1 tly facilitate studies of microglia function
in the developing,
adult, and injured CNS.
2 DNA methylation is highly dynamic
in the developing and adult brain, and is actively regul
3 MiR-211 is one of the most abundant miRNAs
in the developing and adult eye.
4 landscape of distant-acting enhancers active
in the developing and adult human heart, an organ whose
5 of the mesenchymal and epithelial cell types
in the developing and mature mouse ureter.
6 terized homolog, ShcD, is robustly expressed
in the developing and mature nervous system, but its con
7 tivity, probably through distinct mechanisms
in the developing and mature nervous system.
8 ects of neuronal connectivity and plasticity
in the developing and mature nervous system.
9 haemorrhagic hydrocephalus, deregulates Yap
in the developing aqueduct.
10 colorectal cancer is evident in recent years
in the developing Asian nations.
11 In the developing auditory system, inner hair cells (IHC
12 azole group being twisted out of conjugation
in the developing benzylic radical.
13 However, the fate of other cell types
in the developing brain and their contributions to ZIKV-
14 t reduction in synaptic strength also occurs
in the developing brain and there provides an essential
15 Radial glia
in the developing brain extend motile filopodia from the
16 gs in a newborn mouse model of the infection
in the developing brain have indicated that elevated lev
17 The analysis of EGs
in the developing brain identified clusters of coexpress
18 the existence of neural tuning to numerosity
in the developing brain in the youngest sample of childr
19 scriptional activity in cultured neurons and
in the developing brain in vivo We also demonstrate that
20 our results identify a population of neurons
in the developing brain that are acutely dependent on Bc
21 developed a mouse in which WIP1 is expressed
in the developing brain under control of the Neurod2 pro
22 ith MEX1-44, MR-766 grows faster in NPCs and
in the developing brain, and causes more pronounced cell
23 unctions in synapse formation and refinement
in the developing brain, and there is growing evidence t
24 shown that ZIKV can infect neural stem cells
in the developing brain, but infection in the adult brai
25 production of reactive oxygen species (ROS),
in the developing brain, consistent with excitotoxicity;
26 In the developing brain, growth and differentiation are
27 In the developing brain, however, GABAA receptors mediat
28 of either component alone is well tolerated
in the developing brain, in contrast to epithelial tissu
29 In the developing brain, neurons are produced from neura
30 ds to abnormal vascular density and diameter
in the developing brain, resulting in a leaky blood-brai
31 D1B is required for neuronal differentiation
in the developing brain, such as in dendritic arborizati
32 ynaptic connectivity of the cortical neurons
in the developing brain, we used anatomical, ultrastruct
33 s precocious oligodendrocyte differentiation
in the developing brain, whereas genetic inactivation of
34 biases may not directly map onto mechanisms
in the developing brain.
35 regulator of dendrite growth and elaboration
in the developing brain.
36 nication are required for proper myelination
in the developing brain.
37 ho GTPase regulator that is highly expressed
in the developing brain.
38 afferent activation of sensorimotor circuits
in the developing brain.
39 t regulate region-specific enhancer activity
in the developing brain.
40 ith impact on neuronal function and survival
in the developing brain.
41 ses including ZIKV across diverse cell types
in the developing brain.
42 nct from those of its close relative, EphA7,
in the developing brain.
43 the feedback control of long-range coupling
in the developing brain.
44 nal magnetic resonance imaging data acquired
in the developing brain.
45 for NB proliferation and mitotic progression
in the developing brain.
46 tion/differentiation and cortical patterning
in the developing brain.
47 inflammation is a major component of disease
in the developing brain.
48 echanisms regulating cell-fate specification
in the developing brainstem are poorly understood.
49 gulator of cell-fate specification decisions
in the developing brainstem, and as a previously unrecog
50 eficient mice have increased apoptotic cells
in the developing cerebellum and have impaired phagocyto
51 novel role for YB-1 in driving proliferation
in the developing cerebellum and MBCs and they identify
52 of the apical polarity complex protein Pals1
in the developing cerebellum results in a remarkably und
53 produce PCs but rarely Pax2(+) interneurons
in the developing cerebellum, which opposes the "tempora
54 g (Shh) and insulin-like growth factor (IGF)
in the developing cerebellum.
