ライフサイエンスコーパス: in resting

1 mediates activation of ILC2s and Treg cells in resting adipose tissue, but also after helminth infec

2 cell activation, we compared Pol II profiles in resting and activated B cells.

3 hange factor Net1 binds to CARMA1 and CARMA3 in resting and activated cells.

4 estigated the regulation of SRSF1 expression in resting and activated human T cells.

5 ntify the cytoplasmic mRNA targets of CUGBP1 in resting and activated primary human T cells and found

6 subunit of the CD3 T cell receptor complex, in resting and activated primary human T cells.

7 ryanodine receptors (RyR) were investigated in resting and activated primary human T cells.

8 ed by the nature of interdomain interactions in resting and active states, mediates differences in ag

9 een differently tagged flotillin-1 molecules in resting and chemokine-stimulated cells, indicating th

10 a-arterial norepinephrine (NE) were compared in resting and contracting hindlimbs.

11 richia coli AcrAB-TolC multidrug efflux pump in resting and drug transport states, revealing a quater

12 molecular changes underpinning this process in resting and exercising humans.

13 able to provoke greater levels of autophagy in resting and fasting cardiomyocytes in vivo.

14 We quantified the change in resting and hyperemic flow velocity after PCI in sten

15 olysaccharide (LPS) induced hemophagocytosis in resting and IFN-gamma-pretreated macrophages, whereas

16 ASCs were present at normal frequencies in resting and immunized mice and displayed a pattern of

17 ansgenic (TG) mice showed a marked reduction in resting and in maximal heart rate, whereas cardiac ou

18 to examine the behavior of endogenous Tregs in resting and inflamed skin.

19 GFR dynamics on live CHO-K1 plasma membranes in resting and ligand-bound states.

20 examined Stat3 and Stat5 activation patterns in resting and ligand-stimulated primary samples from pe

21 acellularly, with an overall modest increase in resting and odorant-evoked responses during serotoner

22 We found that Hdac3 is highly expressed in resting and prehypertrophic growth plate chondrocytes

23 e testis and the brain, and highly expressed in resting and proliferating chondrocytes of the growth

24 Mitochondria, which are essential organelles in resting and replicating cells, can vary in number, ma

25 poral regulation of cytokine gene expression in resting and restimulated effector T helper 1 (Th1) ce

26 cis, gene expression in resting B cells, and in resting and stimulated monocytes.

27 osphorylation status of presynaptic proteins in resting and stimulated nerve terminals isolated from

28 d oscillatory activity in the 20-40 Hz range in resting and walking states, and increased interhemisp

29 LFPs were recorded in resting and walking states, before and after unilater

30 o constrain the positions of key side chains in resting- and activated-state VS model structures, pro

31 gic signalling and maintained at high levels in resting anergic T cells.

32 a B bind to a wide range of active promoters in resting B and T cells.

33 rly Genes (IEG), which were highly expressed in resting B cell and shifted from non-poised to poised

34 phorylates histone H3S28 at active promoters in resting B cells as well as inhibiting Ring1B-mediated

35 iant that regulates, in cis, gene expression in resting B cells, and in resting and stimulated monocy

36 of Phb1/2 and association with CD86 was low in resting B cells, whereas the level of expression and

37 The latter was used to control for increases in resting blood flow with alpha-adrenergic blockade.

38 can be ruled out as direct pro-FD activators in resting blood; however, active MASP-3 is a very likel

39 functional and is proteolytically processed in resting bone marrow-derived macrophages (BMDMs), dend

40 CD) mapping to study alcohol-related changes in resting brain activity and their association with beh

41 s the hypothesis that pathological increases in resting brain functional connectivity contribute to t

42 s identified KLF2 bound to the CCR5 promoter in resting but not CD3/28 activated T cells, suggesting

43 of DNase I hypersensitive sites genome wide, in resting but not in activated B cells.

44 ntain a poised state at the Il2 target locus in resting but previously stimulated CD4(+) T cells.

45 unchecked, the temperature-driven increases in resting Ca(2+) concentrations and the amounts of ROS

46 l analysis in astrocytes revealed a decrease in resting calcium signaling.

47 ular coupling at rest and that the reduction in resting CBF reflected reduction in synchronized spont

48 that a major restriction on HIV-1 infection in resting CD4 T cells resides in the capacity of these

49 ills (SIVrcm/mnd-2), increased HIV infection in resting CD4 T cells, but not in macrophages, and, une

50 ly induced an increase in HIV RNA expression in resting CD4(+) cells (mean increase, 4.8-fold).

