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1 tly facilitate studies of microglia function in the developing, adult, and injured CNS.
2            DNA methylation is highly dynamic in the developing and adult brain, and is actively regul
3   MiR-211 is one of the most abundant miRNAs in the developing and adult eye.
4 landscape of distant-acting enhancers active in the developing and adult human heart, an organ whose
5 of the mesenchymal and epithelial cell types in the developing and mature mouse ureter.
6 terized homolog, ShcD, is robustly expressed in the developing and mature nervous system, but its con
7 tivity, probably through distinct mechanisms in the developing and mature nervous system.
8 ects of neuronal connectivity and plasticity in the developing and mature nervous system.
9  haemorrhagic hydrocephalus, deregulates Yap in the developing aqueduct.
10 colorectal cancer is evident in recent years in the developing Asian nations.
11                                              In the developing auditory system, inner hair cells (IHC
12 azole group being twisted out of conjugation in the developing benzylic radical.
13        However, the fate of other cell types in the developing brain and their contributions to ZIKV-
14 t reduction in synaptic strength also occurs in the developing brain and there provides an essential
15                                  Radial glia in the developing brain extend motile filopodia from the
16 gs in a newborn mouse model of the infection in the developing brain have indicated that elevated lev
17                          The analysis of EGs in the developing brain identified clusters of coexpress
18 the existence of neural tuning to numerosity in the developing brain in the youngest sample of childr
19 scriptional activity in cultured neurons and in the developing brain in vivo We also demonstrate that
20 our results identify a population of neurons in the developing brain that are acutely dependent on Bc
21 developed a mouse in which WIP1 is expressed in the developing brain under control of the Neurod2 pro
22 ith MEX1-44, MR-766 grows faster in NPCs and in the developing brain, and causes more pronounced cell
23 unctions in synapse formation and refinement in the developing brain, and there is growing evidence t
24 shown that ZIKV can infect neural stem cells in the developing brain, but infection in the adult brai
25 production of reactive oxygen species (ROS), in the developing brain, consistent with excitotoxicity;
26                                              In the developing brain, growth and differentiation are
27                                              In the developing brain, however, GABAA receptors mediat
28  of either component alone is well tolerated in the developing brain, in contrast to epithelial tissu
29                                              In the developing brain, neurons are produced from neura
30 ds to abnormal vascular density and diameter in the developing brain, resulting in a leaky blood-brai
31 D1B is required for neuronal differentiation in the developing brain, such as in dendritic arborizati
32 ynaptic connectivity of the cortical neurons in the developing brain, we used anatomical, ultrastruct
33 s precocious oligodendrocyte differentiation in the developing brain, whereas genetic inactivation of
34  biases may not directly map onto mechanisms in the developing brain.
35 regulator of dendrite growth and elaboration in the developing brain.
36 nication are required for proper myelination in the developing brain.
37 ho GTPase regulator that is highly expressed in the developing brain.
38 afferent activation of sensorimotor circuits in the developing brain.
39 t regulate region-specific enhancer activity in the developing brain.
40 ith impact on neuronal function and survival in the developing brain.
41 ses including ZIKV across diverse cell types in the developing brain.
42 nct from those of its close relative, EphA7, in the developing brain.
43  the feedback control of long-range coupling in the developing brain.
44 nal magnetic resonance imaging data acquired in the developing brain.
45 for NB proliferation and mitotic progression in the developing brain.
46 tion/differentiation and cortical patterning in the developing brain.
47 inflammation is a major component of disease in the developing brain.
48 echanisms regulating cell-fate specification in the developing brainstem are poorly understood.
49 gulator of cell-fate specification decisions in the developing brainstem, and as a previously unrecog
50 eficient mice have increased apoptotic cells in the developing cerebellum and have impaired phagocyto
51 novel role for YB-1 in driving proliferation in the developing cerebellum and MBCs and they identify
52 of the apical polarity complex protein Pals1 in the developing cerebellum results in a remarkably und
53  produce PCs but rarely Pax2(+) interneurons in the developing cerebellum, which opposes the "tempora
54 g (Shh) and insulin-like growth factor (IGF) in the developing cerebellum.
