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1 T laser power (2 W/cm(2)) to achieve maximal induction of apoptosis.
2 ned by a surprising, concentration-dependent induction of apoptosis.
3 cellular defense mechanisms is essential for induction of apoptosis.
4 ll death through activation of caspase-3 and induction of apoptosis.
5 d redox ratio and ROS increased, followed by induction of apoptosis.
6 uced in deguelin-treated mice along with the induction of apoptosis.
7 sed DCs could be partially attributed to the induction of apoptosis.
8 nd an increase in cell death consistent with induction of apoptosis.
9 -driven apoptosis) support a mechanism-based induction of apoptosis.
10 s (PIV5DeltaSH and PIV5VDeltaC) that enhance induction of apoptosis.
11  cells and evaluated the mechanism[s] in the induction of apoptosis.
12 ion of DNA DSBs, G2-M cell-cycle arrest, and induction of apoptosis.
13 ibitors, trigger cellular stress and lead to induction of apoptosis.
14 f the proapoptotic protein, BIM, followed by induction of apoptosis.
15 ndent elevation of cytosolic Ca(2+) prior to induction of apoptosis.
16 bility to secondary bacterial infection, and induction of apoptosis.
17  killing leukemic cells through the forcible induction of apoptosis.
18  enhanced host protein synthesis shutoff and induction of apoptosis.
19 n to be hypersensitive to CPT with prominent induction of apoptosis.
20 se activity, malondialdehyde production, and induction of apoptosis.
21 , accumulation of unrepaired DNA damage, and induction of apoptosis.
22 o dephosphorylation of Akt at serine-473 and induction of apoptosis.
23 ulting from a differentiation defect and the induction of apoptosis.
24  kinase (AMPK), activation of caspase-3, and induction of apoptosis.
25 KT), because PI3K inhibitor results in early induction of apoptosis.
26 orrelate with protein kinase C inhibition or induction of apoptosis.
27 ICD target Hes1 gene in mouse brains without induction of apoptosis.
28 cells were then treated with doxorubicin for induction of apoptosis.
29  by RV metabolite treatments, as well as the induction of apoptosis.
30 cFLIP had been downregulated resulted in the induction of apoptosis.
31 lation of cytotoxic double-strand breaks and induction of apoptosis.
32 ulate Star-PAP control of BIK expression and induction of apoptosis.
33 promotes p53-dependent growth arrest and the induction of apoptosis.
34  a mechanism to promote BOK accumulation and induction of apoptosis.
35 , reduced colony formation in soft agar, and induction of apoptosis.
36 nockdown or inactivation were accompanied by induction of apoptosis.
37 gulation, modulation of differentiation, and induction of apoptosis.
38 nts, may also contribute to piperlongumine's induction of apoptosis.
39 tivation of PI3-kinase (PI3K) signaling, and induction of apoptosis.
40 r of apoptosis, activation of caspase 9, and induction of apoptosis.
41 ctly links ErbB receptor inactivation to the induction of apoptosis.
42 proliferation of pancreatic cancer cells and induction of apoptosis.
43 ion of gamma-tubulin protein levels leads to induction of apoptosis.
44 ing electron transfer, enzyme catalysis, and induction of apoptosis.
45 leading from ligation of the Fas receptor to induction of apoptosis.
46 otes ADAM17 activity in neutrophils upon the induction of apoptosis.
47 horylated on Tyr142, high levels of p53, and induction of apoptosis.
48  the MDA-MB-231 cell line via complement and induction of apoptosis.
49 motes the DNA damage response leading to the induction of apoptosis.
50 esting that kinase activity is essential for induction of apoptosis.
51 active state of the MST kinases and favoring induction of apoptosis.
52 d-type colon cultures respond similarly upon induction of apoptosis.
53 lls in sub-G(1) phase of the cell cycle, and induction of apoptosis.
54 s of the molecular processes involved in the induction of apoptosis.
55 some required EBV to prevent the inefficient induction of apoptosis.
56 iately activated by oncogenic events through induction of apoptosis.
57 ed multimeric IAP antagonist complex for the induction of apoptosis.
58 ibition of JAK-STAT activity, leading to the induction of apoptosis.
59 ression into the cell cycle, followed by the induction of apoptosis.
60 bose) polymerase and caspase cleavage and an induction of apoptosis.
61 asis mainly in the immune system through the induction of apoptosis.
62  of proliferation and survival pathways, and induction of apoptosis.
63 vo, including inhibition of tumor growth and induction of apoptosis.
64 oteins, reduction of cell proliferation, and induction of apoptosis.
65 ay involving cell surface GRP78 receptor for induction of apoptosis.
66 s as well as the JNK1/2/c-Jun pathway to the induction of apoptosis.
67 ibition of soft agar clonogenic capacity and induction of apoptosis.
68 limits both FasL and ceramide analogs in the induction of apoptosis.
69 rowth of the cell line A427, which is due to induction of apoptosis.
70 s results in irreversible DNA damage and the induction of apoptosis.
71 ions in the morphology of this organelle and induction of apoptosis.
72 lular trap formation, bacterial killing, and induction of apoptosis.
73  mediated by both G0/G1 cell cycle block and induction of apoptosis.
74 chondria represents a critical signal in the induction of apoptosis.
75 vity and annexin V-positive cell number upon induction of apoptosis.
76 ells causes G1 cell cycle arrest followed by induction of apoptosis.
77  on cell cycle regulation is followed by the induction of apoptosis.
