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1 neuropathy (CIDP) need long-term intravenous immunoglobulin.
2 r an immunochromatographic assay of specific immunoglobulins.
3 apolipoproteins, complement components, and immunoglobulins.
4 d levels of soluble BAFF, B lymphocytes, and immunoglobulins.
5 rmation for profiling of N-linked glycans on immunoglobulins.
10 sies in children with symptoms and levels of immunoglobulin A against tissue-transglutaminase (TGA-Ig
12 ere seronegative at baseline, anti-rotavirus immunoglobulin A seroconversion rates after 3 vaccine do
13 zation and induction of immunoglobulin G and immunoglobulin A to all antigens tested, while causing n
14 um total free light chain, immunoglobulin G, immunoglobulin A, and immunoglobulin M were measured on
16 ographic assay for the detection of specific immunoglobulins against a target antigen (antibodies) in
17 jection of heat-aggregated human intravenous immunoglobulin and active systemic anaphylaxis after imm
20 , members of the tyrosine kinase family with immunoglobulin and EGF homology domains, are receptor ty
21 tain giant polypeptides composed of multiple immunoglobulin and fibronectin domains and one or two pr
24 racterized by increased expression of T-cell immunoglobulin and mucin domain 3, which down-regulates
25 human syndecan-1 (SDC-1), SDC-2, and T cell immunoglobulin and mucin domain-containing protein 1 (TI
26 lymphocyte-activation gene 3 (LAG-3), T cell immunoglobulin and mucin-3 (TIM-3), and/or programmed ce
28 may indicate only some interactions between immunoglobulins and ABTs clear pneumococcal colonization
29 as lesser allergic phenotypes, reduced serum immunoglobulins and allergic mediators, lower mast cells
31 antation, there is a gradual increase of all immunoglobulins and IgG subclasses, but values were alwa
33 nd the first to study two administrations of immunoglobulins and two doses, showed that both doses of
35 nticoagulation, corticosteroids, intravenous immunoglobulin, and plasma exchange are the most commonl
36 utate viral pathogens and somatically mutate immunoglobulins, and contribute to the diversification a
38 d using the device was consistent with serum immunoglobulin assays that are commonly used in MM diagn
39 he mechanism of conversion of broad-spectrum immunoglobulin-binding proteins, such as HTB1, into targ
40 ve with GABA-enhancing drugs and intravenous immunoglobulin, but some respond poorly and remain disab
43 tential risks and inherent scarcity of human immunoglobulin, careful consideration of its indications
44 d decrease in NHEJ is insufficient to impact immunoglobulin class switching in DEK knockout mice.
45 ivo capacity to induce B cell maturation and immunoglobulin class switching than cells from HIV progr
47 versial, with detection prevalence rates and immunoglobulin classes varying considerably between stud
48 rmal immune cell repertoire, unchanged serum immunoglobulin concentrations and an intact immune respo
51 ding transcripts originating upstream of the immunoglobulin constant region (I transcripts) are requi
53 homa, activating RAS mutations may propagate immunoglobulin-crippled tumour cells, which usually repr
54 including glucocorticosteroids, intravenous immunoglobulins, cyclosporine, plasmapheresis, thalidomi
56 ad lymphocyte-maturation defects that caused immunoglobulin deficiency and intestinal inflammation.
57 globulin (SCIg) is an alternative option for immunoglobulin delivery, but has not previously been inv
59 ated circulating CD4(+) T cells specific for immunoglobulin-derived neoantigens and found these cells
60 rystal structure of TIGIT bound to the first immunoglobulin domain of nectin-2 indicated that the rec
62 ide analysis of members of a small family of immunoglobulin domain proteins, we found that OIG-8, a p
64 ave similar architectures, with the variable immunoglobulin domains of the heavy and light chain each
67 everal extracellular cues, including antigen-immunoglobulin E (IgE) complexes, bacteria, viruses, cyt
69 y fever, allergic sensitization, serum total immunoglobulin E (IgE), forced expiratory volume in one-
71 an emphasis on novel FcepsilonRI regulators, immunoglobulin E (IgE)-independent pathways [e.g., Mas-r
73 kin tests and measurement of serum levels of immunoglobulin E do not accurately identify foods for el
76 efit from targeted anti-interleukin and anti-immunoglobulin E therapies, and in monitoring subsequent
80 rrelated with levels of C5a (P < .01), local immunoglobulins (especially IgM, P < .0001), and anti-do
82 thers and the routine use of cytomegalovirus immunoglobulin for prophylaxis or treatment of infected
83 at daily injections of osteoprotegerin (OPG)-immunoglobulin fragment complex (OPG-Fc) completely rest
84 c IgE, IgG, IgG4, and IgM levels, as well as immunoglobulin free light chains, were measured in both
86 lyte binding interactions between anti-human immunoglobulin G (anti-hIgG) and human immunoglobulin G
90 n the presence of reactive, non-neutralizing immunoglobulin G (IgG) (RNNIg) is the greatest risk fact
91 protective role for both Chlamydia-specific immunoglobulin G (IgG) and polymorphonuclear neutrophils
93 c opsonophagocytic activity (OPA) titers and immunoglobulin G (IgG) concentrations were determined.
