ライフサイエンスコーパス: in ovo

1 hibited the replication of avian RNA viruses in ovo.

2 ogenous NT-3, during early heart development in ovo.

3 gonists, Noggin, to the developing inner ear in ovo.

4 avian replication-incompetent viral vectors in ovo.

5 n cells and cell processes from around day 7 in ovo.

6 stinguishable from neurons only after day 16 in ovo.

7 ations of chick gut from as early as day 4.5 in ovo.

8 dendritic arborization of spinal motoneurons in ovo.

9 in vitro, and reveals proangiogenic effects in ovo.

10 ons using a heterochronic grafting strategy, in ovo.

11 ng and/or maintaining optic cup invagination in ovo.

12 cimol, agents that reduce activation of LMNs in ovo.

13 ity at day 5 while inhibiting them at day 14 in ovo.

14 t implementation of this program until ED 14 in ovo.

15 nd chick in generating mouse-chick chimaeras in ovo.

16 nt to 12:12 hr light-dark cycles in vitro or in ovo.

17 metastatic melanoma cells were transplanted in ovo adjacent to host chick premigratory neural crest

18 Conversely, chronic in ovo administration of d-tubocurarine, which causes an

19 e presence of axons for survival because the in ovo administration of NMDA, which excitotoxically eli

20 vulina (3-1E) was used to vaccinate chickens in ovo against coccidiosis both alone and in combination

21 hybridization until embryonic day (ED) 14-16 in ovo, analysis of VP expression by RT-PCR showed that

22 eas of the posterior tectum of chick embryos in ovo and analyzed the resulting changes of retinal pro

23 jected at specific sites in the dermomyotome in ovo and the fates of dye-labeled cells monitored by c

24 mulating muscarinic receptors with carbachol in ovo and was inhibited by blocking muscarinic choliner

25 tically removed at embryonic day (E)5 and E7 in ovo, and GCs were counted over the following 7 days o

26 ptors, its expression in KS lesions, and its in ovo angiogenic properties.

27 he frequency of bursting activity, caused by in ovo application of the GABA(A) receptor blocker, picr

28 Gypsy insertion sites in ovo are clustered within a region in the promoter of

29 EF5, administered by intravascular infusion in ovo, as an indicator of relative tissue oxygen concen

30 enes during chick embryo retinal development in ovo, as well as in retinal cell cultures treated with

31 t sources of MN trophic support in vitro and in ovo, but only ACM can rescue MNs after unilateral lim

32 xperiments were carried out in chick embryos in ovo, by intraocular overexpression of noggin, a prote

33 Depletion of versican in ovo, by RNA interference, results in retinal arbors w

34 Knockdown of JNK1 in ovo caused defects in spinal cord commissural axon pr

35 SiRNA-mediated knockdown of DSCAM in ovo causes defects in commissural axon projection and

36 ain-like cell arrangements; however, average in ovo cell speeds are as much as 70% faster.

37 When recombinant Del1 was evaluated in an in ovo chick chorioallantoic membrane assay, it was foun

38 e cell formation and cell delamination after in ovo chicken endoderm electroporation.

39 In ovo, cyclopamine treatment before the secondary heart

40 the brainstem auditory nuclei was studied by in ovo drug administration to chick embryos.

41 cking or slowing this bursting activity, via in ovo drug applications at precise developmental period

42 fied antisense oligonucleotides (antagomiRs) in ovo during chick development.

43 were used to overexpress in the chick retina in ovo either follistatin (FS), an activin-binding prote

44 The introduction of in ovo electroporation a decade ago has helped the chick

45 ssed Gdf11 in chick embryonic spinal cord by in ovo electroporation and found that ectopic expression

46 -function experiments in chick embryos using in ovo electroporation and found that Shh signaling is r

47 g a combination of in vitro cell culture and in ovo electroporation assays, we demonstrate that GAS1

48 l overexpression in the developing retina by in ovo electroporation increases the number of RGCs, whe