51 n is prevalent in polyadenylated transcripts in resting CD4(+) T cells and is significantly reduced u

52 oRNAs (miRNAs) repress cyclin T1 translation in resting CD4(+) T cells and that this inhibition is li

53 yeloid cells, also restricts HIV replication in resting CD4(+) T cells by hydrolyzing dNTPs, which ar

54 Furthermore, Naf1 knockdown in resting CD4(+) T cells from HIV-1-infected individual

55 ent classes of latency reversal agents (LRA) in resting CD4(+) T cells from HIV-infected individuals

56 quantified the fraction of HIV-1 proviruses in resting CD4(+) T cells from patients on suppressive a

57 d members were also tested for HIV induction in resting CD4(+) T cells isolated from infected individ

58 vation enhances HIV infection/nuclear import in resting CD4(+) T cells through both T cell activation

59 hieved clinically, only 0.079% of proviruses in resting CD4(+) T cells were reactivated to produce vi

60 The latent HIV-1 reservoir primarily resides in resting CD4(+) T cells which are a heterogeneous popu

61 In resting CD4(+) T cells which are nonpermissive for HI

62 kines suppressed R5 and X4 virus replication in resting CD4(+) T cells, and individually SDF-1beta, C

63 HIV persists in resting CD4(+) T cells, and possibly other cell types

64 to quantify replication-competent latent HIV in resting CD4(+) T cells, both increasing accuracy and

65 n is important: in maintaining HIV-1 latency in resting CD4(+) T cells, potentially affect immune fun

66 This property would be important in resting CD4(+) T cells, where dNTP pools are reduced

67 ts indefinitely in a stable latent reservoir in resting CD4(+) T cells.

68 eases in unspliced cellular HIV-1 RNA levels in resting CD4(+) T cells.

69 infection because latent proviruses persist in resting CD4(+) T cells.

70 ivo and a new mechanism for latent infection in resting CD4(+) T cells.

71 ting a translational repression of cyclin T1 in resting CD4(+) T cells.

72 he HIV field is why HIV-1 fails to replicate in resting CD4(+) T cells.

73 t encode replication-competent virus persist in resting CD4(+) T cells.

74 A latent reservoir for HIV-1 in resting CD4(+) T lymphocytes precludes cure.

75 atent viral reservoirs persist, particularly in resting CD4(+) T lymphocytes.

76 ion require the reactivation of latent HIV-1 in resting CD4+ T cells (rCD4s).

77 triphosphohydrolase that depletes dNTP pools in resting CD4+ T cells and macrophages and effectively

78 HIV persists in a quiescent state in resting CD4+ T cells, where the replicative enzymes t

79 CD28(-) T cells, and inhibiting its activity in resting CD8(+)CD28(+) T cells enhanced glycolytic cap

80 ors in mediating microglial process dynamics in resting cells and alpha2A receptors in activated cell

81 s sufficient to render MRTF-A inactive, both in resting cells and in cells with exogenously activated

82 asma membrane expression of the EGF receptor in resting cells and increased EGF-induced phosphorylati

83 ATM exists in an inactive state in resting cells but can be activated by the Mre11-Rad50

84 nase-3 is a Ser/Thr kinase, tonically active in resting cells but inhibited by phosphorylation of an

85 Instead, we made a novel discovery that in resting cells myoferlin was bound to EHD2 protein and

86 We found that in resting cells p53 is present in a mix of oligomeric s

87 strate that vaccinia-related kinase 1 (VRK1) in resting cells plays an important role in the formatio

88 nt pathway leading to basal Ly49E expression in resting cells that is induced by Pro2-mediated Ly49e

89 pression from integrated proviruses occurred in resting cells that lacked surface CD4, likely resulti

90 In resting cells the B-cell surface antigen CD20 is asso

91 The absence of puncta in resting cells was required to prevent spontaneous sto

92 In resting cells, a flagellar signaling component [22],

93 help maintain low levels of cytosolic cavin1 in resting cells, a prerequisite for cavins acting as si

94 In resting cells, AIM2 physically interacted with and li

95 f endoplasmic reticulum Ca(2+) concentration in resting cells, and for the refilling of Ca(2+) stores

96 In resting cells, Irbit was sequestered by InsP3 recepto

97 In resting cells, IRF8 was mainly bound to composite sit

98 In resting cells, Munc18-2, but not STX3, interacted wit

99 In resting cells, STIM1 diffusion is Brownian, while Ora

100 In resting cells, the protein is present in the cytoplas

101 n of CD45 exon 4 in the final mRNA; however, in resting cells, TRAP150 binds PSF and prevents access

102 eased the formation of LC3(+) autophagosomes in resting cells, while other isoforms promoted autophag

103 e 3 at lysine 27 (H3K27) of the HIV provirus in resting cells.

