ライフサイエンスコーパス: induced hyperthermia

1 +/- 3%CVCmax) sites in response to exercise-induced hyperthermia.

2 n-linear scattering is optimal for NIR laser-induced hyperthermia.

3 ehavior suggests a role for movement in METH-induced hyperthermia.

4 anandamide metabolism, did not alter cocaine-induced hyperthermia.

5 ic-adrenal-pituitary axis activity or stress-induced hyperthermia.

6 he potential morbidity and mortality of drug-induced hyperthermia.

7 ) significantly attenuated the deltorphin-II-induced hyperthermia.

8 DA receptor activation maximizes mu receptor-induced hyperthermia.

9 njection) with a time course similar to PGE2-induced hyperthermia.

10 physiological antagonist to reduce morphine-induced hyperthermia.

11 ranslationally, inhibits IL-1beta and PGE(2)-induced hyperthermia.

12 ere studied under conditions conducive to MA-induced hyperthermia.

13 single alpha-subtype had no effect on stress-induced hyperthermia.

14 Lactulose had no effect on METH-induced hyperthermia.

15 (9 of 21 died) vs. asphyxiated rats without induced hyperthermia (3 of 21) or with hyperthermia indu

16 =+30 s after morphine) can decrease morphine-induced hyperthermia; (3) neither the opioid receptor an

17 4 hrs vs. rats subjected to asphyxia without induced hyperthermia (33 +/- 13 vs. 67 +/- 36; p=.002).

18 ed to: no temperature manipulation (n = 21), induced hyperthermia (40 +/- 0.5 degrees C) for 3 hrs be

19 differ from that in rats asphyxiated without induced hyperthermia (59 +/- 21 vs. 67 +/- 36; p=.885).

20 iffer from those in rats asphyxiated without induced hyperthermia (6.4 +/- 3.0 vs. 6.2 +/- 2.6; p=.90

21 ores than rats subjected to asphyxia without induced hyperthermia (9.3 +/- 1.5 vs. 6.2 +/- 2.6; p=.00

22 ided significant protection against the METH-induced hyperthermia and depletion of DA, DOPAC and HVA.

23 bogaine can completely block methamphetamine-induced hyperthermia and HSP-72 expression in the striat

24 erapies for the treatment of psychostimulant-induced hyperthermia and its sequelae.

25 eview the current literature describing drug-induced hyperthermia and its treatment.

26 ngs indicate that UCP-3 is important in MDMA-induced hyperthermia and point to a new therapeutic dire

27 dicate that NMDA receptors modulate morphine-induced hyperthermia and suggest that increases in gluta

28 tors may contribute to the mediation of MDMA-induced hyperthermia and that drugs targeting these rece

29 As previously reported, METH-induced hyperthermia and the subsequent loss of striatal

30 reduced acoustic startle, attenuated stress induced hyperthermia, and a blunted increase in startle

31 was assessed using acoustic startle, stress-induced hyperthermia, and a challenge with the anxiogeni

32 1 and 3 (UCPDK) fail to show methamphetamine-induced hyperthermia, and have a markedly accelerated lo

33 nnabinoid CB1 receptors to attenuate cocaine-induced hyperthermia, and that dopamine D2 receptor acti

34 METH showed a temporal relationship to METH-induced hyperthermia; and 2.) describe the temporal patt

35 tality rate in asphyxiated rats treated with induced hyperthermia at 24 hrs (9 of 21 died) vs. asphyx

36 cal procedures except asphyxia) treated with induced hyperthermia at 24 hrs (n = 4) or 48 hrs (n = 4)

37 ) for 3 hrs beginning at 24 hrs (n = 21), or induced hyperthermia at 48 hrs (n = 10).

38 investigated which EP receptors mediate PGE2-induced hyperthermia by injecting selective EP receptor

39 l was modestly effective in attenuating MDMA-induced hyperthermia by moderately inhibiting skin vasoc

40 ontrast to the powerful potentiation of MDMA-induced hyperthermia by social interaction and warm ambi

41 D-Fen-induced hyperthermia does not appear to cause generalize

42 Methamphetamine-induced hyperthermia during the 6 h immediately after tr

43 ring core body temperature and preventing MA-induced hyperthermia from exceeding 41.5 degrees C.

44 D-Fen-induced hyperthermia has been shown to coincide with an

45 urrent strategies to treat pathological MDMA-induced hyperthermia in humans are palliative and margin

46 ter maternal separation, to attenuate stress-induced hyperthermia in mice, to increase punished drink

47 circadian rise in Tc or psychological stress-induced hyperthermia in mice.

48 alterations in MDMA pharmacokinetics or MDMA-induced hyperthermia in rats previously exposed to MDMA

49 MDMA-induced hyperthermia is highly variable, unpredictable,

50 However, because cocaine-induced hyperthermia occurs primarily in hot weather, it

51 nd 2.) describe the temporal pattern of METH-induced hyperthermia over an extended dose range.

52 ore temperature increases; the onset of METH-induced hyperthermia ranged from 45 min post-treatment t

53 agents is increasing, the treatment of drug-induced hyperthermia remains unchanged and continues to

54 Acute noradrenaline-induced hyperthermia requires UCP1 but not UCP3.

55 administration, thus indicating that AMG0347-induced hyperthermia results from blockade of tonic TRPV

56 econstruction assistance following microwave-induced hyperthermia to remove the bone tumor.

57 gh fluence rate gave a contribution of laser-induced hyperthermia to the photodamage.

58 uced a marked and immediate reversal of MDMA-induced hyperthermia via inhibition of brain metabolic a

59 The CCK-8 (300 ng)-induced hyperthermia was blocked by pretreatment of rats

60 AMG0347-induced hyperthermia was brought about by one or both of

61 In contrast, methamphetamine-induced hyperthermia was essentially identical in the tw

62 Morphine-induced hyperthermia was first detected at P60 and peake

63 Induced hyperthermia when administered at 24 hrs, but no