49 We demonstrated by in ovo electroporation of a dominant negative retinoid r

50 Finally, in ovo electroporation of axonally targeted GAP-43 mRNA

51 Using in ovo electroporation of chick embryos, we show that ec

52 n altered photoreceptor phenotype, while the in ovo electroporation of chicken embryos resulted in fa

53 In ovo electroporation of Pax2 into the chick ventral op

54 Using in ovo electroporation of PSM cells, we demonstrate that

55 muscle precursor migration, we used targeted in ovo electroporation to express ephrin-A5 ectopically

56 anded RNA (dsRNA) interference combined with in ovo electroporation to knock down RPTP expression lev

57 clin-dependent inducible system Tet-Off with in ovo electroporation to monitor mRNA stability in the

58 Here, we use unilateral in ovo electroporation together with Atoh1 and Neurog1 e

59 To answer this question we combined focal in ovo electroporation with transposon mediated gene tra

60 gain-of-function studies in the chick using in ovo electroporation, and loss-of-function studies in

61 Using in ovo electroporation, we show that misexpressing wild-

62 g EphA4 and disrupting EphA4 signaling using in ovo electroporation.

63 es or in the presumptive neural retina using in ovo-electroporation.

64 ription factor, Chx10, was carried out using in ovo electroporations in chick and transgenic mice.

65 In 1976 an incomplete egg clutch including in ovo embryos of this dinosaur, the oldest known exampl

66 ation (hpf); CYP1 activity was determined by in ovo ethoxyresorufin-o-deethylase (EROD) at 96 hpf, an

67 cities in Xenopus laevis as a consequence of in ovo exposure through maternal transfer of dietary Se.

68 In ovo exposure to SeMet increased mortality and deformi

69 locked glutamatergic or GABA(A) transmission in ovo for 2 days while maintaining relatively normal ne

70 tested in vitro using cells windowed-labeled in ovo for 5 hr on ED 5.

71 todermal cells was traced for the first time in ovo from prestreak stages XI-XII through HH stage 3,

72 We used an in ovo function blocking assay in chick and a conditiona

73 e double mutations E119V I222V) have similar in ovo growth kinetics and infectivity in guinea pigs.

74 ors suggests that no single mechanism guides in ovo hindbrain neural crest cell migration into the br

75 In ovo i.v. injection of PE resulted in deletion of VLR(

76 In ovo imaging allowed us to visualize neural crest cell

77 ing neural crest cells over time using a new in ovo imaging technique.

78 ration strategy used in utero in rodents and in ovo in poultry, and apply it to posthatch zebra finch

79 hh expression, using experimental embryology in ovo in the chick.

80 in the chick embryo retina was investigated in ovo, in dissociated and explant cultures, and in cDNA

81 Furthermore, blockade of trkC in ovo induced reductions in subpopulations of DRG neuro

82 Disruption of that gradient in vitro or in ovo induces changes in hair cell morphologies consist

83 rations of MARIA to developing chick embryos in ovo induces precocious expression of M(2) mAChRs in t

84 ugh AY-9944 does not interrupt Shh signaling in ovo, it blocks the response to Shh-N in explants cult

85 this, we performed fate mapping experiments in ovo, labelling at indifferent stages the coelomic epi

86 ominant chemical species of Se in diets) via in ovo maternal transfer and (2) to investigate the pers

87 In ovo microinjection of PDGF inhibitor causes a similar

88 Following in ovo misexpression of NKL, neuroepithelial cells exit

89 By blocking HH signaling by in ovo misexpression of Ptc1(Delta)(loop2), we show that

90 erimentally generated hypoganglionic hindgut in ovo model.

91 t to specific differentiated fates, using an in ovo modification of the in vitro "window-labeling" te

92 riod, assessing their effects on activity by in ovo motility measurement and muscle nerve recordings

93 There were aspects of the in ovo neural crest cell migration patterning which were

94 When neuropilin-1 function is knocked down in ovo, neural crest cells fail to fully invade the bran

95 Thyroxine (T4) injected in ovo on E9 induces precocious transparency by E12.

96 In ovo, onset of gene expression was asynchronous, becom

97 ation on OP body compartment composition and in ovo or in utero transfer for any given wildlife speci

98 nt to 12-hour light/12-hour dark (LD) cycles in ovo or in vitro.

