1 tions in allergic diseases have been studied
incompletely.
2 ity, physical activity, and asthma have been
incompletely addressed, probably because their time-depe
3 therwise occluding basement membrane remains
incompletely assembled.
4 losteric mTOR inhibitors, such as rapamycin,
incompletely block mTORC1 compared with mTOR kinase inhi
5 However, GPR55 knockout mice remain
incompletely characterized in models of pathological pai
6 ischemia and ventricular dysfunction through
incompletely characterized mechanisms.
7 luable resource for exploring the biology of
incompletely characterized proteins and for elucidating
8 h extracellular vesicles are internalized is
incompletely characterized, it is generally considered t
9 e reaction of these oxidants with PDI remain
incompletely characterized, we investigated the reaction
10 nd implications for subsequent treatment are
incompletely characterized.
11 s that govern Acinetobacter pathogenesis are
incompletely characterized.
12 The integrin-binding sites on iC3b remain
incompletely characterized.
13 cardiac endoplasmic reticulum (ER) stress is
incompletely characterized.
14 rly B cell development and function is still
incompletely characterized.
15 (VEGF) signaling during angiogenesis remain
incompletely characterized.
16 effects at physiological concentrations are
incompletely characterized.
17 lymphoid and myeloid cells, but they remain
incompletely characterized.
18 e release, but the roles of each variant are
incompletely characterized.
19 tatic progression of breast cancer are still
incompletely characterized.
20 sible for the resolution of inflammation are
incompletely characterized.
21 on to variations in dietary Na(+) intake are
incompletely characterized.
22 s (JCV), the sites of JCV persistence remain
incompletely characterized.
23 pplication in postnatal gene editing remains
incompletely characterized.
24 hip between orexin and impulsive behavior is
incompletely characterized.
25 letion mutations at codons 591 to 603 remain
incompletely characterized.
26 resistance and clonal composition of PD are
incompletely characterized.
27 isease, but the underlying mechanism remains
incompletely characterized.
28 mes improve quality of life in patients with
incompletely controlled asthma despite having little eff
29 munological mechanisms underlying C-IRIS are
incompletely defined and no reliable predictive biomarke
30 ost inflammatory disease-associated loci are
incompletely defined, including in the LACC1 (C13orf31)
31 olved in impaired neutrophil function remain
incompletely defined.
32 ledge, the durability of these prostheses is
incompletely defined.
33 s of cancer after kidney transplantation are
incompletely defined.
34 origins and differentiation pathways remain
incompletely defined.
35 ractile gene expression during pregnancy are
incompletely defined.
36 anisms involved in airway remodelling remain
incompletely defined.
37 n, and GATA-2-dependent genetic networks are
incompletely defined.
38 ment of MRAP in adipocyte metabolism remains
incompletely defined.
39 es platelet activation and thrombosis remain
incompletely defined.
40 t, but other aspects of this interaction are
incompletely defined.
41 by NF-kappaB in developing NKT cells remains
incompletely defined.
42 ates multiple insertion pathways that remain
incompletely defined.
43 the drivers of this tolerogenic program are
incompletely defined.
44 es of exacerbation-prone asthma (EPA) remain
incompletely defined.
45 the synergy of prime-boost strategies remain
incompletely defined.
46 ells via metabolic coupling factors that are
incompletely defined.
47 from the female reproductive tract (FRT) are
incompletely defined; in particular, the contribution of
48 mechanisms of work-related asthma (WRA) are
incompletely delineated.
49 dely but inconsistently used technology with
incompletely demonstrated effectiveness.
50 However, recent calendar trends are
incompletely described, and which population subgroups m
51 illness, but its mechanisms of infection are
incompletely described.
52 uries (TBIs), most of which are mild, remain
incompletely described.
53 ant metabolism at the network level is still
incompletely described.
54 late IL-10 production in Th2 cells are still
incompletely described.
55 all GTPase activation by chemokines is still
incompletely described.
56 eractions when the fitness landscape is only
incompletely determined due to imprecise measurements or
57 the biomass-burning aerosol was rapidly but
incompletely displaced, likely by the nitric acid produc
58 Cystinuria is an
incompletely dominant disorder characterized by defectiv
59 which current therapy with beta-blockers is
incompletely effective.
