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1 TSPyV) occur during primary infection of the immunosuppressed host.
2 uce Pcp when the organism is acquired by the immunosuppressed host.
3 mans cells as they produced meningitis in an immunosuppressed host.
4 anting further analysis of virus subtypes in immunosuppressed hosts.
5 in normal hosts cause significant disease in immunosuppressed hosts.
6 viral therapy exists for adenovirus (ADV) in immunosuppressed hosts.
7 ortunistic infection, especially in severely immunosuppressed hosts.
8 n of the JC virus, which generally occurs in immunosuppressed hosts.
9 K variants arising in immunocompetent versus immunosuppressed hosts; and 2) by investigating the effe
10 reactivation of the latent adenovirus in the immunosuppressed host as the source of infection.
11 on of hepatocytes, so it is paradoxical that immunosuppressed hosts have higher rates of fibrosis pro
12                                       In the immunosuppressed host, however, a wide spectrum of infec
13                          Most cases occur in immunosuppressed hosts, including patients with HIV/AIDS
14      Cryptosporidium parvum infection in the immunosuppressed host is frequently complicated by bilia
15 esistance in intensive care units, expanding immunosuppressed host populations, and public health cri
16                          By contrast, in the immunosuppressed host TIMP-1 mRNA was expressed by CNS r
17 for Epstein-Barr virus (EBV) pathogenesis in immunosuppressed hosts, we studied experimental infectio
18                                     Here, in immunosuppressed hosts, we uncover rare naturally arisin
19 lted from incomplete viral suppression in an immunosuppressed host with a high viral load.
20                                           In immunosuppressed hosts with a high organism burden, orga
21 en recognized to be pathogenic in profoundly immunosuppressed hosts with increasing frequency, this i

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