ライフサイエンスコーパス: in vivo,

1 In vivo [(11)C]AZ12204657 bound specifically to GPR44 in

2 structures with good correspondence between in vivo (18) F-flortaucipir and postmortem tau neuropath

3 In vivo, 5-HT4 receptor signaling is also upregulated si

4 trated by animal studies in vitro [3, 4] and in vivo [5, 6], suggesting a causal role of theta activi

5 In vivo (89)Zr-bevacizumab measurements showed heterogen

6 In vivo (89)Zr-bevacizumab PET serves to identify hetero

7 In vivo, a 2.5 mg/kg dose of (+)-pentazocine abolished (

8 d-ligand to promote sensitization to Pru p 3 in vivo, a mouse model of anaphylaxis to peach has been

9 In vivo, a single administration of agrin promotes cardi

10 In vivo, AAV9 (adeno-associated virus serotype 9)-mediat

11 In vivo, activated immune cells secrete cytokines for se

12 disease-initiating and mature leukemic cells in vivo, allowing creation of genetically accurate precl

13 down-regulated FZD4 expression in vitro and in vivo, along with reduced WNT/beta-catenin activity.

14 that, during exposure to an HIV-1 neurotoxin in vivo, alteration of GluN2B-containing NMDAR signaling

15 tative tracking of therapeutic nanoparticles in vivo, an essential step for optimizing light-induced,

16 ox proteins that mediate electron metabolism in vivo, and are also potential components for biologica

17 Thus, our genetic, in vivo, and biochemical data indicate a role for Coy1 i

18 FGF signaling promotes epicardium formation in vivo, and biochemical studies demonstrated that CDC42

19 Plants were examined poststress induction in vivo, and changes in the concentration levels of the

20 ity in vitro and restricts axon regeneration in vivo, and demonstrate a novel, non-traditional role f

21 otoxicity against peptide-expressing targets in vivo, and elicits a better antitumor response in a mo

22 , Gcn5 directly acetylates Rph1 in vitro and in vivo, and Gcn5-containing complex, SAGA, is required

23 ubsequent mechanical activation in vitro and in vivo, and identify the microRNA miR-21 as a long-term

24 on staphopains present in S. aureus biofilms in vivo, and illustrate the complex interplay between bi

25 he expansion and persistence of CTL019 cells in vivo, and immune recovery.

26 analyse the usage of cell nomenclature, both in Vivo, and in Vitro in biomedical literature by using

27 and integrate evidence from epidemiological, in vivo, and in vitro studies.

28 ial for interaction with PI(4)P in vitro and in vivo, and interaction with PI(4)P is required for eff

29 A virus (IAV) were investigated in vitro and in vivo, and mechanisms of action were suggested.

30 few studies have used DNPs to deliver siRNAs in vivo, and none has demonstrated therapeutic efficacy.

31 VACV-E3LDelta83N is attenuated in vivo, and pathogenicity was restored in either RIPK3-

32 ted by parathyroid hormone both in vitro and in vivo, and protects osteocytes from oxidative stress.

33 HSPCs exist in a quiescent state in vivo, and quiescence is correlated with latent infect

34 Differences in the RANKL/OPG Axis in vivo, and RANKL-induced maturation of osteoclast-prec

35 t, loss of Pten or Nf1 increases growth rate in vivo, and reduces survival following cisplatin chemot

36 we report the margination of stiffened RBCs in vivo, and reveal the crucial role of the vessel geome

37 synaptic plasticity in PL-mPFC is reversible in vivo, and suggest a novel strategy that would allow n

38 f controlling treatment-refractory Ph(+) ALL in vivo, and support the development of adoptive immunot

39 ered bacteria for querying pathogen behavior in vivo, and the importance of validating in vitro studi

40 se activities on different immune cell types in vivo, and the need to conduct mechanistic research th

41 matory vascular remodeling of venous thrombi in vivo, and the potential therapeutic application of a

42 dicated that TCTP forms complexes with Rad51 in vivo, and the stability maintenance of Rad51 requires

43 ytoplasm of primary palatal epithelial cells in vivo, and their interaction with IRF6 is significantl

44 ctivated-phy-induced phosphorylation of PIF3 in vivo, and thereby are critical components of a transc

45 eeded to assess the functions of these genes in vivo, and to determine how they cause NS in a timely

46 II, in tubular epithelial cells in vitro and in vivo, and treatment with the small molecule TNFR inhi

47 er F expression, replicated more efficiently in vivo, and was more immunogenic.

