inhibitor], ibuprofen [a nonselective COX inhibitor], neomycine [a p

29 Ibuprofen, a blocker of SLC5A8, inhibited SLC5A8-mediate

30 ered their immune function when treated with ibuprofen, a drug that blocks the formation of cyclooxyg

31 The location and distribution of ibuprofen, a model nonsteroidal anti-inflammatory drug,

32 There was no advantage of ibuprofen, a selective Abeta42-lowering agent (SALA), ov

33 Ibuprofen, a structurally dissimilar COX inhibitor, did

34 The measured ibuprofen accumulation agrees quantitatively with the wa

35 floxacin, piroxicam, theophylline, caffeine, ibuprofen, acetaminophen (paracetamol), sulindac and ind

36 affeine, l-leucine, l-histidine, loratadine, ibuprofen, acetaminophen, acetylsalicylic acid, and coca

37 Ibuprofen activates PPARgamma in neuron-like PC12 and B1

38 Ibuprofen affects the cell transporter function and exhi

39 delayed treatment or symptom management with ibuprofen alone.

40 Ex vivo, ibuprofen also affected Sertoli cell by suppressing AMH

41 bination analgesic group) received 400 mg of ibuprofen and 1000 mg of acetaminophen; 5 mg of oxycodon

42 The combination of ibuprofen and acetaminophen (paracetamol) may represent

43 decreased by 4.3 (95% CI, 3.6 to 4.9) in the ibuprofen and acetaminophen group; by 4.4 (95% CI, 3.7 t

44 biomonitoring, no association was found for ibuprofen and acetaminophen intakes during breastfeeding

45 at 2 hours among single-dose treatment with ibuprofen and acetaminophen or with 3 different opioid a

46 as been successfully demonstrated using both ibuprofen and all-trans retinoic acid; drugs with anti-i

47 isualization of two drugs--anti-inflammatory ibuprofen and anticancer 5-fluorouracil--along with many

48 ealed a potent drug-drug interaction between ibuprofen and aspirin and between naproxen and aspirin b

49 e well correlated with the concentrations of ibuprofen and caffeine, anthropogenic indicators of untr

50 This study compares efficacy of preemptive ibuprofen and dexamethasone protocols for pain preventio

51 Ibuprofen and dexamethasone significantly reduced pain (

52 An ibuprofen and flurbiprofen test mix was utilized to demo

53 ing the nonsteroidal anti-inflammatory drugs ibuprofen and flurbiprofen), as well as mandelic acid de

54 down CRC cells, and inhibition of COX-2 with ibuprofen and indomethacin abrogated STIM1-mediated CRC

55 Ibuprofen and indomethacin are nonselective prostaglandi

56 e and compare the effects of early postnatal ibuprofen and indomethacin on ocular and systemic VEGF,

57 nonsteroidal antiinflammatory drugs (NSAIDs) ibuprofen and indomethacin, the drugs widely used as pai

58 In contrast, ibuprofen and mefenamate must bind in both subunits to i

59 Ibuprofen and mefenamic acid are weak, competitive inhib

60 The non-selective NSAIDs, ibuprofen and naproxen, and a selective COX-2 inhibitor,

61 on-steroidal antiinflammatory drugs aspirin, ibuprofen and naproxen, used as positive controls in the

62 Although ibuprofen and other arylpropionic acid derivatives (APs)

63 oncentrations, physical activity, and use of ibuprofen and other common medications.

64 All patients received ibuprofen and otic analgesic drops for use at home.

65 ty BSA and HSA binding sites for salicylate, ibuprofen and picosulfate by using these sensors to moni

66 umented harmful effects, including naproxen, ibuprofen and rubella live vaccine.

67 smooth muscle cells (SMCs) were tested with ibuprofen and sodium dodecyl sulfate (SDS).

68 ensation that is elicited by oleocanthal and ibuprofen and that is commonly experienced around the wo

69 steroidal antiinflammatory drugs aspirin and ibuprofen and the COX-2 selective inhibitors, Celebrex,

70 mple, we demonstrate that the enantiomers of ibuprofen and the diastereoisomers of one of its main me

71 In the cases of ibuprofen and warfarin, the process with the lowest acti

72 d the relation between frequency of aspirin, ibuprofen, and acetaminophen use and risk of hearing los

73 elled prevention of this memory deficit with ibuprofen, and found that ibuprofen prevented memory imp

74 al anti-inflammatory drugs (such as aspirin, ibuprofen, and indomethacin) failed to influence endotox

75 the presence of the cyclooxygenase inhibitor ibuprofen, and microglial conditioned medium (CM) was us

76 inflammatory drugs, such as indomethacin and ibuprofen, and minocycline, a tetracycline analog with n

77 initiation of NSAID (rofecoxib, diclofenac, ibuprofen, and naproxen compared with celecoxib) therapy

78 ) and synthetic inhibitors (flufenamic acid, ibuprofen, and naproxen) was also readily evaluated.

