ライフサイエンスコーパス: immune disease

1 d in cancer, cardiac, neurodegenerative, and immune disease.

2 bnormal cells in infection, cancer, or other immune disease.

3 owledge has rapidly been translated to human immune disease.

4 nd that decreased Foxp3 expression can cause immune disease.

5 variety of animal models of inflammatory and immune disease.

6 ion of advances in our armamentarium against immune disease.

7 nced understanding of human inflammatory and immune diseases.

8 iety of diseases ranging from cancer to auto-immune diseases.

9 ant IRF3-dependent gene expression linked to immune diseases.

10 ncies, hematological deficiencies, and other immune diseases.

11 seases such as diabetes and inflammatory and immune diseases.

12 es encoding these proteins that give rise to immune diseases.

13 nvariant NKT cells, as well as its impact on immune diseases.

14 gets to treat GVHD and other T-cell-mediated immune diseases.

15 therapeutic potentials for neurological and immune diseases.

16 naling pathways regulating cell migration in immune diseases.

17 and striking association with immune and non-immune diseases.

18 of molecular events underlying vascular and immune diseases.

19 nhibitory molecule for T cell activation and immune diseases.

20 response and pathogen-induced T-cell-driven immune diseases.

21 function and the aetiology of various human immune diseases.

22 paB) for its involvement in inflammatory and immune diseases.

23 ications in therapeutic treatment of various immune diseases.

24 cancers but also for genetic, infectious and immune diseases.

25 in the IL-10 promoter and has been linked to immune diseases.

26 novel target for therapeutic intervention of immune diseases.

27 in novel preventative strategies for chronic immune diseases.

28 o our understanding of immune mechanisms and immune diseases.

29 and are essential for the prevention of auto-immune diseases.

30 isms and contribute to autoantibody-mediated immune diseases.

31 M-1) has been implicated in inflammatory and immune diseases.

32 some also show linkage to other inflammatory immune diseases.

33 n the etiology of cardiovascular, renal, and immune diseases.

34 the hypothesis that myositis may be an 'allo-immune' disease.

35 ar Kv1 antibodies mainly associated with non-immune disease aetiologies, poor longitudinal clinical-s

36 Uveitis is a systemic immune disease and a common cause of blindness.

37 and dysregulation is frequently observed in immune disease and cancer.

38 tibility loci occurs between different human immune diseases and by comparing conserved regions with

39 iduals, might also be relevant in other auto-immune diseases and cancer.

40 otrienes, inflammatory mediators involved in immune diseases and defense.

41 ender rats well suited for reproducing human immune diseases and evaluating therapeutic strategies, d

42 T cell activation, which has implications in immune diseases and immunotherapy.

43 ammation and tissue damage in IL-23-mediated immune diseases and it suggests the GM-CSF-eosinophil ax

44 e levels in predisposition and expression of immune diseases and on mechanisms of glucocorticoid effe

45 We investigated immune diseases and the risk of classical HL in a popula

46 an diseases, including neurodegenerative and immune diseases, and cancer.

47 in resistance, obesity, cardiovascular risk, immune diseases, and even mood and behavior.

48 optosis may cause diseases, such as cancers, immune diseases, and neurodegenerative disorders.

49 been implicated in the pathology of cancers, immune diseases, and neuronal disorders.

50 Concurrent immune diseases are common, and immunoglobulin A nephrop

51 transformative for kinase inhibitors in auto-immune diseases, as several inhibitors have finally prog

52 c approach to modulate immune recognition in immune disease beyond HIV.

53 Thus, decreased Foxp3 expression causes immune disease by subverting the suppressive function of

54 n of IRAK1 and type I interferons in various immune diseases, controlling IRAK1 activation via inhibi

55 DRB4*0103 is associated with concurrent immune diseases, DRB1*0301 with a poor treatment respons

56 ng bacterial genera are also associated with immune diseases in humans, laying a path for future stud

57 that PIDs can also predispose to cancer and immune diseases, including allergy, autoimmunity, and in

58 y a disorder of lipid metabolism but also an immune disease involving CD1.

59 Atherosclerosis is a chronic inflammatory/immune disease involving multiple cell types including m

60 However, the mechanism by which SHED treat immune diseases is not fully understood.

61 zation of molecular trait QTLs at 345 unique immune disease loci.

62 autoimmune hepatitis by male sex, concurrent immune diseases, lower serum gamma-globulin and immunogl

63 ave been found to be elevated in a number of immune disease models.

64 has been found to be elevated in a number of immune disease models.

65 nserved regions with experimental autoimmune/immune disease models.

66 Translational opportunities for immune disease monitoring are driving the rapid developm

67 For old men with an inflammatory or immune disease (n=10), mean old to young ratios of T-cel

68 nventional T cells, which contributed to the immune diseases observed in these mice.

69 ontrol of this potentially debilitating auto-immune disease of the central nervous system.

70 Psoriasis is a chronic inflammatory immune disease of the skin characterized by a complex in

71 o ablate the immune system in viral-mediated immune diseases or virus-initiated autoimmune disease ma

72 site of inflammation as a result of chronic immune diseases, pathogenic infection, and tissue injury

73 aggregation and has been implicated in auto-immune disease pathology.

74 y, we validated our method against the human immune disease pathway.

75 afflicted with certain rheumatological auto-immune diseases produce autoantibodies directed against

76 ppreciated following the genotyping of large immune disease sample sets on a shared SNP array: the 'I

77 A comparative analysis with 15 immune diseases showed that T1D is more similar genetica

78 One obstacle precluding the development of immune-disease specific "humanized" mice is that optimal

79 eceptor (TCR) that is critically involved in immune diseases such as autoimmune arthritis.

80 Chronic immune diseases, such as asthma, are highly prevalent.

81 herapeutic implications for the treatment of immune diseases, such as autoimmunity and allergy.

82 y region, while BATF and ESRRA overlap other immune disease susceptibility regions, validating our ap

83 ine adjuvant development and inflammatory or immune disease therapy.

84 specific subfunctions of Treg cells and the immune diseases they regulate can be influenced by FoxP3

85 We collected information on immune diseases through telephone interviews and perform

86 The comorbidities among the five immune diseases were best explained by biological pleiot