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1 recruitment in vivo (e.g., in B cell-related immune disorders).
2 ned as an allergic Th2-mediated inflammatory immune disorder.
3 F-alpha found in these patients suggested an immune disorder.
4 th development of childhood asthma and other immune disorders.
5 fficacy and underlying mechanisms of MSCs in immune disorders.
6 rincipal driver of multiple inflammatory and immune disorders.
7 ies targeting Bregs for better management of immune disorders.
8 ses, including cancer, neurological and auto-immune disorders.
9 therapies can be applied to treat additional immune disorders.
10 buting to risk, with some loci shared across immune disorders.
11 ontributing risk factor for autoimmunity and immune disorders.
12 for high-risk hematological malignancies and immune disorders.
13 NKG2D and the pathogenesis of organ-specific immune disorders.
14 e effective in the treatment of several auto-immune disorders.
15 athogenesis of various inflammatory and auto-immune disorders.
16 s may lead to new therapeutic strategies for immune disorders.
17 ceptibility to several inflammatory and auto-immune disorders.
18 to new therapies for intestinal and systemic immune disorders.
19 ssociated extraintestinal manifestations and immune disorders.
20 s suis can be beneficial in treating various immune disorders.
21  facilitate the therapeutic use of iTregs in immune disorders.
22  therapeutic agent specific for CTL-mediated immune disorders.
23  novel therapeutic strategy for CTL-mediated immune disorders.
24 eles are functionally significant in complex immune disorders.
25 s cancers, human immunodeficiency virus, and immune disorders.
26 otential therapeutic target for Treg-related immune disorders.
27 ctions in other intestinal and nonintestinal immune disorders.
28 nvolved in the pathogenesis of XLP and other immune disorders.
29 ditions such as liver diseases, diabetes and immune disorders.
30 studies of antiadhesion therapies in various immune disorders.
31 egard to both the initiation and severity of immune disorders.
32 est priority targets for study in other auto-immune disorders.
33 -17 cells) and has been linked to many human immune disorders.
34 or therapeutic intervention in metabolic and immune disorders.
35 uals or the implications of such for chronic immune disorders.
36 lipids and to reduce inflammation in certain immune disorders.
37  Foxp3 expression in T(r) cells with various immune disorders.
38 w therapy for the treatment of several human immune disorders.
39 has been associated with numerous cancer and immune disorders.
40 in transplantation and other T cell mediated immune disorders.
41 a constellation of rare, autosomal recessive immune disorders.
42                                              Immune disorders affecting this pathway are characterize
43 d has been implicated in the pathogenesis of immune disorders and cancer.
44 nditions such as respiratory illnesses, auto-immune disorders and hepatitis.
45 est a wide range of clinical applications in immune disorders and in certain types of cancer.
46 omas in individuals with acquired and innate immune disorders and is strongly associated with Hodgkin
47 ed as a serum marker of T cell activation in immune disorders and of tumor burden in Tac-expressing m
48 atic level) occurs in the context of various immune disorders and plasma cell neoplasia.
49 urthermore, the broad spectrum of underlying immune disorders and the type of graft represent importa
50 ociated with various human diseases, such as immune disorders and tumorigenesis.
51 al immunity, vaccine development strategies, immune disorders, and drug efficacy.
52 tal cytokine linking innate and Th2 adaptive immune disorders, and is upregulated by environmental po
53 ust be tightly controlled to prevent cancer, immune disorders, and other diseases.
54 genases (LO) have been implicated in asthma, immune disorders, and various cancers.
55 ital roles in the immune system, and several immune disorders are associated with disturbances of the
56                                              Immune disorders are common; immunosuppression during ac
57                 Allergic airway diseases are immune disorders associated with heightened type 2 immun
58 s finding opens new avenues for treatment of immune disorders based on selective targeting of activat
59 ere combined immunodeficiency (X-SCID) is an immune disorder caused by mutations in the IL2RG gene an
60 hronic granulomatous disease (CGD), a severe immune disorder characterized by the inability of phagoc
61 s a rare, rapidly fatal, autosomal recessive immune disorder characterized by uncontrolled activation
62  of Usp21 specifically in Treg cells display immune disorders characterized by spontaneous T-cell act
63  lymphoproliferative disease (XLP) is a rare immune disorder commonly triggered by infection with Eps
64            In patients with malignancies and immune disorders expressing Tac (alpha chain of the inte
65 igodendrocytes, may develop in patients with immune disorders following reactivation of chronic benig
66 mammalian CLR, in pneumonic sepsis, a deadly immune disorder frequently associated with a nonresolvin
67       High-throughput TCR sequencing of rare immune disorders has demonstrated that quantitative TCR
68 kage searches of autoimmune and inflammatory/immune disorders have identified a large number of non-m
69 em cells (HSCs) from patients with monogenic immune disorders have not been reported.
