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1 with 20% mannitol solution for reduction of increased intracranial pressure.
2 o increased intracranial venous pressure and increased intracranial pressure.
3 a reduced ammonia-induced brain swelling and increased intracranial pressure.
4 othermia there was no significant relapse of increased intracranial pressure.
5 sts are limited by cerebral vasodilation and increased intracranial pressure.
6 in children with either large hemorrhage or increased intracranial pressure.
7 ) is a syndrome defined by four criteria: 1) Increased intracranial pressure, 2) normal or small vent
8 pected diagnosis of central precocity due to increased intracranial pressure, a diagnosis of congenit
9 ed a primary, closed traumatic brain injury; increased intracranial pressure; an initial head injury
10 nic pulmonary edema is often associated with increased intracranial pressure and can be the initial m
11 g the study, the patients developed signs of increased intracranial pressure and had documented intra
12 d solely on attempts at preventing/reversing increased intracranial pressure, and the treatment of ot
18 he robustness of the model to predict future increased intracranial pressure events 30 minutes in adv
19 Rats undergoing surgically induced acutely increased intracranial pressure (explosive brain death)
20 a, hyperglycemia, fever, blood pressure, and increased intracranial pressure has been insufficiently
22 ents with acute liver failure (ALF) die from increased intracranial pressure (ICP) while awaiting tra
23 here the development of cerebral oedema and increased intracranial pressure in 12 patients with chro
26 A model for early detection of episodes of increased intracranial pressure in traumatic brain injur
27 matoma expansion, perihaematomal oedema with increased intracranial pressure, intraventricular extens
30 merous untoward effects (including promoting increased intracranial pressure), little is known about
31 racranial hypertension is a rare syndrome of increased intracranial pressure manifesting as headache,
32 ongoing pentobarbital infusion, or markedly increased intracranial pressure on interruption of conti
33 her CVI is caused by the seizure disorder or increased intracranial pressure or by the underlying dis
35 esions, worsening hydrocephalus, evidence of increased intracranial pressure, or necessity for surgic
36 arge step toward an early warning system for increased intracranial pressure that can be generally ap
37 ria for poor-prognosis liver failure and had increased intracranial pressure that was unresponsive to
39 f pseudotumor cerebri (PTC), the syndrome of increased intracranial pressure without a brain tumor.
40 g young obese women, producing a syndrome of increased intracranial pressure without identifiable cau
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