79 chemically unrelated NSAIDs, niflumic acid, ibuprofen, and naproxen, acutely increase intracellular

80 to receive premedication with acetaminophen, ibuprofen, and/or 4% lidocaine gel.

81 imental data, we propose a rationale for the ibuprofen antiaggregation effect.

82 rugs, a nonsteroidal anti-inflammatory drug (ibuprofen), antibiotic (azithromycin), antifungal (tolna

83 Rofecoxib and ibuprofen appeared to confer an increased risk in multip

84 Ibuprofen, applied as a close-to-saturated solution in p

85 ng in a lake where diclofenac, naproxen, and ibuprofen are present as pollutants.

86 question whether or not acetaminophen and/or ibuprofen are safe pain medications during pregnancy aro

87 requently used NSAIDs such as diclofenac and ibuprofen are urgently needed.

88 nsteroidal antiinflammatory drugs, including ibuprofen, are among the most commonly used medications

89 and the nonsteroidal anti-inflammatory drug ibuprofen, are described; they produced useful isolated

90 The ibuprofen salt ibuprofen arginate (Spididol) was created to increase so

91 Ibuprofen arginate but not ibuprofen sodium also reverse

92 Here we investigated the idea that ibuprofen arginate can act to simultaneously inhibit COX

93 Ibuprofen arginate functioned similarly to ibuprofen sod

94 These observations show that ibuprofen arginate provides, in one preparation, a COX-2

95 Ibuprofen arginate retains eNOS substrate activity and r

96 or inhibition of mouse/human COX-2, but only ibuprofen arginate served as a substrate for NOS.

97 ng the well-known anti-inflammatory chemical ibuprofen as one example, we demonstrate that the enanti

98 e in-line liquid-liquid separation) provided ibuprofen at a rate of 8.09 g h(-1) (equivalent to 70.8

99 study was designed to measure the effect of ibuprofen at peak plasma levels on intraoperative bleedi

100 in healthy gingival connective tissue, while ibuprofen attained levels of 2.3 mug/mL and 1.5 mug/g, r

101 lidate the presence/absence of acetaminophen/ibuprofen based on known chemical shift and coupling pat

102 Unexpectedly, both toremifene and ibuprofen bind in a cavity between the attachment (GP1)

103 hese observations led us to suggest that the ibuprofen binding mechanism for Abeta fibrils is differe

104 ndow' of sensitivity within first trimester, ibuprofen causes direct endocrine disturbances in the hu

105 For ibuprofen-clarithromycin-furosemide, the elimination pro

106 e promotes the formation of large low-energy ibuprofen clusters, which rarely occur on the surface of

107 gardless of this, it remains unknown whether ibuprofen could act as an endocrine disruptor as reporte

108 isone (NFkappaB translocation inhibitor), or ibuprofen (COX inhibitor) suppressed molecular changes a

109 eus accumbens (NAc), while administration of ibuprofen decreased glial activation with no effect on l

110 High-dose ibuprofen decreased retinal VEGF levels and retinal VEGF

111 tration breakthrough curves (BTC) of BAM and ibuprofen demonstrated neither degradation nor sorption.

112 inhibits several ASIC subtypes, but certain ibuprofen derivatives show some selectivity for ASIC1a o

113 hree widely used pharmaceutical compounds (S-ibuprofen, diclofenac, and S-warfarin) by one of the maj

114 axolide, naproxen, nonylphenol, octylphenol, ibuprofen, diclofenac, bisphenol A, and triclosan.

115 NSAIDs (indomethacin, sulindac, ketoprofen, ibuprofen, diclofenac, ketorolac, etc., cyclooxygenase i

116 uenced hydrocodone actions in this model and ibuprofen did not potentiate fentanyl or morphine analge

117 Preincubation of oocytes with ibuprofen did not significantly alter the effects of NFA

118 Screening toward ibuprofen ester 1, a substrate for which previously used

119 or ibuprofen oxidation in both free acid and ibuprofen ester form.

