ライフサイエンスコーパス: immune abnormality

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1 s strongly correlated with neurohormonal and immune abnormalities.

2 to AIDS, organ transplantation or congenital immune abnormalities.

3 ment strategy to compensate for these innate immune abnormalities.

4 t is characterized by serologic and cellular immune abnormalities and is dependent on the presence of

5 ck of or defective hematopoietic stem cells, immune abnormalities, and disorders of the bone marrow m

6 fants may be capable of producing thymic and immune abnormalities, as suggested by previous reports o

7 odels of lupus has provided insight into the immune abnormalities associated with autoantibody produc

8 ment of barrier abnormalities and downstream immune abnormalities during the elicitation phase of mur

9 pic march." Controversy exists as to whether immune abnormalities, epidermal barrier defects, or both

10 he immune system, as evidenced by the severe immune abnormalities exhibited by patients bearing inact

11 Immune abnormalities have been described in some individ

12 and anxiety-like behaviors, suggesting that immune abnormalities in MIA offspring can contribute to

13 rointestinal tract and to define the mucosal immune abnormalities in patients with and without sympto

14 emerges as a therapeutic target in resetting immune abnormalities in RA.

15 Converging evidence implicates immune abnormalities in schizophrenia (SCZ), and recent

16 ormation will help to elucidate the cellular immune abnormalities leading to production of pathogenic

17 tory conditions suggests a common underlying immune abnormality leading to both conditions.

18 st in investigating the impact of early-life immune abnormalities on later onset of psychosis.

19 cells, we tested the hypothesis that ocular immune abnormalities participate in PG phenotypes.

20 Melanosomal defects along with ocular immune abnormalities play a role in the propagation of p

21 This demonstrates that some of the immune abnormalities previously identified in DBA/2J mic

22 background AD might be explained by baseline immune abnormalities, such as increased TH2, TH17, and n

23 /TSP and underlie many of the characteristic immune abnormalities, such as spontaneous lymphocyte pro

24 rder (PTSD) is associated with endocrine and immune abnormalities that could increase risk for autoim

25 how that if immune biomarkers exist for such immune abnormality, they may be found in raised macropha

26 All of the cellular immune abnormalities were reversed by day 360 in abstine

27 er p21 Ras oncoproteins by causing selective immune abnormalities without general developmental defec

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