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1 local C3 production in a model of glomerular immune complex disease.
2 g/kg) were minimal and related primarily to immune complex disease.
3 otypic abnormalities, including infection or immune complex disease.
4 ular permeability during the early phases of immune complex disease.
5 a mechanism for heritable susceptibility to immune complex disease.
6 angial glomerulopathy into a severe systemic immune complex disease.
7 tibody and circulating immune complexes, and immune complex disease.
8 died within 3 months of age due to AIDS and immune-complex disease.
9 e and in potential involvement in glomerular immune complex diseases.
10 ermine the mechanism by which CRP suppresses immune complex disease, an adoptive transfer system was
11 ons for understanding the pathophysiology of immune complex diseases and for optimizing the efficacy
12 munosuppression, virus-induced autoimmunity, immune complex disease, and virus-lymphocyte and virus-d
13 development of lymphadenopathy or pathogenic immune-complex disease, as assayed by cutaneous, renal,
15 ssociated with circulating immune complexes, immune complex disease, hypergammaglobulinemia, and high
18 pontaneous lupus, as well as the rapid-onset immune complex disease induced in the accelerated nephro
19 Lupus glomerulonephritis is a prototype of immune complex disease mediated by autoantibodies of mul
21 t circulating C3 developed severe glomerular immune complex disease, whereas those with a high level
22 s in neutrophils is critical for stimulating immune complex disease while Vav- and Rac-independent pa
23 Systemic lupus erythematosus is a prototype immune complex disease with circulating autoantibodies t
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