120 ontrast, inhibition is uncompetitive for the ibuprofen-ester substrate, consistent with this ester su

121 Because R-flurbiprofen and ibuprofen exhibited the greatest efficacy, we examined t

122 steroidal anti-inflammatory drugs) naproxen, ibuprofen, flurbiprofen, ketoprofen, and fenoprofen.

123 most authors agree with an initial trial of ibuprofen followed by sumatriptan nasal spray for childr

124 xamethasone is as effective as non-steroidal ibuprofen for preventing or controlling postoperative pa

125 micropollutants diclofenac, gemfibrozil and ibuprofen from EfOM.

126 Four case studies on warfarin, ibuprofen, furosemide and sertraline implied that our me

127 proxen group (1.8%), and 155 patients in the ibuprofen group (1.9%) (hazard ratio for celecoxib vs. n

128 proxen group (2.5%), and 218 patients in the ibuprofen group (2.7%) (hazard ratio for celecoxib vs. n

129 ), the naproxen group (852+/-103 mg), or the ibuprofen group (2045+/-246 mg) for a mean treatment dur

130 erbations in the acetaminophen group vs. the ibuprofen group, 0.94; 95% confidence interval, 0.69 to

131 pared with 47% and 24%, respectively, in the ibuprofen group.

132 Animals treated with high-dose ibuprofen had significantly lower somatic growth and hig

133 Animals treated with ibuprofen had statistically significant decreases in the

134 he practice of alternating acetaminophen and ibuprofen has limited value.

135 temperature after toremifene binding, while ibuprofen has only a marginal effect and is a less poten

136 ver, whereas sucralose, sulfamethoxazole and ibuprofen have always been well correlated in all the in

137 n was reported as a possible risk factor for ibuprofen hepatotoxicity, raising the issue of safe use

138 d environmentally relevant concentrations of ibuprofen (IBF), a non-steroidal anti-inflammatory agent

139 This study explores ibuprofen (IBP) uptake and transformation in the wetland

140 CIP) at ferrate dose of 1 mg Fe/L and 30% of ibuprofen (IBU) at ferrate dose of 2 mg Fe/L.

141 mary carp hepatocytes to the pharmaceuticals ibuprofen (IBU), clotrimazole (CTZ), clofibric acid (CFA

142 c acid (HIPA) and their binding competitors, ibuprofen (IBU), furosemide (FUR) and tryptophan (TRP) w

143 Although ibuprofen impedes fibril growth, it does not significant

144 for urinary metabolites of acetaminophen and ibuprofen improved the accuracy of exposure information.

145 as assessed and challenge was performed with ibuprofen in all cases, and additionally with the culpri

146 concentrations of diclofenac, naproxen, and ibuprofen in bream ranged from 6 to 95 ng mL(-1), 6 to 3

147 sts mimics the RhoA-inhibiting properties of ibuprofen in PC12 cells and, like ibuprofen, promotes ne

148 haracterized model analytes (e.g., caffeine, ibuprofen) in both neat buffer and more complex sample m

149 dely available analgesics (acetaminophen and ibuprofen) in epidemiologic studies has been investigate

150 l antiinflammatory drugs, namely aspirin and ibuprofen, in bilayers of dipalmitoylphosphatidylcholine

151 Naproxen and ibuprofen, in their parent form, were conjugated quickly

152 The results show that both salicylate and ibuprofen increase membrane disorder, as determined from

153 ine or the nonsteroid anti-inflammatory drug ibuprofen increased CPP scores in arthritic rats, sugges

154 Inhibitors of COX (naproxen, diclofenac, or ibuprofen) increased bronchoconstriction in tissue from

155 gradation, and treatment of normal mice with ibuprofen increases the levels of COX-2 in brain tissue.

156 Nonselective (ketorolac tromethamine, ibuprofen, indomethacin), COX-1-selective (SC-560), or C

157 ly, the data suggest that R-flurbiprofen and ibuprofen induce p75(NTR) expression by increased mRNA s

158 showed that treatment with R-flurbiprofen or ibuprofen induced p75(NTR) expression in PC-3 and DU-145

159 Recently, we have demonstrated that ibuprofen inhibits intracellular signaling of RhoA and p

160 Ibuprofen inhibits several ASIC subtypes, but certain ib

161 The molecular mechanisms by which ibuprofen inhibits the RhoA signal in neurons, however,

162 ssociation between prenatal acetaminophen or ibuprofen intake and an increased risk of asthma and inc

163 imes) in early pregnancy or midpregnancy and ibuprofen intake as presence or absence in early pregnan

