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1 ice prevented proteinuria and reduced kidney immune complex formation.
2 eserve CD4(+) T cell memory independently of immune complex formation.
3 une and inflammatory responses via localized immune complex formation.
4 , oligoclonal CD4(+) T cell infiltrates, and immune complex formation.
5 by mediating kidney damage via antibody-DNA immune complex formation.
6 RA patients were also elevated, which led to immune complex formation.
7 get organs and acting as a nidus for in situ immune complex formation.
8 bility to undergo Fab-arm exchange and limit immune complex formation.
9 ssociated with adverse effects suggestive of immune complex formation.
10 MOG cross linking on oligodendrocytes and/or immune complex formation.
12 hibitory KIR with cognate ligands, promoting immune complex formation and NK-cell cytotoxicity specif
13 l inflammation in SLE are closely linked via immune complex formation and systemic complement depleti
15 ata provide a mechanism explaining increased immune-complex formation and disease exacerbation during
16 er characterized by autoantibody production, immune-complex formation, and altered cytokine expressio
17 ntially injurious inflammatory reactions via immune complex formation associated with phagocytosis an
18 dy-mediated brain disease does not depend on immune complex formation but rather on antibody-mediated
20 elopment of antibodies to FCS indicates that immune complex formation could have occurred after the c
21 s on Fcgamma receptor and the requirement of immune complex formation for effector cell activations.
22 ximal tubule antigen-specific antibodies and immune complex formation has not been well characterized
23 ell-driven antibody production that leads to immune complex formation in glomeruli, complement activa
24 glomerular antigens, leading subsequently to immune complex formation in situ and complement activati
25 may be a promising approach to down-regulate immune complex formation in the target organ in individu
26 nd allows the calculation of the kinetics of immune complexes' formation in a continuous-flow system
27 odels of pathogenesis usually assume in-situ immune-complex formation involving an as yet uncharacter
30 min to 3.5 h), different kinetic profiles of immune complex formation of defined geometry were docume
31 In this article, we provide evidence that immune complex formation of serum IgA plays an important
32 odifications, namely aggregation, glycation, immune complex formation, proteoglycan complex formation
33 ntibody immobilization, analyte capture, and immune complex formation with a second biotinylated anti
34 ngly through effects on NK-cell trafficking, immune complex formation with MM cells, and cytotoxicity
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