48 mitochondrial oxidative stress and mitophagy in vivo, and were preceded by increased phosphorylation

49 TOR inhibition potently decreased ALL burden in vivo; antileukemia activity was further enhanced with

50 , and whether this process could be adaptive in vivo, are two major unanswered questions concerning t

51 hannels in TH2 lymphocytes both in vitro and in vivo, as demonstrated by the beneficial effect of Cav

52 ts and selectively binds to TrkB/C receptors in vivo, as evidenced by entrectinib blocking studies.

53 nes are recapitulated in primary neurons and in vivo, as genetic reduction in RanBP9 not only amelior

54 sitivity, millisecond-scale response latency in vivo, as well as adjustable channel off latency.

55 itical bone cells respond to mechanical load in vivo, as well as provide a technique to determine how

56 ulation, hypothesized by Ohno and documented in vivo, balances X gene with autosomal gene output.

57 In vivo, both IL-10 mRNA expression and the number of IL

58 od opens new avenues to study mechanobiology in vivo, both in embryogenesis and in disease processes,

59 MG induced AGEs formation and tumour growth in vivo, both of which can be reversed using a MG scaven

60 oscopy (FLIM) to image microscopic viscosity in vivo, both on a single cell level and in connecting t

61 from mice with long-term morphine treatment (in vivo) but not upon direct exposure of glia to morphin

62 an algorithm that automates "blind" patching in vivo, but full automation of visually guided, targete

63 ll, YKL-40 likely binds many natural ligands in vivo, but its concurrence with physical maladies may

64 fect on homeostatic intestinal proliferation in vivo, but led to marked increases in proliferation du

65 y reversing agents (LRAs) have been examined in vivo, but LRAs alone have not been able to achieve HI

66 (+) MKs, leading to increased platelet yield in vivo, but not in vitro.

67 limited effectiveness against these viruses in vivo, but the viruses were susceptible to a polymeras

68 wn to interact with thousands of RNA species in vivo, but understanding the physiological function of

69 ribosylation is also apparent following DSBs in vivo, by generating a strain with mutations at E18/E1

70 modulation of protein function in vitro and in vivo; (c) labelling of tumour antigens in mouse model

71 To validate outcomes in vivo, C57BL/6 mice were intraperitoneally injected wi

72 In all tumor types tested in vivo, calcium electroporation effectively induced nec

73 In vivo, CFI-402257 reduced MM growth in an orthotopic,

74 In vivo, CL photoactivation could be shown by using the

75 were found to significantly reduce scarring in vivo, compared to hard implants of identical diameter

76 d accumulation of tumor-infiltrating T cells in vivo, compared with the parental virus lacking such e

77 In vivo, compared with wild-type controls, miR106b-93-25

78 In vivo, compound (R,R)-68 significantly lowers plasma g

79 Upon further characterization in vivo, compound 1 demonstrated an unacceptable level o

80 ecreased glucose uptake and glycogen content in vivo, concomitant with decreased abundance of GLUT4.