55       Here, we report that relief of hypoxia in the developing cerebral cortex by ingrowth of blood v
56 s required for efficient NSC differentiation in the developing cerebral cortex by providing oxygen an
57 lood vessels are part of the stem cell niche in the developing cerebral cortex, but their in vivo rol
58                                              In the developing cerebral cortex, radial glia progenito
59                                              In the developing cerebral cortex, sequential transcript
60                                              In the developing cerebral cortex, spindle orientation d
61 ormations due to impaired neuronal migration in the developing cerebral cortex.
62 ey regulators of mitotic spindle orientation in the developing cerebral cortex.
63 d in vitro are capable of targeted migration in the developing chick embryo and extensive colonizatio
64                   Studies on gene expression in the developing claustrum of the mouse have clarified
65 eby analyzed the expression profile of TSG-6 in the developing CNS and following injury.
66 he proliferation and differentiation of OPCs in the developing CNS.
67 terol is rate-limiting for myelin biogenesis in the developing CNS; however, whether cholesterol insu
68  postnatal period when Huwe1 was knocked out in the developing cochlea.
69  maturation of the ascending auditory system in the developing cochlea.
70 ials (APs) arising from the inner hair cells in the developing cochlea.
71 dentifying tip- and stalk-enriched gene sets in the developing collecting duct system.
72 ule-A (JAM-A) as a key target for miR-34/449 in the developing cortex that might be responsible for t
73 sula, one of the most densely connected hubs in the developing cortex, is a major source of the trans
74 nd maintenance of high glycolytic metabolism in the developing cortex.
75 igin and normal progression of this activity in the developing cortex.
76 ning of large patient populations especially in the developing countries that lack sufficient facilit
77 adder schistosomiasis is a prevalent disease in the developing countries, but in non-endemic areas di
78 ubstantially contribute to the food security in the developing countries.
79 t link between GCN5, TACC1, and RA signaling in the developing diencephalon.
80                                              In the developing dorsal spinal cord, multiple BMPs are
81                                              In the developing Drosophila eye, Eyes absent (Eya) and
82                       Through an RNAi screen in the developing Drosophila eye, we found that partial
83 entify SOCE mediated gene expression changes in the developing Drosophila pupal nervous system.
84 edback alters patterns of cell proliferation in the developing Drosophila wing.
85 ed triggers the initiation of glia migration in the developing Drosophila wing.
86  confirmed the endogenous expression of IRF6 in the developing ductal, serous, and mucous acinar cell
87 olymers and nanomaterials, were acknowledged in the developing efficient 3rd generation enzyme bioele
88         The walls of the cerebral ventricles in the developing embryo harbor the primary neural stem
89  animals, primordial germ-cell specification in the developing embryo is driven by maternal messenger
90               However its downstream network in the developing embryo is not fully characterized.
91                                              In the developing embryo, hematopoietic stem cells (HSCs
92                                              In the developing embryo, Hox genes are activated sequen
93                                              In the developing embryo, melanoblasts originating from
94                                              In the developing embryo, primordial germ cells (PGCs) r
95 -assembly of cells that mimics organogenesis in the developing embryo.
96 primed hemogenic endothelial cell population in the developing embryo.
97  temporal differentiation of fields of cells in the developing embryo.
98 for the development of the three germ layers in the developing embryo.
99 ritical for ER homeostasis and NTD formation in the developing embryo.
100 isrupt neuronal maturation and OR expression in the developing embryonic OE.
101 with sensitivity in the pH range that occurs in the developing enamel matrix during amelogenesis.
102 of obesogenic endocrine disrupting chemicals in the developing endocrine system.
