ライフサイエンスコーパス: ileal

1 we suggest a new name for this enzyme: human ileal aminopeptidase (HILAP).

2 iating enterohepatic circulation of BA using ileal and colonic (ascending and sigmoid) biopsies obtai

3 Mice lacking RelA in ileal and colonic enterocytes were born in expected Mend

4 Ex vivo cultures of ileal and colonic mucosa from 10 PI-IBS, diarrhea predom

5 ntisense oligonucleotide, mongersen, targets ileal and colonic SMAD7.

6 aser capture microdissection (LCM)-harvested ileal and colonic tip and crypt epithelial fractions fro

7 Ileal and colonic tissues were collected from patients w

8 insoluble kiwifruit fibre and led to higher ileal and faecal concentrations of short-chain fatty aci

9 fruit inclusion level decreased the apparent ileal and faecal digestibilities of several nutrients (P

10 astric and pancreatic digestion and in vitro ileal and faecal fermentation were combined with Caco-2

11 121 and 193/175, that were found both in the ileal and faecal fermented samples, passed the Caco-2 ce

12 ne of the 2 primary branches of the SMV (the ileal and jejunal branches), with or without involvement

13 ously characterized ontogenic changes in rat ileal and renal ASBT expression.

14 s on gut microbiota and on the expression of ileal antimicrobial peptides.

15 ptor and TGR5, and transporters, such as the ileal apical sodium-dependent bile acid transporter, app

16 esize that pharmacological inhibition of the ileal, apical sodium-dependent bile acid transporter (AS

17 ave a large effect in individuals with extra ileal as well as ileal inflammation.

18 Hur(IKO) mice showed no change in ileal Asbt expression, fecal bile acid excretion, or ent

19 the canalicular membrane, and (3) increased ileal BA absorption.

20 Neomycin also changed the composition of the ileal bacterial community (Proteobacteria became the mos

21 We propose that rifaximin modulates the ileal bacterial community, reduces subclinical inflammat

22 The ileal bacterial richness tended to decrease when the die

23 perimental cholestasis, FXR agonism improved ileal barrier function by attenuating intestinal inflamm

24 IRT1, a key nutrient sensor, is required for ileal bile acid absorption and systemic bile acid homeos

25 The ileal bile acid transporter (IBAT) protein expressed in

26 f GSK2330672, a selective inhibitor of human ileal bile acid transporter (IBAT), in patients with pri

27 iliary cholangitis with pruritus, 14 days of ileal bile acid transporter inhibition by GSK2330672 was

28 rst randomised controlled crossover trial of ileal bile acid transporter inhibitor, a novel class of

29 bt and Mcf2l (encodes Ost) and absorption of ileal bile acids.

30 -Phe-cholyl-insulin had been taken up by the ileal bile salt transporters.

31 We obtained ileal biopsies from sites of inflammation and peripheral

32 ion studies, total RNA was isolated from 168 ileal biopsies of study participants with (34) and witho

33 Real-time PCR analysis of human duodenal and ileal biopsy samples demonstrates that PAT1, TauT and AT

34 In ileal biopsy samples of patients with CD, there was an i

35 Total RNA was extracted from ileal biopsy specimens and genomic DNA was obtained from

36 rrelated with reduced expression of PRDM1 in ileal biopsy specimens and peripheral blood mononuclear

37 H+ exchanger 3 (NHE3) were measured in human ileal biopsy specimens from patients who did and did not

38 TX-ARG rats demonstrated greater jejunal and ileal bowel weight, greater ileal mucosal weight, greate

39 Activation of the ileal brake through the delivery of nutrients into the d

40 branch without reconstruction as long as the ileal branch is intact and of good caliber.

41 Tumors which involve the ileal branch of the SMV, in the rare setting in which th

42 The expression of NHE3 was reduced in the ileal brush border of patients with diarrhea.

43 sequestrant (n = 3,806), diet (n = 458), and ileal bypass surgery (n = 838).

44 xpression (ie, diet, bile acid sequestrants, ileal bypass, and ezetimibe) (between-group difference,

45 cases of MDC and 6214 cases of non-Meckelian ileal cancer, between 1973 and 2006, from the Surveillan

46 Ileal cannulated pigs (50+/-1.9 kg) were fed a fibre-fre

47 Blocking ileal CBA reabsorption in transferred Rag1(-/-) mice res

48 her detected another defect in patients with ileal CD, because the PBMC secretomes derived from patie

49 n expression characteristic of patients with ileal CD.

