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1 00-1.45) compared with those who did not tan indoors.
2 skin cancer screening in individuals who tan indoors.
3 and polypropylene deposition plates deployed indoors.
4 oxyl and peroxy radicals (OH and HO2, "HOx") indoors.
5 ), compared with individuals who did not tan indoors.
6 of respondents were women who usually worked indoors.
7 and organic (57.2+/-3.2mug/kg) compared with indoor (40.2+/-3.1mug/kg) (P<0.001), which was perhaps r
8 because of both the amount of time we spend indoors (~90%) and the ability of buildings to positivel
9 f hyperglycemia based on different levels of indoor activity (<5.0, 5.0-6.9, and >/=7.0 hours/day) we
10 besity, and an inverse association of either indoor activity or sleeping time with the risk of hyperg
11 glycemia associated with different levels of indoor activity, outdoor activity, TV watching, and slee
16 st observation of multiday persistence of an indoor aerosol transformation linked to a kitchen degrea
17 The median PFOA intakes from residential indoor air (5.7 pg kg bw(-1) day(-1)) and personal air (
18 e-DBE-DBCH) were more frequently detected in indoor air (detection frequencies >60%), while less vola
19 etardants (LFRs) were measured in samples of indoor air (n = 35) and indoor dust (n = 77) from UK hom
20 des (FOSAs), were assessed in 61 residential indoor air and 15 personal air samples collected in Oslo
22 ganophosphate esters (OPEs) by clothing from indoor air and transfer via laundering to outdoors were
23 try of squalene contained within skin oil to indoor air chemistry, the surface reaction of squalene w
25 owed a significant concentration increase in indoor air concentrations during computer installation a
26 amples from farmworker households had higher indoor air concentrations of both pesticides than did sa
32 ese "clean" Petri dishes and foil squares to indoor air in two different residences for successively
34 t buildings (n = 37,185; 19% exposed), where indoor air PCB concentrations have been measured were cr
36 with skin aging, but no direct link between indoor air pollutants and skin aging manifestations has
38 community household socioeconomic position, indoor air pollution, previous tuberculosis among househ
39 ry fine particles and their contributions to indoor air quality deterioration were examined by collec
40 ke, an air-filter intervention that improved indoor air quality did not affect quality-of-life measur
42 elds, such as automobile, aerospace, safety, indoor air quality, environmental control, food, industr
47 and the median concentrations in residential indoor air were 2970, 10400, and 3120 pg m(-3) for 6:2,
49 bw(-1) day(-1) were obtained in residential indoor air, while 1.0, 0.10, 3.3, 0.33, 0.88, and 0.09 p
57 ude chronic asthma and exposure to perennial indoor allergens and asthma related to fungal infection.
58 ate climate zones and an important source of indoor allergens causing allergic asthma and rhinitis in
61 ntial solid fuel use contributes to degraded indoor and ambient air quality and may affect global sur
62 lance approach for quantifying exposure from indoor and ground-level urban and rural outdoor sources
67 ion-to-exposure archetypes range from global indoor and outdoor averages, via archetypal urban and in
71 widely recognized as an important source of indoor and outdoor particle and volatile organic compoun
72 ofile of human subjects engaged in prolonged indoor and outdoor physical activities, and to make a re
73 enriched relative to the local soil both in indoor and outdoor PM2.5 demonstrating their noncrustal
75 sure to fine particulate matter (PM2.5) from indoor and outdoor sources is a leading environmental co
78 venting malaria where vectors primarily bite indoors and late at night, but their effectiveness is un
79 h data loggers and mosquitoes were collected indoors and outdoors using Furvela tent traps and were i
80 variety of carboxylic acids were identified indoors and outdoors, including monoacids, diacids, hydr
81 m passive air samplers deployed outdoors and indoors and polypropylene deposition plates deployed ind
82 hours of the evening, before individuals are indoors and protected by bednets, followed by a return t
83 cream made from milk obtained from cows fed indoors, and by early or late grazing, in alpine rangela
84 d exposure indoors, the amount of time spent indoors ( approximately 95%), and the mode and duration
90 ysis is not likely an important fate for NO3 indoors, but NO2 photolysis could be an important source
98 e and two-storey buildings provided a cooler indoor climate than did traditional housing, two-story b
102 amma-GluCys in nonsmoking females exposed to indoor-combustion products in Xuanwei and Fuyuan, China.