81 l growth defects upon translation inhibition in vivo, consistent with its function in transcription-t

82 siologic role of p53 in adult cardiomyocytes in vivo, Cre-loxP-mediated conditional gene targeting in

83 cused on antibacterial activity in vitro and in vivo, culminating in the discovery of unprecedented s

84 Despite often minute concentrations in vivo, d-amino acid containing peptides (DAACPs) are c

85 In vivo, DACH1 was expressed during early arteriogenesis

86 8 cells outcompeted PD-L1-deleted MC38 cells in vivo, demonstrating tumor PD-L1 confers a selective a

87 ghlight striking differences in cell killing in vivo, depending on the cell subset and organs targete

88 rophils exhibit no clear migratory advantage in vivo, despite interruption of in vitro transendotheli

89 aids in intracellular survival, is expressed in vivo, elicits production of antibodies during infecti

90 transcriptional latency may not be possible in vivo, especially in the presence of combination antir

91 f these three mechanisms is more significant in vivo, especially when effects of miRNA on endogenous

92 In vivo, EspL cysteine protease activity contributes to

93 uration: one model based on a combination of in vivo, ex vivo, and clinical patient information (M36I

94 ng approaches were used to identify relevant in vivo, ex vivo, and patient features and their relativ

95 better reflects nutrients available to cells in vivo, exhibit decreased glutamine catabolism and redu

96 antly more active than dentate granule cells in vivo, exhibit spatial tuning during head-fixed spatia

97 In vivo, expanding vessel networks favor interactions wi

98 In vivo, expression of wild-type BRAF delayed melanoma o

99 H9, DH10) and module-11 of MgsG (i.e., DH11) in vivo, followed by structural characterization of the

100 In vivo, Form 1 and Form 2 differently account for 2/3(r

101 In vivo, formate served as electron donor in conjunction

102 and three of the four lines are transformed in vivo, forming large and invasive tumors in immunocomp

103 peptide complexes stimulate T cell responses in vivo, further emphasizing the need to regulate both p

104 l delivery of Cre recombinase to hepatocytes in vivo, GsD is expressed and allows CNO-dependent cAMP

105 In vivo, GSK101 increased the permeability of retinal bl

106 lowing six weeks of repeated LPS stimulation in vivo, hepatic TNF-alpha and PCNA responses subsided i

107 dioligand binding to neuroreceptors in brain in vivo, here applied to noradrenaline receptors in rat

108 he role of Hic-5 in breast tumor progression in vivo, Hic-5(-/-) mice were generated and crossed with

109 ed to modulate the site of protein induction in vivo, highlighting the critical importance of designi

110 methods with applications both in vitro and in vivo, highlighting these advances by describing their

111 ility of bnAbs to mediate protective effects in vivo, however, is undetermined.

112 thylated lysine 4 of histone H3 in vitro and in vivo; however, precise selectivity of this domain has

113 egeneration-associated proteins in vitro and in vivo; however, the regulation of HSJ1 function is lit

114 tes at the 5' and the 3' regions of the mRNA in vivo, identifies export-promoting ALYREF-binding moti

115 -1beta- or LPS-induced systemic inflammation in vivo, IL-1R2(-/-) mice were created and compared with

116 mpetent animal models to study HBV infection in vivo, immunological responses against the virus and v

117 We finish by describing a future trend for in vivo, implantable sensors, which aims to build on cur

118 scale trinucleotide (GAA)n repeat expansions in vivo, implying failed phosphate-steering promotes an

119 In vivo, in a mouse ITP model we observed a short half-l

120 In vivo, in a mouse model of P. gingivalis-induced calva

121 reduced their growth in immunodeficient mice in vivo, in comparison with NIL alone.

122 Exg8 is largely dispensable for virulence in vivo, in contrast to Eng1.

123 anding of microtubule dynamics and functions in vivo, in differentiated cells and tissues, remains un

124 ly, by combining recordings and pharmacology in vivo, in slices, and in human embryonic kidney 293 ce

125 ular effects of deficient CNP/NPR2 signaling in vivo, in the context of both moderate and advanced ao

126 tylation exacerbate alpha-synuclein toxicity in vivo, in the substantia nigra of rats.

127 We then tested the results in vivo; in mice and in human.

128 tective effects of the antibody in vitro and in vivo, including in various preclinical models of CDI.