50 essment of disease activity in patients with ileal CD.

51 ice, Cre activity was detected in the distal ileal, cecal, colonic, and rectal epithelium, with selec

52 Gastric and ileal chymes were kinetically collected over the postpra

53 was maintained throughout the gut up to the ileal compartment.

54 for the efficacy of RARC and Intracorporeal Ileal Conduit (ICIC).

55 comparing various diversion types, including ileal conduit and orthotopic continent neobladder.

56 ed to analyze bacterial 16S ribosomal RNA in ileal contents from the rats.

57 Ninety subjects (45 with active terminal ileal Crohn disease and 45 without Crohn disease) underw

58 of active Crohn disease was active terminal ileal Crohn disease based on ileocolonoscopy or establis

59 nterography for active inflammatory terminal ileal Crohn disease, despite an inferior subjective imag

60 ease and imaging features of active terminal ileal Crohn disease.

61 active inflammatory terminal or neoterminal ileal Crohn disease.

62 mpelling evidence for an association between ileal Crohn's disease (CD) and the IL23R gene using geno

63 that affect innate immunity increase risk of ileal Crohn's disease (CD).

64 Furthermore, individuals with ileal Crohn's disease and in their third trimester of pr

65 n hypomorphic ATG16L1 function and implicate ileal Crohn's disease as a specific disorder of Paneth c

66 Further, a subset of ileal Crohn's disease patients displayed MDR1 loss of fu

67 nction ICOSLG risk allele associated with an ileal Crohn's disease phenotype, similar to polymorphism

68 Ileal Crohn's disease subjects deviated most from the HP

69 The microbiome of ileal Crohn's disease was notable for increases in virul

70 the hypothesis that colonic Crohn's disease, ileal Crohn's disease, and ulcerative colitis are all ge

71 genetic risk factors in the pathogenesis of ileal Crohn's disease, but the exact contributions of No

72 ease, much better explained by three groups (ileal Crohn's disease, colonic Crohn's disease, and ulce

73 colon tissues from patients with UC, but not ileal Crohn's disease, than control tissues; levels of G

74 OD2 gene are strong genetic risk factors for ileal Crohn's disease.

75 to Nod2 function relevant to inflammation of ileal Crohn's disease.

76 erative colitis, colonic Crohn's disease and ileal Crohn's disease.

77 sk score strongly distinguished colonic from ileal Crohn's disease.

78 However, treatment of non-transformed IEC-18 ileal crypt cells with PKC agonists has a biphasic effec

79 ation of 4E-BP1 in IEC-18 nontransformed rat ileal crypt cells.

80 re selectively overexpressed in duodenal and ileal crypts, respectively, in RBP-Jkappa-deficient mice

81 d in vitro hindgut digestibility assay using ileal digesta (sampled from the chicken or rat) pertaini

82 nowledge, there are no systematic studies of ileal digesta endogenous proteins.

83 of the endogenous amino acid composition of ileal digesta in humans, to our knowledge, there are no

84 uly endogenous component within the terminal ileal digesta.

85 with susceptibility to CD, specifically with ileal disease (rs6559629, P = 3.1 x 10(-)(5); odds ratio

86 in Crohn's disease is fibrostenotic terminal ileal disease.

87 of Crohn's disease, with a 2.2-fold risk of ileal disease.

88 s, including the colorectal, cecal, jejunal, ileal, duodenal, and cecal tonsil tissues.

89 the allograft provides access to samples of ileal effluent and mucosal biopsies.

90 gradation products in the urine, plasma, and ileal effluent of healthy volunteers and ileostomists (s

91 and on the resistant starch (RS) content of ileal effluent.

92 Analysis of ileal effluents (at end of small intestine) demonstrated

93 ive proportions of several bacterial taxa in ileal effluents and especially Firmicutes, could be used

94 with changes in the microbial populations in ileal effluents and support microbiota profiling as a po

95 Ileal efflux samples collected after digestion of BB-DSF

96 icant contribution to published estimates of ileal endogenous amino acids and protein.

97 The abnormalities in the ileal enteroendocrine cells appear to be caused by two m

98 rome (IBS), and whether any abnormalities in ileal enteroendocrine cells are correlated with abnormal

99 esults provide a better understanding of the ileal environment in the mouse and the changes that occu

100 e have evaluated gene expression patterns of ileal epithelial cells isolated by laser capture microdi

101 In vitro, cultured ErbB4(-/-) ileal epithelial enteroids had reduced Paneth cell marke

102 Within the ileal epithelium, both ATG16L1 and a second essential au

103 tial external biliary diversions (PEBDs), 11 ileal exclusions (IEs), and seven gallbladder-to-colon (

104 r results were seen after infection of human ileal explants with EHEC.