103                                     YY1 loss in the developing endoderm had no apparent consequences
104  Hor2 expression activated by GAMYB and BPBF in the developing endosperm and the Amy6.4 activation in
105 ance and differentiation of progenitor cells in the developing enteric nervous system are controlled
106 dermal progenitor identity and proliferation in the developing epidermis via Polycomb-dependent and -
107 an embryonic tissue, and observed expression in the developing eye, neural tube, brain and kidney.
108 f membrane voltage potential regionalization in the developing face and disrupts expression of import
109 ecipitating decreased alpha:beta cell ratios in the developing fetal pancreas (P = 0.001), sustained
110 y and tolerance, remain poorly characterized in the developing fetus.
111 nd differentiation of neural precursor cells in the developing fetus.
112 he Drosophila bone morphogenetic protein Dpp in the developing fly wing and that this is necessary fo
113 stand the functions of the ATXN1-CIC complex in the developing forebrain and found that losing this c
114 beta-catenin signaling pathway, is expressed in the developing forebrain and pituitary gland, but its
115 tends our understanding of Dlx gene function in the developing forebrain beyond the regulation of tan
116               We show that deletion of Foxp1 in the developing forebrain leads to impairments in neon
117 iously undescribed mechanism of RA signaling in the developing forebrain that is required to maintain
118 iously reported in human fetal neocortex and in the developing forebrains of other mouse models, incl
119 tides are required in order to replicate DNA in the developing germline.
120  that Shh secretion from a signalling centre in the developing gill arches establishes gill arch ante
121  tyrosine kinase signaling, and is expressed in the developing GU-tract in mice and humans.
122 s that the Wnt9a ligand may play such a role in the developing hearing organ of the bird cochlea.
123                    BMP10 is highly expressed in the developing heart and plays essential roles in car
124 enriched and differential zones of mitophagy in the developing heart and within specific cells of the
125 ascade, inhibits cardiomyocyte proliferation in the developing heart to control heart size and preven
126                                              In the developing heart, heterotypic TF interactions, su
127 s non-CpG methylation of REST-targeted genes in the developing heart.
128 lishing the correct number of cardiomyocytes in the developing heart.
129 mpatible gene programs are repressed by TBX5 in the developing heart.
130 ith accurately quantifying mechanical forces in the developing heart.
131  differentially expressed microRNAs (miRNAs) in the developing hippocampus following SE, including th
132 t and timing of neurogenesis and gliogenesis in the developing hippocampus.
133 egulator of the neuron-glia cell-fate switch in the developing hippocampus.
134 ction of Dmrt5/Dmrta2 as a neurogenic factor in the developing hippocampus.
135 f the production of neurons versus astroglia in the developing hippocampus.Finally, we confirm that L
136            Our characterization of infection in the developing human brain clarifies the pathogenesis
137  it has been challenging to localize regions in the developing human brain that contribute to spontan
138 about the expression and function of lncRNAs in the developing human brain.
139 ated the spatiotemporal expression of CPAMD8 in the developing human eye.
140 n, CFTR, CLCN2 and CLCA1, are also expressed in the developing human fetal lung at gestational stages
141              The direct target cells of ZIKV in the developing human fetus are not clear.
142 at are expressed in a region-specific manner in the developing human intestine.
143 compared to the mother and here we show that in the developing human lung this hypercalcaemia acts on
144 ulated type I adenylate cyclase is expressed in the developing human lung.
145 ule of the genes with the highest expression in the developing human neocortex, but its functions rem
146 ed understanding of the timing of key events in the developing human retina, and in particular the fa
147 al hierarchies governing cell-type diversity in the developing human telencephalon, including distinc
148 demonstrated strong expression of Foxa2 mRNA in the developing hypothalamus, pituitary, pancreas, lun
149                                              In the developing hypothalamus, the fat-derived hormone
150 lin binding to the beta1 proximal NPxY motif in the developing kidney collecting system in mice that
151 genesis; however, the role of these proteins in the developing kidney has not been established.
