ライフサイエンスコーパス: illegitimate recombination

1 duplicated blocks may have been affected by illegitimate recombination.

2 protect chromosome ends against fusions and illegitimate recombination.

3 letions post-polyploidization, and increased illegitimate recombination.

4 oval by unequal homologous recombination and illegitimate recombination.

5 te homologous recombination while preventing illegitimate recombination.

6 ansgene and genomic fragments recombined via illegitimate recombination.

7 me gene, but too much tolerance will lead to illegitimate recombination.

8 hort homology (microhomology), a hallmark of illegitimate recombination.

9 sms that control not only transposition, but illegitimate recombination.

10 eats, a unique signature of intrachromosomal illegitimate recombination.

11 hich may have contributed to DNA breakage or illegitimate recombination.

12 ementary ends can be joined by mechanisms of illegitimate recombination.

13 ntermediates in ERCC1- cells are repaired by illegitimate recombination.

14 ir of double-strand breaks by homologous and illegitimate recombination.

15 amplicon boundaries in 19 mutants reflected illegitimate recombination.

16 A and ligates nonhomologous ends, leading to illegitimate recombination.

17 causing chromosome aberrations by mediating illegitimate recombination.

18 outcomes, and to suppress inappropriate or 'illegitimate' recombination.

19 DNA incorporated into the transgene loci via illegitimate recombination; 50 of the 82 delivered DNA f

20 s for RecQ members in DNA metabolism and the illegitimate recombination and cancer-prone phenotypes a

21 le-strand breaks, is closely associated with illegitimate recombination and chromosomal rearrangement

22 pansion in plants, while DNA removal through illegitimate recombination and intrastrand homologous re

23 tween DNA double-strand break repair (DSBR), illegitimate recombination and plasmid DNA integration.

24 small insertion/deletions, thereby limiting illegitimate recombination and spontaneous mutation.

25 s of magnitude, driven by mechanisms such as illegitimate recombination and transposable element prol

26 ound evidence for important contributions of illegitimate recombination and transposable elements to

27 Duplications and deletions caused by illegitimate recombination and unequal crossing over wer

28 combinant T-DNA molecules were indicative of illegitimate recombination and were similar to left-bord

29 both the frequency of homologous as well as illegitimate recombination, and that RAD18 contributes t

30 te that unequal homologous recombination and illegitimate recombination are primarily responsible for

31 sms of DNA replication, gene correction, and illegitimate recombination at the Ori of PCV1, and it ma

32 diated double-strand breaks in meiosis cause illegitimate recombination between 11q23 and 22q11 resul

33 The deletions can be explained by illegitimate recombination between short (4- to 15-bp) e

34 is an inversion of chromosome 10 mediated by illegitimate recombination between the RET and the H4 ge

35 es can mediate DNA recombination and promote illegitimate recombination by catalyzing the ligation of

36 ude that hPot1 protects chromosome ends from illegitimate recombination, catastrophic chromosome inst

37 DNA end joining is a type of illegitimate recombination characterized by the joining

38 is genomic instability typified by elevated illegitimate recombination events and accelerated loss o

39 Moreover, illegitimate recombination events appear to be an import

40 the tip of the putative hairpin, leading to illegitimate recombination events between similar AT-ric

41 marker system to screen for intrachromosomal illegitimate recombination events in order to assess the

42 We propose that illegitimate recombination events leading to inverted du

43 and HR, shifting the cellular milieu toward illegitimate recombination events such as iHR and CN-LOH

44 is of the relative numbers of homologous and illegitimate recombination events suggests that C. glabr

45 eplication, chimeric gene formation, and the illegitimate recombination events that lead to stoichiom

46 e find that YKU80 plays an essential role in illegitimate recombination events that result in the acc

47 de and replace genomic DNA through two joint illegitimate recombination events.