105 times more FGF19 than FXR agonist-stimulated ileal explants.

106 ese modifications corresponded to an altered ileal expression of C-type lectins Reg3gamma and Reg3bet

107 xpression that was inversely correlated with ileal expression of fibroblast growth factor 15 (FGF15).

108 h, induces metabolic alterations and affects ileal expression of genes involved in immunity.

109 In the postprandial state, activation of ileal farnesoid X receptor (FXR) by bile salts results i

110 Ileal Fgf15 is a potent inhibitor of BA synthesis.

111 t gene) and inversely with Shp mRNA, but not ileal Fgf15 mRNA.

112 isotype that has been proposed to mediate an ileal FGF15/19 to hepatocyte FGFR4 axis in cholesterol h

113 ed hepatic BA synthesis, potentially through ileal fibroblast growth factor 15- and Fxr-mediated inhi

114 Human ileal fluid samples were collected from 20 patients post

115 Here, we characterized the global pattern of ileal gene expression and the ileal microbial community

116 Genotypes, antimicrobial serologies, ileal gene expression, and ileal, rectal, and faecal mic

117 s identified Ahr(-/-)-dependent increases in ileal gene expression, indicating increased inflammatory

118 rations to the murine intestinal microbiota, ileal gene expression, specific intestinal T-cell popula

119 Ileal genes controlling extracellular matrix production

120 rats in vivo We further demonstrate that an ileal glucagon-like peptide-1 receptor-dependent neurona

121 ression was unaltered; however, HS increased ileal GLUT-2 protein expression (P=0.06).

122 AMP1/Fc mice is an increase in the number of ileal goblet and intermediate cells.

123 he small intestine and regulates the jejunal-ileal gradient in absorptive enterocyte gene expression.

124 GM-CSF regulates ileal homeostasis in CD and in mouse models.

125 We show that two ileal IBD-stereoenterotypes ('cobblestones' versus 'vill

126 ays a pivotal role in determining jejunal vs ileal identity.

127 Relative to colonic extent, ileal, ileocolonic, and upper GI extent were significant

128 Disease extent was ileal in 45.1%, colonic in 32.0%, and ileocolonic in 18.

129 pathogen and was associated with more severe ileal inflammation and pathology.

130 We analyzed ileal inflammation in SAMP1/YitFc and SAMP1/YitFc Itgb7(

131 ection of an IL1 receptor antagonist reduced ileal inflammation in SHIP-null mice.

132 e early induction of RELMbeta expression nor ileal inflammation requires the presence of viable intes

133 During ileal inflammation, absorption of nutrients and electrol

134 t in individuals with extra ileal as well as ileal inflammation.

135 on of Il1b, which contributes to spontaneous ileal inflammation.

136 Ileal interposition (IT) is a surgical model that isolat

137 We have investigated the effects of ileal interposition (IT), a surgical relocation of the d

138 eum that acted as a leading point to an ileo-ileal intussusception.

139 L in adult CD serum and were associated with ileal involvement (P<.001).

140 opment of complications were the presence of ileal involvement and perianal disease.

141 Ileal ischemia was accomplished through obstruction of t

142 ng glucose-stimulated GLP-1 release in human ileal L cells.

143 syntaxin-1a (syn1a) was expressed by murine ileal L cells.

144 s to increased free fatty acids reaching the ileal L cells.

145 (+) DCs were defective in migration from the ileal lamina propria to the MLN.

146 Expression of FOXP3 in the ileal lamina propria was significantly decreased at all

147 enal tubular cell damage, hepatocyte damage, ileal leakage of horseradish peroxidase, and bacterial t

148 (AIEC) strains are frequently isolated from ileal lesions of CD patients indicating a potential role

149 Here, we developed the pig ileal-ligated loop model for semi-quantitative analysis

150 Evidence is limited, with support from naso-ileal lipid infusion studies.The objective of the study

151 Ileal location, duration of disease, and increased GM-CS

152 We found evident differences in rabbit ileal loop and catfish ileal loop responses to E. ictalu

153 In vivo mouse ileal loop experiments showed reduced fluid accumulation

154 cause bloody necrotic enteritis in a rabbit ileal loop model and also showed that purified CPB, in t

155 inal loop model and compare it to the rabbit ileal loop model inoculated with Salmonella enterica ser

156 ty is necessary for enterotoxicity, a rabbit ileal loop model was used to compare the in vivo effects

157 d in vivo screening using the rabbit ligated ileal loop model.