129 hA2 effectively suppressed tumor cell growth in vivo, including TNBC patient-derived xenografts.

130 for numerous applications, such as injection in vivo (including with cells and therapeutic molecules)

131 ral immunity and DC-based priming of T cells in vivo, indicating a critical regulatory role.

132 594A) mutation) triggers lung adenocarcinoma in vivo, indicating that BRAF-inactivating mutations are

133 s, while decreasing the levels of Th17 cells in vivo, indicating that CCR2 regulates the immune respo

134 tic with sunitinib in impairing tumor growth in vivo, indicating that these responses are triggered a

135 in vitro but have a prolonged survival time in vivo, indicating that tissue microenvironment plays a

136 f how cancer cells communicate at long range in vivo, inducing intratumor heterogeneity and resistanc

137 e brain endothelial cells (a target of MAV-1 in vivo) infected ex vivo with MAV-1 had no difference i

138 In vivo, injection of human IPF MPCs converted a self-li

139 and affecting Rev-containing HIV transcripts in vivo, interacting directly with the RRE and Rev in vi

140 In vivo, intraarticular injection of lentiviral Wnt7a st

141 In vivo, intracelluar uptake of fluorescently labeled al

142 ating from three compartments in human brain in vivo: intracellular (compartment 1), extracellular (c

143 In vivo, it produced fewer symptoms and lower mortality

144 rogates over time was measured using optical in vivo (IVIS) imaging.

145 In vivo, kidney expression levels of NFkappaB2 p100 and

146 In vivo, LDHA overexpression promoted tumor growth, and

147 s strongly enhance this bacteriocin activity in vivo, leading to greater SGG colonization.

148 licing of P-transposable element transcripts in vivo, leading to the production of the non-transposas

149 ured organic electronic devices and circuits in vivo, leveraging the internal structure and physiolog

150 umulates within HSCs to promote Tet activity in vivo, limiting HSC frequency and suppressing leukaemo

151 In-vivo, liver specific Tp53 loss increases the conversi

152 In CA1-region neurons in vivo, Lphn2 was specifically targeted to dendritic sp

153 In SCLC models in vitro and in vivo, LY2606368 exhibited strong single-agent efficac

154 ne the role of ILK in hepatic insulin action in vivo, male C57BL/6J ILK(lox/lox) mice were crossed wi

155 global plasma protein turnover rates in rats in vivo, measure variability of protein turnover rates i

156 In vivo, Mice were pretreated with FK866 at 24, 12, and

157 In vivo, mice with knockout of SREBP2 in astrocytes have

158 In vivo, miR-29b overexpression is sufficient to promote

159 valuable tool for the detection of microRNAs in vivo, molecular beacons can also be employed to inhib

160 assay), ex vivo (human liver microsomes) and in vivo (mouse) model systems.

161 In vivo, MR-409 mitigated cardiac hypertrophy in mice su

162 In vivo, mutating Indian Hedgehog results in a sex ratio

163 In vivo, mutations in RSV G and PIV5 L were found in ind

164 In vivo, myeloid cells and their progenitors are an impo

165 hich alveolar macrophages mediate protection in vivo, namely antibody-induced inflammation and antibo

166 In vivo, nonproliferating oncogene-expressing melanocyte

167 In this report, we demonstrate systemic, in vivo, nonviral mRNA delivery through lipid nanopartic

168 y to improve antitumor efficacy in vitro and in vivo, offering a new avenue for cancer theranostics.

169 In vivo, oncogenic PIK3CA-driven mouse mammary tumors tr

170 In vivo, only cells expanded from PD-1(+) CD8 TILs conta

171 cles in human coronary artery-sized atheroma in vivo (P<0.05 versus reference segments).

