ライフサイエンスコーパス: immune factor

1 ssociated with higher concentrations of milk immune factors.

2 milk but also by increasing delivery of milk immune factors.

3 eases milk concentrations of certain soluble immune factors.

4 iciency (Imd), JAK/STAT, and Leu-rich repeat immune factors.

5 eals, central nervous system activation, and immune factors.

6 imiting permissive macrophages and on innate immune factors.

7 DefA) coding region, one of the major insect immune factors.

8 ion does not increase milk concentrations of immune factors.

9 However, it is not clear how immune factors affect ER stress and therefore disease sy

10 py; and, 4) investigations of immune and non-immune factors affecting late cardiac allograft outcomes

11 ed ocular cellular, humoral, and nonspecific immune factors allowing viral DNA to persist for slightl

12 mechanisms for bacterial resistance to host immune factors and antibiotics.

13 llectively, these findings suggest that host immune factors and bacterial persisters may directly int

14 NEC with regard to the involvement of innate immune factors and histopathological changes as compared

15 dding IL-25 to PN would restore these innate immune factors and maintain barrier function.

16 ed on identification of relevant genetic and immune factors and understanding serum and hepatic facto

17 ficiency virus (HIV)-endothelial cells, host immune factors, and cytokines are described.

18 d with a local deficiency of multiple innate immune factors, and this deficiency could predispose ind

19 Our results suggest that host immune factors are able to affect bacterial disseminatio

20 thies are changing, as the various toxic and immune factors are modified by new treatment strategies.

21 Immune factors are required for muscle regeneration, but

22 that the mechanism of resistance may involve immune factors, as strains subjected to whole-body irrad

23 Here, we analyzed the immune factors associated with changes in proviral HIV-1

24 ctivities were measured to examine potential immune factors associated with differences in susceptibi

25 Comprehensive analyses of host, viral, and immune factors associated with severe respiratory syncyt

26 ost defense against mucosal candidiasis, the immune factors associated with susceptibility to OPC in

27 ere were no significant treatment effects on immune factors at 3 mo.

28 during systemic spread and killing by innate immune factors at intestinal mucosal surfaces.

29 ostrum contains the highest concentration of immune factors, being the most potent immune booster kno

30 To determine whether immune factors can effect amyloid removal, we developed

31 attempt to delineate the mechanisms by which immune factors can lead to the loss of connective tissue

32 y (defined as an elevated Na:K) and baseline immune factor concentrations were controlled for, there

33 d the elucidation of genetic, immune and non-immune factors contributing to the development of this i

34 Multiple immune factors control host responses to Mycobacterium t

35 However, immune factors determining whether individuals control i

36 transcription factors Dorsal/Dorsal-related immune factor (Dif), was first identified in Drosophila.

37 tion factors, Dorsal (Dl) and Dorsal-related immune factor (Dif).

38 t advances indicate that innate and adaptive immune factors drive the development of diverse subsets

39 mans, less is understood regarding lung host immune factors during early stages of fungal infection.

40 Crucial immune factors emerge that affect macrophage populations

41 lated to lipid metabolism, tyrosine kinases, immune factors, extracellular matrix components, steroid

42 k between a T2S system and the modulation of immune factors following intracellular infection.

43 coring the dependence on different mammalian immune factors for control of mucosal versus systemic Ca

44 tion for transfusion-dependent patients, but immune factors governing risk for alloimmunization are u

45 ve viral strain, while others suggested host immune factors have a key role.

46 Likewise, levels of TGF-beta1 or other immune factors in brains of AD patients may influence th

47 Maternal vaccination to induce anti-HIV immune factors in breast milk is a potential interventio

48 This suggests that immune factors in breast milk of HIV-1-infected mothers

49 ly enhance the expression of these intrinsic immune factors in HIV-1-infected individuals.

50 cle reviews current knowledge of the role of immune factors in inflammatory bowel disease and the pot

51 Conversely, new findings implicate non-immune factors in inflammatory myopathy pathogenesis.

52 ecome infected, raising the possibility that immune factors in milk inhibit mucosal transmission of H

53 ral SIV host species, such as innate SIV/HIV immune factors in milk, as a means of naturally preventi

54 esting the existence of naturally protective immune factors in milk.

