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1 seizure frequency was associated with use of immunomodulatory therapy.
2 e response of multiple sclerosis patients to immunomodulatory therapy.
3 odels, provides a target for diagnostics and immunomodulatory therapy.
4 rome, suggests a potential narrow target for immunomodulatory therapy.
5  combination of systemic corticosteroids and immunomodulatory therapy.
6 ng inflamed eyes being treated with systemic immunomodulatory therapy.
7 g as well as for development of prophylactic immunomodulatory therapy.
8 plastic AIR should be treated with long-term immunomodulatory therapy.
9 IR patients should be treated with long-term immunomodulatory therapy.
10               Thirty-seven patients received immunomodulatory therapy.
11 armacological inhibitors of ion channels for immunomodulatory therapy.
12 nic demyelinating optic neuropathy on stable immunomodulatory therapy.
13  a minimum of 3 months following a switch of immunomodulatory therapy.
14 ance can be overcome by autoantigen-specific immunomodulatory therapy.
15 glecs could provide novel targets for cancer immunomodulatory therapy.
16 ur cohort of patients who received frontline immunomodulatory therapy.
17 uveitis when used as an adjuvant to systemic immunomodulatory therapy.
18 (22/25) when used as an adjuvant to systemic immunomodulatory therapy.
19 bidity despite continuous systemic and local immunomodulatory therapy.
20 fied short-term to chronic antibiotic and/or immunomodulatory therapy.
21                    IVIg is widely used as an immunomodulatory therapy.
22 gets, and as such are attractive targets for immunomodulatory therapy.
23 hosphate lyase may be a potential target for immunomodulatory therapy.
24 ssues to better inform future application of immunomodulatory therapies.
25 ion, suggesting a direction for pro-tolerant immunomodulatory therapies.
26 with OCD and tic disorders will benefit from immunomodulatory therapies.
27  documenting immune responses in studies for immunomodulatory therapies.
28 ing tolerance of skin may require additional immunomodulatory therapies.
29 ction without the requirement of vaccines or immunomodulatory therapies.
30 nment on the final outcome of antibody-based immunomodulatory therapies.
31     These conditions often improve following immunomodulatory therapies.
32  to identify patients who would benefit from immunomodulatory therapies.
33 inflammation continues with greater focus on immunomodulatory therapies.
34 ma either alone or in combination with these immunomodulatory therapies.
35 hich suggests ZAP-70 as a logical target for immunomodulatory therapies.
36 will be additive or synergistic with current immunomodulatory therapies.
37 of human DCs, presenting a unique target for immunomodulatory therapies.
38 sponse and therefore is a logical target for immunomodulatory therapies.
39 ddress this issue in the context of lymphoma immunomodulatory therapy, a workshop was convened to pro
40 new population niches owing to the advent of immunomodulatory therapies and increased numbers of pati
41 ttern, development of effective antiviral or immunomodulatory therapies and vaccines should become sc
42 sence of major comorbidities, treatment with immunomodulatory therapy and disruption of the microbiom
43 onparaneoplastic AIR patients with long-term immunomodulatory therapy and that there is enough equipo
44 e of his or her disease for consideration of immunomodulatory therapy, and will require legislation m
45                           Antiangiogenic and immunomodulatory therapies are gaining momentum in the p
46     However, attempts at introducing various immunomodulatory therapies as a new treatment strategy h
47 c immunoglobulins are used as replacement or immunomodulatory therapy, but can transmit clinically im
48  Prospective controlled clinical trials with immunomodulatory therapy can help define future treatmen
49 heson 2014 criteria and LYmphoma Response to Immunomodulatory therapy Criteria (LYRIC) (2016 revised
50 s of monoclonal antibody therapies and other immunomodulatory therapies currently being contemplated
51                             The responses to immunomodulatory therapies, defined by changes in modifi
52 entially reversible, justifying early use of immunomodulatory therapy directed at lowering IgG levels
53                          Currently available immunomodulatory therapies do not stop the pathogenesis
54                                  Although 12 immunomodulatory therapies exist, they have only modest
55      Despite the immunogenicity of melanoma, immunomodulatory therapies fail in the majority of patie
56 d improved design of cytokine based clinical immunomodulatory therapies for cancer and infectious dis
57 evelopment of specific, clinically available immunomodulatory therapies for Graves eye disease.
58 IDS but also occurs in individuals receiving immunomodulatory therapies for immune-related diseases s
59 by donor human leukocyte antigens (HLA), new immunomodulatory therapies for organ-transplant recipien
60 findings suggest that future studies seeking immunomodulatory therapies for preterm infants should co
61 hese developments highlight the potential of immunomodulatory therapies for treatment of these condit
62 ects of chronic MS and had not been received immunomodulatory therapy for MS.