164 udy, we associate maternal acetaminophen and ibuprofen intake during pregnancy and breastfeeding to i

165 Furthermore, ibuprofen interactions shift the thermodynamic equilibri

166 As a result, ibuprofen interference modifies the free energy landscap

167 Ibuprofen is a non-steroidal anti-inflammatory drug (NSA

168 Ibuprofen is a nonsteroidal anti-inflammatory drug widel

169 Ibuprofen is among those drugs newly described as causin

170 Our results show that ibuprofen is an allosteric inhibitor of ASIC1a, which bi

171 Among pregnant women ibuprofen is one of the most frequently used pharmaceuti

172 of SMCT activity and show its inhibition by ibuprofen (K(i) = 73 +/- 9 microM) in Xenopus laevis ooc

173 te, clofibric acid, diclofenac, gemfibrozil, ibuprofen, ketoprofen, naproxen, sulfamethoxazole, and s

174 Oxidative degradation of ibuprofen led to a shift in its toxicity profile from ma

175 e show that treatment with R-flurbiprofen or ibuprofen led to sustained activation of the p38 mitogen

176 The results suggest that ibuprofen localizes in the hydrophobic acyl chain region

177 Ibuprofen may be more effective than indomethacin for su

178 tified 245 out of 8604 spectra as containing ibuprofen metabolite signals from the global data set af

179 use with (1)H NMR-detected acetaminophen and ibuprofen metabolites among 2,660 Western INTERMAP parti

180 use with (1)H NMR-detected acetaminophen and ibuprofen metabolites among 496 participants from Chicag

181 previously unreported positive mode ions of ibuprofen metabolites with their NMR correlates and sugg

182 ation, in particular, from acetaminophen and ibuprofen metabolites.

183 hen model correctly predicted 98.2%, and the ibuprofen model correctly predicted 99.0% of the urine s

184 g acetaminophen metabolite signals while the ibuprofen model identified 245 out of 8604 spectra as co

185 The acetaminophen and ibuprofen models were subsequently used to predict the p

186 Instead, ibuprofen modulated hippocampal gene expression in pathw

187 gies for SJS/TENS were antibiotics (n = 25), ibuprofen (n = 15), and lamotrigine (n = 11).

188 as well as oral formulations of diclofenac, ibuprofen, naproxen, and a composite of all other NSAIDs

189 Although ibuprofen, naproxen, and celecoxib all had the potential

190 y the propensity of commonly consumed NSAIDs-ibuprofen, naproxen, and celecoxib-to cause a drug-drug

191 ins with nonsteroidal antiinflammatory drugs ibuprofen, naproxen, and flurbiprofen.

192 three nonsteroidal anti-inflammatory drugs: ibuprofen, naproxen, and ketoprofen.

193 nit, we separated a standard mixture of 1 mM ibuprofen, naproxen, and phenylbutazone using a commerci

194 on aspirin, acetaminophen, and other NSAID (ibuprofen, naproxen, indomethacin) use were based on a s

195 ctivity of rapid, reversible COX inhibitors (ibuprofen, naproxen, mefenamic acid, and lumiracoxib) de

196 gyakukagoshuyushokyoto and Jumihaidokuto and ibuprofen ointment since three-month of age, she was ref

197 We examined the effects of ibuprofen on cognitive deficits, Abeta and tau accumulat

198 there was a significant protective effect of ibuprofen on hippocampal volume and NAA loss.

199 Moreover, the effect of ibuprofen on RhoA activity and neurite growth in neurona

200 exchange simulations, we study the impact of ibuprofen on the growth of wild-type Abeta fibrils.

201 inhibition (HR, 1.01; 0.95-1.07; P=0.884 and ibuprofen only: HR, 1.00; 0.93-1.07; P=0.996).

202 In this study, use of ibuprofen or acetaminophen (but not aspirin) 2 or more d

203 ritoneal injections of low and high doses of ibuprofen or indomethacin at birth (postnatal day [P]1)

204 es, celecoxib was found to be noninferior to ibuprofen or naproxen with regard to cardiovascular safe

205 purchases of high-dose aspirin and low-dose ibuprofen or NSAID dosing schedules, there were several

206 taminophen (OR, 1.03; 95% CI, 0.88-1.22) and ibuprofen (OR, 1.19; 95% CI, 1.05-1.36).

207 odds ratio [OR], 1.21; 95% CI 1.04-1.41) and ibuprofen (OR, 1.35; 95% CI, 1.19-1.52) intake.