97 amma receptors (FcgammaRs), the Fc domain of immunoglobulin G (IgG) mediates a wide spectrum of immun
101 ree-dimensional (3D) ordered arrays of human immunoglobulin G (IgG) were fabricated using well-define
104 d interface for the attachment of monoclonal immunoglobulin G (IgGNS1) and to favor specific detectio
107 nasopharyngeal colonization and induction of immunoglobulin G and immunoglobulin A to all antigens te
108 yme-linked immunosorbent assay for anti-HCMV immunoglobulin G and immunoglobulin M and for cIL-10 and
109 nes, correlated directly with B. burgdorferi immunoglobulin G antibodies (P </= .02), suggesting a be
110 our studies on posttransplant production of immunoglobulin G antibodies targeting cell surface antig
111 ntrations and neutralizing activity of serum immunoglobulin G antibodies to the RSV prefusion (pre-F)
112 itis C therapy in patients carrying anti-HEV immunoglobulin G antibodies, raising 2 major questions:
115 Eight patients with endocarditis had phase I immunoglobulin G antibody titers >800 but did not meet t
118 The primary endpoint was serotype-specific immunoglobulin G concentrations values (geometric mean c
119 oconversion, defined as a 4-fold increase in immunoglobulin G directed against Cryptosporidium gp15 a
120 Among 34 patients (91.9%) with monoclonal immunoglobulin G gammopathy, 20 (58.8%) had kappa light
123 croarray antibody capture assay for anti-HDV immunoglobulin G wherein recombinant HDV delta antigen i
124 glucose, ascorbic acid human serum protein, immunoglobulin G, and immunoglobulin M), and demonstrate
126 model protein in human serum, that is, human immunoglobulin G, with the aim to demonstrate a virtuall
128 ELISA) was used to investigate serum anti-CT immunoglobulin G1 (IgG1; long-lived response) and immuno
129 lence of myelin oligodendrocyte glycoprotein immunoglobulin G1 (MOG-IgG) and associated clinical feat
130 exploiting the stable architecture of human immunoglobulin G1 We used iterative experimental validat
132 oglobulin G1 (IgG1; long-lived response) and immunoglobulin G3 (IgG3; short-lived response indicating
133 extended version of our previously published ImmunoGlobulin Galaxy (IGGalaxy) virtual machine that wa
134 with a distinct strand bias, to enlarge the immunoglobulin gene mutation spectrum from G-C to A-T ba
137 dentify a novel susceptibility signal in the immunoglobulin heavy chain (IGH) locus centring on a hap
139 inding protein homologous protein (chop) and immunoglobulin heavy chain binding protein (bip) levels.
140 and antisense strand DNA mutagenesis at the immunoglobulin heavy chain locus and some other regions
142 , we performed high-throughput sequencing of immunoglobulin heavy chain VDJ rearrangements of naive,
144 s were exclusively derived from the lymphoma immunoglobulin heavy- or light-chain variable regions.