55 Here, we report that relief of hypoxia
in the developing cerebral cortex by ingrowth of blood v
56 s required for efficient NSC differentiation
in the developing cerebral cortex by providing oxygen an
57 lood vessels are part of the stem cell niche
in the developing cerebral cortex, but their in vivo rol
58 In the developing cerebral cortex, radial glia progenito
59 In the developing cerebral cortex, sequential transcript
60 In the developing cerebral cortex, spindle orientation d
61 ormations due to impaired neuronal migration
in the developing cerebral cortex.
62 ey regulators of mitotic spindle orientation
in the developing cerebral cortex.
63 d in vitro are capable of targeted migration
in the developing chick embryo and extensive colonizatio
64 Studies on gene expression
in the developing claustrum of the mouse have clarified
65 eby analyzed the expression profile of TSG-6
in the developing CNS and following injury.
66 he proliferation and differentiation of OPCs
in the developing CNS.
67 terol is rate-limiting for myelin biogenesis
in the developing CNS; however, whether cholesterol insu
68 postnatal period when Huwe1 was knocked out
in the developing cochlea.
69 maturation of the ascending auditory system
in the developing cochlea.
70 ials (APs) arising from the inner hair cells
in the developing cochlea.
71 dentifying tip- and stalk-enriched gene sets
in the developing collecting duct system.
72 ule-A (JAM-A) as a key target for miR-34/449
in the developing cortex that might be responsible for t
73 sula, one of the most densely connected hubs
in the developing cortex, is a major source of the trans
74 nd maintenance of high glycolytic metabolism
in the developing cortex.
75 igin and normal progression of this activity
in the developing cortex.
76 ning of large patient populations especially
in the developing countries that lack sufficient facilit
77 adder schistosomiasis is a prevalent disease
in the developing countries, but in non-endemic areas di
78 ubstantially contribute to the food security
in the developing countries.
79 t link between GCN5, TACC1, and RA signaling
in the developing diencephalon.
80 In the developing dorsal spinal cord, multiple BMPs are
81 In the developing Drosophila eye, Eyes absent (Eya) and
82 Through an RNAi screen
in the developing Drosophila eye, we found that partial
83 entify SOCE mediated gene expression changes
in the developing Drosophila pupal nervous system.
84 edback alters patterns of cell proliferation
in the developing Drosophila wing.
85 ed triggers the initiation of glia migration
in the developing Drosophila wing.
86 confirmed the endogenous expression of IRF6
in the developing ductal, serous, and mucous acinar cell
87 olymers and nanomaterials, were acknowledged
in the developing efficient 3rd generation enzyme bioele
88 The walls of the cerebral ventricles
in the developing embryo harbor the primary neural stem
89 animals, primordial germ-cell specification
in the developing embryo is driven by maternal messenger
90 However its downstream network
in the developing embryo is not fully characterized.
91 In the developing embryo, hematopoietic stem cells (HSCs
92 In the developing embryo, Hox genes are activated sequen
93 In the developing embryo, melanoblasts originating from
94 In the developing embryo, primordial germ cells (PGCs) r
95 -assembly of cells that mimics organogenesis
in the developing embryo.
96 primed hemogenic endothelial cell population
in the developing embryo.
97 temporal differentiation of fields of cells
in the developing embryo.
98 for the development of the three germ layers
in the developing embryo.
99 ritical for ER homeostasis and NTD formation
in the developing embryo.
100 isrupt neuronal maturation and OR expression
in the developing embryonic OE.
101 with sensitivity in the pH range that occurs
in the developing enamel matrix during amelogenesis.
102 of obesogenic endocrine disrupting chemicals
in the developing endocrine system.
