ライフサイエンスコーパス: impulsive aggression

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1 familial transmission (eg, mood disorder and impulsive aggression).

2 n and impulsivity and a composite measure of impulsive aggression.

3 centration were accounted for by measures of impulsive aggression.

4 ygdala circuits have long been implicated in impulsive aggression.

5 t model for gene-environment interactions in impulsive aggression.

6 antisuicidal properties through reduction in impulsive aggression.

7 be mediated by the familial transmission of impulsive aggression.

8 smission of suicidal behavior is mediated by impulsive aggression.

9 s the categorical expression of pathological impulsive aggression.

10 ne (m-CPP), was examined in 13 subjects with impulsive aggression and 13 normal controls.

11 Impulsive aggression and borderline personality disorder

12 To examine further the relationship between impulsive aggression and orbital/medial prefrontal dysfu

13 ly treatment of mood disorders and targeting impulsive aggression and sexual trauma may be helpful in

14 order and is also related to the offspring's impulsive aggression and the familial transmission of se

15 We posit that impulsive aggression and violence arise as a consequence

16 isorder, substance abuse disorder, increased impulsive aggression, and a history of sexual abuse.

17 to psychopathology, suicide attempt history, impulsive aggression, and exposure to familial adversity

18 athologic findings, suicide attempt history, impulsive aggression, and exposure to familial environme

19 bused, such steroids can cause irritability, impulsive aggression, and signs of major depression, but

20 on measures of depressed mood, hopelessness, impulsive aggression, and suicidal behavior, including l

21 ior, studies of white matter connectivity in impulsive aggression are warranted.

22 spring mood disorder at each time point, and impulsive aggression as a precursor of mood disorder.

23 suicidal behavior include mood disorder and impulsive aggression as well as parental history of suic

24 nd their offspring reported greater lifetime impulsive aggression compared with each of the other two

25 Unlike normal subjects, patients with impulsive aggression did not show activation specificall

26 A disorder of impulsive aggression has been included in DSM since the

27 Impulsive aggression (IA) in adults is associated with b

28 orts the hypothesis that lithium use reduces impulsive aggression in addition to stabilizing mood.

29 se A (MAOA) gene, previously associated with impulsive aggression in animals and humans.

30 Interventions that target mood disorder and impulsive aggression in high-risk offspring may attenuat

31 cture of the central neuromodulatory role of impulsive aggression in human subjects.

32 cture of the central neuromodulatory role of impulsive aggression in human subjects.

33 munoreactivity concentration and measures of impulsive aggression in human subjects.

34 Mood disorder and self-reported impulsive aggression in offspring and a history of sexua

35 ation of inhibitory regions in patients with impulsive aggression in response to a serotonergic stimu

36 Hopelessness and impulsive aggression independently increase the risk of

37 ic patients with a disorder characterized by impulsive aggression, Intermittent Explosive Disorder (I

38 Impulsive aggression is a prevalent problem and yet litt

39 bsequently, renewed interest in disorders of impulsive aggression led to a recent series of community

40 An increase in either impulsive aggression or hopelessness or a diagnosis of b

41 rs, such as depression, childhood abuse, and impulsive aggression, report inconsistent results.

42 be involved in the association of MAOA with impulsive aggression, suggest neural systems-level effec

43 In the offspring, impulsive aggression was the most powerful predictor of

44 ation of brain lesions that produce acquired impulsive aggression, we hypothesized that IED subjects

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