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1 -specific CD8+ CTL have been suggested to be immunopathogenic.
4 These data suggest a distinct genetic and immunopathogenic basis for AIH and PBC at the MIF locus.
5 n in AC22 mice, thereby providing us with an immunopathogenic basis for the fatal outcome of SARS-CoV
8 he possibility that LIGHT may play a role in immunopathogenic conditions that are associated with loc
9 ignals transduced by HIV-1 envelope may have immunopathogenic consequences, including anergy, syncyti
12 ur knowledge regarding immune-protective and immunopathogenic events in severe acute respiratory synd
13 APCs) known to be critically involved in the immunopathogenic events leading to type 1 diabetes, we h
15 ome serves as a prototype model to study the immunopathogenic features of a human organ-specific auto
16 25(+)Foxp3(+) regulatory T cells (Tregs) are immunopathogenic in cancers by impeding tumor-specific i
18 hepatocellular carcinoma, and virus-mediated immunopathogenic infections, affect billions of people w
19 Animal and clinical studies suggest that the immunopathogenic inflammatory condition of VVC is initia
20 ines of the Th17 lineage, and therefore, the immunopathogenic inflammatory response during VVC occurs
22 ouse model of HSK is often used to delineate immunopathogenic mechanisms and bears many of the charac
23 an avenue to monitor MS and to characterize immunopathogenic mechanisms and therapeutic targets in t
25 plex array of multiphasic and multifactorial immunopathogenic mechanisms are involved in the establis
26 advances have increased understanding of the immunopathogenic mechanisms associated with the formatio
28 litis, tantalizing clues as to the potential immunopathogenic mechanisms in acute transverse myelitis
29 A, will be useful for the fine dissection of immunopathogenic mechanisms in EBA and for the developme
32 ensive cardiac inflammation, suggesting that immunopathogenic mechanisms may promote cardiomyopathy.
38 ease that serves as a model for studying the immunopathogenic mechanisms of uveitis and organ-specifi
39 rting the role of p38 MAPK in regulating key immunopathogenic mechanisms underlying autoimmune inflam
40 the white matter to include a broad range of immunopathogenic mechanisms, axonal damage, and widespre
41 ), in which CD4(+) Th1 and/or Th17 cells are immunopathogenic, mimics various clinical features of no
43 ults also show that beta-gal can serve as an immunopathogenic neural autoantigen, and that T cells ra
44 immune response could explain the increased immunopathogenic potential and associated increase in cl
46 rotein Hsp10 as a potential correlate to the immunopathogenic process in women with tubal factor infe
48 molecular forms of antigen may initiate the immunopathogenic processes in the two forms of anti-GBM
52 be a previously unrecognized Notch-mediated immunopathogenic role for stromal cell niches in seconda
53 bserved in Clcn5 KO mice, suggesting a novel immunopathogenic role for the functional defects that re
56 oth Tfh and Th17 cells from entering the non-immunopathogenic site, the gut, and retained these T eff
57 e transfer of EAU to naive syngeneic mice by immunopathogenic T cells and suppressive effects of p28/
59 mmune activation/suppression associated with immunopathogenic viruses such as hantaviruses; however,
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