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1 ng the cagA-dependent genes, and many of the immune response genes.
2 ontrolling the expression of a wide range of immune response genes.
3 r occupancy is translated into activation of immune response genes.
4 ins, and for the activation of antibacterial immune response genes.
5 activates the transcription of antibacterial immune response genes.
6 ch directs expression of innate and adaptive immune response genes.
7 e it participates in the activation of early immune response genes.
8 cal type 2 genes and decreased expression of immune response genes.
9 t2 could lead to 5hmC alteration at specific immune response genes.
10 rammed breast cancer cells to express innate immune response genes.
11 latent enhancers that modulate expression of immune response genes.
12 -surface receptors to drive transcription of immune response genes.
13 f reactive oxygen species, and expression of immune response genes.
14 ons among sex hormones, sex chromosomes, and immune response genes.
15 nfluenced by host genetic factors, including immune response genes.
16 enhanced transcriptional activation of early immune response genes.
17 with pathogenic mutation in pro-inflammatory immune response genes.
18 cancer are enriched for lipid metabolism and immune response genes.
19 mmalian cells, along with other cellular and immune response genes.
20 ar kinase cascades rapidly activates primary immune response genes.
21 n of PVK cells failed to induce these innate immune response genes.
22 domain that regulates transcription of host immune response genes.
23 le intermediates that alter transcription of immune response genes.
24 reas MCPyV-positive tumors were enriched for immune response genes.
25 ated to muscle structure and development and immune response genes.
26 on of several cytokine, chemokine, and other immune response genes.
27 some 3L, overlapping five known or suspected immune response genes.
28 near the transcriptional initiation sites of immune response genes.
29 LPS) results in the induction of an array of immune response genes.
30 volving activation of apoptotic pathways and immune response genes.
31 heat shock protein, antioxidant, and innate immune response genes.
32 gions important for gene regulation in other immune-response genes.
33 control expression of a variety of inducible immune-response genes.
34 ergistically activate the expression of many immune-response genes.
35 luding several signals in innate or adaptive immune-response genes.
36 gramming through up-regulation of the enzyme immune response gene 1 (IRG1) and its product itaconate
37 gene EDEM1 (3p26; P = 8.9 x 10-7), near the immune response gene ALCAM (3q13; P = 9.3 x 10-7), withi
38 anisms for coagulation, and reprogramming of immune response genes all have critical roles in the dev
41 we observed that aneuploid cells up-regulate immune response genes and down-regulate genes involved i
42 s a key role in the activation of many early immune response genes and is regulated by subcellular lo
43 nvolve an overexpression of inflammation and immune response genes and of genes associated with the l
45 haracterised by an abundance of inflammatory immune response genes and pathways, including many relat
46 and regulatory regions, and upregulation of immune response genes and regulatory regions, which are
47 istic studies reveal that KL1 modulates host immune response genes and suppresses the production of r
48 ociated with elevated expression of adaptive immune response genes and, notably, T cell response gene
50 Furthermore, mitochondria-related genes, immune response genes, and transposable elements are als
51 gulating type I and II interferon and innate immune response genes, and up-regulating B-cell modules
52 ults indicate that under hypoxic conditions, immune response genes are differentially expressed in cu
53 We determined whether SNPs in 112 selected immune response genes are important for HCV clearance, b
55 ntly increased, whereas expression of innate immune response genes are significantly decreased, in hu
56 Grade 1B was associated with upregulated immune response genes, as 1 categorical distinction from
57 bolomic profiles revealed down-regulation of immune response genes associated with reduced levels of
58 increased and accelerated activation of host immune response genes associated with severe pulmonary p
60 ata have emerged in our understanding of the immune response gene associations and the description of
61 erns, including a strong induction of innate immune response genes at early times post-exposure, and
62 pattern in autoimmunity are not necessarily "immune response" genes, but are genes that encode protei
63 ed via IgE cross-linking selectively induced immune response genes Ccl1, Il3, and Il2 compared with I
64 abundance of individual ceRNAs, among three immune response genes (CCL22, IL2RB, and IRF4) is predic
65 y been elucidated, implicating predominantly immune-response genes, changes in environmental factors
66 as syringae in leaves and, accordingly, some immune-response genes, containing repeats in their promo
67 fied sets of Type I IFN and pro-inflammatory immune response genes determined from mRNA sequencing of
68 addition, a small subset of strongly induced immune response genes displayed a noncanonical TF recrui
70 ider the role of altered innate and adaptive immune responses, gene-environment interactions, epigene
71 There was a strong correlation between host immune response gene expression (CXCL13 and IgG) and spi
72 s, including transposable element and innate immune response gene expression involved in viral defens
75 lationship between the epigenetic control of immune response gene expression, exposure to environment
80 n of PD-L1 nuclear localization that governs immune-response gene expression, and thereby advocate ta
81 and chemokines, and the expression of innate immune response genes following Ad injection were TLR2 d
83 action of adenoviral E1A oncoprotein on host immune-response genes has been attributed to interaction
87 oderate associations were found between some immune response genes (ie, IL3 and IL13) and parasite ra
88 ppear specially programmed for expression of immune response genes implicated in immunity and inflamm
89 African malaria vector Anopheles gambiae for immune response genes in adult female mosquitoes, which
91 longevity, and elevated expression of innate immune response genes in glial cells, indicating that a
94 mputational workflow to experimental data of immune response genes in macrophages, we found that a mu
96 and briefly review how alteration of innate immune response genes in murine models can provide insig
98 DGFRbeta signaling also induces a battery of immune response genes in pericytes and mesenchymal cells
99 ducted a comparative expression profiling of immune response genes in peripheral blood mononuclear ce
100 type of expression pattern from a family of immune response genes in single cells has not been ident
102 c studies implicated several genes including immune response genes in the risk of developing type 1 d
103 cription factor that regulates metabolic and immune response genes in the setting of low oxygen tensi
104 ind that Vpr suppresses expression of innate immune response genes in uninfected bystander cells, and
105 Equally significant is the dysregulation of immune response genes in vapers and smokers, modulated b
106 leotides is the induction of a set of innate immune response genes in Xenopus embryos and that splici
108 apid induction of numerous NF-kappaB-induced immune response genes, including antimicrobial peptide g
109 ther sequences commonly found for Drosophila immune response genes, including interferon-related regu
110 vealed global p53/NF-kappaB co-regulation of immune response genes, including several chemokines, whi
111 l immunity to drive the expression of innate immune response genes, including those encoding antivira
112 tensive activation of diverse sets of innate immune response genes, including those that encode multi
113 ctor necessary for expression of host innate immune response genes, including those that target Env.