60 vitreal injections which may be painful, are
incompletely efficacious, and represent a significant tr
61 y checkpoints and CAR T cell therapy remains
incompletely explored.
62 by binding to exposed hydrophobic regions of
incompletely folded or aggregated proteins.
63 e parameters governing allograft outcome are
incompletely identified.
64 nditions, but is represented empirically and
incompletely in today's models.
65 wide and narrow clefts, mutant channels were
incompletely inactivated.
66 contrast with known ASIC1a inhibitors, Hi1a
incompletely inhibits ASIC1a activation in a pH-independ
67 ween Plk1 up-regulation and tumorigenesis is
incompletely investigated.
68 c activation of cardiac CaV1.2, however, are
incompletely known, but are presumed to require phosphor
69 ch coordination, though such entities remain
incompletely known.
70 choice and the underlying neurochemistry are
incompletely known.
71 e components and pathways responsible remain
incompletely known.
72 ntial laser applications, this degeneracy is
incompletely lifted in the valence band, spreading the h
73 atified compartments but as components of an
incompletely mixed bioreactor.
74 ected two bnAbs, PGT121 and 3BNC117, as they
incompletely neutralize the clade C simian-human immunod
75 chniques in flies often reduce gene activity
incompletely or rely on cell division.
76 associated with small nuclear RNAs (snRNAs)
incompletely processed spliceosomal.
77 moradiotherapy (CRT) for locally advanced or
incompletely resected non-small-cell lung cancer (NSCLC)
78 patients with hypertrophic cardiomyopathy is
incompletely resolved.
79 by the thymus through mechanisms that remain
incompletely resolved.
80 pid accumulation through mechanisms that are
incompletely resolved.
81 sms underlying repeat-induced disease remain
incompletely resolved.
82 r observed directly, but only indirectly and
incompletely through noisy measurements based on express
83 c evaluation with a clinical suspicion of an
incompletely treated liver abscess.
84 imulus-secretion pathway in L-cells is still
incompletely understood and a topic of debate.
85 and identity of the cells responsible remain
incompletely understood and contentious.
86 essive malignancy whose histopathogenesis is
incompletely understood and that is largely resistant to
87 tuttering is a neuropsychiatric condition of
incompletely understood brain origin.
88 rization of peroxisomal ABCD transporters is
incompletely understood but is of potential significance
89 unctions have been proposed in yeast but are
incompletely understood in humans.
90 ver, its primary molecular mechanisms remain
incompletely understood in plants and animals.
91 ypoxia) limits cholesterol synthesis through
incompletely understood mechanisms mediated by the oxyge
92 y used analgesic drug with multiple but only
incompletely understood mechanisms of action, including
93 sed analgesic and antipyretic drug with only
incompletely understood mechanisms of action.
94 Through
incompletely understood mechanisms, activated mTORC1 tri
95 ammatory responses and osteoclastogenesis by
incompletely understood mechanisms.
96 n cancer cells contributes to oncogenesis by
incompletely understood mechanisms.
97 insulin-like signaling (ILS) pathway through
incompletely understood mechanisms.
98 ed cardiovascular morbidity/mortality and an
incompletely understood pathophysiology.
99 tics are likely to be important in the still
incompletely understood physiologic mechanisms that regu
100 xtracellular matrix (ECM) has a crucial, but
incompletely understood role during tumor progression an
101 angiectasia mutated (ATM) plays a unique yet
incompletely understood role in gammaherpesvirus infecti
102 e recruited to the lungs, where they have an
incompletely understood role in health and disease.