158 differences in rabbit ileal loop and catfish ileal loop responses to E. ictaluri and S. Typhimurium L

159 In each patient, the terminal ileal loop was imaged with contrast-enhanced US before t

160 In a mouse ligated-ileal-loop assay, 2D6 IgA promoted V. cholerae agglutina

161 reported to enhance enterotoxicity in rabbit ileal loops and the reactogenicity of live cholera vacci

162 Ileal loops from wild-type rabbits and mice and A(2B)AR(

163 We injected TxA into ileal loops in PAR(2) or dipeptidyl peptidase I (DPPI) k

164 nting EPEC-induced fluid secretion in rabbit ileal loops than alpha,beta-methylene-ADP.

165 hagic luminal fluid in duodenal, jejunal, or ileal loops treated for 6 h with purified CPB, while no

166 CT activity against cultured cells and ileal loops was also blocked by GA, as was the ER-to-cyt

167 profile induced by S. typhimurium in ligated ileal loops was dominated by TH17 responses, including t

168 Similar treatment of ileal loops with either of the noncytotoxic rCPE variant

169 ted significant fluid accumulation in rabbit ileal loops, along with severe mucosal damage that start

170 In contrast, in bovine ligated ileal loops, flagellin pattern recognition contributed t

171 testinal epithelial cells, and ligated mouse ileal loops, thereby disrupting barrier function.

172 nti-microRNAs were transferred to mice using ileal loops.

173 inal infection were tested in bovine ligated ileal loops.

174 I), can reproduce type C pathology in rabbit ileal loops.

175 haemorrhagic necrotizing enteritis in rabbit ileal loops.

176 A null mutant of CN3685 remained virulent in ileal loops.

177 ion of about 700-fold was observed in rabbit ileal loops.

178 marker (rs6689879) contributed to increased ileal MAGI3 expression level in non-IBD controls.

179 bal pattern of ileal gene expression and the ileal microbial community in 359 treatment-naive pediatr

180 alpha-defensin deficiency is associated with ileal microbiota alterations, the cecal and colonic micr

181 that the transcriptomes of L. plantarum and ileal microbiota are not altered shortly after SIV infec

182 The ileal microbiota of L. plantarum-containing healthy and

183 gene tags to compare the composition of the ileal microbiota present during nonrejection, prerejecti

184 Fecal and ileal microbiota were analyzed by pyrosequencing of 16S

185 ced a TNM staging classification for jejunal-ileal (midgut) neuroendocrine tumors (NETs).

186 allenge, RB-fed pigs had significantly lower ileal mitotic index and villus width, and significantly

187 to early innate host responses in the bovine ileal mucosa but not in the murine cecal mucosa.

188 are regulated during early infection, human ileal mucosa cultured as explants was infected with C. p

189 We show that the ileal mucosa in CF have a mucus that adhered to the epit

190 Escherichia coli (AIEC), which colonize the ileal mucosa of patients with CD, adhere to and invade i

191 tion of T helper type 17 (TH17) cells in the ileal mucosa of rhesus macaques, thereby impairing mucos

192 Three hours after anti-CD3 mAb injection, ileal mucosa stripped of muscular and serosal layers sho

193 ntestinal epithelial cells and in the bovine ileal mucosa.

194 transits from the intestinal lumen into the ileal mucosa.

195 g agents that actively circulate through the ileal mucosa.

196 these compounds for their ability to prevent ileal mucosal barrier dysfunction induced by subjecting

197 eight, greater ileal mucosal weight, greater ileal mucosal DNA and protein levels, greater villus hei

198 In addition to reproducing healthy ileal mucosal dynamics as well as a series of morphogen

199 rially produced SCFAs, propionate, activates ileal mucosal free fatty acid receptor 2 to trigger a ne

200 In Duoxa(-/-) mice, abnormalities in ileal mucosal gene expression at homeostasis recapitulat

201 The 10% CP dietary treatment damaged ileal mucosal morphology, and decreased the expression o

202 P, brain ghrelin levels, serum HMGB1 levels, ileal mucosal permeability to fluorescein isothiocyanate

203 1960s to 1980s at the luminal surface of the ileal mucosal wall and is a Na(+)- and Cl(-)-dependent t

204 ater jejunal and ileal bowel weight, greater ileal mucosal weight, greater ileal mucosal DNA and prot

205 The properties of the ileal mucus of CF mice were normalized by secretion into

206 The ileal mucus of cystic fibrosis (CF) mice with a nonfunct

207 In isolated guinea pig ileal myocytes, ML204 blocked muscarinic cation currents

208 Here, we use an isogenic human ileal neobladder surgical model and compare global DNA m

209 (18)F-FDOPA sensitivities are highest in ileal NETs and may also be helpful in insulinomas.