114 linc1992 was required for expression of many immune-response genes, including other cytokines and tra
115 rmation also identified correlations between immune response genes, indicating biological coordinatio
116 regulates the expression of a wide range of immune response genes involved in immunity to pathogens.
120 c mechanisms and have been shown to regulate immune response genes, making them prime targets for the
121 nd indicates that differences in the dose of immune response genes may constitute a genetic basis for
122 e (CBD) and sarcoidosis suggest that similar immune-response genes may be involved in susceptibility
123 e-dependent expression of RANTES and related immune-response genes) may more effectively coordinate i
125 ndings reveal a relationship between mCB and immune response gene networks in the placenta as a poten
126 ons made possible the identification of many immune-response genes not previously identified in the c
127 distinguishing threats becomes available to immune response genes; one specific phase could be mappe
128 gside associations with MHC and other canine immune response genes parallel that of different ethnic
130 is from independent groups have demonstrated immune response gene polymorphisms, and particularly in
131 he important role of HLA class-II alleles as immune response genes predisposing their carriers for se
132 similar clinical and serologic features, the immune response genes predisposing to and protecting fro
133 frequency of phenotypic features and in the immune-response genes predisposing to and protecting fro
136 ulatory proteins to nuclear factor kappaB at immune response gene promoters through protein-protein i
137 that the X escapee Kdm6a regulates multiple immune response genes, providing a mechanism for sex dif
138 sses induction of NF-kappaB-regulated innate immune response genes required for host defense in human
139 led that LXRbeta regulates the expression of immune response gene sets and lipids known to be involve
140 with upregulation of cellular metabolism and immune response gene sets, while testosterone upregulate
141 could reduce expression of a stress-related immune response gene signature known as the Conserved Tr
142 s of R4RA synovial biopsies identify humoral immune response gene signatures associated with response
143 pression profiling studies have demonstrated immune response gene signatures that appear predictive o
144 C cohort were performed to determine whether immune response gene signatures were associated with MIB
146 on pathway was the immune response, with 131 immune response genes significantly up- or downregulated
147 c genes such as DEK in superficial tumors or immune response genes such as Cd86 antigen in invasive d
149 tors participates in the regulation of early immune response genes such as IL-2, IL-4, CD40 ligand, a
150 t a 5% false discovery rate, including a few immune response genes such as NLRC5, S100A12, LILRA4 and
151 AT family member (Stat1) and Stat1-dependent immune-response genes such as intercellular adhesion mol
152 ly genes, complement factors, apoptosis, and immune response genes suggests a neuroinflammatory progr
153 ne lupus and suggest that Nba2 may act as an immune response gene that influences Ag-driven B cell re
154 mechanism by which HIV-1 manipulates a host immune response gene that is important in its own replic
155 inflammation and a candidate for epithelial immune response genes that are abnormally programmed in
156 autoantibody status, and expand the list of immune response genes that are possibly important in the
157 ated hCPC performance and identified several immune response genes that correlated with performance.
158 es uncovered a subset of proinflammatory and immune response genes that overlapped with those regulat
159 our RNAseq data, such as Relish, a critical immune response gene, that shows increased expression wi
161 sis localized the positions of these non-MHC immune response genes to genomic intervals on chromosome
162 nsive chromatin remodeling that reprogrammed immune response genes toward a stably maintained primed
164 barrier and the elucidation of cytokine and immune response gene variation in defining posttransplan
166 ysis revealed that the expression of several immune response genes was increased, including CCL5, whi
168 lammatory cytokines, receptors, and Th1-type immune response genes were down-modulated, and some gene
169 er genes, stress response genes, and defense/immune response genes were up-regulated, as anticipated
171 ssociated with elevated expression of innate immune response genes, whereas the hypoinflammatory phen
172 ion significantly suppresses a subset of the immune response genes, which include the IFN-stimulated
173 mice transgenic for the human HLA-DRB1*0101 immune response gene with CII elicits an arthritis (coll
174 including genes involved in acute rejection, immune response genes with an unknown role in rejection,