146 l of EV-D68 infection: (1) human intravenous immunoglobulin (hIVIG), (2) fluoxetine, and (3) dexameth
147 s with nasal polyps is associated with local immunoglobulin hyperproduction and the presence of IgE a
148 protein-protein interfaces of two different immunoglobulin (Ig) constant domain pairs are exchanged
150 cterized transmembrane protein with a single immunoglobulin (Ig) domain, instructs the distinct, neur
151 k binds to the leucine-rich repeat (LRR) and immunoglobulin (Ig) domains of Gpr124, and weakening thi
152 predicted peak of pollen) to determine serum immunoglobulin (Ig) E concentrations and Treg percentage
157 inase (AID) is a mutator enzyme that targets immunoglobulin (Ig) genes to initiate antibody somatic h
158 was largely mediated by germline encoded and immunoglobulin (Ig) heavy-chain complementarity determin
160 iver disease often manifests systemically as immunoglobulin (Ig)-related syndromes due to aberrant B-
161 tromal lymphopoietin, IL-5 and IL-13), serum immunoglobulin (Ig)E and airway hyper-responsiveness (AH
162 omic DNA, and infection elicited significant immunoglobulin (Ig)G and IgM antibody responses, indicat
163 tion, there is a significant decrease of all immunoglobulins, IgG subclasses and pneumococcal polysac
165 e, phenylacetylglycine, alanine) and mucosal immunoglobulin (IgM) and cytokine (IL-10, IL-4) producti
166 to maintain homeostasis is the secretion of immunoglobulins (Igs) across epithelial barriers, which
167 tic epitopes exposed in the hinge regions of immunoglobulins (Igs) and do not bind to the intact Ig c
168 n-1 receptor 8 (IL-1R8, also known as single immunoglobulin IL-1R-related receptor, SIGIRR, or TIR8)
172 volumes were tested for association with the immunoglobulin indices and the frequencies of immune cel
173 usions (IUPT), as well as weekly maternal IV immunoglobulin infusion (IVIG), with or without addition
174 combined with plasmapheresis and intravenous immunoglobulins is an option for patients with AMR.
176 feration and guide their differentiation and immunoglobulin isotype switching by delivering contact-d
181 all trials suggest that low-dose intravenous immunoglobulin (IVIg) may improve the symptoms of comple
186 of recombination signal-binding protein for immunoglobulin Jkappa region (RBPJkappa), a downstream e
187 and recombination signal binding protein for immunoglobulin kappa J region (RBPjkappa), key modulator
190 y inflammation, TH2 cytokine production, and immunoglobulin levels and a modest decrease in the phago
193 tion markers, and proliferation and secreted immunoglobulin levels were analyzed by using flow cytome
199 BCR knock-in mice lacking self-Thy-1 ligand, immunoglobulin light chain editing occurred, generating
200 n both AL and multiple myeloma (MM), soluble immunoglobulin light chains (LC) are produced by clonal
201 arge membrane pores and high permeability to immunoglobulin light chains) or a conventional high-flux
202 al lymphoplasmacytic malignancies, including immunoglobulin light-chain amyloidosis, multiple myeloma
203 tional dynamics of a pathogenic kappa4 human immunoglobulin light-chain variable domain, SMA, associa
204 izing and destabilizing mutations is key for immunoglobulin light-chains populating unfolded intermed
205 for NK cell inhibition via inhibitory killer immunoglobulin-like (KIR) receptors and interrupts their
206 riant of human peroxidasin 1 comprising four immunoglobulin-like domains and the catalytically active
207 e complex shows how AlkC uses unique HLR and immunoglobulin-like domains to induce a sharp kink in th
208 antigenic motifs in a single-domain chimeric immunoglobulin-like fold generated a vaccine that greatl
211 cell alloreactivity mediated by killer cell immunoglobulin-like receptor (KIR)-HLA interactions may
214 a subset of RIFINs binds to either leucocyte immunoglobulin-like receptor B1 (LILRB1) or leucocyte-as
215 ucts (RAGE) is an ubiquitous, transmembrane, immunoglobulin-like receptor that exists in multiple iso
216 e frequently expressed educating killer cell immunoglobulin-like receptors compared with NK cells in
217 immune-cell-specific genes, including novel immunoglobulin-like receptors, and neofunctionalization
218 eater degree with the gB receptor the paired immunoglobulin-like type 2 receptor alpha (PILRalpha) th
219 re composed of alphabeta subunits displaying immunoglobulin-like variable domains that recognize pept
220 We report that certain Sialic-acid-binding immunoglobulin-like-lectins (siglecs) are expressed in h
224 terize the nanoscale spatial organization of immunoglobulin M (IgM) and IgG BCRs on the surfaces of r
225 GC-dependent immune responses, reduces total immunoglobulin M (IgM) and IgG levels, and leads to incr
227 ptor for the crystallizable fragment (Fc) of immunoglobulin M (IgM) can function as a cell-surface re
229 % CI: 0.7-4.7%) for Epstein-Barr virus (EBV) immunoglobulin M (IgM) positivity, 94.7% (95% CI: 90.7-9
230 infections and rule out misleading positive immunoglobulin M (IgM) results in areas with various lev
232 globulinemia (MC), a monoclonal expansion of immunoglobulin M (IgM)(+) autoreactive B cells, and also
233 t expression of the secretory heavy chain of immunoglobulin M (micros), is well-tolerated in HeLa cel
234 ent assay for anti-HCMV immunoglobulin G and immunoglobulin M and for cIL-10 and vIL-10 levels using
235 (-) T cells, and decreases in elevated serum immunoglobulin M and inflammatory markers including inte
236 eroprevalence of ZIKV was 6.2% based on ZIKV immunoglobulin M and negative for dengue reactivity.