63                 Lactate could be a promising immunomodulatory therapy for patients with acute organ i
64 who are considering lymphocytotoxic or other immunomodulatory therapy for RA.
65                  IFN-beta-1b is a first-line immunomodulatory therapy for relapsing-remitting multipl
66 ficant limitations of rhIL-10 as a potential immunomodulatory therapy for sepsis.
67               Recent studies have shown that immunomodulatory therapy for the treatment of rheumatic
68                               The utility of immunomodulatory therapy for this condition remains unkn
69      High-dose i.v. Ig (IVIg) is a prominent immunomodulatory therapy for various autoimmune and infl
70                                              Immunomodulatory therapy has been associated with better
71                             As a result, new immunomodulatory therapies have been used to treat stero
72                                     Existing immunomodulatory therapies have had limited effects on p
73 gs (SAIDs), immunosuppressive therapy drugs (immunomodulatory therapy [IMT]), or biologic response mo
74 in tau and raising concerns about the use of immunomodulatory therapies in AD.
75  the potential to guide optimal selection of immunomodulatory therapies in individual patients and mo
76 om existing experience with the use of these immunomodulatory therapies in other conditions and that
77 firmed by the differential susceptibility to immunomodulatory therapies in vivo.
78 e implications of these cases for the use of immunomodulatory therapy in CD and the questionable asso
79 able in vivo model for examining the role of immunomodulatory therapy in modifying HIV infection.
80 mmunologists prepared to move on to systemic immunomodulatory therapy in those instances where the ch
81                                              Immunomodulatory therapy involving HTLV-1-infected patie
82    RECENT FINDINGS: Use of targeted biologic immunomodulatory therapy is becoming widespread and prov
83                      The issue of attempting immunomodulatory therapy is discussed in view of the pub
84 autoimmune liver disease for which effective immunomodulatory therapy is lacking.
85 detrimental or beneficial, before developing immunomodulatory therapies, it is necessary to better un
86 n of oncogene-targeted therapy together with immunomodulatory therapy may be ideal for the developmen
87                                              Immunomodulatory therapy may not always be required.
88                 When CpG was administered as immunomodulatory therapy prior to allergen sensitization
89 nsight into the design of clinical trials of immunomodulatory therapies, ranging from optimal patient
90                                              Immunomodulatory therapy represents an attractive approa
91 tivitis other than MMP, rather than systemic immunomodulatory therapy, resulting in irreversible clin
92                                              Immunomodulatory therapies should be investigated in adu
93                         17 patients required immunomodulatory therapies, six improved spontaneously,
94  creates new translational possibilities for immunomodulatory therapies, specifically vaginal therapi
95 n inhibition might be a useful supplement to immunomodulatory therapies such as corticosteroids in ON
96 t investigations for diagnosis; conventional immunomodulatory therapies, such as interferon beta and
97 ves have been further studied, and promising immunomodulatory therapies, such as targeted B-cell ther
98                                              Immunomodulatory therapies targeting MMPs in preclinical
99             It highlights targets for future immunomodulatory therapy that may treat and potentially
100 esponse have fueled considerable interest in immunomodulatory therapy, the role of such agents in cli
101  micro- and nanoparticles are often added to immunomodulatory therapies to enhance the triggered immu
102 gun, with the possibility of designing novel immunomodulatory therapies to intervene with neuroinflam
103 gether, our data highlight the potential for immunomodulatory therapies to stimulate alveologenesis i
104 y IL-1 response could be used as a potential immunomodulatory therapy to improve the outcome of asper
105 e spread of nodal metastases and the role of immunomodulatory therapy to prevent or possibly even rev
106 optive T-cell therapy development as well as immunomodulatory therapy tools available for immediate c
107 nd innovative strategies, including targeted immunomodulatory therapies, tyrosine kinase inhibitors a
108              Emerging evidence suggests that immunomodulatory therapies used in nephrotic syndrome di
109 ficantly influences its response to numerous immunomodulatory therapies used in organ transplantation
110            Prednisone doses before and after immunomodulatory therapy were compared.
111 ectious scleritis refractory to conventional immunomodulatory therapy who were seen at the Massachuse
112           The data support the potential for immunomodulatory therapy with CpG in early life to reduc
113 en antibiotic treatment, but not biologic or immunomodulatory therapy, with reduced proportions of 11

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