ライフサイエンスコーパス: inducing factor

1 ondrial intermembrane protein AIF (apoptosis-inducing factor).

2 tress and mitochondrial release of apoptosis-inducing factor.

3 of Toll-like receptor-associated interferon-inducing factor.

4 , to mature caspase-8, which is an apoptosis-inducing factor.

5 the ability to respond to the male soporific-inducing factor.

6 ic apoptosis through caspase-3 and apoptosis-inducing factor.

7 lled and released cytochrome c and apoptosis-inducing factor.

8 ulation of the proapoptotic factor apoptosis inducing factor.

9 n, controls production of a diffusible heart-inducing factor.

10 ic protein with some similarity to apoptosis-inducing factor.

11 eceptor-interacting protein 1, and apoptosis-inducing factor.

12 hondrial proteins cytochrome c and apoptosis-inducing factor.

13 mac/DIABLO, but not the release of apoptosis-inducing factor.

14 tein was termed S. mansoni-derived apoptosis-inducing factor.

15 riosteal cells release a soluble cholinergic inducing factor.

16 /TRX, originally described as an IL-2R alpha-inducing factor.

17 release of cytochrome c (Cyt-c) or apoptosis-inducing factor.

18 hondrial proteins cytochrome c and apoptosis-inducing factor.

19 ptotic pathway associated with the apoptosis-inducing factor.

20 e it also coincided with increased apoptosis-inducing factor.

21 of mitochondrial cytochrome c and apoptosis-inducing factor.

22 for the production of endoderm-derived heart-inducing factors.

23 rasites and reversed the induction of anergy-inducing factors.

24 rounds of stimulation in the presence of Th1-inducing factors.

25 termediates (ROI), and release of cell death-inducing factors.

26 on requires cooperation with differentiation-inducing factors.

27 ntified in studies to reveal cell senescence-inducing factors.

28 h control macrophages or exogenous IFN-gamma-inducing factors.

29 Zea mays) root exudate or purified haustoria-inducing factors.

30 expression of miR-200 in the absence of EMT-inducing factors.

31 fect of HDLs on beta-cells against ER stress-inducing factors.

32 Differentiation-inducing factor 1 (DIF-1) is a polyketide-derived morpho

33 to the prestalk cell inducer Differentiation inducing factor 1 (DIF-1), or are subjected to hyper-osm

34 ette-inducing small molecule, dubbed rosette-inducing factor 1 (RIF-1), produced by the Gram-negative

35 re the release of cytochrome C and apoptotic inducing factor-1 (AIF1) by Western blotting.

36 Differentiation-inducing factor-1 (DIF-1) is a polyketide that induces D

37 DNA arrays were used to identify the hypoxia-inducing factor-1 (HIF-1) as a downstream target of TGF-

38 looxygenase-1 (COX-1) and COX-2, and hypoxia-inducing factor-1 alpha (HIF-1 alpha) and vascular endot

39 of several stress pathways, such as hypoxia-inducing factor-1-alpha, receptor-interacting protein 1,

40 cancer specimens and correlates with hypoxia-inducing factor 1alpha (HIF1alpha) expression.

41 Hypoxia-inducing factor 1alpha (HIF1alpha) is known to be hypera

42 Glycolysis enhancement required hypoxia-inducing factor-1alpha to up-regulate glucose transporte

43 A well-characterized inducing factor, 2, 6-dimethoxy-p-benzoquinone (DMBQ), c

44 rocess involving secretion of the cell death-inducing factor 24p3 by mouse leukemia cells, raising th

45 nsity, release of cytochrome C and apoptosis inducing factor, (4) chromatin condensation, nuclear lam

46 derived activator of caspases, and apoptosis-inducing factor, accompanied by a proteolytic cascade wi

47 survival factors or overexpression of death-inducing factors accounted for the phenotypes observed.

48 sed on our initial finding that the mesoderm-inducing factor activin A is suppressed by DeltaNp63 in

49 sue explants (animal caps) with the mesoderm-inducing factor activin induces tissue elongation and de

50 ECs released B cell-activating AID-inducing factors after sensing microbial products throug

51 Apoptosis-inducing factor (AIF) and AMID (AIF-homologous mitochond

52 ted Bcl-2, resulting in release of apoptosis-inducing factor (AIF) and cytochrome c from mitochondria

53 e of proapoptotic factors, such as apoptosis-inducing factor (AIF) and cytochrome c.

