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1 ggesting that there is no healthy pattern of increased weight.
2 ine also increased hypoglycemia and modestly increased weight.
3 e with increased expenditure associated with increased weight.
4 d from malalignment and the excess load from increased weight.
5 the vehicle or MS1 conditions significantly increased weight (9-20 g) and produced smaller reduction
6 1-null mice grow longer than wild type, with increased weight and adiposity, when restricted in vitam
9 may increase dieting, a risk factor for both increased weight and eating disorders in adolescents.
10 and pediatric isolates were associated with increased weight and gestational age of newborns (P </=
11 osure to reduced rations was associated with increased weight and greater indexes of fat deposition a
13 tica pathogenesis, mice lacking Gal-1 showed increased weight and survival, lower bacterial load, and
14 hort-term adverse events-acne, night sweats, increased weight, and altered mood and libido-are recogn
15 e, which results in flies with larger cells, increased weight, and decreased life span compared to wi
17 cerebellar Purkinje neurons reduced ataxia, increased weight, and prolonged life, but it did not pre
18 ies also support the notion that fatigue and increased weight are linked to higher osteoarthritis pai
19 Hand OA was significantly associated with increased weight at ages 26 years, 43 years, and 53 year
21 he single mutants had higher oil content and increased weight compared to those of the wild type, wit
22 , and/or lifestyle behaviors account for the increased weight, fat mass, and central adiposity experi
24 pothalamic satiation signaling, hyperphagia, increased weight gain and adiposity, and enhanced lipoge
25 leptin signaling (Leprdb/db mice), including increased weight gain and adiposity, hyperphagia, cold i
26 hat these Npc1(+/-) mice were susceptible to increased weight gain characterized by increased whole b
28 glucose intolerance, insulin resistance, and increased weight gain during, but not prior to, pregnanc
31 ght gain (diet-induced obese [DIO] rats) had increased weight gain in response to consuming saccharin
32 that daily insulin injections significantly increased weight gain in the transgenic lines expressing
35 ng, whereas group-housed female mice display increased weight gain on high-fat diet, reduced behavior
37 ted protein 2 antibodies was associated with increased weight gain with either intensive or conventio
39 hort day hamsters were also characterized by increased weight gain, and heavier adrenal glands (p < 0
40 nd cardiovascular disease-related mortality, increased weight gain, and high risk for severe hypoglyc
42 rdination and cognitive function but exhibit increased weight gain, elevated white adipose tissue dep
45 g insulin concentrations are associated with increased weight gain, whereas insulin resistance seems
47 essive gestational weight gain may result in increased weight in children; however, studies have not
50 order of magnitude variation in body weight; increased weight is supported solely through disproporti
52 ggest that, despite a known association with increased weight, long-term sulfonylurea therapy may red
54 MC4R (I251L), a rare variant associated with increased weight loss after RYGB and increased basal act
55 ulted in more severe disease, as measured by increased weight loss and airway resistance, as compared
56 istering DSS to IL-17R(-/-) mice resulted in increased weight loss and more severe intestinal inflamm
57 en in mice infected with M. tuberculosis and increased weight loss and mortality in mice infected wit
58 ore susceptible to endotoxemia, evidenced by increased weight loss and serum TNF-alpha, IL-6, and IL-
59 and intestinal-secreted IgA correlated with increased weight loss at the end of DSS administration.
60 a significantly reduced survival percentage, increased weight loss, and a more-rapid increase in bact
61 ctivated RSV-vaccinated children, as well as increased weight loss, clinical illness, and enhanced pa
62 zed with vacvG results in the development of increased weight loss, clinical illness, and Penh simila
63 s in exacerbated disease severity, including increased weight loss, morbidity, and enhanced airway re
68 stature, black race, age 55 years or older, increased weight, rapid pulse, and smoking history (< or
70 -fast blood glucose normally associated with increased weight, suggesting a role for resistin in medi
72 ethoxy-4-iodophenyl-2-aminopropane), further increased weight-supported stepping in transplant rats.
73 rease of energy intake needed to sustain the increased weight (the maintenance energy gap) has amount
75 ce estimates using Gray's k-sample P values, increased weight was ascribed to the earlier data becaus
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