105 AER (Tier 1), infiltration factors (Tier 2), indoor concentrations (Tier 3), personal exposure factor
106 In addition, some carboxylic acids measured indoors correlated to CO2 in daytime, suggesting that hu
108 wheezing, including airborne viruses, smoke, indoor dampness, cockroaches, and poor access to health
112 /electronic equipment that contained RDP and indoor dust collected on and around surfaces of this equ
116 e to the microbial and allergenic content of indoor dust is associated with adverse and protective he
118 3T3-L1 cells were exposed to extracts of indoor dust or individual SVOCs and assessed for triglyc
123 current state of science with respect to the indoor environment and its effects on the development an
124 ance for future research in the study of the indoor environment and on environmental interventions as
130 tion in national asthma guidelines to target indoor environmental exposures, most insurers generally
131 payment of the goods and services needed for indoor environmental interventions for their patients wi
136 ionizing, organic chemical concentrations in indoor environments resulting in delivered tissue doses
137 cts of cooking, heating, and wood burning in indoor environments, and, more recently, products genera
145 the biologic properties of indoor exposures, indoor exposure assessment, and exposure reduction techn
151 new insights into the biologic properties of indoor exposures, indoor exposure assessment, and exposu
154 h yet to be discovered about their impact on indoor health and the unique ecological dynamics within
155 Participants completed questionnaires on indoor heating and cooking practices for both their enro
158 Analytes included four insecticides sold for indoor home use during our study period (carbaryl, cyper
161 ought to evaluate the adipogenic activity of indoor house dust extracts and a suite of semivolatile o
162 ent tubes will be important to room-averaged indoor HOx levels due to the strong distance dependence
164 o charge the battery under both full sun and indoor illumination conditions, and the addition of the
168 ur results indicated that the level of Cr in indoor industrial dust was more than twice, compared to
170 Among high-risk inner-city children, higher indoor levels of pet or pest allergens in infancy were a
172 ed photon fluxes and distance dependences of indoor light sources including halogen, incandescent, an
175 al/annual) in the laboratory, in tanks in an indoor mesocosm facility, and in coastal mesocosms under
176 were close to unity providing evidence that indoor metal-bearing PM2.5 had predominantly outdoor ori
177 to characterize the microbial communities of indoor microcosms that were either exposed to each pesti
184 ids are ubiquitous in ambient air, yet their indoor occurrence and abundance are poorly characterized
185 ime for oxidation was the same as that in an indoor office with an MRO3 between 22 and 32 ppb, sugges
187 nclusions and Relevance: Individuals who tan indoors often exhibited a concurrent tendency to sunburn
189 as realized contact between a person and an indoor or outdoor environment that poses a risk of incre
191 e UNEP/SETAC Life Cycle Initiative a coupled indoor-outdoor emission-to-exposure framework to provide
192 pants ages 13 to 20 years matched on sex and indoor-outdoor location at the time of each index case p
193 and rural outdoor sources using an effective indoor-outdoor population intake fraction and a system o
195 eased in association with <800mug FA and any indoor pesticide exposure {adjusted odds ratio [OR]=2.5
196 d with low FA [OR=1.2 (95% CI: 0.7, 2.2)] or indoor pesticides [OR=1.7 (95% CI: 1.1, 2.8)] alone.