54 dependent mitochondrial release of apoptosis-inducing factor (AIF) and cytochrome complex (Cyt c) is

55 ax and Bak were enhanced, and both apoptosis-inducing factor (AIF) and endonuclease G (Endo G) were r

56 ation of the proapoptotic factors, apoptosis-inducing factor (AIF) and endonuclease G (EndoG), throug

57 bserved a nuclear translocation of apoptosis-inducing factor (AIF) and endonuclease G in CNGA3(-/-)/N

58 h involve release of mitochondrial apoptosis-inducing factor (AIF) and its translocation to the nucle

59 cellular activities, we identified apoptosis-inducing factor (AIF) as an XIAP binding protein.

60 We further show that apoptosis-inducing factor (AIF) cooperated with Bnip3 to promote l

61 nylate kinase-2, cytochrome c, and apoptosis-inducing factor (AIF) during apoptosis and compared the

62 lease and nuclear translocation of apoptotis-inducing factor (AIF) followed by irreversible caspase-i

63 he release of cytochrome c and the apoptosis-inducing factor (AIF) from mitochondria in HL-60 and HL-

64 The translocation of apoptosis-inducing factor (AIF) from mitochondria to the nucleus h

65 Release of cytochrome c and apoptosis-inducing factor (AIF) from mitochondria was observed in

66 sociated with the translocation of apoptosis-inducing factor (AIF) from the cytoplasm to nuclei.

67 ctivation and the translocation of apoptosis-inducing factor (AIF) from the mitochondria to the nucle

68 n is required for translocation of apoptosis-inducing factor (AIF) from the mitochondria to the nucle

69 ich was followed by the release of apoptosis-inducing factor (AIF) from the mitochondria, large-scale

70 ion as a proviral insertion in the apoptosis-inducing factor (Aif) gene, causing about an 80% reducti

71 duced the nuclear translocation of apoptosis-inducing factor (AIF) in A2058 and SKMEL5 cells, and the

72 on in the mitochondrial release of apoptosis-inducing factor (AIF) in cisplatin-treated renal tubular

73 ized by activation of calpains and apoptosis-inducing factor (Aif) in dying photoreceptors.

74 The role of apoptosis inducing factor (AIF) in promoting cell death versus sur

75 ndrial release of cytochrome c and apoptosis-inducing factor (AIF) in the penumbra region were reduce

76 Apoptosis-inducing factor (AIF) is a bifunctional mitochondrial fl

77 Mitochondrial apoptosis-inducing factor (AIF) is a central player in the caspase

78 Apoptosis-inducing factor (AIF) is a mitochondrial flavoprotein th

79 Apoptosis-inducing factor (Aif) is a mitochondrial flavoprotein th

80 Apoptosis-inducing factor (AIF) is a mitochondrial flavoprotein wi

81 Apoptosis-inducing factor (AIF) is an evolutionarily conserved, ub

82 We report here that apoptosis-inducing factor (AIF) mediates PARP-1-dependent glutamat

83 93Val) in AIFM1, the gene encoding apoptosis-inducing factor (AIF) mitochondrion-associated 1.

84 tivation, and cleavage, as well as apoptosis-inducing factor (AIF) nuclear translocation and executio

85 recently discovered mitochondrial apoptosis-inducing factor (AIF) on activation is translocated to t

86 Apoptosis-inducing factor (AIF) promotes cell death yet also contr

87 dependent cell death, triggered by apoptosis-inducing factor (AIF) release from mitochondria and its

88 nction suppressed cell killing and apoptosis-inducing factor (AIF) release from mitochondria.

89 sm of cisplatin-induced apoptosis, apoptosis-inducing factor (AIF) release into the cytosol was obser

90 tor Bax in mitochondria, while the apoptosis-inducing factor (AIF) remains unchanged.

