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1 njury remains challenging due to circulatory immune clearance.
2 nd resensitizing the pathogen to host innate immune clearance.
3 oevasive strategy used by the virus to avoid immune clearance.
4 C3-coated particles, a function critical for immune clearance.
5 sponse, employs multiple mechanisms to evade immune clearance.
6 ivating inflammation and by interfering with immune clearance.
7 sues, adhesion to host cells, and evasion of immune clearance.
8 CR1, and suggest that C1q may participate in immune clearance.
9 contribute to the ability of HCMV to escape immune clearance.
10 nce represent functional mechanisms to evade immune clearance.
12 ariation allows microbial pathogens to evade immune clearance and establish persistent infection.
13 igenically variant surface proteins to evade immune clearance and establish persistent infection.
14 m used by bacterial pathogens to escape host immune clearance and establish persistent infection.
15 critical functions of its host cell to avoid immune clearance and gain access to nutritional resource
16 rferi to determine the role of complement in immune clearance and joint histopathology during experim
17 display no additional properties related to immune clearance and persistence within relevant hosts.
18 onized particles and provide a mechanism for immune clearance and phagocytosis of IgG-coated particle
20 rier, the use of stealth coatings can reduce immune clearance and prolong circulatory times, thereby
21 developed a sophisticated machinery to evade immune clearance and promote survival in the infected ce
23 hesized that individual TprK variants escape immune clearance and seed new disseminated lesions to ca
24 lly relevant model of HBV infection to study immune clearance and test therapeutic and curative appro
25 ate immune function allows them to undermine immune clearance and thereby increase their persistence
26 axis allowed it to escape IL-12p70-dependent immune clearance and to cause inflammatory bone loss in
27 d core protein of wild-type genotype C delay immune clearance and trigger the subsequent emergence of
28 associated with failures of T-cell-mediated immune clearance and with abnormal B-cell growth and act
29 fect the gastric mucosal surface, evade host immune clearance, and cause peptic ulcer disease or gast
30 fection in the host, most likely in avoiding immune clearance by cytotoxic lymphocytes and natural ki
32 The virus has evolved strategies to escape immune clearance by remaining latent in most infected ce
33 tance factor, which is involved in resisting immune clearance, contributes to E faecalis fitness.
35 80 should render the parasite susceptible to immune clearance from the host by both providing a focus
36 as consisting of 4 phases: immune tolerance, immune clearance [hepatitis B e antigen (HBeAg)-positive
37 g successfully developed mechanisms to evade immune clearance, hepatitis C virus (HCV) establishes pe
38 sions and Relevance: Increased resistance to immune clearance in an ESBL E coli lineage already known
39 the ability of P. aeruginosa to avoid innate immune clearance in mammals is a type III secretion syst
40 t mice to examine the role of FcgammaRIIA in immune clearance in the absence of functional FcgammaRI
42 sized that this flagellated pathogen escapes immune clearance, in part, by avoiding detection by the
43 vel strategy for Salmonella evading the host immune clearance, in which Salmonella produce microRNA-l
44 robably require potent suppression of innate immune clearance mechanisms (e.g., by replacement of ade
45 isease pathogenesis by interfering with host immune clearance mechanisms or by exhibiting pro-inflamm
48 egs, protect breast cancer cells (BCCs) from immune clearance MSCs suppressed the proliferation of PB
49 nes and decrease proapoptotic genes, improve immune clearance of Abeta, and are associated with an im
50 mitochondrial depolarization, apoptosis, and immune clearance of cancer cells by antitumor leukocytes
53 mber of groups have investigated whether the immune clearance of infection can be achieved through a
56 or more of the following processes: adaptive immune clearance of merozoites and parasitised red blood
57 he cytokine interferon (IFN)-gamma regulates immune clearance of parasitic, bacterial, and viral infe
59 ing evidence for a role of chemokines in the immune clearance of rickettsiae from the vasculature.
61 ntly increase as infection progresses toward immune clearance of the pathogen, suggesting a role for
62 stabilised luciferase expression, suggesting immune clearance of the transfected cells occurs in immu
68 levels are associated with the course of the immune-clearance phase in chronic HBV infection, and are
71 proportions of these cells, T-cell-dependent immune clearance relies on them, and abundance of their
72 responses, Borrelia burgdorferi often evades immune clearance through largely unknown mechanisms.
73 er investigate the mechanism of BcfA-induced immune clearance, we examined the role of neutrophils an
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