104 as PRC2-Ezh1 is required for its maintenance in resting cells.

105 ted at the lower dNTP concentrations present in resting cells.

106 ter in actively growing bacterial cells than in resting cells.

107 P-TEFb activity and expression are regulated in resting central memory CD4(+) T cells, one of the mai

108 tate, whereas F57Bpa interacts predominantly in resting channel conformations.

109 These GPVI dimers in resting circulating platelets will enable them to bin

110 ther astroglial Cx43 hemichannels are active in resting conditions and regulate basal synaptic transm

111 In resting conditions, estrogens have strong regulatory

112 In resting conditions, NK3Rs were predominantly located

113 e, RyR-mediated Ca(2+) sparks are suppressed in resting conditions, whereas application of transient

114 to inhibit HIV-1 activation and replication in resting conventional T cells in vitro.

115 scle metaboreflex activation on ventilation, in resting COPD patients and healthy participants.

116 to ET-1 (0.1 nM) with a 40 +/- 6% reduction in resting diameter in 20 minutes.

117 or Erg, the most highly expressed ETS member in resting EC, controls quiescence by repressing proinfl

118 Finally, we found that ECs in resting endothelial monolayers use lamellipodial prot

119 he energy they contain, a possible increment in resting energy expenditure, and an augmentation of fa

120 Changes in resting energy expenditure, cardiac function, and bod

121 to demonstrate that patterns of correlation in resting fMRI are closely aligned with functional arch

122 is used to quantify the functional symmetry in resting fMRI data throughout the cortex.

123 LBNP) evoked decreases in muscle oxygenation in resting forearm (-12 +/- 1%) that were attenuated dur

124 al activity induced a long-lasting reduction in resting free astrocyte Ca(2+) and that this phenomeno

125 y role in LLD even in the absence of changes in resting functional connectivity and gray matter.

126 We suggest that reduction in resting GABAergic inhibition triggers homeostatic pla

127 raphe stimulation elicited a strong increase in resting GCaMP fluorescence with only a slight enhance

128 We found increased ADP content in resting Glu(-) cells, a condition that counteracts th

129 port fear of terror-induced annual increases in resting heart rate (pulse), a notable risk factor of

130 is demonstrated that for 1 beat/min increase in resting heart rate, there was a 4% greater adjusted r

131 idues [S-nitrosothiol (SNO)] on 491 proteins in resting hearts from a mouse model of DMD (dmd(mdx):ut

132 Dynamin 1xa is highly phosphorylated at S857 in resting hippocampal neurons and in a hippocampal cell

133 r classes of drug transporters, SLC and ABC, in resting human blood neutrophils.

134 ophilin A, and 12-kDa FK506-binding protein, in resting human Jurkat T cells.

135 that LFA-1 has a low ligand-binding activity in resting human NK cells, but it can be stimulated by t

136 Importantly, SLC1A5 failed to be upregulated in resting human T cells kept under low tryptophan condi

137 Telomerase activity is not readily detected in resting human T lymphocytes, however upon antigen pre

138 ton has been reported to restrict signalling in resting immune cells.

139 ute to the increased reliance on fatty acids in resting KO animals.