91 nsity and nuclear translocation of apoptosis-inducing factor (AIF) suggesting caspase-independent cel

92 hat the intramitochondrial protein apoptosis-inducing factor (AIF) translocates to the nucleus and pr

93 ndent neuronal death that involves apoptosis-inducing factor (AIF) translocation from mitochondria to

94 through a unique pathway involving apoptosis-inducing factor (AIF) translocation into the nucleus.

95 d with the mitochondrial localized apoptosis inducing factor (AIF) under both normal and oxidant stre

96 Induction in apoptosis inducing factor (AIF) was observed, suggesting a paralle

97 Apoptosis-inducing factor (AIF) was originally discovered as a mit

98 Cytosolic cytochrome c and nuclear apoptosis-inducing factor (AIF) were increased 3 h after OGD, and

99 uction increased the activation of apoptosis-inducing factor (AIF), a caspase-independent cell death

100 Apoptosis-inducing factor (AIF), a mitochondrial oxidoreductase, i

101 Apoptosis-inducing factor (AIF), a mitochondrial oxidoreductase, i

102 sitive for nuclear localization of apoptosis-inducing factor (AIF), an early event in apoptosis.

103 d AIFM1 gene encodes mitochondrial apoptosis-inducing factor (AIF), an FAD-containing and NADH-specif

104 rylation, as well as cytochrome c, apoptosis-inducing factor (AIF), and endonuclease G (EndoG) releas

105 tivation, nuclear translocation of apoptosis-inducing factor (AIF), and induction of p53, and prevent

106 nd modified caspase-3, Bcl-2, Bad, apoptosis-inducing factor (AIF), and PARP were quantified by immun

107 apoptogenic factors cytochrome c, apoptosis-inducing factor (AIF), and proinflammatory high-mobility

108 of cytochrome c, Smac/DIABLO, and apoptosis-inducing factor (AIF), but not endonuclease G.

109 etion and mitochondrial release of apoptosis-inducing factor (AIF), but the causal relationships betw

110 ction in the mitochondrial protein apoptosis-inducing factor (AIF), exhibited signs of oxidative stre

111 of caspase-independent apoptosis, apoptosis-inducing factor (AIF), from mitochondria is induced by H

112 ear translocation of mitochondrial apoptosis-inducing factor (AIF), known to trigger both apoptotic m

113 ial release of cytochrome c, Smac, apoptosis-inducing factor (AIF), or loss of mitochondrial membrane

114 iators, including cytochrome c and apoptosis-inducing factor (AIF), was studied in the absence and pr

115 zed a human gene homologous to the apoptosis-inducing factor (AIF), which is named AIF-like (AIFL).

116 X, and release of cytochrome c and apoptosis-inducing factor (AIF), which was translocated to the nuc

117 AMID is an apoptosis-inducing factor (AIF)-homologous and mitochondria-associ

118 ential, reduced levels of ATP, and apoptosis-inducing factor (AIF)-induced apoptosis.

119 Although hsp70 antagonizes apoptosis-inducing factor (AIF)-mediated cell death, the relative

120 type of neuronal PCD involving the apoptosis-inducing factor (AIF).

121 e-independent mechanism, involving apoptosis-inducing factor (AIF).

122 defective nuclear translocation of apoptosis-inducing factor (AIF).

123 rmined by nuclear translocation of apoptosis-inducing factor (AIF).

124 ding cytochrome c, Smac/DIABLO and apoptosis-inducing factor (AIF).

125 but not endonuclease G (EndoG) and apoptosis-inducing factor (AIF).

126 in the release of cytochrome c and apoptosis-inducing factor (AIF).

127 ition and nuclear translocation of apoptosis-inducing factor (AIF).

128 ates in the nucleus, together with apoptosis-inducing factor (AIF).

129 DP-ribose) polymerase (PARP-1) and apoptosis-inducing factor (AIF).

130 dentified mitochondrion-associated apoptosis inducing factor (AIFM1) have roles in the induction of a

131 pathway involving translocation of apoptosis-inducing factor and caspase 12 to the nucleus.