210 TLR2 distribution and function in the ileal neuromuscular layer of mice were determined by imm

211 ndent mechanisms; in contrast, cAMP inhibits ileal NHE3 only by a PKA-dependent pathway, which is ind

212 ls were assessed, in addition to colonic and ileal nitrergic myenteric neuron quantifications and mot

213 and urea nitrogen, represented >68% of total ileal nitrogenous losses.

214 . coli-laden and unladen LP macrophages from ileal or colonic biopsies.

215 scence staining for CD40 in IECs of inflamed ileal or colonic mucosa.

216 Suggested treatment algorithms for NETs of ileal or jejunal origin and of pancreatic origin are pre

217 ppeared to be an enteric duplication cyst of ileal origin.

218 Undigested food in the ileal output was examined microscopically to identify ce

219 6A variant showed defective clearance of the ileal pathogen Yersinia enterocolitica and an elevated i

220 o be possibly related to meropenem (isolated ileal perforation and an episode of fungal sepsis).

221 t following oral inoculation, prions bind to ileal Peyer patch and cecal patch microfold cells (M cel

222 The continuous ileal Peyer's patches (IPP) of sheep are regarded as a t

223 , resulting from a failure of the gastric or ileal phase of physiological B12 absorption, best exempl

224 Restorative proctocolectomy with ileal pouch anal anastomosis (IPAA) is associated with t

225 Ileal pouch anal anastomosis (IPAA) is the treatment of

226 For patients who require colectomy, the ileal pouch anal anastomosis operation has alleviated th

227 multivariable analyses among patients in the ileal pouch cohort (odds ratio, 1.38; 95% CI, 1.05-1.82)

228 ications were observed among patients in the ileal pouch cohort who received anti-TNF agents preopera

229 Studies evaluating ileal pouch formation and antireflux surgery showed conf

230 olitis (UC) who required partial or complete ileal pouch reconstruction due to poor function or infec

231 Pelvic sepsis developing after redo ileal pouch surgery was the primary indicator of pouch f

232 o determine whether immediate colectomy with ileal pouch-anal anastamosis (IPAA) after diagnosis of s

233 s (48%), 118 subtotal colectomies (19%), 134 ileal pouch-anal anastomoses (21%), 23 segmental colecto

234 , single-center experience of transabdominal ileal pouch-anal anastomoses (IPAA) redo surgery for a f

235 ith end ileostomy, and 1172 (47.3%) received ileal pouch-anal anastomoses.

236 A prospectively collected ileal pouch-anal anastomosis (IPAA) database was reviewe

237 Restorative proctocolectomy with ileal pouch-anal anastomosis (IPAA) has substantially re

238 choice in ulcerative colitis, performance of ileal pouch-anal anastomosis (IPAA) is controversial in

239 Although restorative proctocolectomy with ileal pouch-anal anastomosis (IPAA) substantially reduce

240 Pouchitis is common after ileal pouch-anal anastomosis (IPAA) surgery for ulcerati

241 ims to compare surgical outcome of transanal ileal pouch-anal anastomosis (ta-IPAA) with transabdomin

242 Total proctocolectomy with ileal pouch-anal anastomosis is considered the procedure

243 Although ileal pouch-anal anastomosis is recommended after colect

244 days of surgery among patients who underwent ileal pouch-anal anastomosis was associated with higher

245 se patients, 71 (65.7%) underwent subsequent ileal pouch-anal anastomosis, 2 died of other causes, an

246 omy, or a combined total proctocolectomy and ileal pouch-anal anastomosis.

247 emains an important issue even in the era of ileal pouch-anal anastomosis.

248 atients with restorative proctocolectomy and ileal pouch-anal anastomosis.

249 colitis (UC) undergoing proctocolectomy with ileal pouch-anal anastomosis.

