ライフサイエンスコーパス: immunoglobulin V

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1 ell repertoire is generated by combinatorial immunoglobulin V(D)J gene segment rearrangements that oc

2 A-breaks induce error-prone DNA synthesis in immunoglobulin V(D)J regions by error-prone DNA polymera

3 tigen receptors through the rearrangement of immunoglobulin V, D, and J gene fragments, whereas jawle

4 l hotspot in the Ser31 codon of a transgenic immunoglobulin V gene allowed us to use PCR to detect tr

5 cell lymphoma leads to a failure to perform immunoglobulin V gene conversion.

6 blated the pattern of diversification of the immunoglobulin V gene in the chicken DT40 B-cell lymphom

7 Leukemic cells of the immunoglobulin V gene mutation subgroups differ in telom

8 henotypes of B-CLL patients with and without immunoglobulin V gene mutations suggests that the 2 subg

9 thways for processing a common lesion in the immunoglobulin V gene.

10 gnition, VRC01 contacts gp120 mainly through immunoglobulin V-gene regions substantially altered from

11 al cases of CLL cells that expressed mutated immunoglobulin V genes and ZAP-70 also experienced highe

12 After gene rearrangement, immunoglobulin V genes are further diversified by either

13 lines that constitutively hypermutate their immunoglobulin V genes during culture can be exploited t

14 that major intrinsic mutational hotspots in immunoglobulin V genes in vivo might reflect favoured si

15 We find that although the codon usage in immunoglobulin V genes renders them distinct among trans

16 action, random mutations are introduced into immunoglobulin V genes to increase the affinity of antib

17 of human heavy, kappa and lambda light chain immunoglobulin V-genes are in reasonably good agreement

18 Mutations in immunoglobulin V-genes can be due to gene multiplication

19 ytes and that the B-CLL cells that differ in immunoglobulin V genotype may have different antigen-enc

20 reaks specifically in the variable region of immunoglobulin (V(H)) gene locus, leading to hypermutati

21 raction induces hypermutation of heavy-chain immunoglobulin (V(H)) in B cells.

22 shown to express a restricted repertoire of immunoglobulin V(H) and V(L) genes, V(H)1-69 and VkappaA

23 Although immunoglobulin V(H) mutation status (IgV(H) MS) is progn

24 hisms in a human autosomal region (i.e., the immunoglobulin V(H) region), through use of single haplo

25 activating function for Pax5 in RAG-mediated immunoglobulin V(H)-to-DJ(H) recombination.

26 mutation (SHM) process that normally targets immunoglobulin V (IgV) genes in B cells appears to misfi

27 immunotherapy with steroids and intravenous immunoglobulins vs. late immunotherapy), and a low white

28 f ligands by the presence of a single set of immunoglobulin-V-like and immunoglobulin-C-like (VC) dom

29 s that have failed to productively rearrange immunoglobulin V region genes encoding a functional B-ce

30 Previous analyses of immunoglobulin V region promoters has led to the discove

31 equent DNA repair generates mutations within immunoglobulin V-regions.

32 e the altered alternative splicing of CD33's Immunoglobulin V-set domain 2 and ultimately influence A

33 e, using NMR spectroscopy we showed that the immunoglobulin V-type domain of RAGE is responsible for

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