ライフサイエンスコーパス: increase in blood pressure

1 ETA antagonist did not completely block the increase in blood pressure.

2 g childhood may help suppress the adolescent increase in blood pressure.

3 els, leading to negative effects, such as an increase in blood pressure.

4 n fibre muscle afferents and evokes a reflex increase in blood pressure.

5 ar bed induces sympathetic activation and an increase in blood pressure.

6 ation rate, salt and water retention, and an increase in blood pressure.

7 e baroreceptor reflex which could lead to an increase in blood pressure.

8 hedra alkaloids and water produced a greater increase in blood pressure.

9 A) decreased reflexively as the result of an increase in blood pressure.

10 xide-induced vasodilation and the subsequent increase in blood pressure.

11 e is secreted by the heart in response to an increase in blood pressure.

12 0.01) but was not correlated to the absolute increase in blood pressure.

13 in PVN neurons attenuated the Ang II-induced increases in blood pressure.

14 ells and abrogation of alpha(1D)-AR-mediated increases in blood pressure.

15 ssures increased dramatically in response to increases in blood pressure.

16 D in renovascular hypertension halts further increases in blood pressure.

17 ods and dairy and meat products may moderate increases in blood pressure.

18 arsenic exposure may accelerate age-related increases in blood pressure.

19 bin-based oxygen carriers often induces mild increases in blood pressure.

20 peripheral nerve, blood flow increased with increases in blood pressure.

21 n (400 mg/kg iv) in control rats produced an increase in blood pressure (43%) and total peripheral re

22 osslinked hemoglobin (400 mg/kg iv) produced increases in blood pressure (81%), cardiac output (36%),

23 There was no increase in blood pressure after HM administration of PE

24 e 35 d CIH-exposed mice showed a significant increase in blood pressure and an increased density of N

25 the GFAP-hAGT transgene exhibited a 15-mm Hg increase in blood pressure and an increased preference f

26 ce of most cardiac changes and a significant increase in blood pressure and aortic intima-media thick

27 DP-choline caused a dose- and time-dependent increase in blood pressure and decrease in heart rate.

28 crosslinked hemoglobin produces an immediate increase in blood pressure and marked regional circulato

29 Linear increases in blood pressure and an exponential increase

30 The increases in blood pressure and cardiac output are due t

31 deled the effect of treating obesity-related increases in blood pressure and dyslipidemia.

32 heat, restraint, inflation), caused similar increases in blood pressure and heart rate in control an

33 od pressure and heart rate in humans, caused increases in blood pressure and heart rate, while modest

34 ncluding reductions in fat mass; significant increases in blood pressure and in total, LDL-, and HDL-

35 s rise in plasma cortisol was accompanied by increases in blood pressure and plasma concentrations of

36 urons attenuate psychological stress-induced increases in blood pressure and promote neuroendocrine a

37 strictor reactivity and hypertension-induced increases in blood pressure and renal injury in mice.

38 rtain forms of stress can induce significant increases in blood pressure and sympathetic nerve activi

39 There were significant increases in blood pressure and systemic vascular resist

40 tion of baseline serum aldosterone levels to increases in blood pressure and the incidence of hyperte

41 eased endogenous H2S level, an age-dependent increase in blood pressure, and impaired endothelium-dep

42 It was also associated with an increase in blood pressure, and there was no significant

43 Altogether, 12 patients had abrupt increases in blood pressure, and 8 had some impairment o

44 We defined an increase in blood pressure as an increment of at least o

45 nd individually adjusted to achieve the same increase in blood pressure as was observed in a matched

46 and treadmill exercise caused an 11% higher increase in blood pressure, both suggesting impaired vas

47 he nucleus tractus solitarius (NTS) after an increase in blood pressure (BP) and prolonged baroreflex

48 t afferent mechanisms to those eliciting the increase in blood pressure (BP).

49 ory cardiovascular reflexes characterized by increases in blood pressure (BP) and heart rate (HR), wh

50 ug) or fostriecin (15 pg) caused significant increases in blood pressure (BP) and potentiated the mag

51 that individuals who exhibit frequent, large increases in blood pressure (BP) during psychological st

52 The tasks elicited prompt increases in blood pressure (BP), heart rate, cortisol,

53 ductions in vascular function independent of increases in blood pressure (BP).

