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1 red among 54%, but only 3% were positive for immunoglobulin M.
2 of the CSF samples tested positive for ZIKV immunoglobulin M.
3 IL-7R and EBF but not B220, and can produce immunoglobulin M.
4 ng antibody and T-cell-independent antiviral immunoglobulin M.
5 uction of early T-cell-independent antiviral immunoglobulin M.
6 ion of Bam32 after ligation of CD40, but not immunoglobulin M.
7 ytes produce only the membrane-bound form of immunoglobulin M.
8 as used to screen serum samples for anti-CMV immunoglobulin M.
11 globulins enriched with immunoglobulin A and immunoglobulin M alleviated the symptoms of sickness beh
12 ox/lox)-CD19(+/Cre) mice have an increase in immunoglobulin M and CD43 and a decrease in CD5 expressi
14 ent assay for anti-HCMV immunoglobulin G and immunoglobulin M and for cIL-10 and vIL-10 levels using
15 n in 67% and complement fixation in 70%, and immunoglobulin M and G antibodies were demonstrated by e
16 immune markers of mortality, while anti-LVS immunoglobulin M and IL-1beta were associated with survi
17 r periorbital edema and Trichinella-specific immunoglobulin M and immunoglobulin G antibodies after e
18 tion, and intrathecal synthesis of borrelial immunoglobulin M and immunoglobulin G antibody in 100%,
20 ces of galactose expression or deposition of immunoglobulin M and immunoglobulin G were found in xeno
21 LD), an enzyme immunoassay (EIA) followed by immunoglobulin M and immunoglobulin G Western blots, per
22 it is characterized by low levels of surface immunoglobulin M and impairment of calcium mobilization
23 (-) T cells, and decreases in elevated serum immunoglobulin M and inflammatory markers including inte
24 gulated the expression of cell surface CD22, immunoglobulin M and major histocompatibility complex cl
25 eroprevalence of ZIKV was 6.2% based on ZIKV immunoglobulin M and negative for dengue reactivity.
28 d human serum protein, immunoglobulin G, and immunoglobulin M), and demonstrated a high correlation w
29 and bilirubin, lower serum concentrations of immunoglobulin M, and greater occurrence of autoantibodi
30 iters of serum and cerebrospinal fluid (CSF) immunoglobulin M, and occasionally positive results of W
31 circulating transitional B cells, increased immunoglobulin M, and reduced immunoglobulin G2 levels i
33 on, we found that Ad is opsonized by natural immunoglobulin M antibodies and complement and that thes
34 orn to yaws-infected mothers did not produce immunoglobulin M antibodies and their organs, examined b
40 in G antibodies, and sometimes low levels of immunoglobulin M antibodies, to Borrelia burgdorferi (Bb
42 donors identified with early WNV infection (immunoglobulin M antibody negative) and those with uncon
44 ble case required fever and a positive rapid immunoglobulin M antibody test for typhoid (TUBEX TF); a
47 in the United States, confirmed by high ZIKV immunoglobulin M antibody titers in serum and cerebrospi
49 he infection when low levels of neutralizing immunoglobulin M antibody were detected in wild-type mic
50 fected CD28(-/-) mice produced primarily the immunoglobulin M antibody, which upon passive transfer p
51 ipid rafts in response to ligation with anti-immunoglobulin M (as a surrogate for antigen) are featur
54 IL-21 integrated signals mediated by surface immunoglobulin M (B-cell receptor) and Toll-like recepto
55 previously unidentified and unique subset of immunoglobulin M(+) B cells that present with an AA4.1(-
56 vation of cells lacking ASF/SF2 through anti-immunoglobulin M-B-cell receptor cross-linking rescued v
57 h Laboratory (VDRL) test and a specific IgM (immunoglobulin M) capture enzyme-linked immunosorbent as
58 en VWF levels < 130 U/dL and both monoclonal immunoglobulin M concentration (mIgMC) and viscosity.
