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1 2 mRNA rhythms were delayed by 0.97 +/- 0.29 hr (p < 0.01), indicating that human molecular clocks may be regulated by fe
2  Bouin's solution (18/18) than in buffered formalin (0/18), indicating that Bouin's solution is more appropriate for dete
3 aximum gape also scales with positive allometry (SL(1.20)), indicating that larger neonates are capable of greater jaw ex
4 njections resulted in light-like phase delays and advances, indicating that the neurotransmitter of the compound eyes par
5 pertussis toxin as well as by wortmannin but not by AG1478, indicating that Gi/o and phosphoinositide 3-kinase (PI3K) med
6         The results well matched the pathological analysis, indicating that PCA-tdTHz is a quick, powerful, evolving tool
7 ntly eliminated the excessive ROS and suppressed autophagy, indicating that the increased generation of ROS was associate
8 ization dependent on MinD and the DivIVA-like protein Cdv3, indicating that two distinct pools of MinC are coordinated in
9 y demonstrates the highly volatile nature of the beta cell, indicating that acquiring and sustaining beta cell identity a
10 ly, the extracellular mouth of the ion pore remains closed, indicating that local movements of the transmembrane helices
11 expansion activity and elevated levels of MutSbeta complex, indicating that MutSbeta abundance drives expansions.
12 d in consistently inactive and frequently active compounds, indicating that the structural context in which PAINS occur m
13 r-deprived but not under food- or salt-depleted conditions, indicating that the hedonic value of water-evoked responses i
14 CTD, but the HIV-1 CA NTD cannot replace the HTLV-1 CA CTD, indicating that the HTLV-1 CA subdomains provide distinct con
15                             In this study, we describe data indicating that the yeast family members Ltc1 and Ltc3/4 func
16 ensitivity to the mTORC1 signaling pathway and cause death, indicating that persistent signaling supports senescent cell
17 opic MYCN partially reversed the effects of MDM2 depletion, indicating that MYCN is an important MDM2 target.
18  GS and innexin2 are increased following sleep deprivation, indicating that GS and innexin2 genes are dynamically linked
19 e present in DCIS before the emergence of invasive disease, indicating that the malignant nature of the DCIS is defined b
20  between our 'top-down' and combined 'bottom-up' estimates, indicating that large CH4 emissions from trees adapted to per
21                      In the present study, we show evidence indicating that SIDT1 and SIDT2 not only do not transport RNA
22 -dependent reduction in ICa was not mimicked by Gbetagamma, indicating that this effect was independent of Gbetagamma.
23 ent, but TCF-independent TSSs exhibited the same hierarchy, indicating that it reflects gene activation per se.
24 rapidly fragment when GTP hydrolysis of Sey1p is inhibited, indicating that network maintenance requires continuous membr
25 ed with neutralizing anti-IL-2 Ab or STAT5/JAK3 inhibitors, indicating that STAT5 signaling drives RICD in TEM cells.
26 tic change in concentrations during equipment installation, indicating that no sources of target compounds were introduce
27 ocated downstream of the motif show even lower MGPA levels, indicating that the GCN4/SKn1/RY motif is not the only elemen
28 ased at 9 d after therapy but was not seen macroscopically, indicating that SPECT/CT imaging might be more sensitive than
29  migration in Xenopus embryos in a Snail1-dependent manner, indicating that the mechanism of action for NA in cell migrat
30             We observed a high degree of parental mosaicism indicating that a large fraction of these de novo mutations o
31 ding to experimentally-induced stress in desiccated mosses, indicating that spectral imaging is an effective tool to moni
32 ealing after MI by activating reparative Ly6C(low) Mos/Mps, indicating that Ep3 receptor activation may be a promising th
33  within residues 44-65 against reconstituted HDL particles, indicating that these mAbs specifically recognize the epitope
34 derably compared to G1-phase, but were resumed in G2-phase, indicating that specific interactions need to be dissolved fo
35 ERK1/2) was blocked by the Src family kinase inhibitor PP2, indicating that the action of pilocarpine on endogenous M3R i
36 letal reorganization, resulted in reduced RVFV replication, indicating that this pathway is important for viral replicati
37 responses did not affect any of the CCK-8-evoked responses, indicating that rather than acting on the nerves within the p
38 te for the full expression of sensory-evoked NVC responses, indicating that ACh may alter the fidelity of hemodynamic sig
39 ose, the grik1-2 grik2-1 mutant was sensitive to high salt, indicating that GRIKs are also involved in salinity signallin
40          However, viruses lacking vp3 have abnormal shapes, indicating that the vp3 gene is important for virus structure
41 cid significantly reduced hamster ear sebaceous gland size, indicating that this pro-drug approach was critical to obtain
42 GDS and promotes proteasome-mediated degradation of SmgGDS, indicating that nucleolar sequestration of SmgGDS by UBF stab
43 )F]8 showed rapid brain uptake and high target specificity, indicating that [(18)F]8 is a promising PDE4B-preferring radi
44 ned optimization eliminates intermediate metastable states, indicating that flux is enhanced in molecular machines with f
45 ers a selective defect in POLA2-binding and PP stimulation, indicating that these activities are critical for the in vivo
46 al number of repeats varies under different types of stress indicating that natural variation in repeat numbers may optim
47 Htt is present largely in a nonaggregated form at synapses, indicating that cytosolic forms of the protein are likely to
48 toration of bladder integrity following chitosan treatment, indicating that chitosan can be used to effectively combat re
49 BRAF(D594A) mutation) triggers lung adenocarcinoma in vivo, indicating that BRAF-inactivating mutations are initiating ev
50 T cells, while decreasing the levels of Th17 cells in vivo, indicating that CCR2 regulates the immune response by modulat

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