99 tive vasculogenic cells in the proepicardium in ovo or tagging dissected proepicardial cells in vitro

100 -function Msx2 expression within rhombomeres in ovo prior to the emigration of cranial neural crest c

101 the M1 layer in mature virions and inhibited in ovo propagation of multiple IAV strains including H1N

102 we show that ectopic expression of ephrin-A5 in ovo reduced arborization of cutaneous axons in skin o

103 fold increase in the efficiency of infection in ovo relative to infection with virus particles bearin

104 al canal of the developing chick spinal cord in ovo resulted in guidance errors for commissural axons

105 in the AV canal and overexpression of Wnt-9a in ovo results in enlarged endocardial cushions and AV i

106 he chicken (Gallus domesticus) and performed in ovo retroviral transduction and plasmid electroporati

107 this amphibian species is less sensitive to in ovo Se exposure than most of the fish species studied

108 that Raman spectroscopy enables contactless in ovo sex determination of the domestic chicken (Gallus

109 Existing experimental methods for in ovo sexing require taking samples and are applicable

110 In ovo studies involving shifts from the male- or female

111 In contrast, marking experiments in ovo suggest that the atrioventricular canal, atria an

112 Here in atrial cells 5 days in ovo, TGFbeta increased carbamylcholine inhibition of

113 In manipulated embryos cultured for 3 days in ovo, the mesonephros as well as the pronephros failed

114 recently in neuronal cell death in vitro and in ovo, the role of specific genes belonging to this fam

115 appear in the pia on embryonic day (E)13-14 in ovo, then along blood vessels extending from the pia

116 observed in F1-generation zebrafish exposed in ovo to 6.8 and 12.7 mug Se/g d.m and raised to adulth

117 rotations, and translocations were performed in ovo to identify the tissues that control inner ear mo

118 current study, 100 ng nicotine applied daily in ovo to yolk during embryonic days (E) 1-7 mimicked ma

119 whole egg (1.2 mug), although depuration via in ovo transfer was substantial.

120 in atrial cells cultured from chicks 14 days in ovo, transforming growth factor beta (TGFbeta) decrea

121 Through a combination of in ovo transplantation, co-culture and electroporation t

122 in E11 K(Ca) density was found after chronic in ovo treatment with the neuronal nicotinic antagonist

123 est cells were labeled with DiI and followed in ovo using a new approach for long-term time-lapse con

124 We provide evidence that targeted infection in ovo using adenovirus containing Msx2 and a reporter m

125 Shh overexpression, achieved in ovo using Shh-encoding retrovirus and in organ cultur

126 hese results provide the first evidence that in ovo vaccination with the recombinant 3-1E Eimeria pro

127 eptor to limited numbers of progenitor cells in ovo, we examined the effects of disrupted trk C signa

128 myocytes cultured from chick embryos 14 days in ovo were used as an in vitro model for ischemic preco

129 also promoted tumor formation and metastasis in ovo, where depleting ILK significantly abrogated the

130 t pathway was identified and tested using an in ovo whole chick embryo culture assay.

131 Chick epicardial cells labeled in ovo with DiI invaded the subepicardial extracellular

132 ival, we chronically treated chicken embryos in ovo with either d-tubocurarine (dTC) or muscimol duri

133 Similarly, treatment of embryos in ovo with exogenous HGF/SF rescues lumbar but not othe

134 oporated channelrhodopsin-2 can be activated in ovo with light flashes to drive waves at precise inte

135 report here that treatment of chick embryos in ovo with muscarinic agonist causes decreases in mRNA

136 ling, chick embryos were chronically treated in ovo with picrotoxin to block GABA(A) receptors, while

137 D, we transduced the embryonic chicken heart in ovo with recombinant adenovirus expressing a death (F

138 A single-dose immunization in ovo with the att H5N1 vaccine virus in 18-day-old chi

139 in the presomitic mesoderm of chick embryos in ovo, Wnt-1 differentially affects the expression of d