1  prompted us, in an earlier study, to inject in the border zone of acute infarcts Lin(-) c-kit(POS) B

2                   Late potentials were noted in the border zones of both surfaces of the scar.

3 ctor (TGF)-beta/Smad3 signaling is activated in the border zone of healing infarcts and induces fibro

4                                     Peak Ecc in the border zone of inducible patients (-11.42% +/- 0.

5 s obtained from cloned embryos were injected in the border zone of infarcted mice to induce tissue re

6  cardiac interstitial fibrosis were analyzed in the border zone of infarcted myocardium.

7                    Injection of DNA for VEGF in the border zone of MI in rat hearts induced angiogene

8 scular endothelial growth factor (phVEGF165) in the border zone of myocardial infarct tissue in rat h

9  Optical mapping showed increased activation in the border zone of PPy-chitosan-treated rats.

10 autophagy (mitophagy) were rapidly increased in the border zone of the infarct in wild type mice.

11 ogenesis and decreased myofibroblast density in the border zone of the infarct support the beneficial

12                 Cells were injected directly in the border zone of the infarcted heart or in correspo

13  This was accompanied by increased apoptosis in the border zone of the MI.

14 s, myofibroblasts, and c-Kit + cells present in the border zone of the remodeling infarct.