103 YY1 loss
in the developing endoderm had no apparent consequences
104 Hor2 expression activated by GAMYB and BPBF
in the developing endosperm and the Amy6.4 activation in
105 ance and differentiation of progenitor cells
in the developing enteric nervous system are controlled
106 dermal progenitor identity and proliferation
in the developing epidermis via Polycomb-dependent and -
107 an embryonic tissue, and observed expression
in the developing eye, neural tube, brain and kidney.
108 f membrane voltage potential regionalization
in the developing face and disrupts expression of import
109 ecipitating decreased alpha:beta cell ratios
in the developing fetal pancreas (P = 0.001), sustained
110 y and tolerance, remain poorly characterized
in the developing fetus.
111 nd differentiation of neural precursor cells
in the developing fetus.
112 he Drosophila bone morphogenetic protein Dpp
in the developing fly wing and that this is necessary fo
113 stand the functions of the ATXN1-CIC complex
in the developing forebrain and found that losing this c
114 beta-catenin signaling pathway, is expressed
in the developing forebrain and pituitary gland, but its
115 tends our understanding of Dlx gene function
in the developing forebrain beyond the regulation of tan
116 We show that deletion of Foxp1
in the developing forebrain leads to impairments in neon
117 iously undescribed mechanism of RA signaling
in the developing forebrain that is required to maintain
118 iously reported in human fetal neocortex and
in the developing forebrains of other mouse models, incl
119 tides are required in order to replicate DNA
in the developing germline.
120 that Shh secretion from a signalling centre
in the developing gill arches establishes gill arch ante
121 tyrosine kinase signaling, and is expressed
in the developing GU-tract in mice and humans.
122 s that the Wnt9a ligand may play such a role
in the developing hearing organ of the bird cochlea.
123 BMP10 is highly expressed
in the developing heart and plays essential roles in car
124 enriched and differential zones of mitophagy
in the developing heart and within specific cells of the
125 ascade, inhibits cardiomyocyte proliferation
in the developing heart to control heart size and preven
126 In the developing heart, heterotypic TF interactions, su
127 s non-CpG methylation of REST-targeted genes
in the developing heart.
128 lishing the correct number of cardiomyocytes
in the developing heart.
129 mpatible gene programs are repressed by TBX5
in the developing heart.
130 ith accurately quantifying mechanical forces
in the developing heart.
131 differentially expressed microRNAs (miRNAs)
in the developing hippocampus following SE, including th
132 t and timing of neurogenesis and gliogenesis
in the developing hippocampus.
133 egulator of the neuron-glia cell-fate switch
in the developing hippocampus.
134 ction of Dmrt5/Dmrta2 as a neurogenic factor
in the developing hippocampus.
135 f the production of neurons versus astroglia
in the developing hippocampus.Finally, we confirm that L
136 Our characterization of infection
in the developing human brain clarifies the pathogenesis
137 it has been challenging to localize regions
in the developing human brain that contribute to spontan
138 about the expression and function of lncRNAs
in the developing human brain.
139 ated the spatiotemporal expression of CPAMD8
in the developing human eye.
140 n, CFTR, CLCN2 and CLCA1, are also expressed
in the developing human fetal lung at gestational stages
141 The direct target cells of ZIKV
in the developing human fetus are not clear.
142 at are expressed in a region-specific manner
in the developing human intestine.
143 compared to the mother and here we show that
in the developing human lung this hypercalcaemia acts on
144 ulated type I adenylate cyclase is expressed
in the developing human lung.
145 ule of the genes with the highest expression
in the developing human neocortex, but its functions rem
146 ed understanding of the timing of key events
in the developing human retina, and in particular the fa
147 al hierarchies governing cell-type diversity
in the developing human telencephalon, including distinc
148 demonstrated strong expression of Foxa2 mRNA
in the developing hypothalamus, pituitary, pancreas, lun
149 In the developing hypothalamus, the fat-derived hormone
150 lin binding to the beta1 proximal NPxY motif
in the developing kidney collecting system in mice that
151 genesis; however, the role of these proteins
in the developing kidney has not been established.