55 To determine the role of viral and immune factors in monocyte penetration across the BBB, H

56 To explore the host immune factors in selection of the emergence of this new

57 milk exposure, raising the possibility that immune factors in the breast milk confer protection to t

58 he transmission rate is low, suggesting that immune factors in the breast milk of HIV-1-infected moth

59 Additional support for the role of altered immune factors in the etiology of schizophrenia comes fr

60 against the HMW1 and HMW2 adhesins and other immune factors in the lower respiratory tracts of patien

61 nalysed the genetic, microenvironmental, and immune factors in tumours derived from a transgenic brea

62 y inactive cells, and direct effects of host immune factors (in the absence of immune cells) on persi

63 om inevitable destruction by cellular innate immune factors including the APOBEC3 DNA mutating enzyme

64 ins and the production of viral and cellular immune factors injurious to human neurons.

65 , our understanding of the functions of many immune factors involved in fighting infection is limited

66 rus-induced increased expression of numerous immune factors involved in myeloid-derived suppressor ce

67 The review discusses immune factors involved in the response to vascular inju

68 levant in light of the increasing reports of immune factors involved in vascular disease and interven

69 bits innate immune responses by targeting an immune factor, IRF7.

70 ly and genotypically distinct cohort, a host immune factor is highly associated with restriction of v

71 ssion is the greatest risk factor, recipient immune factors likely also play a role in the pathogenes

72 Although a variety of downstream immune factors likely regulate inhibitor formation, the

73 tant protein A (SP-A) as an important innate immune factor of the lungs, amniotic fluid, and the vagi

74 To examine the effect of individual host immune factors on MeV load dynamics further, we develope

75 To identify immune factors present during the acute rash phase of me

76 tion of a long-term association between host immune factors, 'red complex' pathogens and periodontal

77 annose-binding lectin (MBL) is a circulating immune factor responsible for opsonization of pathogens

78 These results imply the importance of immune factors such as cytokines and autoantibodies in d

79 erium must circumvent killing by host innate immune factors, such as cationic antimicrobial peptides

80 Interestingly, we found that innate immune factors, such as natural killer cells, plasmacyto

81 Expression of 15 immune factors (TGFbeta, IL-1beta, IL-4, IL-6, IL-10, GM

82 Apobec3/Rfv3 is an innate immune factor that promotes the neutralizing Ab response

83 ain-containing protein 1 (SAMHD1), an innate immune factor that suppresses HIV replication, was ident

84 ompletely understood, particularly regarding immune factors that are harmful rather than protective t

85 asure the concentrations of innate pulmonary immune factors that are important in lung defense agains

86 surface recognition ligands as well as host immune factors that bind sialic acid.

87 in mice, we discuss the innate and adaptive immune factors that control CHIKV dissemination and clea

88 The immune factors that control DEN infection or contribute

89 The chemokines CCL21 and CXCL13 are immune factors that dictate homing and motility of lymph

90 However, the immune factors that drive the formation of a protective

91 nomodulators is highlighted by the number of immune factors that have been associated with increased

92 This model reveals important immune factors that influence susceptibility to infectio

93 treatment, it is vital that we identify the immune factors that limit virus spread in the heart and

94 s in understanding early immune events, oral immune factors that modulate HIV/SIV susceptibility (inc

95 f enteric viruses, little is known about the immune factors that restrict persistent enteric infectio

96 or to the MFA, since there may be additional immune factors that synergize the transmission-blocking

97 nsplant microenvironment as well as the host immune factors that underlie the pathophysiology of poly

98 Among host immune factors, the cytokine TNF-alpha has previously be

99 Enhancement of these protective immune factors through maternal vaccination will be a cr

100 The increasing need to identify immune factors to devise host-targeted therapies against

101 Much work has focused on the contribution of immune factors to the pathogenesis of hemorrhage, but ho

102 e must overcome numerous innate and adaptive immune factors, traverse the genital epithelium, and est

103 kely to be heterogenous and may involve host immune factors, viral factors, or both.

104 Additional transfusion and patient immune factors were examined.

105 Cytokines, chemokines, and other soluble immune factors were measured in longitudinal samples fro

106 pylori, Streptococcus pneumoniae, and innate immune factors were not lower among HIV-infected women t

107 some nutrients can modulate inflammatory and immune factors, which are altered in women with endometr

108 of interferon regulatory factor 7 (IRF7), an immune factor with a pivotal role in viral pathogenesis.

109 sum, Sec13 regulates specific expression of immune factors with key functions in inflammation.