48 This motif was shown to be a target for illegitimate recombination events.

49 3)] were isolated as the apparent result of "illegitimate" recombination events on intrahelical pseud

50 homologous recombination and high levels of illegitimate recombination found in the tubercle bacillu

51 Illegitimate recombination fused normally distant chromo

52 through unequal homologous recombination and illegitimate recombination have attenuated the growth of

53 ylation and their removal from the genome by illegitimate recombination have been well documented, th

54 ared boundary sequences of module junctions, illegitimate recombination in a non-sequence-directed pr

55 provides compelling evidence for the role of illegitimate recombination in horizontal genetic exchang

56 ions or other target DNA sites implicated in illegitimate recombination in mammalian cells.

57 However, large-scale evaluation of illegitimate recombination in plant genomes has not been

58 appears to be involved in the suppression of illegitimate recombination in plant mitochondria.

59 Our data strongly suggest that illegitimate recombination in plants is mediated by a DN

60 ontributions of homologous recombination and illegitimate recombination in the repair process.

61 verexpression of the topoisomerase I gene on illegitimate recombination in the yeast Saccharomyces ce

62 ence and structure-specific requirements for illegitimate recombination in tobacco.

63 hows that these activities together suppress illegitimate recombination in vivo, whereas unregulated

64 We designed substrates representing illegitimate recombination intermediates formed when a d

65 omosomal homology, the plasmid integrated by illegitimate recombination into random sites in the geno

66 edominantly reflected microhomology mediated illegitimate recombination involving short complementary

67 n events near the IS1236 elements arise from illegitimate recombination involving transposase-mediate

68 This suggests that random DNA breaks attract illegitimate recombination (IR) events that compete with

69 gh unequal homologous recombination (UR) and illegitimate recombination (IR) is proposed to be the ma

70 r direct DNA delivery methods occurs through illegitimate recombination (IR).

71 that removal of retrotransposon sequences by illegitimate recombination is also operating more slowly

72 These results suggest that illegitimate recombination is an important competing pat

73 Sequence analysis suggested that illegitimate recombination is nonrandom at the single-ge

74 Our results indicate that illegitimate recombination is the driving force behind g

75 Illegitimate recombination is the prevailing molecular m

76 We propose that illegitimate recombination, not positive selection, has

77 Nested insertions and illegitimate recombination occurred extensively between

78 erted repeat correction (or conversion), and illegitimate recombination of any circular DNA molecule

79 Illegitimate recombination of fragments that encode prot

80 A model is proposed in which the illegitimate recombination of the cps island into the ga

81 he frequency of transformation due to random illegitimate recombination of transfected DNA into the g

82 operon and could have moved there either by illegitimate recombination or more plausibly via integra

83 ought to stimulate homologous recombination, illegitimate recombination, or both in mammalian cells.

84 induce a genome-wide microhomology-mediated illegitimate recombination pathway that facilitates inte

85 or two distinct and evolutionarily conserved illegitimate recombination pathways.

86 ependent events both mediated by independent illegitimate recombination processes.

87 In 12 illegitimate recombination products analysed, we found t

88 quired from the host bacterial chromosome by illegitimate recombination, providing further evidence t

89 ated by its slow growth and its high rate of illegitimate recombination relative to homologous DNA ex

90 The mechanism, short-patch double illegitimate recombination (SPDIR), facilitates short si

91 rtant implications in terms of mechanisms of illegitimate recombination that can result in chromosoma

92 years, the two chromosomes have experienced illegitimate recombination that has been temporally rest

93 these small fragments provided evidence that illegitimate recombination was most likely mediated by a

94 ogous recombination about 100-fold; however, illegitimate recombination was stimulated more than 1,00

95 equal homologous recombination compared with illegitimate recombination were highly variable between

96 evolutionary innovations depend much more on illegitimate recombination, which makes novel genes by g

97 ITRs in the helper plasmid were involved in illegitimate recombination with AAV ITRs, deletions of w

98 Illegitimate recombination within direct pentameric DNA

99 DNA double-strand breaks can be repaired by illegitimate recombination without extended sequence hom