59 urrence with these findings, postvaccination immunoglobulin M concentrations were not significantly i
60 oint swelling and higher anti-B. burgdorferi immunoglobulin M cross-reactive responses than other str
61 -)), CD8-deficient (CD8(-/-)), and secretory immunoglobulin M-deficient (sIgM(-/-)) mice and wild-typ
64 hrough dengue nonstructural protein 1 and/or immunoglobulin M detection, were included in this study
66 an lipopolysaccharide-binding protein (LBP), immunoglobulin M endotoxin core antibodies to lipopolysa
67 n, WNV-specific B cells were not detected by immunoglobulin M enzyme-linked immunospot analysis until
69 ding alpha/beta interferon (IFN-alpha/beta), immunoglobulin M, gammadelta T cells, and complement aga
70 infection (viral nonstructural protein 1 and immunoglobulin M) has greatly simplified laboratory-base
71 the stability of the alternatively expressed immunoglobulin M heavy chain secretory mRNA is developme
73 Escherichia coli K1 bacteria, we produced 12 immunoglobulin M hybridomas secreting monoclonal antibod
74 BV infection in nine cases of X-linked hyper-immunoglobulin M (hyper-IgM) syndrome who, due to a muta
76 ated transgenic zebrafish in which the major immunoglobulin M (IgM(+)) B-cell subset expresses green
79 ntial for generating acute pathogen-specific immunoglobulin M (IgM) and early IgG, which reduced path
80 d splicing substrates derived from the mouse immunoglobulin M (IgM) and human immunodeficiency virus
81 nosorbent assays for anti-H. pylori-specific immunoglobulin M (IgM) and IgA established using bacteri
82 hose obtained by BLOTrix analysis of MarBlot immunoglobulin M (IgM) and IgG and by ViraScan analysis
83 immunosorbent assay (ELISA) that can detect immunoglobulin M (IgM) and IgG antibodies in patients wi
87 terize the nanoscale spatial organization of immunoglobulin M (IgM) and IgG BCRs on the surfaces of r
88 gingivalis CPS developed elevated levels of immunoglobulin M (IgM) and IgG in serum that reacted wit
90 GC-dependent immune responses, reduces total immunoglobulin M (IgM) and IgG levels, and leads to incr
91 eatures, including continuous high titers of immunoglobulin M (IgM) and IgG reactivity throughout all
92 ssociated with higher brain CFU, lower serum immunoglobulin M (IgM) and IgG responses to glucuronoxyl
93 ocococcal pulmonary infection elicited serum immunoglobulin M (IgM) and IgG to the capsular polysacch
96 components of humoral immunity, particularly immunoglobulin M (IgM) and IgG, have critical roles in p
97 /-) mice produced low levels of WNV-specific immunoglobulin M (IgM) and IgG, viral clearance from the
99 abeta(+) and gammadelta(+) T cells generated immunoglobulin M (IgM) and IgG3 B. burgdorferi-reactive
100 se sensitivity and permit the measurement of immunoglobulin M (IgM) and immunoglobulin G (IgG) classe
103 ) enzyme-linked immunosorbent assay (ELISA), immunoglobulin M (IgM) and immunoglobulin G (IgG) Wester
104 lyzed for (1) viral DNA loads, (2) anti-B19V immunoglobulin M (IgM) and immunoglobulin G (IgG), (3) a
105 gy prior to termination showed both specific immunoglobulin M (IgM) and immunoglobulin G (IgG), where
106 a positive correlation between BA plasma CMV immunoglobulin M (IgM) and liver T-cell CMV reactivity w
107 rated reporter and two endogenous rat genes, Immunoglobulin M (IgM) and Rab38, we demonstrate that a
108 y to such xenobiotics would be predominantly immunoglobulin M (IgM) and using sera from a large cohor
109 at the majority of Lyme disease patients had immunoglobulin M (IgM) and/or IgG responses to p66 and t
114 the identification of rubella virus-specific immunoglobulin M (IgM) antibodies in serum, but approxim
115 or this included elevated levels of anti-HEL immunoglobulin M (IgM) antibodies in the serum of CD5(-/
116 athies associated with persistently elevated immunoglobulin M (IgM) antibodies that react with GQ1b,
117 of persons with suspected HEV was tested for immunoglobulin M (IgM) antibodies to HEV to confirm infe
119 day prior to the onset of clinical disease; immunoglobulin M (IgM) antibodies were detected at low l
122 shown to induce the production of anti-oxLDL immunoglobulin M (IgM) antibodies, due to molecular mimi
125 eguides for the detection of dengue-specific immunoglobulin M (IgM) antibody, and we demonstrate dete
126 ing-mouse brain WN virus antigen used in the immunoglobulin M (IgM) antibody-capture and indirect IgG
129 enzyme or crude lysates generated from anti-immunoglobulin M (IgM) antibody-treated RAMOS cells.