70 ent cytotoxicity led to the life-threatening immune disorder hemophagocytic lymphohistiocytosis (HLH)
71 rtant role in the most frequent drug-induced immune disorder, heparin-induced thrombocytopenia (HIT).
72  1 led to failed Treg homeostasis and lethal immune disorder in mice.
73 dings that CD is primarily a T cell-mediated immune disorder in which CD4(+) T cells that recognize g
74 ugh successful therapies have been found for immune disorders in animal studies, few have passed the
75                      The presence of various immune disorders in families with several members with m
76 NSTs are linked to several developmental and immune disorders in humans, and in pathogenic microbes t
77 ld establish therapeutic targets for several immune disorders in which estrogens play a prominent rol
78 that has been reported to be associated with immune disorders, in the modulation of carotid vasodilat
79 T) lymphocytes underlie various inflammatory immune disorders including autoimmunity.
80  and might be developed for the treatment of immune disorders, including inflammatory bowel diseases.
81 ons including oculocutaneous telangiectasia, immune disorders, increased susceptibly to cancer and re
82 orary thought holds that asthma is a complex immune disorder involving innate as well as adaptive imm
83 une development and response and can lead to immune disorders like autoimmunity and cancer.
84 hogens found in patients with AIDS and other immune disorders, N-[(2,4-diaminopteridin-6-yl)methyl]di
85 tigen (HLA) and non-HLA genes and with other immune disorders, notably juvenile diabetes and thyroid
86 -gamma are implicated in the pathogenesis of immune disorders of the central nervous system (CNS).
87 ) may have a role in modulating allergic and immune disorders of the skin.
88 different causes, including inflammatory and immune disorders, oxidative stress, and nephrotoxins, am
89 1 psychiatric diagnoses or cardiovascular or immune disorders performed a functional neuroimaging tas
90 IL-2 as a therapeutic agent for a variety of immune disorders ranging from AIDS to cancer.
91 inked to various metabolic, inflammatory and immune disorders, regulatory proteins controlling macrop
92 fector functions in the pathogenesis of this immune disorder remains unclear.
93                 In contrast with pleiotropic immune disorders reported in obese mice with leptin or L
94 wing evidence that rituximab has efficacy in immune disorders resulting from autoantibody formation.
95 pportunities to develop future therapies for immune disorders such as autoimmunity and cancer.
96 s are widely used agents in the treatment of immune disorders such as rheumatoid arthritis and inflam
97 lay a role in the initiation of Th2-mediated immune disorders, such as asthma.
98 hat may be exploited in treatment of chronic immune disorders, such as multiple sclerosis and inflamm
99 ommon variable immunodeficiency (CVID) is an immune disorder that not only causes increased susceptib
100 enetically heterogeneous autosomal recessive immune disorder that results when the critical regulator
101  gut microbiota dysbiosis is associated with immune disorders, the underlying mechanism remains unkno
102        Although wasted mice display a severe immune disorder, they express normal levels of NFAT1 pro
103 ota, known as dysbiosis, can trigger several immune disorders through the activity of T cells that ar
104   A connection between OST dysregulation and immune disorders was demonstrated in Trex1(-/-) mice, TR
105              Missense mutants that cause the immune disorder Wiskott-Aldrich Syndrome (WAS) map prima
106  patients with systemic scleroderma, an auto-immune disorder with clinical fibrotic features.
107  Tec family member Btk, cause the hereditary immune disorder, X-linked agamaglobulinemia.
108 ucosal surface is illustrated by the primary immune disorder, X-linked agammaglobulinemia in which pa

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