132 expression of apoptotic proteins (apoptosis-inducing factor and cleaved caspase-3) and autophagy pro

133 rleukin (IL)-18 is an interferon (IFN)-gamma-inducing factor and contributes to the Th1 immune respon

134 rial depolarization and release of apoptosis-inducing factor and cytochrome c Furthermore, this prote

135 in both cell types, but release of apoptosis-inducing factor and endonuclease G was detected only in

136 ed from the mitochondrion, such as apoptosis-inducing factor and endonuclease G, may induce caspase-9

137 CIMD also depends on apoptosis-inducing factor and endonuclease G, which are effectors

138 ribofuranosylbenzimidazole (DRB) sensitivity-inducing factor and found that the IC(50) determined was

139 ented intranuclear localization of apoptosis-inducing factor and protected neurons from excitotoxic i

140 ssing the mitochondrial release of apoptosis-inducing factor and Smac/DIABLO.

141 further examined the association of Th1/Th2 inducing factors and allergic disease and intestinal inf

142 of the antagonistic roles played by secreted inducing factors and their soluble inhibitory binding pr

143 under inflammatory conditions; however, the inducing factors and underlying mechanisms remain unknow

144 mitochondrial caspase-independent (apoptosis-inducing factor) and caspase-dependent (Smac/Diablo and

145 in (IL)-1beta, IL-18 (interferon (IFN)-gamma inducing factor) and IFN-gamma, but not tumour-necrosis

146 g., cytochrome c, Smac/DIABLO, and apoptosis-inducing factor), and caspase activation.

147 irectly the release of Cyt c, AIF (apoptosis-inducing factor), and Smac (second mitochondria-derived

148 elongation factor) and DSIF (DRB sensitivity-inducing factor)--and P-TEFb (positive elongation factor

149 ndria, release of cytochrome c and apoptosis-inducing factor, and activation of caspase-9 and caspase

150 translocation of cytochrome c and apoptosis inducing factor, and active caspases 3 and 7, consistent

151 release of mitochondrial proteins, apoptosis-inducing factor, and cytochrome c.

152 itochondrial proapoptotic factors, apoptosis inducing factor, and endonuclease G.

153 osolic release of cytochrome c and apoptosis-inducing factor, and mitochondrial membrane potential ch

154 t stress, mitochondrial release of apoptosis inducing factor, and nuclear DNA fragmentation resulting

155 respiration, prevented release of apoptosis-inducing factor, and reduced neuronal cell death trigger

156 e leakage of both cytochrome c and apoptosis-inducing factor, and significantly improved cell surviva

157 ded MHC class II molecules, interferon-gamma inducing factor, apolipoprotein E, and cathepsin S.

158 In this study, we show that the CCR8-inducing factors are heat stable and protease resistant

159 illustrates the complex regulation to which inducing factors are subject.

160 and the pausing factor DSIF (DRB sensitivity-inducing factor) are still present at the hsp70 loci in

161 CCK-induced caspase 3 activation, apoptosis-inducing factor, as well as X-linked inhibitor of apopto

162 ation and mitochondrial release of apoptosis-inducing factor at low microM concentrations.

163 Total and modified apoptosis-inducing factor, Bcl-2 family proteins, phospho-PKC-alph

164 otic cells and expression of total apoptosis-inducing factor, Bcl-2, Bak, and Bax in the pre-CP/Rep a

165 One model for NMD suggests that decay-inducing factors bound to mRNAs during early processing

166 NGF was found to be a proliferation-inducing factor but not a survival factor.

167 variation in the recognition of a haustorial-inducing factor by a parasitic member of the Scrophulari

168 varian cancer, we show that "helper" NK cell-inducing factors can be used to enhance local production

169 d key apoptotic mediators, such as apoptosis-inducing factor, caspase 3, caspase 8, caspase 9, poly(a

170 Bcl-2 and increased expression of apoptosis-inducing factor, caspase-3, and cleavage of BID, c-IAP-1

171 mediated by the enhanced induction of anergy-inducing factors cbl-b, c-cbl (cbl is abbreviation for C

172 FLP represses the expression of the mitosis-inducing factor CDKB1;1, which, along with CDKB1;2, is s

173 tic organs develop in response to haustorial-inducing factors contained in host root exudates.