131 , we report that ST6Gal-I deficiency induces immunoglobulin M (IgM) antigen receptor endocytosis in t
132 ed by a selective pressure to retain surface immunoglobulin M (IgM) BCR despite an active class-switc
133 Between 5- and 10-fold more purified human immunoglobulin M (IgM) but not IgG was deposited onto ds
134 ptor for the crystallizable fragment (Fc) of immunoglobulin M (IgM) can function as a cell-surface re
137 y of undetermined significance (MGUS) of the immunoglobulin M (IgM) class, which progresses to lympho
139 Passive transfer of normal mouse serum, immunoglobulin M (IgM) from normal mouse serum, or IgG f
141 rpretation of positive cytomegalovirus (CMV) immunoglobulin M (IgM) in the first trimester of pregnan
144 significantly higher and the endotoxin core immunoglobulin M (IgM) level lower in cases, compared wi
145 ated with hu5C8 secreted adenovirus-specific immunoglobulin M (IgM) levels comparable to control anim
150 lved in the pathogenesis of premalignant non-immunoglobulin M (IgM) monoclonal gammopathy of undeterm
153 detect human parvovirus B19 (B19V)-specific immunoglobulin M (IgM) or IgG in the sera of 198 pregnan
155 enstrom macroglobulinemia (WM), which has an immunoglobulin M (IgM) paraproteinemia, is classified as
156 antibody library obtained from a human donor Immunoglobulin M (IgM) pool was determined to be at leas
157 % CI: 0.7-4.7%) for Epstein-Barr virus (EBV) immunoglobulin M (IgM) positivity, 94.7% (95% CI: 90.7-9
158 gh TACI and BCMA, and each ligand stimulated immunoglobulin M (IgM) production by peripheral blood B
159 Both up-regulation of CD22 expression and immunoglobulin M (IgM) production, markers of CLL differ
160 lymphoma characterized by hypersecretion of immunoglobulin M (IgM) protein and tumor infiltration in
163 (>/=7 days), respiratory manifestations, an immunoglobulin M (IgM) response in peripheral blood, and
165 cked VN activity; moreover, purified natural immunoglobulin M (IgM) restored VN activity to antibody-
168 infections and rule out misleading positive immunoglobulin M (IgM) results in areas with various lev
175 ompared the results of a serum-based measles immunoglobulin M (IgM) test with results of tests using
178 1) integrins reduced B. burgdorferi-specific immunoglobulin M (IgM) titers, enhanced pathogen burden,
180 icient mice were unable to class switch from immunoglobulin M (IgM) to IgG virus-neutralizing antibod
181 by a switch in anti-PPS14 from predominantly immunoglobulin M (IgM) to IgG1 by day 25 following prima
182 t assay (ELISA) for the detection of chicken immunoglobulin M (IgM) to WN virus was developed, standa
183 re to oral pathogens was determined by fetal immunoglobulin M (IgM) umbilical cord seropositivity.
184 In Yaounde, Cameroon, parasite-specific immunoglobulin M (IgM) was detected in 14% of cord blood
185 globulinemia (MC), a monoclonal expansion of immunoglobulin M (IgM)(+) autoreactive B cells, and also
186 these primary immunodeficiencies have fewer immunoglobulin M (IgM)(+)IgD(+)CD27(+) B cells, a popula
187 V(+)MC(+) subjects have clonal expansions of immunoglobulin M (IgM)(+)kappa(+)IgD(low/-)CD21(low)CD27
188 Levels of total immunoglobulin G (IgG), immunoglobulin M (IgM), and IgG subtypes against the fol
189 a direct relationship between surface CD79B, immunoglobulin M (IgM), and IgM-induced signaling levels
190 transfer of normal mouse serum or polyclonal immunoglobulin M (IgM), but not IgG, into B-cell-deficie
191 kin lymphoma that features overproduction of immunoglobulin M (IgM), clearly has a familial component
192 des stercoralis was shown to be dependent on immunoglobulin M (IgM), complement activation, and granu
195 enzyme-linked immunospot assay, we evaluated immunoglobulin M (IgM), IgA, and IgG antibody-secreting
197 serum or plasma samples were tested for WNV immunoglobulin M (IgM), IgA, and IgG by using enzyme-lin
199 All animals had high prechallenge levels of immunoglobulin M (IgM), IgG, IgG1, and IgG2 serum antibo
201 , as well as higher concentrations of plasma immunoglobulin M (IgM), relative to individuals with the
202 WM) is characterized by an overproduction of immunoglobulin M (IgM), which can lead to a hyperviscosi
207 on of EBV latency III LCL gene expression to immunoglobulin M (IgM)-stimulated B cells, germinal-cent
211 tion in bone marrow, lymph node development, immunoglobulin M (IgM)/IgG production, and humoral immun
213 58.95; P = .001) and high levels of phase II immunoglobulin M (IgM; AHR, 6.59; 95% CI, 1.37-31.62; P
214 further into the immune response by studying immunoglobulin-M (IgM) and IgG subclass 3 (IgG3) DSA to
215 the mechanisms behind WM transformation from immunoglobulin-M (IgM) monoclonal gammopathy of undeterm
216 njugates, but the Inaba-specific antibodies (immunoglobulin M [IgM] and IgG1) were neither vibriocida