172 In vivo, perfusion of a nonhuman primate kidney TLN-supp

173 r-selective engorge-and-accumulate processes in vivo, phagocytosis in vitro inhibited macrophage migr

174 In vivo, pharmacological inhibition of HDAC6 improved es

175 properties and dendrite morphology, studied in vivo, play a role in selective sensory processing in

176 PLIP1 is a phospholipase A1 In vivo, PLIP1 hydrolyzes polyunsaturated acyl groups fr

177 In vivo, poly(I.C)-induced neutrophilia and mucosal chem

178 dies (DMAbs) can be produced by muscle cells in vivo, potentially allowing prevention or treatment of

179 about immune-modulatory effects of nilotinib in vivo, potentially predicting response to therapy.

180 tical step in the assembly of elastic fibers in vivo, preceding chemical cross-linking.

181 ration in vitro, supports allograft survival in vivo, prevents corneal transplant rejection, and atte

182 In vivo, promoter responses to TAF mutations correlate w

183 Ex vivo and in vivo, protamine abolishes well-known apelin effects,

184 As a result, we now know that in vivo, protein folding requires assistance by a comple

185 repair (HDR) in targeted cells, particularly in vivo, provides an invaluable tool for biomedical rese

186 In vivo, Rac1 deletion in SMCs or pharmacological Rac1 i

187 In vivo, recombinant HpARI abrogated IL-33, group 2 inna

188 h distinct transcriptional signatures in DCs in vivo, reflecting the diverse environments in which Th

189 ion, trafficking, and function, particularly in vivo, remain poorly understood, though recent studies

190 In vivo, restoration of TTP expression enhances anti-tum

191 sence of CD200L signaling, both in vitro and in vivo, resulted in a higher inflammatory response of t

192 leads to degenerate DNA-binding specificity in vivo, resulting in ectopic transcription and anemia i

193 acerbates disruption of dopaminergic neurons in vivo, resulting in the neurodegeneration characterist

194 In vivo, RGC-32(-/-) mice display an attenuated experime

195 throughput sequencing to monitor translation in vivo, ribosome profiling can provide critical insight

196 In vivo, Runx2 was induced by 6.2-fold in pulmonary epit

197 , we find that during infection in vitro and in vivo, RVs significantly deplete IFN type I, II, and I

198 iposomal formulations when tested in rabbits in vivo (sc and iv application).

199 We show that, in vivo, short-term plasticity of excitatory inputs to C

200 , and non-specific DNA binding modes of LacI in vivo, showing that all these modes are operational in

201 Clozapine, to which CNO rapidly converts in vivo, shows high DREADD affinity and potency.

202 chronic and acute mouse models of CCM3 loss in vivo, significantly reducing lesion burden and extend

203 (1, 3, at 20 mug/mL) retain potent activity in vivo, significantly reducing the burden of VRE in inf

204 In vivo, Sse1sbd successfully heterodimerized with the y

205 mal transition (EMT) of ovarian cancer cells in vivo, STAT4 failed to induce EMT directly in vitro, s

206 hesize that a similar OCP assembly can occur in vivo, substantiating specific roles of the COCP carot

207 ibrotic response and myofibroblast formation in vivo, suggesting a novel therapeutic approach with p3

208 volved in the VtE-induced amelioration of DN in vivo, suggesting that DGKalpha is an attractive thera

209 than either agent alone both in culture and in vivo, suggesting that strategies that simultaneously

210 ducing the conversion of Mcc(a) into Mcc(ia) in vivo, suggesting that this region corresponds to the

211 stingly, monocyte depletion was not observed in vivo, suggesting their suitability for future testing

212 nfirm the robust antiviral activity of N6-LS in vivo, supporting the further clinical development of

213 In vivo, T-cell-specific ablation of murine Gclc prevent

214 In vivo, T-cell-specific GR deletion in pregnant animals

215 In vivo, targeting RAGE shRNA knockdown in human and mou

216 n an IFN-gamma-rich inflammatory environment in vivo, Th22 cells displayed marked plasticity toward I