174 ial meningitis and induces a novel apoptosis-inducing factor-dependent (AIF-dependent) form of brain

175 ath proceeded predominately via an apoptosis-inducing factor-dependent pathway in XY neurons versus a

176 sion is induced by the stalk differentiation-inducing factor DIF-1 and is restricted to a subset of p

177 osed to the prestalk inducer differentiation inducing factor (DIF-1), a chlorinated hexaphenone.

178 DRB sensitivity-inducing factor (DSIF or Spt4/5) is a conserved transcri

179 eta-d-ribofuranosylbenzimidazole sensitivity-inducing factor (DSIF) and negative elongation factor (N

180 eta-D-ribofuranosylbenzimidazole sensitivity-inducing factor (DSIF) are involved in pausing RNA Polym

181 hloro-1-beta-D-ribobenzimidazole-sensitivity inducing factor (DSIF).

182 reversed by the presence of DRB sensitivity-inducing factor (DSIF).

183 ribofuranosylbenzimidazole (DRB) sensitivity-inducing factor (DSIF).

184 ribofuranosylbenzimidazole (DRB) sensitivity inducing factors (DSIF) and the negative elongation fact

185 by the retinoid in cells exposed to the EMT-inducing factors EGF and heregulin-beta1.

186 her mitochondrial related factors (apoptosis-inducing factor, endonuclease G, and HtrA2/Omi).

187 itochondria, and cytochrome c, and apoptosis-inducing factor escaped from mitochondria to the cytopla

188 or Toll-like receptor-associated interferon-inducing factor expression does not affect the LPS-trigg

189 rental cells, whereas knockdown of apoptosis-inducing factor expression suppressed lapatinib toxicity

190 pase inhibitor and by knockdown of apoptosis-inducing factor expression.

191 itioned medium (MG63CM) contains a migration-inducing factor for MCF-7 cells.

192 brane permeability, and release of apoptosis-inducing factor from mitochondria are partially blocked

193 AD(+) blocked translocation of the apoptosis-inducing factor from mitochondria to nuclei, a step prev

194 phate levels, and translocation of apoptosis-inducing factor from mitochondria to the nucleus, result

195 ing cytochrome c and translocating apoptosis inducing factor from mitochondria to the nucleus.

196 of cytochrome c, Smac/DIABLO, and apoptosis inducing factor from mitochondria, and reduced mitochond

197 before release of cytochrome c and apoptosis-inducing factor from mitochondria.

198 nd the release of cytochrome c and apoptosis inducing factor from mitochondria.

199 hondrial mu-calpain and release of apoptosis-inducing factor from the mitochondrial intermembrane spa

200 sary for the complete discharge of apoptosis-inducing factor from the mitochondrial intermembrane spa

201 Isolation of CD1-inducing factors from M. tuberculosis using normal phase

202 sion of caspase 4, cathepsin B, or apoptosis-inducing factor function significantly suppressed cell k

203 ced immunotherapeutic efficacy; (c) the MDSC-inducing factors G-CSF and GM-CSF facilitated IRF-8 down

204 e feedback loop with Dronc and the apoptosis-inducing factors Hid and Reaper.

205 Loss of the C. elegans hypoxia-inducing factor HIF-1, which regulates iron homeostasis,

206 inase (PDK1) and EGFR along with the hypoxia-inducing factor (HIF)-1alpha in human glioblastoma multi

207 are also induced by the exudate (hormogonium-inducing factor [HIF]) of a symbiotic plant partner.

208 es of T. versicolor roots to these haustoria inducing factors (HIFs) included localized swelling and

209 AMID (apoptosis-inducing factor-homologous mitochondrion-associated indu

210 The equine herpesvirus 1 (EHV-1) alpha-trans-inducing factor homologue (ETIF; VP16-E) is a 60-kDa vir

211 We investigated the effects of IFN-gamma-inducing factor (IL-18) in a ragweed (RW) mouse model of

212 tes recruitment of Smac/Diablo and apoptosis-inducing factor in chronic neurodegeneration.

213 dependent nuclear translocation of apoptosis-inducing factor in NMDA-treated neurons and reduced tPA-

214 hanism independent of caspase- and apoptosis-inducing factor in nonproliferating U937 cells.

