ライフサイエンスコーパス: impaired healing

1 process and deficiencies in the MMP9 lead to impaired healing.

2 een associated with chronic inflammation and impaired healing.

3 is associated with chronic inflammation and impaired healing.

4 agents, is giving new hope to the problem of impaired healing.

5 clinical studies and in our animal models of impaired healing.

6 which suffer from neutrophil persistence and impaired healing.

7 iabetic complications, such as infection and impaired healing after tissue damage.

8 tailored "living bandage" for patients with impaired healing and can serve as prototype for the deve

9 Corneal injury in HO-2 null mice leads to impaired healing and chronic inflammatory complications,

10 urnal rhythm disruption immediately after MI impaired healing and exacerbated maladaptive cardiac rem

11 of numerous tissues, and they contribute to impaired healing and fibrosis.

12 ic wounds may serve as a molecular marker of impaired healing and may provide future targets for ther

13 nds is believed to play an important role in impaired healing and the development of infection-relate

14 FAs exhibited positive correlations with OA, impaired healing, and inflammatory adipokines.

15 Prolonged inflammation and impaired healing are hallmarks of the diabetic response

16 Mechanisms underlying impaired healing are not defined.

17 beta-catenin/c-myc pathway(s) contributes to impaired healing by inhibiting keratinocyte migration an

18 in, thus providing a potential mechanism for impaired healing by local B2AR activation in wound-edge

19 to the persistent inflammatory response and impaired healing characteristic of these wounds.

20 Interleukin-23p19-/- mice showed impaired healing compared with wild-type mice, as seen b

21 migration by impairing connexin 43, and that impaired healing during NEC is associated with reduced g

22 how that local injection of DHEA accelerates impaired healing in an ageing mouse colony.

23 DHEA accelerated impaired healing in an impaired healing model (mice rend

24 actors in chronic wounds and the reversal of impaired healing in animal models, pressure ulcer trials

25 the molecular mechanisms and pathogenesis of impaired healing in chronic ulcers is a serious health i

26 The EP4 agonist markedly improved the impaired healing in COX-2(-/-) mice, as evidenced by res

27 und macrophage phenotype that contributes to impaired healing in diabetes.

28 ive shift in wound microbiota coincides with impaired healing in diabetic mice (Lepr(db/db); db/db).

29 ecifically, we show that in vivo FMT detects impaired healing in FXIII-/- mice.

30 Cited2 deficient cornea exhibited impaired healing in response to corneal epithelial debri

31 aging to compare the course of efficient and impaired healing in wild-type and FXIII-/- mice, respect

32 mice but reduced by 90% in CD73 mutants, (3) impaired healing involves M1-driven immune response with

33 issue formation in healing gingiva, and that impaired healing is associated with factors that decreas

34 Impaired healing is common in wounds infected with the m

35 und healing complications (wound dehiscence, impaired healing, lymphocele, and incisional hernia) wit

36 Patients at risk for impaired healing may benefit from prophylactic measures

37 DHEA accelerated impaired healing in an impaired healing model (mice rendered hypogonadal) assoc

38 peptic ulcer or troublesome dyspepsia led to impaired healing of gastric ulcers and did not affect th

39 In addition, TNF-alpha mice displayed impaired healing of incisional and excisional skin wound

40 We investigated whether impaired healing of injured PHT gastric mucosa is due to

41 ntium Correspondingly, AQP3(-/-) mice showed impaired healing of superficial wounds in the colon and

42 function but is insufficient to correct the impaired healing of the HO-2(-/-) cornea, suggesting tha

43 uring the past 3 decades presented issues of impaired healing or increased risk of cardiac rupture or

44 akage and reduced fibroplasia, indicating an impaired healing response consistent with angiogenesis b

45 d2(-/-) skin microbiome dominated and caused impaired healing, shown in cross-fostering experiments o

46 Impaired healing states are characterized by excessive p

47 ith chronic Ly-6C(hi) monocytosis results in impaired healing, underscoring the need for a balanced a

48 Local and systemic factors can contribute to impaired healing, with the potential to prolong function