ライフサイエンスコーパス: immunopathogenesis

1 two independent processes essential for the immunopathogenesis.

2 tion of B cells in our overall picture of MS immunopathogenesis.

3 e important roles in Chlamydia infection and immunopathogenesis.

4 njury and significantly contributes to early immunopathogenesis.

5 g functions shaping the normal course of EAE immunopathogenesis.

6 s effector responses and eventually leads to immunopathogenesis.

7 ocal regulatory roles for B cells during EAE immunopathogenesis.

8 upstream mediator of innate immunity, in WNV immunopathogenesis.

9 ggesting a role for these cytokines in HIV-1 immunopathogenesis.

10 oth of which could be associated with C-IRIS immunopathogenesis.

11 are supportive of a primary T-cell-mediated immunopathogenesis.

12 e proinflammatory responses that can lead to immunopathogenesis.

13 ve been hypothesized to participate in lupus immunopathogenesis.

14 local inflammation, thereby contributing to immunopathogenesis.

15 ung transplantation with a poorly understood immunopathogenesis.

16 argeted by CTL that may be relevant for KSHV immunopathogenesis.

17 nt to the interpretation of murine models of immunopathogenesis.

18 CTL triggering and proliferation to prevent immunopathogenesis.

19 ine model may provide valuable insights into immunopathogenesis.

20 used to define the influence of G priming on immunopathogenesis.

21 considerations in the development of dengue immunopathogenesis.

22 lpha production that may be a factor in AIDS immunopathogenesis.

23 SS and reveal a novel role for IL-17A in TSS immunopathogenesis.

24 a sequence and expression level modulate T1D immunopathogenesis.

25 nent of Pneumocystis pneumonia (PcP)-related immunopathogenesis.

26 ry response, which is the key feature of its immunopathogenesis.

27 lexity fits better with our understanding of immunopathogenesis.

28 mune diseases might have opposite effects on immunopathogenesis.

29 derstood as is the precise role of DCs in AC immunopathogenesis.

30 the loss of Clc-5 1) exhibits IL-6-mediated immunopathogenesis, 2) significantly exacerbated DSS-ind

31 earch implicated cellular immunity in dengue immunopathogenesis, a wealth of newer data demonstrated

32 Despite advances in understanding its immunopathogenesis, an effective treatment remains to be

33 ive PcP, MyD88 signaling contributes to both immunopathogenesis and control of fungal burden.

34 plications for understanding type 1 diabetes immunopathogenesis and for designing antigen-based immun

35 m for the analysis of virus transmission and immunopathogenesis and for the generation of novel "bar-

36 portant information to facilitate studies of immunopathogenesis and HCV vaccine design and evaluation

37 e that sex is an important variable in tumor immunopathogenesis and immunotherapy responses through d

38 onary infection will deepen our knowledge of immunopathogenesis and is crucial for developing effecti

39 Studies of the immunopathogenesis and mechanisms of protective immunity

40 y play an important role in HIV-1-associated immunopathogenesis and NK cell dysfunction observed duri

41 w will describe current understanding of the immunopathogenesis and pathophysiology of anaphylaxis, f

42 s an altered host immune response, modifying immunopathogenesis and protecting from cerebral disease.

43 the P2X7 receptor has a role in periodontal immunopathogenesis and suggest that targeting of the P2X

44 CTLs should extend our understanding of the immunopathogenesis and the mechanism of clearance or per

45 rate the most severe consequences of malaria immunopathogenesis and the prospect of host survival.

46 and shed light on the role of pDCs in HIV-1 immunopathogenesis and therapy.

47 tant for understanding type 1 diabetes (T1D) immunopathogenesis and to design immune monitoring and i

48 models that could prove useful for studying immunopathogenesis and vaccine development against hepat

49 d phenomenon with important implications for immunopathogenesis and vaccine development.

50 Our data suggest a role for Notch-1 in SLE immunopathogenesis, and for the first time, we present m

51 tudies of hepatitis C virus (HCV) infection, immunopathogenesis, and resulting liver diseases have be

52 Yet, many aspects of its immunopathogenesis are not well understood.

53 Aspects of H pylori immunopathogenesis are reviewed and perspectives on the

54 elucidating the mechanisms of protection and immunopathogenesis associated with dengue virus infectio

55 Differences in immunopathogenesis between MCL and LCL may result from a

56 Monkeys are useful for studies of measles immunopathogenesis, but virus strains must be carefully

57 with our hypothesis that SigF may mediate TB immunopathogenesis by altering cell membrane properties,

58 effector cells, CXCL10 may contribute to the immunopathogenesis by recruiting inflammatory cells.