152 arks a renal stem/progenitor cell population
in the developing kidney that in adult kidney contribute
153 the regulation of beta-catenin target genes
in the developing kidney, but will also advance our unde
154 al questions regarding human gene expression
in the developing kidney, essential signaling crosstalk
155 uncoupling Crkl from its signaling pathways
in the developing kidney, including a fivefold up-regula
156 We previously showed that
in the developing kidney, Wnt9b regulates distinct beta-
157 a homeodomain transcription factor expressed
in the developing lateral craniofacial mesenchyme, retin
158 transmembrane protein, is strongly expressed
in the developing lens and its mutation causes ocular di
159 curate, genome-wide predictions of enhancers
in the developing limb, available through a user-friendl
160 We show that Zak is expressed
in the developing limbs and that a CRISPR/Cas-mediated k
161 amily of transcription factors are expressed
in the developing limbs and their function is required f
162 Loss of Hdac3
in the developing lung epithelium leads to a reduction o
163 et al. (2016) identify IL-33 as a key player
in the developing lung for sensitization to environmenta
164 ed to restrict smooth muscle differentiation
in the developing lung mesothelium.
165 ventions that restore elastic fiber assembly
in the developing lung.
166 determine alternative transcription products
in the developing macronucleus; some even contain free-s
167 Mapping cortical connectivity
in the developing mammalian brain has been an intractabl
168 In the developing mammalian brain, differentiating neuro
169 Although caspase-2 is expressed
in the developing mammalian brain, little is known about
170 l neuronal migration and cortical lamination
in the developing mammalian brain.
171 hogenic mechanisms underlying ZIKV infection
in the developing mammalian brain.
172 binding selectivity modifies gene expression
in the developing mammalian brain.
173 regulates DNA binding and proneural activity
in the developing mammalian neocortex.
174 We observed that Robo1 ablation
in the developing mammary gland compromised actin stress
175 s in regulating adipocyte fate determination
in the developing mammary gland.
176 in source of CXCL12 and Kitl producing cells
in the developing marrow.
177 ct the smooth muscle gene expression program
in the developing mesothelium and allow appropriate cell
178 Loss of Ezh2 specifically
in the developing mesothelium reveals a mesothelial cell
179 d the consequences of aberrant Ras signaling
in the developing mouse brain and uncovered several crit
180 Acute knockdown of Myt1l
in the developing mouse brain mimicked a Notch gain-of-f
181 Novel in vivo imaging reveals that,
in the developing mouse brain, strong and localized GCaM
182 ial for migration of neurons and glial cells
in the developing mouse brain.
183 ity and aberrant corticocortical projections
in the developing mouse brain.
184 n Smad anchor for receptor activation (SARA)
in the developing mouse brain.
185 t ARID1B regulates dendritic differentiation
in the developing mouse brain.
186 ical cell proliferation and radial migration
in the developing mouse brain.
187 n-specific function and sumoylation of FOXP2
in the developing mouse cerebellum.
188 Here, we report
in the developing mouse cochlea that deleted in colorect
189 In the developing mouse embryo, the first hematopoietic
190 Slc26a4 expression
in the developing mouse endolymphatic sac is required fo
191 d Brahma-associated factor (BAF170, SMARCC2)
in the developing mouse forebrain.
192 athway underlying astrocytic differentiation
in the developing mouse neocortex.
193 pithelial cells is required for angiogenesis
in the developing mouse pituitary gland.
194 n-of-function alleles BrafV600E and KrasG12D
in the developing mouse pituitary, results in severe hyp
195 the function of the six Hox genes expressed
in the developing mouth and trunk of the amphipod Parhya
196 BCL11A; hereafter CTIP1) is highly expressed
in the developing murine epidermis.
197 In the developing murine eye, melanin synthesis in the r
198 deficiency, suggesting a local role for MGP
in the developing nasal septum.
