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1 ry protein (FLIP) were all down-regulated by indole-3-carbinol.
2 mpounds ascorbigen (13.0 mumol/100 g FW) and indole-3-carbinol (4.52 mumol/100g FW) with their higher
3 l diet experiments show that the reaction of indole-3-carbinol, a breakdown product of indol-3-ylmeth
5 ethane (DIM) is a major digestive product of indole-3-carbinol, a potential anticancer component of c
7 nts showed that oviposition was increased by indole-3-carbinol and decreased by indole-3-acetonitrile
9 toxin, quercetin, flavone, chlorogenic acid, indole-3-carbinol, and rutin) and three insecticides (di
10 S breakdown products (indole-3-acetonitrile, indole-3-carbinol, ascorbigen and 3,3'-diindolylmethane
12 production is induced by herbivory, such as indole-3-carbinol, function not only to repel herbivores
16 Current chemical and biological interest in indole-3-carbinol (I3C) and its metabolites has resulted
17 ic MDA-MB-231 human breast cancer cells with indole-3-carbinol (I3C) directly inhibited the extracell
21 We tested the chemopreventive efficacy of indole-3-carbinol (I3C), a constituent of Brassica veget
28 established the cellular mechanism by which indole-3-carbinol (I3C), a promising anticancer phytoche
31 ked whether the antiestrogenic phytochemical indole-3-carbinol (I3C), found in cruciferous vegetables
33 vide several classes of compounds, including Indole-3-carbinol (I3C), may have chemopreventive activi
34 tables provides phytochemicals, particularly indole-3-carbinol (I3C), which may be responsible for th
37 e tested the ability of dietary AhR ligands (indole-3-carbinol [I3C] and 3,3'-diindolylmethane [DIM])
38 multaneous determination of sulforaphane and indole-3-carbinol in broccoli using UPLC-HRMS/MS is desc
39 w that OSU-A9 was 100 times more potent than indole-3-carbinol in suppressing the viability of Hep3B,
40 ients that inhibit carcinogenesis, including indole-3-carbinol, indole-3-carboxaldehyde, ferulic acid
41 iety of indole-containing chemicals, such as indole-3-carbinol, indolo[3, 2-b]carbazole, and UV photo
44 actor-kappaB (NF-kappaB), we postulated that indole-3-carbinol must mediate its activity through NF-k
45 ased protein interaction studies showed that indole-3-carbinol perturbs the auxin-dependent interacti
49 ely, re-addition of the plant-derived ligand indole-3-carbinol rescued the barrier deficiency even in
50 n machinery was suggested, as application of indole-3-carbinol rescues auxin-induced root phenotypes.
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