59 r, our data suggest that despite the risk of immunopathogenesis, CD8+ T cells use a perforin-dependen

60 current information about the epidemiology, immunopathogenesis, clinical presentations, diagnosis, a

61 ed mouse model should be useful for studying immunopathogenesis, diagnostic markers, and therapy of h

62 can illuminate studies of HCV transmission, immunopathogenesis, drug resistance development, and vac

63 that regulatory T (Treg) cells prevent liver immunopathogenesis during HIV-1 infection in a humanized

64 ide new insight linking Treg cells and liver immunopathogenesis during HIV-1 infection.

65 es which mediate protective immunity or host immunopathogenesis during M. tuberculosis infection in t

66 urn affects the adaptive immune response and immunopathogenesis during murine NCC.

67 neonatal RSV infection decreased Th2-biased immunopathogenesis during reinfection.

68 neonatal RSV infection decreased Th2-biased immunopathogenesis during reinfection.

69 including contributions to colonization and immunopathogenesis during vulvovaginal candidiasis.

70 r results demonstrate that the extent of the immunopathogenesis established early in HIV infection pr

71 r M1 and M2 alike) to a resting state, cease immunopathogenesis even as bacteria are eliminated by ot

72 ), potentially suggesting different roles in immunopathogenesis for complexes arising at different st

73 As insight in fungal immunopathogenesis forges ahead, interventions for boost

74 lexity of human immunodeficiency virus (HIV) immunopathogenesis has prompted multiple strategic appro

75 f experimental data on dry eye disease (DED) immunopathogenesis have been derived from a murine model

76 isms that contribute to dengue infection and immunopathogenesis have hampered the development of vacc

77 s is still unknown, recent insights into its immunopathogenesis have moved investigators closer to fi

78 trials, more sophisticated understanding of immunopathogenesis, histology, and specific autoantibody

79 and for the analysis of mechanisms of viral immunopathogenesis in AIDS.

80 oyed the db/db mouse model to understand WNV immunopathogenesis in diabetics.

81 eneity at the HLA region suggests a distinct immunopathogenesis in DR2 negative patients.

82 e the exploratory strategies used to dissect immunopathogenesis in human tuberculosis with the aim to

83 immune activation, and CD4(+) T cell death, immunopathogenesis in LTs during early infection remains

84 tance, we examined whether a SIV that causes immunopathogenesis in pigtail macaques could be made sen

85 pidly, elicited enhanced IFN-gamma-dependent immunopathogenesis in the form of acute fatty necrosis.

86 ter insight into the molecular correlates of immunopathogenesis in this disease model.

87 To advance studies of immunopathogenesis in this murine model, we immortalized

88 atory cells (Tregs) overlap or influence EAE immunopathogenesis independently has remained unanswered

89 In healthy individuals such immunopathogenesis is avoided by the presence of regulat

90 However, their role in immunopathogenesis is controversial.

91 resenting features, organ complications, and immunopathogenesis is important for timely diagnosis and

92 inflammasome complexes, but its role in WNV immunopathogenesis is not defined.

93 frequently fatal opportunistic mycosis whose immunopathogenesis is poorly understood.

94 luding hepatitis C virus (HCV) infection and immunopathogenesis, is essential for the study of hepati

95 may indicate a contribution of grancalcin to immunopathogenesis, it is not essential for vital leukoc

96 The resulting immunopathogenesis leads to lesions in epithelial lining

97 studies focusing on better understanding of immunopathogenesis may facilitate vaccine development an

98 uggests that slightly distinct mechanisms of immunopathogenesis may operate in susceptible C57BL/6 an

99 tigen-dependent phenotype may be relevant to immunopathogenesis mediated by certain microbial toxins.

100 f celiac disease and how current concepts of immunopathogenesis might provide alternative approaches

101 The evolving epidemiology, immunopathogenesis, molecular virulence studies, early d

102 rhesus macaque model closely resembles HIV-1 immunopathogenesis, neuropathogenesis, and disease progr

103 is providing important new concepts in viral immunopathogenesis, new anti-inflammatory drug leads and

104 udies are the first to examine the molecular immunopathogenesis of a hamster model of VL infection an

105 self-antigens may play a central role in the immunopathogenesis of AAA.