217 mice produced human monoclonal paraprotein (immunoglobulin M [IgM] and/or kappa or lambda chain) det
219 resistance of monoclonal antibodies B6.1 (an immunoglobulin M [IgM]) and C3.1 (an IgG3) against exper
220 e same HA copy number), the highest primary (immunoglobulin M [IgM]) and secondary (IgG) anti-HA humo
221 CAR bacillus-specific serum immunoglobulins (immunoglobulin M [IgM], IgG1, IgG2a, IgG2b, IgG3, and Ig
222 use mice (transgenic mice that express human immunoglobulin M [IgM], IgG2, and kappa) which were immu
223 three populations: somatically hypermutated immunoglobulin M(+) (IgM(+)) and IgG(+) MBC subsets and
227 re stained for galactose-alpha1,3-galactose, immunoglobulin M, immunoglobulin G, complement, fibrin,
228 l enzyme-linked immunosorbent assay kits for immunoglobulin M/immunoglobulin G antibodies to herpes s
229 globulins enriched with immunoglobulin A and immunoglobulin M improve the integrity of the blood-brai
232 detectable levels of immunoglobulin G and/or immunoglobulin M in human plasma samples after treatment
233 In Gp1, anti-A/B and cytotoxic anti-pig immunoglobulin-M increased steadily during the first yea
237 s and secreting Stx1-specific Hu-MAbs (seven immunoglobulin M(kappa)() [IgM(kappa)] elements [one spe
238 8 pneumococcal capsular polysaccharide D11 [immunoglobulin M(kappa)] protects wild-type and compleme
239 h nonsense CXCR4 mutations have higher serum immunoglobulin M levels and incidence of symptomatic hyp
240 w lower bone marrow disease burden and serum immunoglobulin M levels but show an increased risk of de
241 declined from 55% to 10% (P = .0004), serum immunoglobulin M levels declined from 4,830 to 1,115 mg/
242 bone marrow (BM) disease involvement, serum immunoglobulin-M levels, and symptomatic disease requiri
243 WILDTYPE (WT)) had intermediate BM and serum immunoglobulin-M levels; those with MYD88(WT)CXCR4(WT) s
244 enes (>5% deviation from germ line), surface immunoglobulin M ligation failed to induce receptor tran
245 were immunized with melanin ghosts, and two immunoglobulin M MAbs to melanin were generated from the
246 derly individuals have a lower percentage of immunoglobulin M memory B cells than healthy young adult
247 t expression of the secretory heavy chain of immunoglobulin M (micros), is well-tolerated in HeLa cel
252 DS Eligible patients had measurable disease (immunoglobulin M monoclonal protein > 1,000 mg/dL with >
254 mpared to those for a cohort of B19-specific immunoglobulin M-negative (IgM(-)), IgG(+) remotely infe
255 lored whether the presence of lipid-specific immunoglobulin M oligoclonal bands in cerebrospinal flui
256 globulins enriched with immunoglobulin A and immunoglobulin M on blood-brain barrier integrity and su
258 at IFNalpha2b does not significantly inhibit immunoglobulin M or G serologic responses to the vaccine
261 ad no detectable mature T and B cells, serum immunoglobulin M, or even Thy-1(+) and B220(+) precursor
264 ients had significant increase in polyclonal immunoglobulin M (P = .005), indicating innate immune sy
265 rom their original discovery 20 years ago in immunoglobulin M paraproteinaemic neuropathy through to
267 based on seroconversion for HCMV and/or HCMV immunoglobulin M-positive and low or moderate HCMV immun
269 s (P < 0.05) were evident in CD4(+), CD8(+), immunoglobulin M-positive, and CD172a(+) cell fractions
275 >/=0.2 g/24 h), absence of intact monoclonal immunoglobulin (M protein) in the serum, and no evidence
283 erm persistence of anti-viral capsid antigen immunoglobulin M responses in children from the high-mal
285 versus postoutbreak sample, and near-farm by immunoglobulin M seroprevalence in a municipal populatio
286 B cells from apoptosis and increases surface immunoglobulin M (sIgM) expression on murine splenic B c
287 ates of anergy, indicated by reduced surface immunoglobulin M (sIgM) levels and signaling, consequent
288 ivation also correlated closely with surface immunoglobulin M (sIgM) signaling capacity in vitro in b
289 lls were stimulated with antibody to surface immunoglobulin M (sIgM), while in B cells from uninfecte
290 ps infection can be made by the detection of immunoglobulin M-specific antibodies to mumps virus in a
291 intracellular calcium that accompanies anti-immunoglobulin M stimulation, and this effect mediates t
292 TFE3 and TFEB in T cells resulted in a hyper-immunoglobulin M syndrome due to impaired expression of
295 globulins enriched with immunoglobulin A and immunoglobulin M treatment, no ultrastructural evidence
298 produced immunoglobulin G predominantly, but immunoglobulin M was predominant in younger children.
299 ain, immunoglobulin G, immunoglobulin A, and immunoglobulin M were measured on ICU days 1, 3, and 7.
300 in ZAP-70-positive CLL trigger secretion of immunoglobulin M, which then serves as (auto-) antigen f
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