103 phic lateral sclerosis (ALS) are complex and
incompletely understood, although contributions to patho
104 The underlying causes of cachexia are
incompletely understood, and currently no therapeutic ap
105 e mechanisms of these adaptive responses are
incompletely understood, and it is also unclear why thes
106 tes metabolism in epidermal keratinocytes is
incompletely understood, and it is unknown whether glyco
107 rending foods and dietary patterns are still
incompletely understood, and nutritional science continu
108 is that most of its molecular processes are
incompletely understood, and suggest a number of key que
109 he early stages of protein misfolding remain
incompletely understood, as most mammalian proteinopathi
110 nces in central nociceptive signaling remain
incompletely understood, as nothing is known about how s
111 The role of tyrosine-1 (Tyr1) remains
incompletely understood, as, for example, mutating all T
112 letion processes deplete non-EV particles is
incompletely understood, but depleted serum is associate
113 opulation data are scant and pathogenesis is
incompletely understood, but enteral feed exposures are
114 e of celiac disease and geographic region is
incompletely understood, but the occurrence of several a
115 Although HFpEF pathophysiology remains
incompletely understood, endothelial inflammation is sta
116 val under oxidative stress conditions remain
incompletely understood, especially for the translationa
117 heritable forms of cardiovascular disease is
incompletely understood, even in patients with strong fa
118 PK and its potential as a drug target remain
incompletely understood, largely because of a lack of op
119 ructure and dynamic properties of CSP remain
incompletely understood, limiting our understanding of i
120 pathogenesis of Parkinson's disease (PD) is
incompletely understood, mitochondrial dysfunction is th
121 s nitroxyl (HNO) production and action being
incompletely understood, pharmacological donors show bro
122 though its neurobiological underpinnings are
incompletely understood, the dopamine hypothesis has bee
123 steps in FVIII inhibitor development remain
incompletely understood, we sought to define early regul
124 output of regulatory small RNAs (sRNAs) are
incompletely understood.
125 abetes mellitus, although the mechanisms are
incompletely understood.
126 he challenging gut environment still remains
incompletely understood.
127 l thromboembolism in patients with cancer is
incompletely understood.
128 he biology underlying this treatment remains
incompletely understood.
129 he mechanisms connecting these processes are
incompletely understood.
130 cell homeostasis after thymic emigration are
incompletely understood.
131 g proteins on SK2 channel trafficking remain
incompletely understood.
132 the mechanisms of action of liraglutide are
incompletely understood.
133 ion and propagation of coagulation are still
incompletely understood.
134 regulatory mechanism of this process remains
incompletely understood.
135 e in white adipose tissue physiology remains
incompletely understood.
136 ut this definition and how to achieve it are
incompletely understood.
137 redox processes control neuroinflammation is
incompletely understood.
138 t the underlying mechanisms and function are
incompletely understood.
139 tex on muscles during different behaviors is
incompletely understood.
140 in cancer, a setting in which predictors are
incompletely understood.
141 bolic factors that affect its processing are
incompletely understood.
142 ly used antianginal drug, nitroglycerin, are
incompletely understood.
143 me infected or access viral antigens remains
incompletely understood.
144 by peroxisomes, PBD pathology is complex and
incompletely understood.
145 ng events, but the underlying mechanisms are
incompletely understood.
146 , their role in disease pathogenesis remains
incompletely understood.
147 al immune system in chronic lung diseases is
incompletely understood.
148 versity captures antigen specificity remains
incompletely understood.
149 epair, the role of DEK in DNA repair remains
incompletely understood.
150 tect peripheral nervous system injury remain
incompletely understood.
151 components, but the underlying mechanism is
incompletely understood.
152 wever, the underlying neurocircuitry remains
incompletely understood.
153 ms governing KCNQ trafficking to the AIS are
incompletely understood.
154 t the underlying molecular mechanism remains
incompletely understood.
155 ctional integrity in atherosclerosis remains
incompletely understood.
156 rotective effects of SGLT2 inhibitors remain
incompletely understood.
157 he biological functions of PC4, which remain
incompletely understood.
158 e optimal levels of viral gene expression is
incompletely understood.
159 are the primary site of CO2 elimination, are
incompletely understood.
160 ty, the neurobiological basis of EDs remains
incompletely understood.
161 opathy, but the underlying mechanism remains
incompletely understood.
162 s and mechanisms in brain development remain
incompletely understood.
163 ion, especially the initiation step, remains
incompletely understood.
164 ng their activities in time and space remain
incompletely understood.
165 he relationships between these processes are
incompletely understood.
166 which mPFC regulates reward seeking remains
incompletely understood.
167 dynamics of these facultative pathogens are
incompletely understood.
168 e (HR), but the underlying mechanism remains
incompletely understood.
169 reproductive development, a process that is
incompletely understood.
170 axis and obesity-associated pathologies are
incompletely understood.