199 transcriptional networks regulated by Foxp1
in the developing neocortex and found that such networks
200 hanistic insights into the function of FOXP1
in the developing neocortex and may reveal molecular pat
201 hlight a TBR1-regulated network of ASD genes
in the developing neocortex that are relatively intolera
202 human brains and laminar-expression profiles
in the developing neocortex, highlighting their importan
203 fects neuronal differentiation and migration
in the developing neocortex.
204 milarly with corresponding ligands expressed
in the developing neocortex.
205 ment of FoxO6 for a correct radial migration
in the developing neocortex.
206 phrin/Eph signaling controls neuronal ploidy
in the developing neocortex.
207 he proliferation and differentiation of NPCs
in the developing neocortex.
208 rect migration and axon extension of neurons
in the developing nervous system is essential for the ap
209 (EPO) and its receptor are highly expressed
in the developing nervous system, and exogenous EPO ther
210 lar matrix protein Reelin play crucial roles
in the developing nervous system.
211 oteins that regulate APP-dependent responses
in the developing nervous system.
212 c dissemination of mutant Ras-induced tumors
in the developing nervous system.
213 l and arise from distinct progenitor domains
in the developing neural tube.
214 Although widely expressed
in the developing normal mouse head, Opa3 expression was
215 oop-helix transcription factor, is expressed
in the developing OE.
216 grams that lead to the different ORN classes
in the developing olfactory system are unknown.
217 rier protein(s) and sequestered by vitellins
in the developing oocytes.
218 Radial glia
in the developing optic tectum extend highly dynamic fil
219 ders, yet little is known about its function
in the developing or adult mammalian brain in vivo.
220 initially formed but are lost during meiosis
in the developing ovary, leading to adult female sterili
221 Sema3d, and Sema3e, is ectopically activated
in the developing palatal mesenchyme in Osr2(-/-) embryo
222 ndogenous target of canonical Wnt signaling,
in the developing palatal mesenchyme, particularly in th
223 uding Bmp3, Bmp5, Bmp7, Mef2c, Sox6, and Sp7
in the developing palatal mesenchyme.
224 tic inactivation of Wise, which is expressed
in the developing palatal shelves and encodes another se
225 Vax1 is not expressed
in the developing palate and mutant palatal shelves elev
226 he earliest endocrine cell type to be formed
in the developing pancreas.
227 s the level of STERILE APETALA (SAP) protein
in the developing petals.
228 vely dividing cambial cells, cytokinins peak
in the developing phloem tissue of a Populus trichocarpa
229 ols carbon allocation and biomass production
in the developing plant.
230 r, manipulating MET signaling levels in vivo
in the developing prefrontal projection neurons disrupts
231 lar and whole body), the role of epigenetics
in the developing prenatal and postnatal brain, and in m
232 Inflammation
in the developing preterm lung leads to disrupted airway
233 fr1/2/3 and Fgf7/9/10/22 mRNAs are expressed
in the developing primary somatosensory (S1) barrel cort
234 ynthesis and deposition, which occurred only
in the developing protuberance.
235 oper cell cycle exit of neuronal progenitors
in the developing rat and mouse EGL, which is stimulated
236 e investigated the effects of HIF activation
in the developing renal stroma, which also essentially m
237 aling controlling adipogenic differentiation
in the developing reticular dermis.
238 plasticity shapes cell-type-specific wiring
in the developing retina to stabilize visual information
239 In the developing retina, multipotent neural progenitors
240 tegrity of the inner limiting membrane (ILM)
in the developing retina.
241 vealed that inaa was distributed dynamically
in the developing retina.
242 ays specify retinal ganglion cell (RGC) fate
in the developing retina?
243 e duration for which RSL4 protein is present
in the developing root hair.
244 bsence of both OsPCS1 and OsPCS2 transcripts
in the developing seeds coupled with the significant red
245 Interestingly, LRE expression
in the developing seeds was primarily from the matrigeni
246 Concurrently, cellular differentiation
in the developing shoot is coordinated with the environm
247 Here we show that
in the developing skin, epidermal progenitor cells of mi
248 the periodic pattern of hair or feather buds
in the developing skin.