140 The rise in resting levels of Ca(2+) may not alter the processes

141 M but not Ca(2+)-free CaM were preassociated in resting live cells, while capsaicin activation induce

142 re, the anabolic function of PTEN deficiency in resting liver is transformed into catabolic activitie

143 Monocytes in resting lung and LN had similar gene expression profi

144 Mer acted as a tolerogenic receptor in resting macrophages and during immunosuppression.

145 lpha, AS-IL1alpha is expressed at low levels in resting macrophages and is induced following infectio

146 TDB/TDM caused only weak Syk-phosphorylation in resting macrophages, consistent with low basal Mincle

147 We show that, in resting macrophages, some IRF8 is conjugated to small

148 Although TRPML2 expression was negligible in resting macrophages, TRPML2 mRNA and protein levels d

149 While the receptors exist as monomers in resting macrophages, two distinct populations were di

150 omoted phagocytosis of apoptotic neutrophils in resting macrophages, whereas immobilized HC-HA promot

151 A transwall gradient in resting membrane potential (RMP) exists across the ci

152 commodation response mode induced by changes in resting membrane potential (RMP) or added neurotrophi

153 iculum via IP3Rs contributes to the decrease in resting membrane potential, prolongation of the actio

154 ifetime, benign, persistent latent infection in resting memory B cells in vivo, where the virus is qu

155 evels of viral entry and early DNA synthesis in resting memory CD4 T cells.

156 deficiency virus type 1 (HIV-1) DNA persists in resting memory CD4(+) T cells despite antiretroviral

157 V-1 latency to show that P-TEFb availability in resting memory CD4(+) T cells is governed by the diff

158 The latent reservoir for HIV-1 in resting memory CD4(+) T cells is the major barrier to

159 The latent reservoir (LR) of HIV-1 in resting memory CD4(+) T cells serves as a major barri

160 rge the persistent reservoir of latent virus in resting memory CD4(+) T cells, but the degree of rese

161 viremia or depletion of the latent reservoir in resting memory CD4(+) T cells.

162 IV-1 replication, and targeting latent HIV-1 in resting memory CD4(+) T cells.

163 remarkably stable reservoir of latent HIV-1 in resting memory CD4(+) T cells.

164 ists in a stable latent reservoir, primarily in resting memory CD4(+) T cells.

165 s transcriptionally silent latent infections in resting memory T cells and hematopoietic stem and pro

166 nt effects on body composition or any change in resting metabolic rate (stable within 8 kcal/d).

167 overall dietary energy intake but no change in resting metabolism.

168 bserved no significant extrinsic sialylation in resting mice, suggesting that extrinsic sialylation i

169 rrow (BM) produces the majority of serum IgM in resting mice.

170 In resting microglia, butaprost induced cAMP formation a

171 PO, which is expressed in activated, but not in resting, microglia.

172 asma membrane P2X4 receptor lateral mobility in resting microglial processes is largely random, consi

173 hibits growth of L. braziliensis amastigotes in resting monocytes, and that classical monocytes are p

174 NAIP1 is poorly expressed in resting mouse bone marrow-derived macrophages; howeve

175 Interestingly, this occurs in resting MPhis through tempering of steady-state mitoc

176 p53 protein was increased in resting Mule-deficient mouse embryonic fibroblasts (M

177 ciency severe enough to impair fat oxidation in resting muscle causes an increase, not a decrease, in

178 We propose that the increase in resting muscle metabolism contributes to these positi

179 1)P MRS magnetization transfer has been used in resting muscle to assess what is claimed to be mitoch

180 sed state, thereby inhibiting Ca(2+) leakage in resting muscle.

181 In resting muscles, there was no difference between geno

182 The primary efficacy end point was change in resting myocardial perfusion over 6 months.

183 Mean iodine concentration in resting myocardium was 2.19 mg/mL +/- 0.57.

184 In resting myocytes, RyR2s can also open spontaneously g

185 d by that OM induces a continuous activation in resting myosin ATPase.

186 OH)(4)(-) did not induce significant changes in resting [Na(+)(i)] or the amplitude and rate of acidi

187 rmore, transient induction of actin dynamics in resting naive T cells rescues HIV latent infection fo

188 ces accumulation of depolarized mitochondria in resting neonatal rat cardiac myocytes, as well as in

189 ause larger changes in transmembrane voltage in resting neurons with low membrane conductances than i

190 In resting neutrophils, we found that A2A receptors are

191 se antigens are cytoplasmic and inaccessible in resting neutrophils.

192 e cytokine production and cytolytic activity in resting NK cells.