217 In vivo, the alf4 mutation destabilizes the CUL1 subunit

218 In vivo, the atropine-dependent prolongation of heart ra

219 In vivo, the combination of compound 28 and ibrutinib le

220 In vivo, the DDC-stimulated number of cytokeratin-19-pos

221 ected mutagenesis and expressed in vitro and in vivo, the preparation of proteins phosphorylated at p

222 In vivo, the processing is required for target regulatio

223 f dynamic regulation of protein interactions in vivo, there is a need for techniques that can yield t

224 In vivo, these IRE1 inhibitors led to a significant decr

225 In vivo, these mice exhibited reduced severity of experi

226 njected intraperitoneally, or were activated in vivo, they migrated simultaneously to the lung, splee

227 Under conditions where it was well tolerated in vivo, this FOXO4 peptide neutralized doxorubicin-indu

228 with GroEL(SR) to perform chaperone function in vivo: three GroES(7) variants functioned with GroEL(S

229 and AR inhibition activates the PARP pathway in vivo, thus inhibition of both AR and PARP is required

230 al-1 suppressed iNOS expression in vitro and in vivo, thus reducing the production of NO.

231 This is the first in vivo, transcriptome-scale investigation of genes regu

232 In vivo, using a clinically relevant orthotopic resectio

233 ounts for the complex nuclear movements seen in vivo, using a minimal set of experimentally determine

234 c potential of GHRH stimulation in vitro and in vivo, using GHRH or its agonistic analog MR-409.

235 papulosum cyprini [EPC] fish cell line) and in vivo (using rainbow trout fry) in a dose-dependent an

236 In vivo, vascular smooth muscle cell/mesangial cell-spec

237 sive and antimetastatic effects in vitro and in vivo, warranting further study in clinical settings.

238 rker of type I IFN-induced immune activation in vivo, was enhanced in lymph nodes from pigtailed maca

239 stration and susceptibility to IAV infection in vivo, we administered KGF 120, 48, 24, and 0 h before

240 In vivo, we also found TE-specific endosiRNAs present du

241 investigate activin signaling in osteoblasts in vivo, we analyzed the skeletal phenotypes of mice lac

242 neurons is sufficient to attenuate fertility in vivo, we artificially activated them over a sustained

243 understand their development and functioning in vivo, we concomitantly inactivated CD4 and CD8 genes

244 the role of PRMT1 in innate immune responses in vivo, we created a cell type-specific knock-out mouse

245 ous effects on loss of B cell self-tolerance in vivo, we depleted neutrophils at different stages of

246 To enable precision analysis of R-loops in vivo, we develop an RNase-H-based approach; this reve

247 number of nanoparticles that could be tested in vivo, we developed a method to simultaneously measure

248 echanisms that occur by compartmentalization in vivo, we developed a technique based on single-cell e

249 To assess the effects of miR-32 in vivo, we developed transgenic mice overexpressing miR

250 osphorylation is necessary for PhyR function in vivo, we did not detect expected changes in inter-dom

251 live-imaging oligodendrocyte Ca(2+) activity in vivo, we find that high-amplitude, long-duration Ca(2

252 To better understand the function of PR3 in vivo, we generated a human PR3 transgenic mouse (hPR3

253 To investigate the effects of ARHGAP29 in vivo, we generated a novel murine allele by inserting

254 To examine effects of increased WISP2 in vivo, we generated an aP2-WISP2 transgenic (Tg) mouse

255 To determine the role of podocyte Shp2 in vivo, we generated podocyte-specific Shp2 knockout (p

256 validating our predictions both in vitro and in vivo, we identified a set of compounds with the desir

257 In vivo, we measured interactions on length scales of 80

258 To establish their relative contribution in vivo, we quantified arterial-venous concentration gra

259 n the adult neural subventricular zone (SVZ) in vivo, we show distinct responses to ionising radiatio

260 In vivo, we show enhanced expansion and CAR T cell antit

261 Furthermore, by implementing GBAi in vivo, we show that GBA-dependent signaling modulates

262 r either siRNAs or small molecule inhibitors in vivo, we showed that suppressing these 5 genes with P