152 arks a renal stem/progenitor cell population in the developing kidney that in adult kidney contribute
153  the regulation of beta-catenin target genes in the developing kidney, but will also advance our unde
154 al questions regarding human gene expression in the developing kidney, essential signaling crosstalk
155  uncoupling Crkl from its signaling pathways in the developing kidney, including a fivefold up-regula
156                    We previously showed that in the developing kidney, Wnt9b regulates distinct beta-
157 a homeodomain transcription factor expressed in the developing lateral craniofacial mesenchyme, retin
158 transmembrane protein, is strongly expressed in the developing lens and its mutation causes ocular di
159 curate, genome-wide predictions of enhancers in the developing limb, available through a user-friendl
160                We show that Zak is expressed in the developing limbs and that a CRISPR/Cas-mediated k
161 amily of transcription factors are expressed in the developing limbs and their function is required f
162                                Loss of Hdac3 in the developing lung epithelium leads to a reduction o
163 et al. (2016) identify IL-33 as a key player in the developing lung for sensitization to environmenta
164 ed to restrict smooth muscle differentiation in the developing lung mesothelium.
165 ventions that restore elastic fiber assembly in the developing lung.
166 determine alternative transcription products in the developing macronucleus; some even contain free-s
167                Mapping cortical connectivity in the developing mammalian brain has been an intractabl
168                                              In the developing mammalian brain, differentiating neuro
169              Although caspase-2 is expressed in the developing mammalian brain, little is known about
170 l neuronal migration and cortical lamination in the developing mammalian brain.
171 hogenic mechanisms underlying ZIKV infection in the developing mammalian brain.
172 binding selectivity modifies gene expression in the developing mammalian brain.
173 regulates DNA binding and proneural activity in the developing mammalian neocortex.
174              We observed that Robo1 ablation in the developing mammary gland compromised actin stress
175 s in regulating adipocyte fate determination in the developing mammary gland.
176 in source of CXCL12 and Kitl producing cells in the developing marrow.
177 ct the smooth muscle gene expression program in the developing mesothelium and allow appropriate cell
178                    Loss of Ezh2 specifically in the developing mesothelium reveals a mesothelial cell
179 d the consequences of aberrant Ras signaling in the developing mouse brain and uncovered several crit
180                     Acute knockdown of Myt1l in the developing mouse brain mimicked a Notch gain-of-f
181          Novel in vivo imaging reveals that, in the developing mouse brain, strong and localized GCaM
182 ial for migration of neurons and glial cells in the developing mouse brain.
183 ity and aberrant corticocortical projections in the developing mouse brain.
184 n Smad anchor for receptor activation (SARA) in the developing mouse brain.
185 t ARID1B regulates dendritic differentiation in the developing mouse brain.
186 ical cell proliferation and radial migration in the developing mouse brain.
187 n-specific function and sumoylation of FOXP2 in the developing mouse cerebellum.
188                              Here, we report in the developing mouse cochlea that deleted in colorect
189                                              In the developing mouse embryo, the first hematopoietic
190                           Slc26a4 expression in the developing mouse endolymphatic sac is required fo
191 d Brahma-associated factor (BAF170, SMARCC2) in the developing mouse forebrain.
192 athway underlying astrocytic differentiation in the developing mouse neocortex.
193 pithelial cells is required for angiogenesis in the developing mouse pituitary gland.
194 n-of-function alleles BrafV600E and KrasG12D in the developing mouse pituitary, results in severe hyp
195  the function of the six Hox genes expressed in the developing mouth and trunk of the amphipod Parhya
196 BCL11A; hereafter CTIP1) is highly expressed in the developing murine epidermis.
197                                              In the developing murine eye, melanin synthesis in the r
198  deficiency, suggesting a local role for MGP in the developing nasal septum.