215 is the major cyclooxygenase-2 mRNA and PGE2-inducing factor in pulmonary edema fluid and accounts fo

216 failed to release cytochrome c or apoptosis-inducing factor in response to recombinant Bax or trunca

217 ls did not release cytochrome c or apoptosis-inducing factor in response to recombinant Bax or trunca

218 t an additional function of EHD1 as a tubule-inducing factor in the Arf6 pathway for recycling of pla

219 ble protein 3alpha was the most active mucin-inducing factor in the RSV-infected human small airway e

220 f interleukin-6-like factor and nitric oxide-inducing factor in vitro.

221 One candidate for an endogenous mesoderm-inducing factor in Xenopus is derriere, a member of the

222 growth by down-regulating other angiogenesis-inducing factors in addition to VEGF and that the centra

223 Heart-inducing factors in amphibians are not known, and althou

224 is because of the presence of early obesity-inducing factors in the milk of obese F1 dams.

225 omous manner, indicating a role for secreted inducing factors in the response to FoxD3.

226 Both types of endoderm express cardiac-inducing factors, including WNT antagonists Dkk1 and Sfr

227 1) peptide has been reported as an autophagy-inducing factor inhibiting the replication of pathogens

228 resulting in release of the interferon gamma inducing factor interleukin-18 (IL-18).

229 transfer experiments reveal the PS30-derived inducing factor is soluble and promotes mitogenic ERK an

230 and Toll-like receptor-associated interferon-inducing factor, is blocked by both FADD and phosphatidy

231 eling and in cytosolic and nuclear apoptosis-inducing factor labeling within 60 min.

232 oresis, suggesting a predominantly apoptosis-inducing factor-mediated cell death process.

233 ormation on whether Smac/Diablo or apoptosis-inducing factor might play a role in chronic neurodegene

234 ndrial release of cytochrome c and apoptosis-inducing factor, mitochondrial membrane depolarization,

235 ) for the phosphorylation of DRB sensitivity-inducing factor, negative elongation factor, and C-termi

236 Accordingly, ablation of the primary pause-inducing factor NELF does not increase expression of lin

237 ss of pausing through knockdown of the pause-inducing factor NELF leads to broadly attenuated immune

238 e earliest identified BMP antagonists/neural-inducing factors, noggin and chordin, were expressed in

239 se) polymerase) cleavage, and AIF (apoptosis-inducing factor) nuclear translocation.

240 and efflux rate modulation as the strongest inducing factor of chronic inflammation for a wide range

241 e the activity of Nodal, a secreted mesoderm-inducing factor of the transforming growth factor-beta (

242 The existence of an ovulation-inducing factor (OIF) in seminal plasma has broad implic

243 be maintained by continued expression of the inducing factor or by a mechanism that confers a stable

244 enotype after in vitro treatment with neural-inducing factors or after delivery into the brain.

245 induction of autophagy, release of apoptosis-inducing factor, or opening of the mitochondrial permeab

246 the elongation factors DSIF (DRB Sensitivity-Inducing Factor), P-TEFb (Positive Transcription Elongat

247 ng RNA to reduce the expression of apoptosis-inducing factor partially inhibited CDDO-induced apoptos

248 o occur by a caspase 3-independent apoptosis-inducing factor pathway.

249 f biphenyl dioxygenase (BphA4) and apoptosis-inducing factor, Pdr lacks one of the arginine residues

250 ep, the amount of apoptotic cells, apoptosis-inducing factor, phospho-Bad, phospho-PKC-alpha, phospho

251 ukin (IL) -1beta, TNF-alpha, and proteolysis-inducing factor (PIF) by cancer cells used in this study

252 A novel protein, proteolysis-inducing factor (PIF), has been isolated from the urine

253 d delta-toxin as the mast cell degranulation-inducing factor produced by S. aureus.

254 ells to determine quantitatively if mesoderm-inducing factors promote hematopoietic lineage developme

255 es transcriptionally regulated by haustorium-inducing factors provides a framework for dissecting gen

256 the c18 NC1 domain functioned as a motility-inducing factor regulating the extracellular matrix (ECM

257 ollagen XVIII (NC1) functioned as a motility-inducing factor regulating the extracellular matrix-depe

258 y transition, and cytochrome C and apoptosis-inducing factor release from isolated mitochondria.