106 ce DCs via CCR7 expression contribute to the immunopathogenesis of AC, thereby allowing significant i

107 has been postulated to be a cofactor in the immunopathogenesis of acquired immunodeficiency syndrome

108 static IL-10 responses may contribute to the immunopathogenesis of active tuberculosis and propagatio

109 The role for IL-10 in the immunopathogenesis of acute toxoplasmosis following pero

110 ese findings support a role for IL-17 in the immunopathogenesis of acute vascular rejection and demon

111 ults demonstrate for the first time that the immunopathogenesis of adhesion formation is under the co

112 effector mechanism(s) that fails during the immunopathogenesis of AIDS to allow onset and progressio

113 The immunopathogenesis of AIDS-associated hepatitis was expl

114 In the present study, pathways in the immunopathogenesis of ALI were evaluated.

115 al and cellular responses and focused on the immunopathogenesis of allergic diarrhea.

116 litates studies on the role of lipids in the immunopathogenesis of allergy.

117 ss has been made in our understanding of the immunopathogenesis of ANCA-associated vasculitides.

118 forward and broaden the traditional focus of immunopathogenesis of arterial disease, with the goal of

119 er, we describe the role of vitamin D in the immunopathogenesis of atopic dermatitis and other allerg

120 past year, with particular attention to the immunopathogenesis of atopic dermatitis, as well as some

121 c engineering of RSV, while knowledge of the immunopathogenesis of augmented disease gives hope that

122 review will focus on recent advances in the immunopathogenesis of autoimmunity and will review preli

123 nd fibroproliferation, the central events in immunopathogenesis of BOS following human lung transplan

124 t anti-AEC antibodies may play a role in the immunopathogenesis of BOS in the absence of anti-HLA ant

125 ead toward new insights into the fundamental immunopathogenesis of brucellosis.

126 se as important virulence components for the immunopathogenesis of Candida vaginitis, with implicatio

127 The immunopathogenesis of cardiomyopathy, the main clinical

128 ttle is known about their partnership in the immunopathogenesis of Chagas disease, the chronic infect

129 The immunopathogenesis of Chlamydia trachomatis-induced ovid

130 results have important implications for the immunopathogenesis of chlamydial disease and new vaccine

131 rahepatic lymphocytes is associated with the immunopathogenesis of chronic hepatitis C disease.

132 gest that they play an important role in the immunopathogenesis of chronic hepatitis C.

133 Available data on the immunopathogenesis of chronic inflammatory demyelinating

134 are believed to be directly involved in the immunopathogenesis of chronic liver diseases.

135 en hypothesized to play an important role in immunopathogenesis of chronic rejection of transplanted

136 sponses to self-antigens that contributes to immunopathogenesis of chronic rejection.

137 ng complement, an important component in the immunopathogenesis of CIA.

138 The immunopathogenesis of CM immune reconstitution inflammat

139 role for chemokines in immune evasion and/or immunopathogenesis of CMV retinitis.

140 trophil recruitment, and contributing to the immunopathogenesis of crescentic GN.

141 of effector responses may play a role in the immunopathogenesis of DEN hemorrhagic fever.

142 lymphocytes is thought to contribute to the immunopathogenesis of dengue disease during secondary in

143 cific antibodies have been implicated in the immunopathogenesis of dengue during infancy.

144 infection are thought to play a role in the immunopathogenesis of dengue hemorrhagic fever (DHF) dur

145 IL-18 are two cytokines associated with the immunopathogenesis of diabetes in NOD mice.

146 should lead to a better understanding of the immunopathogenesis of disease and to more specifically t

147 s) are known to play a prominent role in the immunopathogenesis of disease.

148 TLR) 4 and determine its contribution to the immunopathogenesis of dry eye disease (DED).

149 ributes to the predominantly T-cell-mediated immunopathogenesis of dry eye disease.

150 Advances in understanding the immunopathogenesis of each of these entities offer many

151 s GP which may play an important role in the immunopathogenesis of Ebola virus infection.

152 pheral nerve damage in leprosy patients, the immunopathogenesis of ENL remains ill-defined.