237 h nonsense CXCR4 mutations have higher serum immunoglobulin M levels and incidence of symptomatic hyp
238 w lower bone marrow disease burden and serum immunoglobulin M levels but show an increased risk of de
239 ain, immunoglobulin G, immunoglobulin A, and immunoglobulin M were measured on ICU days 1, 3, and 7.
240 infection (viral nonstructural protein 1 and immunoglobulin M) has greatly simplified laboratory-base
241 d human serum protein, immunoglobulin G, and immunoglobulin M), and demonstrated a high correlation w
242 based on seroconversion for HCMV and/or HCMV immunoglobulin M-positive and low or moderate HCMV immun
244 athy (C3G) emphasizes the role of monoclonal immunoglobulin (MIg) in the occurrence of renal disease
247 ression of tumorigenicity-2 (ST2) and T-cell immunoglobulin mucin-3 (TIM3) at day 28 correlated with
248 tor accessory protein (IL1RAP), CD99, T-cell immunoglobulin mucin-3, and CD123 have begun to differen
249 ed of steroids (n = 61/74; 82%), intravenous immunoglobulins (n = 71/74; 96%), and plasmapheresis (n
250 f this strategy to human lymphoma implicates immunoglobulin neoantigens as targets for lymphoma immun
251 osuppressive therapy and chronic intravenous immunoglobulin or plasma exchange for 12 months without
252 nths before screening, or use of intravenous immunoglobulin or plasma exchange within 4 weeks before
255 rogeneous syndrome characterized by impaired immunoglobulin production and usually presents with a no
256 L-2 receptor alpha chain levels and in vitro immunoglobulin production by cultured B cells were quant
257 erresponsiveness (AHR), airway inflammation, immunoglobulin production, TH2-associated cytokine synth
258 iP (glucose-regulated protein 78 kDa/binding immunoglobulin protein) modulates protein folding in rep
261 IgA into the intestinal lumen, the polymeric immunoglobulin receptor, was also dependent on IL-17RA s
262 CD28 and CTLA-4 are members of a family of immunoglobulin-related receptors that are responsible fo
264 ad persistent medical issues, mainly ongoing immunoglobulin replacement (14; 45%), cutaneous viral wa
269 rder to diminish the requirements for rabies immunoglobulin (RIG) and multiple vaccinations for effec
271 tective antibody levels for years due to the immunoglobulin-secreting activity of long-lived plasma c
272 ey inhibited B-cell differentiation, impeded immunoglobulin secretions, and expanded Foxp3(+)CXCR5(+)
274 ononuclear cells (PBMCs), and used available immunoglobulin sequences and 5' and 3' RACE to clone and
275 oles in gene expression and recombination at immunoglobulin sites, their persistence is thought to in
276 ell-surface and secreted proteins containing immunoglobulin superfamily (IgSF) domains discovered a n
278 ession screens and proteomics, we identified immunoglobulin superfamily member 21 (IgSF21) as a neure
279 Programmed death one homolog (PD-1H) is an immunoglobulin superfamily molecule and primarily acts a
281 Junctional adhesion molecule C (JAM-C) is an immunoglobulin superfamily protein expressed in epitheli
283 discontinue immunosuppression and exogenous immunoglobulin support, with improvement in vasculitic d
285 ontaining substrates mimicking the mammalian immunoglobulin switch regions are particularly good AID
286 esults with standard MRD monitoring based on immunoglobulin/T-cell receptor (Ig/TCR) gene rearrangeme
287 sent antigens to CD4 T cells and produce IgE immunoglobulins that arm effector cells; however, mouse
297 immunotherapy with steroids and intravenous immunoglobulins vs. late immunotherapy), and a low white
298 st-HCT median CD4 counts and freedom from IV immunoglobulin were improved after the use of preparativ
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