259 x translocation, cytochrome c, and apoptosis-inducing factor release) and apoptosis by imatinib mesyl

260 tivation) and caspase-independent (apoptosis-inducing factor release) pathways, and limited neuronal

261 g., cytochrome c, Smac/DIABLO, and apoptosis-inducing factor release), caspase activation, and apopto

262 P binding protein with low pI, and apoptosis-inducing factor release), caspase activation, poly(ADP-r

263 rial dysfunction (cytochrome c and apoptosis-inducing factor release), caspase-3 and -8 activation, a

264 rial dysfunction (cytochrome c and apoptosis-inducing factor release), caspase-3 and -8 activation, a

265 ificantly reduced cytochrome c and apoptosis-inducing factor release, loss of mitochondrial membrane

266 ed by the mitochondrial release of apoptosis-inducing factor, resulting in caspase-independent cell d

267 ng the release of cytochrome c and apoptosis-inducing factor, resulting in DNA fragmentation.

268 in some chicken embryos and named resistance-inducing factor (RIF) because it interferes with RSV.

269 Here we show that a rosette inducing factor (RIF-1) produced by A. machipongonensis

270 ergizes with activating sulfonolipid rosette-inducing factors (RIFs) to recapitulate the full bioacti

271 The keratinocyte-derived CCR8-inducing factor(s) were soluble, and independent of vita

272 These exosomes increased chemoresistance-inducing factor, Snail, in recipient epithelial cells an

273 cells were hypersensitive to various stress-inducing factors, such as salts, SDS, and H(2)O(2), espe

274 The presence of cleaved apoptosis inducing factor (tAIF) and the absence of activated casp

275 Furthermore, differentiation-inducing factors that elevate MITF expression in melanom

276 melanocortin peptide agonists act as satiety-inducing factors that mediate their action through the m

277 ial-derived signal in the intestinal stroma, inducing factors that restrict epithelial proliferation

278 Although IL-12 is also an IFN-gamma-inducing factor, the activity of IL-18 (but not IL-12) i

279 ly delivering a specific combination of Treg inducing factors through degradable polymer microspheres

280 The first candidate mesoderm-inducing factor to be identified was activin, a member o

281 lease and nuclear translocation of apoptosis-inducing factor to initiate chromatinolysis and cell dea

282 s activation, the translocation of apoptosis-inducing factor to the nucleus, and DNA fragmentation in

283 bose) polymerase, translocation of apoptosis-inducing factor to the nucleus, and morphological eviden

284 e permeability or translocation of apoptosis-inducing factor to the nucleus.

285 ted, at least in part, by specific fugetaxis-inducing factors to which only mature cells respond.

286 , and Egf and up-regulation of the apoptosis-inducing factor Trail in the oviduct.

287 lycolysis in response to the differentiation-inducing factor transforming growth factor beta1 (TGF-be

288 ough mitochondrial dysfunction and apoptosis-inducing factor translocation from the mitochondria to t

289 induced mitochondrial dysfunction, apoptosis-inducing factor translocation, and subsequent cell death

290 yD88 adaptor-like protein, Toll receptor IFN-inducing factor (Trif), and Trif-related adaptor molecul

291 Interferon (IFN)-gamma-inducing factor was previously termed interleukin (IL)-1

292 However, apoptosis-inducing factor was released from mitochondria into the

293 eta-synuclein can act as a neurodegeneration-inducing factor, we demonstrated that wild-type beta-syn

294 d release from mitochondria of the apoptosis-inducing factor were selectively abrogated in iNOS(-/-)

295 Glial-derived barrier-inducing factors were characterized using size-exclusion

296 polarization and relocalization of apoptosis-inducing factor, whereas the BRAF-V600E-mutated melanoma

297 lease and nuclear translocation of apoptosis-inducing factor with subsequent cell death.

298 ation of caspases-1, -3, and -8 or apoptosis-inducing factor within MNs, with a blockade of apoptosis

299 ion in ECs infected with the TB40E or fusion-inducing factor X (FIX) HCMV strains.

300 ndothelial cells by BADrUL131 and the fusion-inducing factor X clinical human cytomegalovirus isolate