ライフサイエンスコーパス: immunopathological

1 es where the immune response to pathogens is immunopathological.

2 We performed extensive immunopathological analyses of airway tissue inflammatio

3 Immunopathological analysis of heart tissues obtained at

4 Immunopathological analysis revealed that the cells in t

5 Therefore, the immunopathological and antiviral effects of CTLs can be

6 normally have a positive effect by limiting immunopathological and autoreactive immune responses, bu

7 e combination of the clinical, histological, immunopathological and immunogenetic features indicate t

8 This disease is considered to be immunopathological and mediated primarily by CD4+ T cell

9 milarities with lesions in humans suggest an immunopathological basis for HGE.

10 irst to demonstrate cellular immunity as the immunopathological basis of periodontal diseases, recurr

11 action of antiviral therapy in ameliorating immunopathological brain injury and ADC.

12 This may protect lymphoid tissues from immunopathological changes but compromise immune defense

13 To investigate time-related immunopathological changes in the lacrimal glands (LGs)

14 g a self-regulatory mechanism that minimizes immunopathological changes.

15 We describe for the first time the immunopathological characteristics of a novel inflammato

16 eases, but it remains unknown whether lethal immunopathological characteristics, caused by Schistosom

17 shown to ameliorate and reverse an existing immunopathological condition by inducing tolerance.

18 derlie the emerging role of NKG2D in various immunopathological conditions and define new therapeutic

19 rge amounts of IL-10, and suppress excessive immunopathological conditions elicited by recall expansi

20 eful therapeutic option for the treatment of immunopathological conditions including graft-versus-hos

21 NKT cells also regulate a range of immunopathological conditions, but the mechanisms and th

22 the development of HLH and potentially other immunopathological conditions.

23 nses, and the balance between protective and immunopathological consequences during the chronic stage

24 focusing which evidence suggests could have immunopathological consequences from cross-reactive reco

25 Our findings thus reveal unique immunopathological consequences of MyD88 signaling in B

26 ion in microbial burden while minimizing the immunopathological consequences of the host inflammatory

27 -MHC restriction and contribute to undesired immunopathological consequences.

28 Immunopathological damage in schistosomiasis consist of

29 ital tract to control infection and minimize immunopathological damage to vital reproductive structur

30 ation of a balanced T-cell response limiting immunopathological damage.

31 T cell responses, which sometimes results in immunopathological damage.

32 Clinical, radiological, and immunopathological data suggest that NMO-IgG might be pa

33 important but poorly understood roles in the immunopathological demyelinating disease multiple sclero

34 Infectious mononucleosis (IM) is an immunopathological disease caused by EBV that occurs in

35 st immunity and how control of parasitic and immunopathological disease might be achieved.

36 control lesion severity in a virally induced immunopathological disease.

37 control lesion severity in a virally induced immunopathological disease.

38 illustrate how aberrant immune responses and immunopathological diseases could be generated by EBV in

39 unopathogenesis of Sezary syndrome and other immunopathological diseases.

40 implications for understanding and treating immunopathological diseases.

41 various pathological processes, from chronic immunopathological disorders to cognitive and psychiatri

42 in the clinic for the treatment of different immunopathological disorders.

43 effective strategies to circumvent excessive immunopathological effects.

44 nervous system (CNS), is believed to have an immunopathological etiology arising from gene-environmen

45 terns of oligodendrocyte destruction and the immunopathological evidence of complement activation.

46 erns of oligodendrocyte destruction, and the immunopathological evidence of complement activation.

47 timicrobic susceptibility testing or in vivo immunopathological experiments.

48 The concomitant presence of these immunopathological features in the transgenic mice sugge

49 nism of Brucella-induced T-cell responses to immunopathological features is not known.

50 oter polymorphisms and CD4 decline and other immunopathological features like immune activation and C

51 romosomal regions (NOD.Abd3), reproduces the immunopathological features of NOD ABD.

52 to support the concept of patient-dependent immunopathological heterogeneity in early MS and suggest

53 Multiple sclerosis (MS) lesions demonstrate immunopathological heterogeneity in patterns of demyelin

54 Stromal keratitis is a chronic immunopathological lesion of the eye caused by HSV-1 inf

55 Stromal keratitis (SK) is a chronic immunopathological lesion of the eye caused by HSV-1 inf

56 n corneal neovascularization during blinding immunopathological lesion stromal keratitis (SK).

57 e have evaluated the role of T(reg) in viral immunopathological lesion stromal keratitis.

