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1 uten protein structure, using SEM, light and immunofluorescent microscopy.
2 tected in infected cells either by RT-PCR or immunofluorescent microscopy.
3 yuridine, and Hoechst 33342 as visualized by immunofluorescent microscopy.
4 transcriptase polymerase chain reaction, and immunofluorescent microscopy.
5 f these proteins with caveolin-1 as shown by immunofluorescent microscopy.
6 ed macrophages using both immunoblotting and immunofluorescent microscopy.
7 at stained for Annexin V [CD146(AnnV+)]) and immunofluorescent microscopy.
8 transcription polymerase chain reaction and immunofluorescent microscopy.
9 ent of compound action potentials (CAPs) and immunofluorescent microscopy.
10 t analysis and either immunohistochemical or immunofluorescent microscopy.
11 by electrophoretic mobility shift assay and immunofluorescent microscopy.
12 timates of glomerular IgG deposition seen by immunofluorescent microscopy.
13 anslocation was determined by confocal laser immunofluorescent microscopy.
14 zed to the cytoplasm as detected by indirect immunofluorescent microscopy.
16 calization was experimentally verified using immunofluorescent microscopy and a cell-surface biotinyl
19 uscle was demonstrated using double labeling immunofluorescent microscopy and immunoelectron microsco
26 ion, surface protein biotinylation, confocal immunofluorescent microscopy, and functional measurement
35 ( approximately 8,500 dimers per cell), and immunofluorescent microscopy (IFM) located MreCD(Spn) to
38 ult rat CF to myofibroblasts, as assessed by immunofluorescent microscopy, immunoblotting, and collag
39 ogenous Aalpha and Galpha(12) colocalized by immunofluorescent microscopy in Caco-2 cells and in neur
41 detected by immunoblot analysis and confocal immunofluorescent microscopy in fibrotic livers from mic
43 confirmed by Western blot analysis, confocal immunofluorescent microscopy in vitro, and on cultured o
51 scence-activated cell sorting (FACS), and by immunofluorescent microscopy of tissue sections and isol
55 -immunoprecipitated with Cyp60, and confocal immunofluorescent microscopy revealed intracellular co-l
61 brains sectioned and prepared for dual label immunofluorescent microscopy, single label bright field
64 uginosa infection, and (iii) high-resolution immunofluorescent microscopy to monitor ExoS translocati
66 existence of this mechanism was provided by immunofluorescent microscopy, using fluorescently labele
68 iotinylation coupled with immunoblotting and immunofluorescent microscopy, we assessed the kinetics o
73 pression of AP-2 and PAR-1 simultaneously by immunofluorescent microscopy with an automated quantific
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