153 les encoded by these genes contribute to the immunopathogenesis of experimental autoimmune encephalom

154 investigated the role of gelatinase B in the immunopathogenesis of experimental BP using mice contain

155 termine if autoantibodies play a role in the immunopathogenesis of experimental dry eye disease.

156 patterns of sequence change might reveal the immunopathogenesis of fibrosis progression.

157 n the host-parasite relationship, especially immunopathogenesis of filarial infection, may improve ou

158 from basic research primarily related to the immunopathogenesis of H pylori that have advanced the pr

159 he goal of this study was to investigate the immunopathogenesis of H. pylori-induced gastritis and de

160 ings may have important implications for the immunopathogenesis of HCV in both immune and hepatic cel

161 age underpinning the role of NK cells in the immunopathogenesis of HCV.

162 ection and dysfunction may contribute to the immunopathogenesis of HIV disease.

163 evels of CD8+ T cells may play a role in the immunopathogenesis of HIV in the female genital tract.

164 role of opioids, including methadone, in the immunopathogenesis of HIV infection and AIDS.

165 ay have significant in vivo relevance to the immunopathogenesis of HIV infection and AIDS.

166 that meth may function as a cofactor in the immunopathogenesis of HIV infection and may lead to the

167 The results suggest that the immunopathogenesis of HIV infection may not depend solel

168 irm that the GI tract is a key player in the immunopathogenesis of HIV infection, and they reveal a s

169 have been implicated as contributing to the immunopathogenesis of HIV infection.

170 ggest a potential unique role of pDCs in the immunopathogenesis of HIV-1 infection by inducing the de

171 Ls) have important and opposing roles in the immunopathogenesis of HIV-1 infection.

172 inflammation in lymphoid tissues and in the immunopathogenesis of HIV-1/SIV, and suggest that TLR3 l

173 CD4+ T cells play a central role in the immunopathogenesis of HIV/AIDS, and their depletion duri

174 hinders a more complete understanding of the immunopathogenesis of HTLV-1 infections.

175 generated significant new insights into the immunopathogenesis of human celiac disease.

176 ation as well as their potential role in the immunopathogenesis of human diseases.

177 tic cells (DC) play an important role in the immunopathogenesis of human immunodeficiency virus (HIV)

178 can exacerbate bacterial infections and the immunopathogenesis of human immunodeficiency virus type

179 s on current approaches to understanding the immunopathogenesis of human T cell lymphotropic virus (H

180 ity of powerful new tools for dissecting the immunopathogenesis of human tuberculosis, the generation

181 okines which are known to be involved in the immunopathogenesis of IBD.

182 pus have provided important insight into the immunopathogenesis of IgG autoantibody production and lu

183 The immunopathogenesis of immune reconstitution inflammatory

184 tumor-infiltrating lymphocytes (TILs) in the immunopathogenesis of individual cancer is a challenge f

185 phocytes (CTLs) play a prominent role in the immunopathogenesis of infection by the noncytolytic, per

186 L specificity may have a pivotal role in the immunopathogenesis of infection, and that simple quantit

187 eukocytes is known to strongly influence the immunopathogenesis of infection.

188 To investigate the immunopathogenesis of inflammation-associated fibrosis,

189 n, but little is known about its role in the immunopathogenesis of inflammatory bowel disease (IBD).

190 ng other cytokines likely have a role in the immunopathogenesis of IRIS.

191 learly identified as a Th1 inflammation, the immunopathogenesis of its counterpart inflammatory bowel

192 ationship of Th1/Th2 immune responses in the immunopathogenesis of JRA, and their effect on cytokine

193 d LTB4 mediator pathways are involved in the immunopathogenesis of LPS-induced experimental ALI.

194 ify a complex role for MMPs and TIMPs in the immunopathogenesis of lung allograft rejection, and indi

195 The complement pathway was implicated in the immunopathogenesis of lupus and other autoimmune disorde

196 The exact role of these cells in the immunopathogenesis of lupus-like disease in PN mice rema

197 se model provides a means to investigate the immunopathogenesis of M. pneumoniae infection and its po

198 (MIF) place it in a central position in the immunopathogenesis of many diseases.