199  transcriptional networks regulated by Foxp1 in the developing neocortex and found that such networks
200 hanistic insights into the function of FOXP1 in the developing neocortex and may reveal molecular pat
201 hlight a TBR1-regulated network of ASD genes in the developing neocortex that are relatively intolera
202 human brains and laminar-expression profiles in the developing neocortex, highlighting their importan
203 fects neuronal differentiation and migration in the developing neocortex.
204 milarly with corresponding ligands expressed in the developing neocortex.
205 ment of FoxO6 for a correct radial migration in the developing neocortex.
206 phrin/Eph signaling controls neuronal ploidy in the developing neocortex.
207 he proliferation and differentiation of NPCs in the developing neocortex.
208 rect migration and axon extension of neurons in the developing nervous system is essential for the ap
209  (EPO) and its receptor are highly expressed in the developing nervous system, and exogenous EPO ther
210 lar matrix protein Reelin play crucial roles in the developing nervous system.
211 oteins that regulate APP-dependent responses in the developing nervous system.
212 c dissemination of mutant Ras-induced tumors in the developing nervous system.
213 l and arise from distinct progenitor domains in the developing neural tube.
214                    Although widely expressed in the developing normal mouse head, Opa3 expression was
215 oop-helix transcription factor, is expressed in the developing OE.
216 grams that lead to the different ORN classes in the developing olfactory system are unknown.
217 rier protein(s) and sequestered by vitellins in the developing oocytes.
218                                  Radial glia in the developing optic tectum extend highly dynamic fil
219 ders, yet little is known about its function in the developing or adult mammalian brain in vivo.
220 initially formed but are lost during meiosis in the developing ovary, leading to adult female sterili
221 Sema3d, and Sema3e, is ectopically activated in the developing palatal mesenchyme in Osr2(-/-) embryo
222 ndogenous target of canonical Wnt signaling, in the developing palatal mesenchyme, particularly in th
223 uding Bmp3, Bmp5, Bmp7, Mef2c, Sox6, and Sp7 in the developing palatal mesenchyme.
224 tic inactivation of Wise, which is expressed in the developing palatal shelves and encodes another se
225                        Vax1 is not expressed in the developing palate and mutant palatal shelves elev
226 he earliest endocrine cell type to be formed in the developing pancreas.
227 s the level of STERILE APETALA (SAP) protein in the developing petals.
228 vely dividing cambial cells, cytokinins peak in the developing phloem tissue of a Populus trichocarpa
229 ols carbon allocation and biomass production in the developing plant.
230 r, manipulating MET signaling levels in vivo in the developing prefrontal projection neurons disrupts
231 lar and whole body), the role of epigenetics in the developing prenatal and postnatal brain, and in m
232                                 Inflammation in the developing preterm lung leads to disrupted airway
233 fr1/2/3 and Fgf7/9/10/22 mRNAs are expressed in the developing primary somatosensory (S1) barrel cort
234 ynthesis and deposition, which occurred only in the developing protuberance.
235 oper cell cycle exit of neuronal progenitors in the developing rat and mouse EGL, which is stimulated
236 e investigated the effects of HIF activation in the developing renal stroma, which also essentially m
237 aling controlling adipogenic differentiation in the developing reticular dermis.
238  plasticity shapes cell-type-specific wiring in the developing retina to stabilize visual information
239                                              In the developing retina, multipotent neural progenitors
240 tegrity of the inner limiting membrane (ILM) in the developing retina.
241 vealed that inaa was distributed dynamically in the developing retina.
242 ays specify retinal ganglion cell (RGC) fate in the developing retina?
243 e duration for which RSL4 protein is present in the developing root hair.
244 bsence of both OsPCS1 and OsPCS2 transcripts in the developing seeds coupled with the significant red
245                Interestingly, LRE expression in the developing seeds was primarily from the matrigeni
246       Concurrently, cellular differentiation in the developing shoot is coordinated with the environm
247                            Here we show that in the developing skin, epidermal progenitor cells of mi
248 the periodic pattern of hair or feather buds in the developing skin.