193 the longitudinal cohort, abnormal increases in resting NMRP expression were evident at the earliest

194 (2+) chelator BAPTA caused smaller increases in resting open probability in Bth mutant OHCs than in w

195 In resting or "immature" DCs, ubiquitinated MHC II molec

196 hi) monocytes patrol the extravascular space in resting organs, and Ly6c(lo) nonclassical monocytes p

197 in linear and nonlinear age-related changes in resting oscillatory power and network synchrony were

198 Deletion of Drosha or Dicer in resting phase follicles did not affect follicular str

199 JAM-A associates with integrin alphaIIbbeta3 in resting platelets and dissociates upon platelet activ

200 show that SPL/RGS/SHP1 complexes are present in resting platelets where constitutive phosphorylation

201 TYMP-associated Lyn was inactive in resting platelets, and TYMP trapped and diminished ac

202 ript-1, which is localized to alpha-granules in resting platelets, binds fibrinogen, and acts as a po

203 The results show that, in resting platelets, free RGS18 levels are relatively l

204 In resting platelets, the cytoplasmic tail of PEAR1 was

205 t GPVI dimers account for ~29% of total GPVI in resting platelets, whereas activation by either colla

206 A recruits Csk to the integrin-c-Src complex in resting platelets.

207 itotic catastrophe, but the function of MDM2 in resting podocytes has not been explored.

208 Whether the depolarizing shift in resting potential and enhanced spontaneous firing are

209 Instructive signals mediated by changes in resting potential control proliferation, differentiat

210 re, we report excessive NF-kappaB signalling in resting primary bronchial epithelial cells from ZZ pa

211 In resting primary CD4(+) T cells, where levels of P-TEF

212 howed low levels of tyrosine phosphorylation in resting primary mouse CD4(+) T cells; the levels of t

213 We find that Orai1 is a dimer in resting primary mouse embryonic fibroblasts but displ

214 Despite a half-life of approximately 2 h in resting primary T cells, the covalent inhibition of I

215 We show that CLEC3A is present in resting, proliferating, and hypertrophic growth-plate

216 ughout the entire length of apical dendrites in resting pyramidal neurons.

217 d approximately 9% to 10% of total variation in resting QTc in EA individuals and approximately 12% t

218 agonist association is slower than diffusion in resting receptors but nearly diffusional in active re

219 the presence of pentobarbital or gabazine or in resting receptors.

220 in labeling to measure IN-OT-induced changes in resting regional cerebral blood flow (rCBF) in 32 hea

221 had no obvious effect on F-actin arrangement in resting RPE but prevented recruitment of F-actin to s

222 access to its binding site is extremely slow in resting RyR2 but is accelerated by promoting RyR open

223 ter-hemispheric symmetry, typically observed in resting sensorimotor networks, depends on coordinated

224 IM) 2, but not STIM1, was arranged in puncta in resting shTRPC2 cells but not in control cells.

225 However, the measured Pi-->ATP flux in resting skeletal muscle is far higher than the true r

226 alpha-adrenoceptors elicits vasoconstriction in resting skeletal muscle that is blunted during exerci

227 to blunt alpha1 -adrenergic vasoconstriction in resting skeletal muscle would be independent of KIR ,

228 This conformation is present in resting skeletal muscle, but it is not known how exit

229 t affecting AMP content or the AMP/ATP ratio in resting skeletal muscle.

230 s involved in maintaining tissue homeostasis in resting skin and hint at their contribution to signal

231 TR1 knockout mice had a normal phenotype in resting skin, whereas GPx4 loss in the epidermis caus

232 This study aimed to investigate changes in resting state brain activity in remissive Crohn's Dis

233 rate that the global directionality patterns in resting state brain networks can be predicted solely

234 r (ASD) has been associated with a reduction in resting state functional connectivity, though this as

235 ionships across the entire brain connectome, in resting state functional magnetic resonance imaging d

236 Despite advances in resting state functional magnetic resonance imaging i

237 al methods to explore brain network topology in resting state functional MRI data acquired from 17 pa

238 amework for analysing effective connectivity in resting state functional MRI with strong a priori hyp

239 Here we assess LRTCs in resting state human EEG data during a 40-hour sleep d

240 Our findings identify large-scale changes in resting state network interactions that are a physiol

241 standard deviation of BOLD signal amplitude) in resting state networks (RSNs) associated with cogniti

242 nt and dose-dependent longitudinal increases in resting state rCBF in brain regions intrinsic to memo

243 clusterin gene (CLU) on longitudinal changes in resting state regional cerebral blood flow (rCBF) dur

244 heorized that topological network alignments in resting state scans predict psychologically condition

245 e-producing adenomas conducts omega-currents in resting state, but not during voltage-sensing domain

246 shift that is observed in depressed subjects in resting-state activities between the perigenual anter

247 ly demonstrate that the FC patterns observed in resting-state BOLD-fMRI are dictated by the underlyin

248 have reported functionally localized changes in resting-state brain activity following a short period

249 Although changes in resting-state brain connectivity are a transdiagnosti

250 he results suggest that individual variation in resting-state brain dynamics offer a neural explanati

251 Here, we examine changes in resting-state brain function in humans from the Balti

252 ns are associated with widespread reductions in resting-state CBF.