171 programs drive this regionalization remains
incompletely understood.
172 ays to repress self-reactive T cells remains
incompletely understood.
173 ations impact tumorigenesis in ways that are
incompletely understood.
174 changes underpinning disease progression are
incompletely understood.
175 and the consequences of their disruption are
incompletely understood.
176 tivity; however, the underlying mechanism is
incompletely understood.
177 genetic mechanisms of transformation remain
incompletely understood.
178 rapidly develops through mechanisms that are
incompletely understood.
179 tivity-dependent molecular mechanisms remain
incompletely understood.
180 opriate growth of intact malignant tumors is
incompletely understood.
181 nes, yet their molecular equivalence remains
incompletely understood.
182 that control cellular action potentials are
incompletely understood.
183 mechanisms responsible for these effects are
incompletely understood.
184 receptors and the underlying mechanisms are
incompletely understood.
185 association with center characteristics, is
incompletely understood.
186 e immune response, but the mechanisms remain
incompletely understood.
187 ow PDAC cells to endure these conditions are
incompletely understood.
188 chronic lung disease after preterm birth are
incompletely understood.
189 on in reconstructs used for skin grafting is
incompletely understood.
190 l constituents of surrounding stroma remains
incompletely understood.
191 h mutations affect synaptic function remains
incompletely understood.
192 ent, but their functions in this process are
incompletely understood.
193 isms by which these actions are executed are
incompletely understood.
194 ctions below the detection limit of RDTs are
incompletely understood.
195 cell-cycle progression in human cells remain
incompletely understood.
196 ineages, the underlying mechanisms are still
incompletely understood.
197 o impaired resolution in atherosclerosis are
incompletely understood.
198 t the requirements for spore germination are
incompletely understood.
199 sses; however, their molecular regulation is
incompletely understood.
200 ch, the cause and pathogenesis of TTS remain
incompletely understood.
201 organ and induce sustained inflammation are
incompletely understood.
202 trol these divergent functions of TF remains
incompletely understood.
203 pond to bile is remarkably complex and still
incompletely understood.
204 sis of human resistance to infection remains
incompletely understood.
205 of doxorubicin-induced cardiotoxicity remain
incompletely understood.
206 directly affect neutrophil function remains
incompletely understood.
207 wever, their roles in health and disease are
incompletely understood.
208 nctions that regulate reactivation are still
incompletely understood.
209 The biology of HIV latency is complex and
incompletely understood.
210 mechanisms by which they are established are
incompletely understood.
211 transcriptionally upon the initial insult is
incompletely understood.
212 ning tumour exosome release, however, remain
incompletely understood.
213 eutic implications of these mutations remain
incompletely understood.
214 responses regulating antifungal immunity are
incompletely understood.
215 ctors determining the risk of recurrence are
incompletely understood.
216 isms mediating RSK-dependent motility remain
incompletely understood.
217 lying prostate cancer lineage plasticity are
incompletely understood.
218 mode of action of the ubiquitin coat remain
incompletely understood.
219 e of these proteins in viral pathogenesis is
incompletely understood.
220 n broad, general clinical use is complex and
incompletely understood.
221 ce of CDK4 amplicon in GBM formation remains
incompletely understood.
222 The mechanism of axon degeneration is
incompletely understood.
223 y partitioned onto a postsynaptic arbor, are
incompletely understood.
224 ts biochemical and phenotypic effects remain
incompletely understood.
225 and disease conditions for reasons that are
incompletely understood.
226 owever, their role in progenitor function is
incompletely understood.
227 differentiation into these two lineages are
incompletely understood.
228 gating and proton conductance mechanisms are
incompletely understood.
229 Many aspects of allosteric regulation remain
incompletely understood.
230 ellular signaling, but its mechanism remains
incompletely understood.
231 t pAPCs maintain mucosal homeostasis remains
incompletely understood.
232 eat containing receptor NLRP10 in disease is
incompletely understood.
233 g to the emergence of dominant clones remain
incompletely understood.
234 th of Proteobacteria during inflammation are
incompletely understood.
235 iation in circulating PTH concentrations are
incompletely understood.
236 cells resist HIV-induced apoptosis is still
incompletely understood.