199 istance genes and mechanisms relevant to the immunopathogenesis of mercury-induced autoimmunity shoul

200 microglia/macrophages play a key role in the immunopathogenesis of MS and its corresponding animal mo

201 The immunopathogenesis of multiple sclerosis (MS) has always

202 o the CNS is a prominent feature driving the immunopathogenesis of multiple sclerosis and its animal

203 An evolving understanding of the immunopathogenesis of multiple sclerosis suggests that d

204 plicate antibody-dependent mechanisms in the immunopathogenesis of multiple sclerosis.

205 thogen interactions and participating in the immunopathogenesis of mycobacterial infections.

206 implicate IL-6 as a critical molecule in the immunopathogenesis of NMO, and a critical role for T cel

207 ines should facilitate future studies of the immunopathogenesis of NSDC in the B10.D2-->BALB/c murine

208 ion of immune responses to self-antigens and immunopathogenesis of OAD following the administration o

209 tivation and alloantibody development in the immunopathogenesis of OAD.

210 d other inflammatory cells essential for the immunopathogenesis of ocular atopy.

211 M has emerged as an important host factor in immunopathogenesis of ocular HSV type 1 (HSV-1) infectio

212 The immunopathogenesis of paradoxical tuberculosis-associate

213 D8(+) T cells play a significant role in the immunopathogenesis of PBC.

214 te that mast cells and TNF contribute to the immunopathogenesis of periodontitis and may offer therap

215 Similarities exist in the epidemiology and immunopathogenesis of periodontitis and rheumatoid arthr

216 s jiroveci (P. jiroveci) is important in the immunopathogenesis of Pneumocystis pneumonia (PcP), but

217 Autoimmunity plays a key role in the immunopathogenesis of psoriasis; however, little is know

218 Recognition of the immunopathogenesis of psoriatic arthritis, as with rheum

219 approaches to define factors underlying the immunopathogenesis of pulmonary diseases including sarco

220 e notion of a potential role of IP-10 in the immunopathogenesis of RA.

221 prevalent African allele HLA-B*53:01 in the immunopathogenesis of raltegravir-induced DRESS syndrome

222 o play an important role in the immunity and immunopathogenesis of respiratory syncytial virus (RSV)

223 emokine that potentially plays a role in the immunopathogenesis of rheumatoid arthritis (RA).

224 ibute to disease susceptibility, whereas the immunopathogenesis of RPF is mediated by different immun

225 is model provides a means to investigate the immunopathogenesis of RSV infection and its association

226 Although T cells have been implicated in the immunopathogenesis of secondary infections with heterolo

227 The immunopathogenesis of severe dengue is poorly understood

228 ole for cross-reactive memory T cells in the immunopathogenesis of severe disease.

229 ce will provide additional insights into the immunopathogenesis of severe EBOV disease.

230 polymorphic variability in conditioning the immunopathogenesis of severe malarial anemia (SMA) remai

231 ral models have been proposed to explain the immunopathogenesis of severe T cell-mediated drug HSRs,

232 aberrant cytokine secretion that drives the immunopathogenesis of Sezary syndrome and other immunopa

233 The observations strengthen the immunopathogenesis of sIBM, and offer the basis for futu

234 arallels the increasing understanding of the immunopathogenesis of SLE and looks promising.

235 strate an extended role for CREMalpha in the immunopathogenesis of SLE because it contributes to incr

236 pression of multiple genes implicated in the immunopathogenesis of SLE suggests that this pharmacolog

237 al functions, exerts a dual influence on the immunopathogenesis of SLE.

238 ays and provide mechanistic insight into the immunopathogenesis of SLE.

239 amage that are the essential features of the immunopathogenesis of SLE.

240 Although the immunopathogenesis of SMA is largely undefined, we have

241 ctors VEGF and GM-CSF may be involved in the immunopathogenesis of subjects with CRS and nasal polyps

242 body-mediated neuronal cell signaling in the immunopathogenesis of Sydenham chorea and will lead to a

243 Much less is known about the immunopathogenesis of syndromes that affect the central

244 A peptide binding groove is important in the immunopathogenesis of T-cell mediated drug hypersensitiv

245 am-negative pathogens to host cells, but the immunopathogenesis of TAAs remains unknown.

246 n update of the current understanding of the immunopathogenesis of TED and explore these mechanisms f

247 ressive advances in the understanding of the immunopathogenesis of TED continue to spur clinical tria

248 (+) CTLs, thought to be major players in the immunopathogenesis of the disease.