250 demonstrate that heterologous rAd41 oral or ileal priming with rAd5 intramuscular boosting elicits e

251 508) have no lung phenotype but show similar ileal problems to humans.

252 -dependent neuronal network is necessary for ileal propionate and long chain fatty acid sensing to re

253 obial serologies, ileal gene expression, and ileal, rectal, and faecal microbiota were assessed.

254 the effects of a single dose of encapsulated ileal-release glutamine (6 g) and placebo (microcrystall

255 pass (DJB), jejunal resection (jejunectomy), ileal resection (ileectomy), pair-fed sham-operated, and

256 h Crohn's disease who had undergone terminal ileal resection between 1981 and 2009.

257 luence the rate of recurrence after terminal ileal resection for Crohn's disease.

258 Ileal samples from patients with CD have increased level

259 Ileal samples were collected after sacrifice.

260 Faecal and ileal samples were collected on days 5, 11, 21, 35, 49 a

261 In ileal samples, the genus Clostridium sensu stricto was d

262 rotein expressions, leading to a decrease in ileal secretion of chloride, likely responsible for mass

263 In this study, surgically isolated ileal segments in newborn calves (n = 5) were used to es

264 After imaging, scanned ileal segments were analyzed ex vivo both for inflammati

265 was at least 70 times higher than any other ileal site.

266 RLC phosphorylation and force development in ileal smooth muscle appear to be dependent on MLCK and M

267 signaling pathways, induce inhibition of the ileal smooth muscle contractions, and affect distinct ph

268 RLC phosphorylation and force development in ileal smooth muscle depend on myosin light chain kinase

269 role of the MLCP regulatory subunit MYPT1 in ileal smooth muscle in adult mice with (1) smooth muscle

270 edicted to inhibit MLCP activity in isolated ileal smooth muscle tissues, with additional phosphoryla

271 increased mRNA and protein expression of the ileal sodium-dependent bile acid transporter (ISBT) in t

272 TGF-beta1, collagen, and CTGF production in ileal strictures.

273 Ileal substrate organic matter digestibility (OMD) incre

274 in the Probiotic group coincided with higher ileal T regulatory cells (Tregs) before and after challe

275 and villus epithelial cell lineages in human ileal tissue provides novel insights into the molecular

276 Ex-vivo mouse ileal tissue sections were treated with either EcN or th

277 We analyzed jejuno-ileal tissue specimens from 14 patients with familial SI

278 the Vi capsular antigen was detected in calf ileal tissue via fluorescence microscopy.

279 t, when supernatant recovered from untreated ileal tissue was pre-incubated with EcN, the derivative

280 Using cultures of human ileal tissue, we showed that the secretome of peripheral

281 evate 5-HT overflow when used to treat fresh ileal tissue.

282 s, T cells, and fibroblasts are also eQTL in ileal tissue.

283 Inflamed ileal tissues and PBMCs from patients with CD had lower

284 culture, and C-type lectins were assessed in ileal tissues by reverse transcriptase-quantitative poly

285 tome (mRNAs) and the microRNAome (miRNAs) of ileal tissues collected at one-month post-infection.

286 rt that NAP conjugate positive APCs in human ileal tissues from individuals with ulcerative colitis a

287 N is able to enhance 5-HT bioavailability in ileal tissues through interaction with compounds secrete

288 in reduced phosphorylation of S6 and AKT in ileal tissues, indicating inhibition of the mTOR complex

289 y-maximal force responses to EFS in isolated ileal tissues.

290 L-arginine or L-citrulline reduced levels of ileal transcripts encoding interleukin-4 (IL-4), a key m

291 intestinal reabsorption due to induction of ileal transporters (Slc10a2, Slc51a) and increases in wh

292 ty-six lesions were analyzed, including four ileal, two ileocecal, three cecal, three appendicular, a

293 ty for non-invasive assessment of pathologic ileal vascularization in the course of Crohn's disease.

294 l mucosal cellular immunity and that oral or ileal vector delivery for primary immunization facilitat

295 L-arginine deficiency blunts mastocytosis in ileal villi as well as bacterial translocation, measured

296 t of rapamycin complex 2 also contributes to ileal villus maintenance and goblet cell size.

297 Contrast-enhanced US of the ileal wall is a promising method for objective, reproduc

298 Ileal wall segments thicker than 3 mm were examined with

299 Ileal wall thickening was observed in all patients.

300 on as measured by changes in body weight and ileal wet weight.