237 ute dependence of PDAC cells on KRAS remains
incompletely understood.
238 the interstitial space of inflamed lungs are
incompletely understood.
239 and cells with high CD44 expression remains
incompletely understood.
240 hanism for this cross-talk, however, remains
incompletely understood.
241 w into tissue-destructive metastases remains
incompletely understood.
242 itself is regulated in different lineages is
incompletely understood.
243 lls (ILC) in different microenvironments are
incompletely understood.
244 reas the mechanisms of their efficacy remain
incompletely understood.
245 y which extracellular signals control it are
incompletely understood.
246 ries of cell-cycle-regulated events that are
incompletely understood.
247 ctivation, but the underlying mechanisms are
incompletely understood.
248 feeding than male mice, for reasons that are
incompletely understood.
249 ivate NEUROG1 and adopt a neuroblast fate is
incompletely understood.
250 d reentry (LR) in atrial tachycardia remains
incompletely understood.
251 rs linking obesity to insulin resistance are
incompletely understood.
252 tricular hypertrophy in patients with CKD is
incompletely understood.
253 tic underpinning for this state has remained
incompletely understood.
254 c mechanisms underlying this disorder remain
incompletely understood.
255 t interactions and roles in pathobiology are
incompletely understood.
256 erlapping roles during normal physiology are
incompletely understood.
257 regulated alternative splicing have remained
incompletely understood.
258 ligins and their molecular mechanisms remain
incompletely understood.
259 ever, how the secretome is regulated remains
incompletely understood.
260 Mechanisms of DVT remain
incompletely understood.
261 nking proteins in cortical dynamics is still
incompletely understood.
262 AS) with preserved ejection fraction (EF) is
incompletely understood.
263 sive strategies, the pathophysiology remains
incompletely understood.
264 oliferation in this inflammatory process are
incompletely understood.
265 ovascular associations of these drugs remain
incompletely understood.
266 f myeloproliferative neoplasms (MPN) remains
incompletely understood.
267 signaling, but the underlying mechanisms are
incompletely understood.
268 ss scarring, but the mechanisms involved are
incompletely understood.
269 ecular mechanisms of this suppression remain
incompletely understood.
270 ut the mechanisms driving this evolution are
incompletely understood.
271 ges CBF by 3%-4%), the coupling mechanism is
incompletely understood.
272 the molecular functionality of CD147 remains
incompletely understood.
273 ogical interactions between DM and TB remain
incompletely understood.
274 , factors that underlie Abeta deposition are
incompletely understood.
275 pin-containing transcripts in the genome are
incompletely understood.
276 rly the function of primary cilia, are still
incompletely understood.
277 is of major clinical relevance, yet remains
incompletely understood.
278 l effector versus memory differentiation are
incompletely understood.
279 of FLG deficiency in AE pathogenesis remains
incompletely understood.
280 rease neurogenesis and modulate behavior are
incompletely understood.
281 et the factors affecting their formation are
incompletely understood.
282 ium; however, the mechanisms involved remain
incompletely understood.
283 sible for flow-induced beneficial effects is
incompletely understood.
284 ure, but regulation of their biosynthesis is
incompletely understood.
285 d consequences of tubule fission failure are
incompletely understood.
286 y affect the function of neural circuits are
incompletely understood.
287 alterations that promote tumor formation are
incompletely understood.
288 ration in humans through mechanisms that are
incompletely understood.
289 or protein is spatiotemporally controlled is
incompletely understood.
290 eduction of arsenite, As(III), by pyrite are
incompletely understood.
291 that promote cardiomyocyte cell cycle remain
incompletely understood.
292 in human urine, where their functionality is
incompletely understood.
293 t trafficking of nascent KRAS4B to the PM is
incompletely understood.
294 nisms to prevent aberrant activation remains
incompletely understood.
295 ISM), and some combustion conditions remains
incompletely understood.
296 in the pathogenesis of allergic diseases is
incompletely understood.
297 the specificity of the subunits mutated are
incompletely understood.
298 al mechanisms of narcotic bowel syndrome are
incompletely understood; however, opioid-induced hyperal
299 ment, maintenance and function so far remain
incompletely unexplored.
300 ss of potential therapeutics are complex and
incompletely validated.