249 the understanding of the immunogenetics and immunopathogenesis of the IIMs may in the future provide

250 a2(+) T-cell responses may contribute to the immunopathogenesis of the SIV-related tuberculosis-like

251 hough there are important differences in the immunopathogenesis of these 2 viral pathogens, little is

252 In just over 20 years, many aspects of the immunopathogenesis of these diseases have been dissected

253 gaps in knowledge remain with regard to the immunopathogenesis of these diseases.

254 AR, suggesting potential differences in the immunopathogenesis of these diseases.

255 rides in our understanding of the underlying immunopathogenesis of these disorders, which have led to

256 nsight into the mechanisms that underlie the immunopathogenesis of these persistent viral infections

257 or a model system to investigate the complex immunopathogenesis of this chronic disease and subsequen

258 Further understanding of the immunopathogenesis of this condition will facilitate dev

259 on caused by Mycobacterium tuberculosis, the immunopathogenesis of this disease is poorly understood.

260 tes and their functional relationship in the immunopathogenesis of this disease model by using mice d

261 on could provide important insights into the immunopathogenesis of this disease.

262 to deletion of MTB-reactive T cells and the immunopathogenesis of this disease.

263 To define the immunopathogenesis of this malaria-induced pregnancy com

264 ells and enterocytes) may play a role in the immunopathogenesis of this pathogen-driven experimental

265 To characterize the immunopathogenesis of this syndrome, we studied viral lo

266 The immunopathogenesis of this tissue response to infection

267 However, the immunopathogenesis of TSS and other diseases involving S

268 The immunopathogenesis of tuberculosis-associated immune rec

269 is surface are important determinants in the immunopathogenesis of tuberculosis.

270 d migrating capacities and their role in the immunopathogenesis of tuberculosis.

271 ould have implications for their role in the immunopathogenesis of tuberculosis.

272 sly unsuspected role for this protein in the immunopathogenesis of tuberculosis.

273 The immunopathogenesis of type 1 diabetes mellitus is associ

274 The immunopathogenesis of type 1 diabetes mellitus is associ

275 disease-relevant CD8 T cells that inform the immunopathogenesis of type 1 diabetes.

276 regulation, implying a role for CLC-5 in the immunopathogenesis of UC.

277 response is important for understanding the immunopathogenesis of VL.

278 The molecular immunopathogenesis of West Nile virus (WNV) infection is

279 geneic bone marrow transplantation (BMT) the immunopathogenesis of which is not well understood.

280 provides unique mechanistic insight into the immunopathogenesis of WNVE observed in diabetics and can

281 The impact of HIV-associated immunopathogenesis on B cells has been largely associate

282 st exclusively been held responsible for its immunopathogenesis, partly because certain MHC class II

283 CD8(+) T suppressor cell recruitment, limits immunopathogenesis, preserves lung function, and enhance

284 stain the autoreactive T cells essential for immunopathogenesis, promoting their accumulation and dis

285 and highlight a significant aspect of viral immunopathogenesis related to the progression of adult T

286 rucellosis diagnostics and understanding the immunopathogenesis related to this intracellular pathoge

287 g monocyte apoptosis response may inform HIV immunopathogenesis, retention of infected macrophages, a

288 Immunopathogenesis studies employing West Nile virus (WN

289 of viral replicative capacity (vRC) on HIV-1 immunopathogenesis that is independent of viral load (VL

290 letion in human immunodeficiency virus (HIV) immunopathogenesis, their homeostasis in peripheral bloo

291 llenges, epidemiology, aetiology, pathology, immunopathogenesis, therapy, genetic influences, and the

292 w regulatory mechanisms to be overridden and immunopathogenesis to proceed.

293 his nonhuman primate model of HIV-associated immunopathogenesis, using a virus derived from a human p

294 Respiratory syncytial virus (RSV) immunopathogenesis was evaluated in 129xC57BL/6, C57BL/6

295 AC immunopathogenesis was evaluated via clinical examinatio

296 ole of mycobacterial antigens in sarcoidosis immunopathogenesis, we assessed the immune recognition o

297 evaluate the importance of this mechanism of immunopathogenesis, we determined the relative contribut

298 role of each immune component in human tumor immunopathogenesis, we have studied the interaction betw

299 ds to identify genetic factors, studying the immunopathogenesis with human tissue and animal models,

300 e of MyD88 in the development of PcP-related immunopathogenesis, WT and MyD88(-/-) mice were rendered