200 nitial examination group, we showed that the indoor PM2.5 exposure levels were positively associated
201 owed a 67% (95% CI: 50% to 77%) reduction in indoor PM2.5, but no change was observed with the improv
204 nic compounds (SVOCs), an important class of indoor pollutants that may have significant adverse effe
205 red PM2.5 exposure and questionnaire data of indoor pollution sources, we built a regression model to
208 Long-lasting insecticidal nets (LLINs) and indoor residual spraying (IRS) have contributed substant
210 s (LLINs) alone or LLINs in combination with indoor residual spraying (IRS) with a pyrethroid (deltam
211 ng-lasting insecticide treated nets (LLINs), indoor residual spraying (IRS), and artemisinin combinat
212 al, with seasonal malaria chemoprevention or indoor residual spraying added second depending on seaso
214 e points (95% CI 0.86-15.83) and coverage of indoor residual spraying increased by 6.63 percentage po
215 to its detrimental impact on the ecosystem, indoor residual spraying of DDT is still recommended for
216 Long-lasting insecticidal nets (LLINs) and indoor residual spraying of insecticide (IRS) are the pr
218 ed pregnant women in an area of Uganda where indoor residual spraying of insecticide had recently bee
219 IV-infected pregnant women in the setting of indoor residual spraying of insecticide, adding monthly
221 on of long-lasting insecticide-treated nets, indoor residual spraying, and focal mass drug administra
222 , gender, insecticide-treated net (ITN) use, indoor residual spraying, household wealth, and geograph
224 ol tools such as insecticide treated nets or indoor residual sprays target mosquitoes in human dwelli
229 d outdoor averages, via archetypal urban and indoor settings, to 3646 real-world cities in 16 paramet
230 oupled plasma-mass spectrometry, we captured indoor signatures of outdoor episodic emissions arising
231 a dynamic process that can serve as a major indoor source of biological particulate matter (bioPM).
233 fractions from residential and occupational indoor sources range from 470 ppm to 62000 ppm, mainly a
235 " (shared conversation) and "casual" (shared indoor space) social contacts in 16 Zambian communities
239 roduction system (organic vs. free range vs. indoor), supermarket and purchase date on the concentrat
243 ng individuals aged 18 to 34 years, frequent indoor tanners (>/=10 times in the past year) were more
244 justed model: 82.3% (95% CI, 77.9%-86.0%) of indoor tanners had at least 1 sunburn during the precedi
246 years, P < .001) and reported more frequent indoor tanning (median number of sessions, 100 vs 40, P
249 ongitudinal study of the association between indoor tanning and melanoma in a large cohort of Norwegi
250 d age- and sex-specific associations between indoor tanning and melanoma to determine if these trends
251 dence of a dose-response association between indoor tanning and risk of melanoma and supports the hyp
252 f indoor tanning and the association between indoor tanning and sunburn among US high school students
253 e needed to further reduce the prevalence of indoor tanning and sunburn and thus prevent future cases
255 To examine the trends in the prevalence of indoor tanning and the association between indoor tannin
256 years, women younger than 40 years initiated indoor tanning at a younger age (16 vs 25 years, P < .00
258 ents in the United States, the prevalence of indoor tanning decreased from 15.6% (95% CI, 13.7%-17.6%
260 were calculated for melanoma in relation to indoor tanning exposure for men and women by diagnosis o
261 bjective: To examine the association between indoor tanning frequency and behaviors related to skin c
268 risk and linear regression to examine age of indoor tanning initiation in relation to age at diagnosi
277 2015 among high school students nationwide, indoor tanning remains commonplace among certain subgrou
278 confidence intervals for the relationship of indoor tanning to melanoma risk and linear regression to
283 ted the associations of age at initiation of indoor tanning, duration of tanning-device use, and dose
286 and cooling is a strategy by which to expand indoor temperature setpoint range for large energy savin
287 ates reflect the effects of reduced exposure indoors, the amount of time spent indoors ( approximatel
288 humans spend approximately 90% of their time indoors, the built environment exposome merits particula
289 semivolatile organic compounds (SVOCs) from indoors to outdoors through accumulation from air and th
291 oncentrations of other insecticides sold for indoor use (carbaryl, cypermethrin, and permethrin) and
292 opoxur) and four that are no longer sold for indoor use including dichlorodiphenyltrichloroethylene (
293 mixed with new scenes and classified them as indoor versus outdoor (encoding task) or old versus new
295 en though this source clearly contributes to indoor volatile organic compounds (VOCs) and influences
296 nt concentrations in bars were modeled using indoor volumes, air exchange rates and the number of hou
297 18 to 60 years, those who frequently tanned indoors were more likely to rarely/never use sunscreen (
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