249                  Accordingly, IMP2 knockdown in the developing spinal cord led to strong defects in c
250  regulation of the transcription factor NFIA in the developing spinal cord, we identified long-range
251 ng of AqJAG revealed a wide range of defects in the developing stems, leaves and flowers; strongest p
252 h the developmental state of the chromoplast in the developing stigma.
253      Lesser starch accumulation was observed in the developing storage roots at the initiation stage
254 ressed with the dSPN markers, Isl1 and Ebf1, in the developing striatum.
255 bventricular zone (SVZ), the function of ECM in the developing SVZ remains unknown.
256 36 tumor suppressor that regulates apoptosis in the developing sympathetic nervous system.
257 ption factor 1 (Ascl1) plays important roles in the developing telencephalon, whether Ascl1 regulates
258          Individual radial glial progenitors in the developing thalamus actively divide and produce a
259  steadily from 4-6 d postfertilization (dpf) in the developing thymus, with il-2rgammac.a expression
260 d that, whereas expression of Inhba and Bmp4 in the developing tooth mesenchyme is independent of eac
261 d Wnt antagonists, including Dkk2 and Sfrp2, in the developing tooth mesenchyme.
262 ria with their mtDNA payload are transferred in the developing tumour, and provide functional evidenc
263             In contrast, mice lacking talins in the developing ureteric bud developed kidney agenesis
264 itu hybridization confirmed Nrip1 expression in the developing urogenital system of the mouse.
265 ation and differentiation of nascent neurons in the developing vertebrate brain.
266 d suggest that alternative splicing of C-Src in the developing vertebrate nervous system evolved to r
267 ound impact on the responsiveness of neurons in the developing visual cortex.
268  the persistent impact of MD on synapse loss in the developing visual cortex.
269  is normally present below saturating levels in the developing visual system of the Xenopus tadpole.
270                                              In the developing visual system, visual deprivation earl
271 r 4 but instead target subplate neurons deep in the developing white matter.
272                  We found calcium transients in the developing wing, and inhibition of Irk channels r
273 EC) cause more than 500,000 deaths each year in the developing world and are characterized on a molec
274 n infections represent a major health burden in the developing world and contribute significantly to
275 % and topical antibiotics commonly available in the developing world for treatment of bacterial kerat
276             Surgical repair of TGA performed in the developing world is associated with an early surv
277 ination of dairy cattle is a common practice in the developing world that can improve farmer incomes
278                                              In the developing world, billions of individuals are chr
279 e data on economic livelihoods remain scarce in the developing world, hampering efforts to study thes
280                                              In the developing world, we found a non-significant 2 po
281                      Cassava is a major crop in the developing world, with its production in Africa b
282 or transposition of the great arteries (TGA) in the developing world.
283 alnutrition and growth faltering in children in the developing world.
284 asitic fluke that infects millions of people in the developing world.
285 harness science in support of sustainability in the developing world.
286 ly since 1990 in the developed world but not in the developing world.
287 bidity and mortality among women, especially in the developing world.
288  to global health and are particularly acute in the developing world.
289 ine suspension, may be of particular utility in the developing world.
290  as well as prevention of corneal ulceration in the developing world.
291 ide protein for a billion people, especially in the developing world.
292 vement efforts may benefit patients with TGA in the developing world.
293  over 100,000 people a year, mostly children in the developing world.
294 n and an important cause of death in infants in the developing world.
295 ormation of hindbrain segments (rhombomeres) in the developing zebrafish as an example, but the mecha
296                          Functional analysis in the developing zebrafish embryo demonstrated that bot
297                We also track migrating cells in the developing zebrafish embryo, demonstrating the ut
298 nt vascular morphogenesis, both in vitro and in the developing zebrafish embryo.
299                                              In the developing zebrafish larvae, in vivo monitoring o
300                        However, here we show in the developing zebrafish that topographic decoding pe

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