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1 ; also known as Jo-1) in the pathogenesis of idiopathic inflammatory myopathy.
2 muscle inflammation that is the hallmark of idiopathic inflammatory myopathy.
3 d be undertaken in any patient with presumed idiopathic inflammatory myopathy.
4 ate immune mechanisms in the pathogenesis of idiopathic inflammatory myopathies.
5 the optimal design of clinical trials in the idiopathic inflammatory myopathies.
6 the development of better therapies for the idiopathic inflammatory myopathies.
7 ggest that non-immunological factors promote idiopathic inflammatory myopathies.
8 mune responses and clinical syndromes in the idiopathic inflammatory myopathies.
9 ting the serial CMAS scores of children with idiopathic inflammatory myopathies.
10 sorders than the currently used designation, idiopathic inflammatory myopathies.
11 skeletal muscle involvement in patients with idiopathic inflammatory myopathies.
12 s of strength and endurance in children with idiopathic inflammatory myopathies.
13 assessing disease activity and damage in the idiopathic inflammatory myopathies and speculates on pos
18 Inasmuch as the clinical features of the idiopathic inflammatory myopathies are not easily differ
19 eurin inhibitors are being used to treat the idiopathic inflammatory myopathies, but their efficacy h
20 teroviruses as potential etiologic agents of idiopathic inflammatory myopathy, but their relationship
22 ing myopathy (IMNM) is considered one of the idiopathic inflammatory myopathies, comprising dermatomy
23 the pathogenesis and course of the juvenile idiopathic inflammatory myopathies continued this year.
24 dermatomyositis, polymyositis, and the other idiopathic inflammatory myopathies focus primarily on fe
25 nces in the development of therapies for the idiopathic inflammatory myopathies have been enabled by
27 tions into possible infectious causes of the idiopathic inflammatory myopathies have focused on retro
28 our understanding of the pathogenesis of the idiopathic inflammatory myopathies have served to identi
30 the case for other autoimmune diseases, the idiopathic inflammatory myopathies (IIM) develop as a re
31 f patients with systemic sclerosis (SSc) and idiopathic inflammatory myopathies (IIM), and HLA-DQA1*0
32 ease damage, have not been developed for the idiopathic inflammatory myopathies (IIM), thus limiting
34 rovement in clinical trials of patients with idiopathic inflammatory myopathy (IIM) has varied to dat
36 date the reader on immunogenetic advances in idiopathic inflammatory myopathy (IIM) over the past 18
40 Mechanisms driving the autoimmune process in idiopathic inflammatory myopathies (IIMs) have not been
41 en made in understanding the genetics of the idiopathic inflammatory myopathies (IIMs) in the past 2
44 y contribute to the etiology of the juvenile idiopathic inflammatory myopathies (IIMs), which are sys
47 most striking humoral characteristics of the idiopathic inflammatory myopathies is the specific targe
52 Advances in dermatomyositis and the juvenile idiopathic inflammatory myopathies over the past year in
55 es should be distinguished from the commoner idiopathic inflammatory myopathies such as polymyositis
56 ve assessments of therapeutic strategies for idiopathic inflammatory myopathies were recently publish
57 were performed in 19 patients with juvenile idiopathic inflammatory myopathy who performed stair-ste
58 Methods for assessing target organs of the idiopathic inflammatory myopathies will likely evolve as
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