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1 ptors and, therefore, a potential remedy for idiopathic epilepsy.
2 in SCN1A and SCN2A cause several subtypes of idiopathic epilepsy.
3 EJM1, the first gene identified for a common idiopathic epilepsy.
4 than recurrent seizures reminiscent of human idiopathic epilepsy.
5 ly identified in a rare Mendelian variety of idiopathic epilepsy.
6 nderlie the functional abnormalities in this idiopathic epilepsy.
7 mon neuronal excitability disorder, sporadic idiopathic epilepsy.
8 at position 43 (R43Q), is linked to familial idiopathic epilepsies.
9 ues for research on pathogenic mechanisms of idiopathic epilepsies.
10 ve children (age 8-18) with new/recent onset idiopathic epilepsy and 62 healthy controls underwent st
11 logistic regression analysis identified non-idiopathic epilepsy and abnormal motor-sensory (neurolog
12 te to common paroxysmal disorders, including idiopathic epilepsy and migraine, that share features wi
13 se the largest subset of monogenic causes of idiopathic epilepsy, and pathogenic variants contribute
14 neural development may be a cause of common idiopathic epilepsy, and the findings have implications
17 ADHD is a prevalent comorbidity of new onset idiopathic epilepsy associated with a diversity of salie
18 We generated a digenic mouse model of human idiopathic epilepsy by combining two epilepsy-associated
19 d SCN2A are responsible for several dominant idiopathic epilepsy disorders, including generalized epi
23 he molecular genetic basis of these familial idiopathic epilepsies is entirely unknown, but a mutatio
24 ociated with hip dysplasia, elbow dysplasia, idiopathic epilepsy, lymphoma, mast cell tumour and gran
25 Children (age: 8-18 years) with recent-onset idiopathic epilepsy (n = 53) and healthy controls (n = 5
26 To date 5% of the imaged cohort or 8% of non-idiopathic epilepsy patients have undergone surgical pro
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