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1 apacity and quality of life in patients with idiopathic pulmonary arterial hypertension.
2 n the changing demographics of patients with idiopathic pulmonary arterial hypertension.
3 on of a novel gene, KCNK3, with familial and idiopathic pulmonary arterial hypertension.
4 arterial hypertension and 230 patients with idiopathic pulmonary arterial hypertension.
5 ptions for treatment of medically refractory idiopathic pulmonary arterial hypertension.
6 pulmonary vascular lesions of patients with idiopathic pulmonary arterial hypertension.
7 ptosis and the loss of small vessels seen in idiopathic pulmonary arterial hypertension.
8 P) provide such information in patients with idiopathic pulmonary arterial hypertension.
9 nguishable from those found in patients with idiopathic pulmonary arterial hypertension.
10 current treatment options for children with idiopathic pulmonary arterial hypertension.
11 of endothelial cells in plexiform lesions in idiopathic pulmonary arterial hypertension.
12 ence and point prevalence of 0.7 and 4.4 for idiopathic pulmonary arterial hypertension and 2.2 and 1
13 thin the pulmonary adventitia of humans with idiopathic pulmonary arterial hypertension and animals w
14 without anemia is prevalent in patients with idiopathic pulmonary arterial hypertension and associate
15 ein levels were elevated in lungs from human idiopathic pulmonary arterial hypertension and in lungs
16 B, MANTIS was downregulated in patients with idiopathic pulmonary arterial hypertension and in rats t
17 derived endothelial cells from patients with idiopathic pulmonary arterial hypertension and in the pu
19 d retinoid levels in plasma of patients with idiopathic pulmonary arterial hypertension and the effec
20 the walls of small intrapulmonary vessels in idiopathic pulmonary arterial hypertension and the rare
21 survival in transplantation candidates with idiopathic pulmonary arterial hypertension, as is being
22 derived endothelial cells from patients with idiopathic pulmonary arterial hypertension attenuated th
23 and in lungs of patients with emphysema and idiopathic pulmonary arterial hypertension comparably ex
24 ervation that lung tissue from patients with idiopathic pulmonary arterial hypertension demonstrated
26 rrent diagnostic and treatment strategies of idiopathic pulmonary arterial hypertension in children h
28 olic volume is a strong prognostic marker in idiopathic pulmonary arterial hypertension, independent
34 ted an altered hyaluronan (HA) metabolism in idiopathic pulmonary arterial hypertension (IPAH) lung t
35 AR-2 and tryptase expression in the lungs of idiopathic pulmonary arterial hypertension (IPAH) patien
37 opathic pulmonary fibrosis (IPF) and 38 with idiopathic pulmonary arterial hypertension (IPAH), the l
38 lays an important role in the development of idiopathic pulmonary arterial hypertension (IPAH), where
39 (SScPAH) has a worse prognosis compared with idiopathic pulmonary arterial hypertension (IPAH), with
54 We show that cultured ECs from patients with idiopathic pulmonary arterial hypertension (IPAH-ECs) ha
55 ally classified as pre-capillary (as seen in idiopathic pulmonary arterial hypertension [IPAH]) or po
56 nd transplantation outcomes in patients with idiopathic pulmonary arterial hypertension is poorly des
57 Although several studies have focused on idiopathic pulmonary arterial hypertension, less is know
61 othelial cells (PAECs) from patients who had idiopathic pulmonary arterial hypertension (PAH) in comp
62 nt in the vascular lesions of PH, whether in idiopathic pulmonary arterial hypertension (PAH) or PAH
66 aph-mass spectrometer technique in plasma of idiopathic pulmonary arterial hypertension patients and
69 andidates for treatments previously used for idiopathic pulmonary arterial hypertension patients.
70 th severe hypoxia-induced PH and humans with idiopathic pulmonary arterial hypertension (PH-Fibs) dis
73 ns in the BMP type II receptor (BMPRII) with idiopathic pulmonary arterial hypertension suggests an i
75 well as increased survival in patients with idiopathic pulmonary arterial hypertension using current
76 l cells cultured from lungs of patients with idiopathic pulmonary arterial hypertension versus contro
77 ist mortality has decreased in patients with idiopathic pulmonary arterial hypertension wait-listed f
78 hildren diagnosed between 1982 and 1995 with idiopathic pulmonary arterial hypertension was followed
80 seven control subjects and six patients with idiopathic pulmonary arterial hypertension were analyzed
83 patients with HRPH, but not in patients with idiopathic pulmonary arterial hypertension, were also Ne
84 e (CMR) is useful for risk stratification in idiopathic pulmonary arterial hypertension, whereas it h
85 ovascular magnetic resonance measurements in idiopathic pulmonary arterial hypertension, with no stud
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