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   1 2) are the most common cause of familial and idiopathic Parkinson's disease.                         
     2 ld improve postural control in patients with idiopathic Parkinson's disease.                         
     3 nsation for dopamine loss in experimental or idiopathic Parkinson's disease.                         
     4 ofile similar to that previously reported in idiopathic Parkinson's disease.                         
     5 t the causative role of paraquat exposure in idiopathic Parkinson's disease.                         
     6 pamine neurons is characteristic of advanced idiopathic Parkinson's disease.                         
     7 ) has an increasing role in the treatment of idiopathic Parkinson's disease.                         
     8 tation, and had typical clinical findings of idiopathic Parkinson's disease.                         
     9 veries and to discuss their relationships to idiopathic Parkinson's disease.                         
    10 tern of nigrostriatal dysfunction typical of idiopathic Parkinson's disease.                         
    11 substitution at codon 2019 (Gly2019 ser), in idiopathic Parkinson's disease.                         
    12 physiology of resting and postural tremor in idiopathic Parkinson's disease.                         
    13 ut the Lewy body pathology characteristic of idiopathic Parkinson's disease.                         
    14 e and a clinical syndrome closely resembling idiopathic Parkinson's disease.                         
    15  diseases, including Alzheimer's disease and idiopathic Parkinson's disease.                         
    16 ly indistinguishable from most patients with idiopathic Parkinson's disease.                         
    17  lower than those reported for patients with idiopathic Parkinson's disease.                         
    18  changes reminiscent of those which occur in idiopathic Parkinson's disease.                         
    19 ciated with GBA mutations, and modestly with idiopathic Parkinson's disease.                         
    20 he specific mutation (A53T) in a 40 cases of idiopathic Parkinson's disease, 3 cases of multisystem a
  
    22 pared to controls persisted in patients with idiopathic Parkinson's disease (after exclusion of all G
    23 ganglia was investigated in 25 patients with idiopathic Parkinson's disease and 14 matched healthy co
  
  
    26 e non-demented subjects with newly diagnosed idiopathic Parkinson's disease and 37 healthy matched co
    27  movement have been studied in patients with idiopathic Parkinson's disease and age-matched healthy i
    28 idal disorders: idiopathic torsion dystonia, idiopathic Parkinson's disease and Huntington's disease,
    29 resynaptic dopamine storage in patients with idiopathic Parkinson's disease as a function of the cate
    30 Eligible patients were aged 25-75 years, had idiopathic Parkinson's disease as measured by Queen Squa
    31 en described with a phenotype much closer to idiopathic Parkinson's disease, but it does not appear t
    32 how that fibroblasts from five patients with idiopathic Parkinson's disease can be efficiently reprog
  
    34 d DJ-1 to be an abundant protein in control, idiopathic Parkinson's disease, cases with clinical and 
    35 gid syndromes of less than 5 years duration: idiopathic Parkinson's disease corticobasal degeneration
  
  
    38 esponses to clonidine clearly differentiated idiopathic Parkinson's disease from MSA-C and MSA-P.    
  
    40 ed 86 deceased older Catholic clergy without idiopathic Parkinson's disease from the Religious Order 
  
    42 se association between cigarette smoking and idiopathic Parkinson's disease has been reported in seve
  
    44 e, 26 female) cases were diagnosed as having idiopathic Parkinson's disease (IPD) and 70 (42 male, 28
    45 lsy (PSP), multiple system atrophy (MSA) and idiopathic Parkinson's disease (IPD) can be clinically i
  
    47 uptake (P = 0.0008) compared with a group of idiopathic Parkinson's disease (IPD) patients who had ba
    48 y the role of neuronal complex 1 activity in idiopathic Parkinson's disease (IPD), expression of mito
  
  
  
  
  
    54 rlier in the cancer trajectory but rarely in Idiopathic Parkinson's Disease(IPD), Progressive Supranu
  
  
    57 y syndrome, peduncular hallucinosis, treated idiopathic Parkinson's disease, Lewy body dementia witho
    58  pars compacta of controls and subjects with idiopathic Parkinson's disease matched for age and postm
    59 sed serum GH concentrations in patients with idiopathic Parkinson's disease (median increase 8.98 [IQ
    60 re to differentiate individual patients with idiopathic Parkinson's disease, multiple system atrophy,
  
  
    63 peripheral lymphocytes of 43 newly diagnosed idiopathic Parkinson's disease (PD) cases and 62 age- an
    64 ny compelling basis for a heritable basis of idiopathic Parkinson's disease (PD) has focused attentio
  
  
    67  loss of nigral dopaminergic (DA) neurons in idiopathic Parkinson's disease (PD) is believed to resul
  
    69 on of nigrostriatal dopamine (DA) neurons in idiopathic Parkinson's disease (PD) is still unknown.   
  
    71 or many years, visual hallucinations (VH) in idiopathic Parkinson's disease (PD) were thought to be a
    72 atients with parkinsonian disorders: 86 with idiopathic Parkinson's disease (PD), 30 with progressive
    73 sm, and through analysis of a subcohort with idiopathic Parkinson's disease (PD), we have demonstrate
  
  
  
  
  
    79 uscles in the forearm of seven patients with idiopathic Parkinson's disease, studied with and without
  
    81 tals, we randomly assigned 299 patients with idiopathic Parkinson's disease to undergo either pallida
    82 ver study, 19 patients with mild-to-moderate idiopathic Parkinson's disease underwent functional magn
  
  
    85 esting and postural tremors of patients with idiopathic Parkinson's disease were monitored using tran
  
    87 l as significant reductions in patients with idiopathic Parkinson's disease, which correlates with th
    88 -75 years, had been diagnosed with bilateral idiopathic Parkinson's disease with motor symptoms for m
  
    90 d the effects of clonidine on 14 people with idiopathic Parkinson's disease (without autonomic defici
    91 nucleus (STN DBS) improves motor symptoms in idiopathic Parkinson's disease, yet the mechanism of act
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