249 Accordingly, IMP2 knockdown
in the developing spinal cord led to strong defects in c
250 regulation of the transcription factor NFIA
in the developing spinal cord, we identified long-range
251 ng of AqJAG revealed a wide range of defects
in the developing stems, leaves and flowers; strongest p
252 h the developmental state of the chromoplast
in the developing stigma.
253 Lesser starch accumulation was observed
in the developing storage roots at the initiation stage
254 ressed with the dSPN markers, Isl1 and Ebf1,
in the developing striatum.
255 bventricular zone (SVZ), the function of ECM
in the developing SVZ remains unknown.
256 36 tumor suppressor that regulates apoptosis
in the developing sympathetic nervous system.
257 ption factor 1 (Ascl1) plays important roles
in the developing telencephalon, whether Ascl1 regulates
258 Individual radial glial progenitors
in the developing thalamus actively divide and produce a
259 steadily from 4-6 d postfertilization (dpf)
in the developing thymus, with il-2rgammac.a expression
260 d that, whereas expression of Inhba and Bmp4
in the developing tooth mesenchyme is independent of eac
261 d Wnt antagonists, including Dkk2 and Sfrp2,
in the developing tooth mesenchyme.
262 ria with their mtDNA payload are transferred
in the developing tumour, and provide functional evidenc
263 In contrast, mice lacking talins
in the developing ureteric bud developed kidney agenesis
264 itu hybridization confirmed Nrip1 expression
in the developing urogenital system of the mouse.
265 ation and differentiation of nascent neurons
in the developing vertebrate brain.
266 d suggest that alternative splicing of C-Src
in the developing vertebrate nervous system evolved to r
267 ound impact on the responsiveness of neurons
in the developing visual cortex.
268 the persistent impact of MD on synapse loss
in the developing visual cortex.
269 is normally present below saturating levels
in the developing visual system of the Xenopus tadpole.
270 In the developing visual system, visual deprivation earl
271 r 4 but instead target subplate neurons deep
in the developing white matter.
272 We found calcium transients
in the developing wing, and inhibition of Irk channels r
273 EC) cause more than 500,000 deaths each year
in the developing world and are characterized on a molec
274 n infections represent a major health burden
in the developing world and contribute significantly to
275 % and topical antibiotics commonly available
in the developing world for treatment of bacterial kerat
276 Surgical repair of TGA performed
in the developing world is associated with an early surv
277 ination of dairy cattle is a common practice
in the developing world that can improve farmer incomes
278 In the developing world, billions of individuals are chr
279 e data on economic livelihoods remain scarce
in the developing world, hampering efforts to study thes
280 In the developing world, we found a non-significant 2 po
281 Cassava is a major crop
in the developing world, with its production in Africa b
282 or transposition of the great arteries (TGA)
in the developing world.
283 alnutrition and growth faltering in children
in the developing world.
284 asitic fluke that infects millions of people
in the developing world.
285 harness science in support of sustainability
in the developing world.
286 ly since 1990 in the developed world but not
in the developing world.
287 bidity and mortality among women, especially
in the developing world.
288 to global health and are particularly acute
in the developing world.
289 ine suspension, may be of particular utility
in the developing world.
290 as well as prevention of corneal ulceration
in the developing world.
291 ide protein for a billion people, especially
in the developing world.
292 vement efforts may benefit patients with TGA
in the developing world.
293 over 100,000 people a year, mostly children
in the developing world.
294 n and an important cause of death in infants
in the developing world.
295 ormation of hindbrain segments (rhombomeres)
in the developing zebrafish as an example, but the mecha
296 Functional analysis
in the developing zebrafish embryo demonstrated that bot
297 We also track migrating cells
in the developing zebrafish embryo, demonstrating the ut
298 nt vascular morphogenesis, both in vitro and
in the developing zebrafish embryo.
299 In the developing zebrafish larvae, in vivo monitoring o
300 However, here we show
in the developing zebrafish that topographic decoding pe