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1 ons among sex hormones, sex chromosomes, and immune response genes.
2 ar kinase cascades rapidly activates primary immune response genes.
3 e it participates in the activation of early immune response genes.
4 n of PVK cells failed to induce these innate immune response genes.
5 domain that regulates transcription of host immune response genes.
6 reas MCPyV-positive tumors were enriched for immune response genes.
7 ated to muscle structure and development and immune response genes.
8 on of several cytokine, chemokine, and other immune response genes.
9 some 3L, overlapping five known or suspected immune response genes.
10 near the transcriptional initiation sites of immune response genes.
11 LPS) results in the induction of an array of immune response genes.
12 volving activation of apoptotic pathways and immune response genes.
13 heat shock protein, antioxidant, and innate immune response genes.
14 ng the cagA-dependent genes, and many of the immune response genes.
15 ontrolling the expression of a wide range of immune response genes.
16 r occupancy is translated into activation of immune response genes.
17 ins, and for the activation of antibacterial immune response genes.
18 activates the transcription of antibacterial immune response genes.
19 ch directs expression of innate and adaptive immune response genes.
20 control expression of a variety of inducible immune-response genes.
21 ergistically activate the expression of many immune-response genes.
22 gions important for gene regulation in other immune-response genes.
23 anisms for coagulation, and reprogramming of immune response genes all have critical roles in the dev
26 s a key role in the activation of many early immune response genes and is regulated by subcellular lo
27 nvolve an overexpression of inflammation and immune response genes and of genes associated with the l
29 and regulatory regions, and upregulation of immune response genes and regulatory regions, which are
31 Furthermore, mitochondria-related genes, immune response genes, and transposable elements are als
32 ults indicate that under hypoxic conditions, immune response genes are differentially expressed in cu
33 We determined whether SNPs in 112 selected immune response genes are important for HCV clearance, b
35 Grade 1B was associated with upregulated immune response genes, as 1 categorical distinction from
36 increased and accelerated activation of host immune response genes associated with severe pulmonary p
37 ata have emerged in our understanding of the immune response gene associations and the description of
38 erns, including a strong induction of innate immune response genes at early times post-exposure, and
39 pattern in autoimmunity are not necessarily "immune response" genes, but are genes that encode protei
40 ed via IgE cross-linking selectively induced immune response genes Ccl1, Il3, and Il2 compared with I
41 abundance of individual ceRNAs, among three immune response genes (CCL22, IL2RB, and IRF4) is predic
42 y been elucidated, implicating predominantly immune-response genes, changes in environmental factors
43 as syringae in leaves and, accordingly, some immune-response genes, containing repeats in their promo
44 ider the role of altered innate and adaptive immune responses, gene-environment interactions, epigene
45 There was a strong correlation between host immune response gene expression (CXCL13 and IgG) and spi
51 and chemokines, and the expression of innate immune response genes following Ad injection were TLR2 d
52 action of adenoviral E1A oncoprotein on host immune-response genes has been attributed to interaction
56 oderate associations were found between some immune response genes (ie, IL3 and IL13) and parasite ra
57 ppear specially programmed for expression of immune response genes implicated in immunity and inflamm
58 African malaria vector Anopheles gambiae for immune response genes in adult female mosquitoes, which
59 longevity, and elevated expression of innate immune response genes in glial cells, indicating that a
62 and briefly review how alteration of innate immune response genes in murine models can provide insig
63 DGFRbeta signaling also induces a battery of immune response genes in pericytes and mesenchymal cells
64 type of expression pattern from a family of immune response genes in single cells has not been ident
66 c studies implicated several genes including immune response genes in the risk of developing type 1 d
67 cription factor that regulates metabolic and immune response genes in the setting of low oxygen tensi
69 apid induction of numerous NF-kappaB-induced immune response genes, including antimicrobial peptide g
70 ther sequences commonly found for Drosophila immune response genes, including interferon-related regu
71 vealed global p53/NF-kappaB co-regulation of immune response genes, including several chemokines, whi
72 l immunity to drive the expression of innate immune response genes, including those encoding antivira
73 tensive activation of diverse sets of innate immune response genes, including those that encode multi
74 linc1992 was required for expression of many immune-response genes, including other cytokines and tra
75 regulates the expression of a wide range of immune response genes involved in immunity to pathogens.
78 nd indicates that differences in the dose of immune response genes may constitute a genetic basis for
79 e (CBD) and sarcoidosis suggest that similar immune-response genes may be involved in susceptibility
80 e-dependent expression of RANTES and related immune-response genes) may more effectively coordinate i
82 ons made possible the identification of many immune-response genes not previously identified in the c
83 gside associations with MHC and other canine immune response genes parallel that of different ethnic
84 is from independent groups have demonstrated immune response gene polymorphisms, and particularly in
85 similar clinical and serologic features, the immune response genes predisposing to and protecting fro
86 frequency of phenotypic features and in the immune-response genes predisposing to and protecting fro
88 ulatory proteins to nuclear factor kappaB at immune response gene promoters through protein-protein i
89 sses induction of NF-kappaB-regulated innate immune response genes required for host defense in human
90 pression profiling studies have demonstrated immune response gene signatures that appear predictive o
92 on pathway was the immune response, with 131 immune response genes significantly up- or downregulated
93 c genes such as DEK in superficial tumors or immune response genes such as Cd86 antigen in invasive d
95 tors participates in the regulation of early immune response genes such as IL-2, IL-4, CD40 ligand, a
96 t a 5% false discovery rate, including a few immune response genes such as NLRC5, S100A12, LILRA4 and
97 AT family member (Stat1) and Stat1-dependent immune-response genes such as intercellular adhesion mol
98 ne lupus and suggest that Nba2 may act as an immune response gene that influences Ag-driven B cell re
99 mechanism by which HIV-1 manipulates a host immune response gene that is important in its own replic
100 inflammation and a candidate for epithelial immune response genes that are abnormally programmed in
101 autoantibody status, and expand the list of immune response genes that are possibly important in the
102 es uncovered a subset of proinflammatory and immune response genes that overlapped with those regulat
103 our RNAseq data, such as Relish, a critical immune response gene, that shows increased expression wi
105 sis localized the positions of these non-MHC immune response genes to genomic intervals on chromosome
106 nsive chromatin remodeling that reprogrammed immune response genes toward a stably maintained primed
107 barrier and the elucidation of cytokine and immune response gene variation in defining posttransplan
109 ysis revealed that the expression of several immune response genes was increased, including CCL5, whi
110 lammatory cytokines, receptors, and Th1-type immune response genes were down-modulated, and some gene
111 er genes, stress response genes, and defense/immune response genes were up-regulated, as anticipated
112 mice transgenic for the human HLA-DRB1*0101 immune response gene with CII elicits an arthritis (coll
113 including genes involved in acute rejection, immune response genes with an unknown role in rejection,
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