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1 cked at the progenitor B cell stage prior to immunoglobulin gene rearrangement.
2 on from B-0 (also known as B-2) occurs after immunoglobulin gene rearrangement.
3 mammalian DNA double-strand break repair and immunoglobulin gene rearrangement.
4 ressed the transgene and excluded endogenous immunoglobulin gene rearrangements.
5 reaction for clone-specific T-cell receptor/immunoglobulin gene rearrangements.
6 nds, and Rag1 is a transposase that mediates immunoglobulin gene rearrangements.
7 is that should occur following nonproductive immunoglobulin gene rearrangements.
8 ssociated antigens (11/15), exhibited clonal immunoglobulin gene rearrangements (13/13), contained Ep
9 ll antigen receptor through nested secondary immunoglobulin gene rearrangements, a process termed rec
11 for DNA double-strand break (DSB) repair and immunoglobulin gene rearrangement and may play a role in
12 nal center B cells creates the potential for immunoglobulin gene rearrangement and the generation of
16 t microbes can induce specific signatures of immunoglobulin gene rearrangements and that pathogen exp
18 nt is a highly ordered process that involves immunoglobulin gene rearrangements, antigen receptor exp
19 ne conversion but also prevents the aberrant immunoglobulin gene rearrangements associated with RAD18
21 cillary testing included PCR for heavy chain immunoglobulin gene rearrangements, immunohistochemistry
22 ents occur in developing macronuclei, as for immunoglobulin gene rearrangements in mammals, but not d
28 machinery that is responsible for secondary immunoglobulin gene rearrangement, we examined the expre
29 Clonal CD27(+)ALDH(high) B cells, sharing immunoglobulin gene rearrangements with lymph node HRS c
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