253 PTSD differences in resting-state connectivity of BLA complex could be bi

254 law scaling in neuronal LRTCs and avalanches in resting-state data and during the performance of audi

255 Mean HEP amplitudes in resting-state electroencephalograms and their correla

256 paper, topological organisation was examined in resting-state fMRI data obtained from 32 ASPD patient

257 In resting-state fMRI, hemodynamic fluctuations have bee

258 of functional connectivity networks detected in resting-state fMRI.

259 we have demonstrated age-related decrements in resting-state functional connectivity across most par

260 ratings was also correlated with an increase in resting-state functional connectivity between the mid

261 We found that post-learning increases in resting-state functional connectivity between the SN/

262 Capitalizing on recent advances in resting-state functional connectivity magnetic resona

263 Fluctuations in resting-state functional connectivity occur but their

264 Changes in resting-state functional connectivity, focusing on th

265 Major advances in resting-state functional magnetic resonance imaging (

266 We observed reduced effective connectivity in resting-state functional magnetic resonance imaging b

267 studies suggest circumscribed abnormalities in resting-state functional magnetic resonance imaging c

268 Cerebral connectivity was quantified in resting-state functional magnetic resonance imaging t

269 Increases in resting-state heart rate and decreases in its variabi

270 tionships between interregional correlations in resting-state measures of activity, neuronal function

271 tensor imaging, and functional connectivity in resting-state networks using functional MRI.

272 litus (T1DM) are associated with alterations in resting-state neural connectivity and that these chan

273 ons are a hallmark of amplitude fluctuations in resting-state neuronal oscillations.

274 ults demonstrate that intrinsic fluctuations in resting-state variability exhibit unique maturation t

275 the activity of astroglial Cx43 hemichannels in resting states regulates basal excitatory synaptic tr

276 Further, there is a marked increase in resting sympathetic nervous system (SNS) activity wit

277 d if the virus is eradicated from reservoirs in resting T cells and possibly other hematopoietic cell

278 ng that PAG is constitutively phosphorylated in resting T cells and rapidly dephosphorylated once the

279 In resting T cells cholesterol keeps TCRs in the resting

280 l lines and primary CTCL cells but is absent in resting T cells from healthy donors and B-cell lympho

281 ssfully produce infectious offspring virions in resting T cells that become activated after infection

282 In resting T cells treated with the intrinsic apoptosis

283 CTPS1 expression was found to be low in resting T cells, but rapidly upregulated following TC

284 acellular store in activated T cells but not in resting T cells, indicating that RyR are functionally

285 In resting T cells, T-bet levels directly correlate to s

286 In resting T cells, the CD3epsilon cytoplasmic tail asso

287 HDAC11 at the Eomes and Tbet gene promoters in resting T cells, where it rapidly disassociated follo

288 Because miR-28-3p is highly expressed in resting T cells, which are resistant to HTLV-1 infect

289 the balance between active and inactive Lck in resting T cells, which guarantees operative T cell ef

290 l stimuli, which would not induce a response in resting T cells.

291 reactivity was detected in activated but not in resting T cells.

292 vivo, Zap70 is bound to phosphorylated ITAMs in resting T cells.

293 The extent of VM-PM contact areas in resting terminals correlated with eccentricity in ves

294 sed at the TNF locus and other related genes in resting Th1 cells and released in a myosin VI-depende

295 ehavior superimposed on drift, whereas cells in resting tissue did not show significant displacements

296 cle is blunted relative to that which occurs in resting tissue; however, the mechanisms underlying th

297 ulatory elements in DNA by Foxp3 was similar in resting Treg cells and those activated in vivo, Foxp3

298 we can achieve almost 50% average reduction in resting tremor amplitude and in so doing form the bas

299 y regions of genes involved in TLR signaling in resting UCB monocytes.

300 es and lymphotoxin (LT) alphabeta dependency in resting versus TCR-activated intraepithelial gammadel