208 he addition of the cyclooxygenase inhibitor, ibuprofen, or a DP1 receptor antagonist (MK0524).

209 al anti-inflammatory treatment with aspirin, ibuprofen, or indometacin.

210 were randomly assigned to receive celecoxib, ibuprofen, or naproxen.

211 acetaminophen and 0.87 per participant with ibuprofen over 46 weeks of follow-up (relative rate of a

212 urvival, in rank-order, were R-flurbiprofen, ibuprofen, oxaprozin, fenoprofen, naproxen, and ketoprof

213 erefore acting as an effective inhibitor for ibuprofen oxidation in both free acid and ibuprofen este

214 ith celecoxib than with naproxen (P=0.01) or ibuprofen (P=0.002); the risk of renal events was signif

215 significantly lower with celecoxib than with ibuprofen (P=0.004) but was not significantly lower with

216 amined the associations of acetaminophen and ibuprofen (per unit increase in exposure category) durin

217 While PGE2 production was suppressed by ibuprofen, PGD2 production was not.

218 ugh the membrane is permeable to both drugs, ibuprofen preferentially accumulates in the membrane, wh

219 Mepacrine or ibuprofen pretreatments almost completely blocked the pr

220 emory deficit with ibuprofen, and found that ibuprofen prevented memory impairment without producing

221 The subjects were instructed to take ibuprofen prior to one of the surgeries.

222 perties of ibuprofen in PC12 cells and, like ibuprofen, promotes neurite elongation in primary cultur

223 Ibuprofen protected the increase of myo-inositol at six

224 osition of Abeta40-42 in the hippocampus and ibuprofen protects against multiple components of the AD

225 In addition, another study suggests that ibuprofen reduces generation of amyloid-beta42 peptide v

226 ylactic treatment of young 3xTg-AD mice with ibuprofen reduces intraneuronal oligomeric Abeta, reduce

227 /v% of a linear graft copolymer could extend ibuprofen release over three-fold (from 3days to >9days)

228 Whereas treatment with R-flurbiprofen or ibuprofen resulted in a massive induction of p75(NTR) pr

229 Ibuprofen's anti-aggregation effect is explained by its

230 Ibuprofen's clearance varies with CYP2C9*3 genotype.

231 The ibuprofen salt ibuprofen arginate (Spididol) was created

232 Hence, R-flurbiprofen and ibuprofen selectively induce p75(NTR)-dependent decrease

233 ested compounds, the 7-coumarine hybrid with ibuprofen showed potent and persistent antihyperalgesic

234 In general, ibuprofen shows a preference to bind to those regions of

235 tinct anti-inflammatory drugs, cortisone and ibuprofen, significantly inhibited angiogenesis, indicat

236 inhibitors (C3-exoenzmye, fasudil, Y-27632, ibuprofen, siRhoA, and p21) in experimental spinal cord

237 Ibuprofen arginate but not ibuprofen sodium also reversed the inhibitory effects of

238 The blood plasma ibuprofen sodium concentrations achieved in rats (263mug

239 mg ibuprofen sodium, the theoretical mass of ibuprofen sodium contained within the dry MN alone, was

240 Ibuprofen arginate functioned similarly to ibuprofen sodium for inhibition of mouse/human COX-2, bu

241 r therapeutically-relevant concentrations of ibuprofen sodium in humans.

242 Ibuprofen sodium was used as the model here and was succ

243 The delivery of 1.5mg ibuprofen sodium, the theoretical mass of ibuprofen sodi

244 inhibition is competitive for the free acid ibuprofen substrate, no doubt because this substrate can

245 3xTg-AD mice were fed ibuprofen-supplemented chow between 1 and 6 months.

246 Ibuprofen suppressed testosterone and Leydig cell hormon

247 substrates as well as commercial aspirin and ibuprofen tablets.

248 ngredients, including caffeine, paracetamol, ibuprofen, tamoxifen, BAY 11-7082 and fluorescein, with

249 f the groove on the CV fibril edge, in which ibuprofen tends to accumulate.

250 as significantly higher binding affinity for ibuprofen than the convex edge.

251 anti-inflammatory drugs, such as aspirin or ibuprofen, that block prostaglandin synthesis.

252 For ibuprofen, the multivariate-adjusted relative risk of he

253 We show that binding of ibuprofen to Abeta destabilizes the interactions between

254 Furthermore, binding of ibuprofen to Abeta monomers, as compared to fibrils, res

255 implicit solvent model we probed binding of ibuprofen to Abeta(10-40) monomers and amyloid fibrils.