263 ive effects even after single administration in vivo, we tested the hypothesis that these effects may

264 ardiovascular role for these candidate genes in vivo, we used morpholinos to reduce SYNE1, NUP37, and

265 To test whether this occurs in vivo, we used the binding of growth factor receptor b

266 to gain insight into how ApoL1 may function in vivo, we used vesicle-based ion permeability, direct

267 ic approach to control mutant MEK expression in vivo, we were able to induce tumor regression and sig

268 This was validated in vivo, where deletion of trkB.T1 in astrocytes reduced

269 ma cells in vitro This effect was replicated in vivo, where Ifit2 knockout mice displayed a dramatica

270 ates seeding and growth of peritoneal tumors in vivo, where its expression corresponds to hypoxic reg

271 gulating canonical Wnt/beta-catenin activity in vivo, where lentiviral shRNA depletion of Ror2 expres

272 nt activity of the ligand was also confirmed in vivo, where the complex of Pru p 3-ligand induced hig

273 hypertrophy and left ventricular dysfunction in vivo, whereas genetic knockdown or pharmacological bl

274 ated SMCs is essential for SMC reprogramming in vivo, whereas in vitro approaches demonstrate that Kl

275 of the 49-mer Phe521Leufs peptide identified in vivo, whereas its absence abrogates fibril formation.

276 tion of beige adipocytes to white adipocytes in vivo, whereas loss of Lsd1 precipitates it.

277 Myogenic Differentiation 1 (MyoD) transcript in vivo, whereas MyoD protein is absent.

278 ADAM10 were committed to becoming MZB cells in vivo, whereas T1 B cells lacking expression of ADAM10

279 together suppress illegitimate recombination in vivo, whereas unregulated duplex unwinding is detrime

280 as the dominant if not exclusive dimer found in vivo, whether SecA was cytosolic or in lipid or SecYE

281 nding of the fate and transformation of IAPP in vivo, which are expected to have consequential bearin

282 ter gene into adult hematopoietic stem cells in vivo, which are predominantly quiescent, by generatin

283 ngStar, a platform for perturbation analysis in vivo, which combines live imaging, real-time image an

284 olled by the periductal extracellular matrix in vivo, which contributes to a dampened circadian rhyth

285 bited ductal branching and MEC proliferation in vivo, which corroborated the in vitro anti-proliferat

286 -depleted mice exhibit delayed wound closure in vivo, which could be rescued by topical IL-27 treatme

287 hagocytosis of tumor cells both in vitro and in vivo, which defines the MHC class I-LILRB1 signaling

288 st large-scale sensing of sarcomere dynamics in vivo, which is a necessary first step to understand m

289 The cleaved ends are difficult to repair in vivo, which may indicate their biological significanc

290 n, and invasion of cancer cells in vitro and in vivo, which were confirmed by colony formation assay,

291 1-mediated profibrotic pathways in vitro and in vivo, while esculetin significantly inhibited Wnt/bet

292 and greatly reduces their long-term survival in vivo, while having no effect on the survival of centr

293 entify RNAs that interact directly with CsrA in vivo, while ribosome profiling and RNA-seq uncover th

294 response toward either M1 or M2 macrophages in vivo, wild-type mice were injected with gamma interfe

295 Bre5 binds RNA in vivo, with a preference for exon 2 regions of intron-

296 ds can be effective at targeting tumor PD-L1 in vivo, with good specificity and rapid clearance.

297 track molecular processes, both in vitro and in vivo, with high sensitivity and specificity.

298 n predicting extrusion hotspots and dynamics in vivo, with potential applications to tissue regenerat

299 To specifically study the function of LCs in vivo, without altering the DC subset composition in t

300 rus, rLS/AMPV-C F&G, was slightly attenuated in vivo, yet maintained similar characteristics in vitro