58 se corneas can lead to the development of an immunopathological lesion, termed herpetic stromal kerat

59 tuents impacts the subsequent development of immunopathological lesions.

60 haracterized by severe hyperinflammation and immunopathological manifestations in several tissues.

61 the MHC/CD8 complex is considered a specific immunopathological marker because it distinguishes the a

62 distinct sets of such strains did not elicit immunopathological markers at levels above those observe

63 Immunopathological mechanisms appear to contribute to th

64 to be elucidated, the evidence suggests that immunopathological mechanisms are involved.

65 mon skin and synovial antigens led to shared immunopathological mechanisms at these different sites,

66 cal manifestations of AMR, and discusses the immunopathological mechanisms contributing to antibody-m

67 om direct ehrlichia-mediated injury but from immunopathological mechanisms initiated by ehrlichial in

68 knowledge of costimulatory molecule-mediated immunopathological mechanisms of uveitis and suggests a

69 Dissection of the underlying immunopathological mechanisms revealed the elaborate Tre

70 ing CVB4-V infection is able to suppress the immunopathological mechanisms that lead to chronic disea

71 eater understanding of time and place in the immunopathological mechanisms underlying human tuberculo

72 The immunopathological mechanisms underlying TB-IRIS are inc

73 ronic pancreatitis, in this model, is due to immunopathological mechanisms.

74 bundant ELSs and found that they constituted immunopathological microniches wherein malignant hepatoc

75 and highlight the importance of LPDA1 as an immunopathological molecule.

76 lly, this may leave them more susceptible to immunopathological morbidity when attempting to clear ex

77 trated that one lung infection modulates the immunopathological outcome to a subsequent unrelated pat

78 hat infect the intestine but differ in their immunopathological outcomes.

79 Four distinct immunopathological patterns have been described in multi

80 y-one patients (95%) showed a persistence of immunopathological patterns in tissue sampled from diffe

81 a profound, interindividual heterogeneity in immunopathological patterns of active demyelination, whi

82 The structural and immunopathological patterns of demyelination suggest tha

83 active multiple sclerosis lesions show four immunopathological patterns of demyelination.

84 l nervous system is characterized by several immunopathological patterns that suggest the involvement

85 ilies that influence susceptibility to other immunopathological phenotypes, including survival after

86 ntial role of CTLs as final effectors of the immunopathological process in narcolepsy.

87 tax gene expression provokes and sustains an immunopathological process that progressively destroys m

88 gression, and various cytokines increase the immunopathological process.

89 We conclude that different immunopathological processes (endotypes) may underlie ty

90 have emerged as important in the control of immunopathological processes.

91 tution inflammatory syndrome (TB-IRIS) is an immunopathological reaction to mycobacterial antigens in

92 ous system (CNS) is relatively intolerant of immunopathological reactions.

93 may also initiate negative feedback against immunopathological reactions.

94 applied as a means to control virus-induced immunopathological reactions.

95 this protozoan parasite induces an abnormal immunopathological response consisting of pancreatic tis

96 dengue virus infection, is believed to be an immunopathological response to a secondary infection wit

97 odeficiency virus pneumonia may represent an immunopathological response to macrophage-tropic virus.

98 immunoinflammatory lesion that represents an immunopathological response to the infection.

99 hat both T-cell subsets were involved in the immunopathological response.

100 environmental promoting factors provokes an immunopathological response.

101 molecular circuits that prevent or regulate immunopathological responses against one parasite is lik

102 egulatory T (T(R)) cells can inhibit harmful immunopathological responses directed against self or fo

103 e modulation that we might apply to diminish immunopathological responses that cause human diseases.

104 development of novel interventions to limit immunopathological responses to infection.

105 is model should generate novel insights into immunopathological responses to viral ocular infection.

106 onses to sequential infection, and excessive immunopathological responses, co-infections are noted by

107 efinitive evidence that T(reg) cells have an immunopathological role in human cancer is lacking.

108 ersial, but most studies admit the important immunopathological role of infiltrating CD8+ (cytotoxic)

109 ion between chlamydial hsp60 and destructive immunopathological sequelae in infected patients.

110 sis of TBM to discover strategies to prevent immunopathological sequelae.

111 and suggesting the potential development of immunopathological side effects when interfering with th

112 Clinically, immunopathological staining showed strong PTGR2 expressi

113 Immunopathological studies demonstrate the deposition of

114 Immunopathological studies indicated that TCRdelta(-/-)

115 Immunopathological studies suggest that the target of im

116 Advanced immunopathological techniques hold promise for more prec