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1 ent in a mouse model of cryoglobulin-induced immune complex glomerulonephritis.
2 phoid hyperplasia, B cell activation, and an immune complex glomerulonephritis.
3 ion of autoantibodies and the development of immune complex glomerulonephritis.
4 ntaneous lupus-like disease characterized by immune complex glomerulonephritis.
5 erized by IgG autoantibody production and an immune complex glomerulonephritis.
6 h levels of autoantibodies in the serum, and immune complex glomerulonephritis.
7 mmunity, characterized by DNA antibodies and immune complex glomerulonephritis.
8 A subpopulation of the mice also develops immune complex glomerulonephritis.
9 lvement by monoclonal gammopathy that mimics immune-complex glomerulonephritis.
10 ed by anti-nRNP/Sm and Su autoantibodies and immune complex glomerulonephritis after a single i.p. pr
11 puslike disease with anti-DNA antibodies, an immune complex glomerulonephritis and pulmonary alveolit
12 sults in increased systemic inflammation and immune complex glomerulonephritis, despite intact TLR si
13 le stranded DNA antibodies, proteinuria, and immune complex glomerulonephritis in irradiated BALB/c n
14 s play a central role in the pathogenesis of immune complex glomerulonephritis in lupus, the specific
16 Icos(-/-) MRL(lpr) mice, the progression of immune complex glomerulonephritis is only modestly dimin
17 Twenty-five patients had immune-complex glomerulonephritis: membranoproliferative
18 nephropathy (HIVAN) (n = 13), HIV-associated immune complex glomerulonephritis (n = 3), HIV-associate
21 ious process, and renal biopsies revealed an immune complex glomerulonephritis with features of PIGN.
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