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1 ion into the effector subsets Th1, Th17, and induced regulatory T cells.
2 ed methylation of FOXP3 CpG sites in antigen-induced regulatory T cells.
3 d to allow conversion of CD4(+) T cells into induced regulatory T cells.
4 s to the retinoic acid expansion of TGF-beta-induced regulatory T cells.
5 t Th cell lineages, including Th17, Th9, and induced regulatory T cells.
6 he pro-inflammatory phenotype of Aspergillus-induced regulatory T-cells.
7 tes the differentiation of anti-inflammatory induced regulatory T cells and proinflammatory Th1 cells
8 ligation of OX40 inhibited the production of induced regulatory T cells and the T(H)17 subset of help
9 nversion of conventional CD4(+) T cells into induced regulatory T cells and with the expression of IF
10 nversely, liver DCs with low levels of lipid induced regulatory T cells, anergy to cancer, and oral t
13 nsion of P. gingivalis-specific Th cells and induced regulatory T cells does not depend on mucosal LC
14 d antigen specificity of both endogenous and induced regulatory T cells during these interactions.
15 omotes the differentiation of Th1, Th17, and induced regulatory T cells, implying that under certain
21 rated that galectin-9 is highly expressed by induced regulatory T cells (iTreg) and was crucial for t
25 roperties and therapeutic potential of these induced regulatory T cells (iTreg), we examined their im
27 the feasibility and efficacy of using murine-induced regulatory T cells (iTregs) for the induction of
28 ressed TIM-4 leads to increased induction of induced regulatory T cells (iTregs) from naive CD4(+) T
29 l (in this issue) reporting an enrichment in induced regulatory T cells (iTregs) in normal pregnancy
30 promote differentiation of Foxp3-expressing induced regulatory T cells (iTregs), high levels of Ag h
32 early therapeutic group, the frequencies of induced regulatory T cells (iTregs: CD4(+)CD25(-)Fopx3(+
36 ells) and discrimination between natural and induced regulatory T cells (nT(reg) and iT(reg) cells).
37 t naive T cells can also differentiate into 'induced' regulatory T cells or inflammatory T cells that
38 d not upregulate costimulatory molecules and induced regulatory T cells rather than effector T cells.
40 matinib therapy activated CD8(+) T cells and induced regulatory T cell (T(reg) cell) apoptosis within
44 In addition to early developmental links to induced regulatory T cells (Tregs) reflected in the shar
46 d box protein 3 (FOXP3) CpG sites in antigen-induced regulatory T cells were significantly different
47 cells: TGF-beta induces Foxp3 and generates induced regulatory T cells, whereas IL-6 inhibits TGF-be
48 in CD103(+) DCs inhibited the generation of induced regulatory T cells while promoting Th1 cell deve
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