54 trapib treatment was associated with a small increase in blood pressure, but was devoid of major side

55 Norepinephrine led to an increase in blood pressure by augmenting cardiac indices

56 efferent sympathetic nerve activity and the increase in blood pressure by CsA seen in control are gr

57 The increases in blood pressure, cardiac output, stroke volu

58 population scale, such air pollution-related increases in blood pressure could, in part, account for

59 AdN17Rac1 abolished the increase in blood pressure, decrease in heart rate, and

60 The increase in blood pressure, despite the decrease in HREN

61 ments of nitrosyl hemoglobin, and blunts the increase in blood pressure due to DOCA-salt hypertension

62 Increases in blood pressure due to intravenous phenyleph

63 r =70 bpm, but it was not associated with an increase in blood pressure during exercise in persons wi

64 These results were primarily explained by an increase in blood pressure during sleep opportunities (S

65 , where acute systemic phenylephrine-induced increases in blood pressure evoked a significant increas

66 TP receptor agonist U-46619 causes transient increases in blood pressure followed by cardiovascular c

67 2 was required for LPA to elicit a transient increase in blood pressure following intravenous adminis

68 Hg systolic and 10 mm Hg diastolic increment increase in blood pressure from age 30 to 65 years.

69 estrogen and 1 to 4 mg progestin, and small increases in blood pressure have been reported even amon

70 timuli from contracting muscles evoke reflex increases in blood pressure, heart rate and sympathetic

71 h both mental stress tests, characterized by increases in blood pressure, heart rate, rate-pressure p

72 ent in nontransgenic mice led to significant increases in blood pressure, hypothalamic angiotensin II

73 ctivity increases and this contributes to an increase in blood pressure (i.e. exercise pressor reflex

74 e important implications because even modest increases in blood pressure impact cardiovascular diseas

75 nced ET-1 vasoconstriction and prolonged the increase in blood pressure in anaesthetised rats.

76 gonist BQ-788 caused a small but significant increase in blood pressure in both groups.

77 has been associated with obesity and a small increase in blood pressure in children; however, whether

78 potency was the observation of a significant increase in blood pressure in conscious telemeterized ra

79 ating or tissue angiotensinogen may cause an increase in blood pressure in humans, even in the presen

80 nhance channel activity may contribute to an increase in blood pressure in individuals with essential

81 1 receptor antagonist, SR141716A, elicits an increase in blood pressure in rats subjected to haemorrh

82 The increase in blood pressure in spontaneously hypertensive

83 20 (3%) patients were withdrawn because of increases in blood pressure; in the sibutramine group, s

84 was performed to determine whether the acute increase in blood pressure induced by breathing through

85 sted whether the antinociception produced by increases in blood pressure is dependent on changes in t

86 at neonate lays the foundation for sustained increases in blood pressure later in life.

87 Rats exposed to CIHH had increases in blood pressure, leading to hypertension, an

88 The RR per 10-mm Hg increase in blood pressure measured at 1 year after tran

89 PSNs) was correlated to neither the absolute increase in blood pressure nor the rate of increase in m

90 A dose-related increase in blood pressure occurred with diaspirin cross

91 Importantly, we observed no increases in blood pressure or heart rate with BIM-22493

92 with lower blood pressure and an attenuated increase in blood pressure over time.

93 ted administration of BK elicited consistent increases in blood pressure over a 100 min period while

94 arsenic exposure was associated with greater increases in blood pressure over the course of pregnancy

95 the effects of angiotensin II, which include increase in blood pressure, peripheral vasoconstriction,

96 We report that the increase in blood pressure produced by a 2 week administ

97 or OFF cells was recorded in the RVM during increases in blood pressure produced by intravenous infu

98 (n = 23) or discontinued (n = 10) to manage increases in blood pressure, pulse rate, or other sympto

99 h groups exhibited a significant and similar increase in blood pressure ratio, angiographic score, an

100 iogenic agent sodium lactate elicited robust increases in blood pressure, respiratory rate, and heart

101 exhibited either transient or no significant increase in blood pressure showed tighter .NO binding af

102 at Type A participants would exhibit greater increases in blood pressure than Type B participants ove

103 ular coupling contributes to the age-related increase in blood pressure that occurs in women only.

104 Despite the increase in blood pressure, there is no decrease in eith

105 area rich in 5-HT receptors, would attenuate increases in blood pressure to intravenous 5-HT.

106 PS rats demonstrated a greater increase in blood pressure variability compared with con

107 It is concluded that PVN neurones may elicit increases in blood pressure via excitatory connections w

108 When the cocaine-induced increase in blood pressure was minimized (nitroprusside)

109 ressure and AT1R protein expression, and the increase in blood pressure was reversed by AT1R blockade

110 eceptor antagonist eliminated Ang II-induced increases in blood pressure, water intake, and sympathoa

111 al Npr1 expression leads to a salt-sensitive increase in blood pressure, whereas above normal Npr1 ex

112 ACE2 deficiency was associated with a modest increase in blood pressure, whereas the absence of ACE2

113 clooxygenase, however, prevented the initial increase in blood pressure without affecting the delayed