256 gamma (PPARgamma) is essential for coupling ibuprofen to RhoA inhibition and subsequent neurite grow

257 on of the novel molecular mechanisms linking ibuprofen to RhoA inhibition may provide additional ther

258 Systemic administration of ibuprofen to spinal cord-lesioned rodents reverses the a

259 (DT50) as short as 1.8 days (acetaminophen, ibuprofen) to not degradable (chlorthalidone, fluconazol

260 that the perception of heat stimuli for the ibuprofen treated worms is lower than the wild-type.

261 Ibuprofen-treated transgenic mice showed a significant d

262 We test ibuprofen-treated worms and a TRPV (transient receptor p

263 3xTg-AD mice was significantly improved with ibuprofen treatment compared to untreated 3xTg-AD mice.

264 We have previously reported that chronic ibuprofen treatment improves cognition and decreases int

265 n p75(NTR) protein due to R-flurbiprofen and ibuprofen treatment was accompanied by an increase in p7

266 of either cell line before R-flurbiprofen or ibuprofen treatment with a dominant negative form of p75

267 ulate experiments of (13)C-labeled 2,4-D and ibuprofen turnover.

268 we evaluated the metabolism of naproxen and ibuprofen, two of the most-used human drugs from the Pro

269 Using MD simulations, the neutral ibuprofen, typically observed at low pH and inaccessible

270 During 764,247 person-years of follow-up, ibuprofen use and acetaminophen use were independently a

271 ed an inverse association between aspirin or ibuprofen use and disease (odds ratio = 0.67, 95% confid

272 e within 24 hours after cessation of regular ibuprofen use in healthy individuals.

273 (1)H NMR-detected acetaminophen and ibuprofen use was low ( approximately 4%) among East Asi

274 omparison of self-reported acetaminophen and ibuprofen use with (1)H NMR-detected acetaminophen and i

275 ckness, start month of acetaminophen use and ibuprofen use).

276 etal testes (7-17 gestational weeks (GW)) to ibuprofen using ex vivo culture and xenograft systems.

277 Subcutaneous injections of ibuprofen via minipumps to rats with a thoracic spinal c

278 serum albumin (HSA) and three known ligands (ibuprofen, warfarin, and phenytoin) are involved to demo

279 In a total residence time of three minutes, ibuprofen was assembled from its elementary building blo

280 n five of the bream and roach samples, while ibuprofen was detected in three bream and two roach samp

281 naproxen and dexketoprofen with tolerance to ibuprofen was found in 16 of 20 cases.

282 No other treatment but ibuprofen was given, and it was administered when the an

283 The gene most highly downregulated by ibuprofen was neuronal tryptophan 2,3-dioxygenase (Tdo2)

284 owed a significant increase in bleeding when ibuprofen was preadministered.

285 ity appeared to be driven by neuropathology; ibuprofen was preferred for traumatic brain injury, post

286 urthermore, the well tolerated COX inhibitor ibuprofen was protective against IL-1beta-induced defici

287 BAM), and the pharmaceuticals diclofenac and ibuprofen was released into a mesoscale aquifer with qua

288 orobenzoic acid (p-CBA), atrazine, DEET, and ibuprofen] was not significantly inhibited in the presen

289 Na(+)-activation kinetics, and inhibition by ibuprofen were analyzed by monitoring OTC-induced curren

290 , the binding properties toward naproxen and ibuprofen were measured for two combinatorial libraries

291 d the percentage of reactions induced by ASA/ibuprofen were significantly lower in Group A (P=.005 an

292 id, acetaminophen, atenolol, diclofenac, and ibuprofen were tested as target analytes.

293 recoveries for acidic drugs (ketoprofen and ibuprofen) were in the range of 76%-86%.

294 h low D(ow) values, i.e., clofibric acid and ibuprofen, were not removed from water, while the organi

295 gned them to receive either acetaminophen or ibuprofen when needed for the alleviation of fever or pa

296 nstrate that the over-the-counter analgesic, ibuprofen, which elicits the same restricted pharyngeal

297 nces were detected between acetaminophen and ibuprofen with respect to the percentage of asthma-contr

298 tive agents with cox-1>cox-2 inhibition (eg, ibuprofen) with the primary outcome.

299 inistration of the cyclooxygenase inhibitor, ibuprofen, with the TRPV1 antagonist decreased inflammat

300 oidal antiinflammatory drugs, salicylate and ibuprofen, with vesicles prepared from 1,2-dimyristoyl-s