78  due to cell-cycle regulation leading to the induction of apoptosis.
79 the onset of irreversible DNA damage and the induction of apoptosis.
80 g of gene expression, cell cycle arrest, and induction of apoptosis.
81 d SHP-1 activity, inhibition of p-STAT3, and induction of apoptosis.
82 by immune cells to kill cancer cells through induction of apoptosis.
83 erlying mechanism for this by using H2O2 for induction of apoptosis.
84 alpha, other interferon-stimulated genes and induction of apoptosis.
85 a mechanism of inhibiting metastasis via the induction of apoptosis.
86 ading to cessation of cell proliferation and induction of apoptosis.
87 3 activation and either cell cycle arrest or induction of apoptosis.
88 believed to result directly or indirectly in induction of apoptosis, a better understanding of the bi
89                                              Induction of apoptosis activates Yorkie through the Jun
90 r-bearing mice with LV-tSMAC resulted in the induction of apoptosis, activation of antitumor immunity
91 PARP) cleavage were performed to measure the induction of apoptosis after nifurtimox treatment.
92 esolution by reducing neutrophil numbers via induction of apoptosis after tailfin injury.
93  least partially associated with an enhanced induction of apoptosis although triggered by distinct ap
94 mpairment of granule neuron differentiation, induction of apoptosis and abnormal localization of Purk
95 ogether with inhibited HDAC activity, led to induction of apoptosis and autophagy in WM cells.
96        6b leads to cell death by concomitant induction of apoptosis and autophagy, shows good pharmac
97 feration, motility, and invasion, as well as induction of apoptosis and autophagy.
98 o control OS tumor growth which involved the induction of apoptosis and cell cycle arrest, supporting
99                                              Induction of apoptosis and cell death by MLN8237 were co
100  degradation by PS-341 occurred early before induction of apoptosis and could not be inhibited by a p
101 BP2 had reduced viability and proliferation, induction of apoptosis and cytotoxicity, and up-regulati
102 testing of their biological activity such as induction of apoptosis and evaluation of anti-cancer eff
103 grees than controls, which was attributed to induction of apoptosis and G2 cell cycle arrest.
104 downregulation of MYC expression, as well as induction of apoptosis and induces differentiation of pr
105                      This was accompanied by induction of apoptosis and inhibition of cell growth and
106 y small-interfering (si) RNA caused a strong induction of apoptosis and inhibition of cell-cycle prog
107  stress and p53 activation, resulting in the induction of apoptosis and inhibition of proliferation,
108                     Further, the synergistic induction of apoptosis and inhibition of tumor growth we
109 n of pancreatic islet beta cell mass through induction of apoptosis and lack of regeneration.
110                                  Strikingly, induction of apoptosis and necroptosis effectors was sha
111 00E mutant melanoma xenograft tumors through induction of apoptosis and necrosis.
112 n mouse model of prostate cancer through the induction of apoptosis and proliferation block.
113 eckpoint both contributed to the synergistic induction of apoptosis and proliferation inhibition in A
114 gh PERK-eIF2alpha-dependent pathways, of the induction of apoptosis and reduction of autophagy by Sub
115                                          The induction of apoptosis and repression of the cell adhesi
116 lective DNA damage at telomeric level and to induction of apoptosis and senescence on tumor cells is
117 A] and its downstream substrate FOXO1 in the induction of apoptosis and the synthesis of catalase.
118 nteracts with E2F1, and is required for E2F1 induction of apoptosis and transcription of a number of
119  cytotoxic effect appeared to be mediated by induction of apoptosis and was inhibited in a concentrat
120 GI-1776 results in a concentration-dependent induction of apoptosis and we investigated its effect on
121    DCP inhibitory effects were attributed to induction of apoptosis and/or increased production of in
122               Cell-intrinsic effects such as induction of apoptosis and/or inhibition of cell prolife
123 ell-based assays to evaluate cell viability, induction of apoptosis, and accumulation of ROS followin
124 on of Sp-regulated genes, growth inhibition, induction of apoptosis, and antiangiogenic responses.
125  inhibition of cancer cell and tumor growth, induction of apoptosis, and antiangiogenic responses.
126 alization along with osteonectin expression, induction of apoptosis, and cytotoxicity assays were per
127  cells by RNAi resulted in aberrant mitoses, induction of apoptosis, and decreased ability of human b
128 he most potent effects on growth inhibition, induction of apoptosis, and delay of acquired resistance
129 DO or CDDO-Me resulted in growth inhibition, induction of apoptosis, and down-regulation of cyclin D1
130    The mechanism of action of IAD is largely induction of apoptosis, and each mechanism of resistance
131 luding loss of epithelial barrier functions, induction of apoptosis, and inflammation.
132 4 results in efficient degradation of Mcl-1, induction of apoptosis, and inhibition of tumor growth i
133 ulting in a shut-off of general translation, induction of apoptosis, and inhibition of virus replicat
134 al dysfunction, generation of peroxynitrite, induction of apoptosis, and oxidative stress due to disr
135 ociated with the inhibition of angiogenesis, induction of apoptosis, and reduction in keloid phenotyp
136 rosurvival Bcl-2 proteins leads to effective induction of apoptosis, and strongly support the concept
137 d cells, which were also associated with the induction of apoptosis, and these results were consisten
138 ibition-induced down-regulation of c-Myc and induction of apoptosis are mitigated when the cells are
139  correction of mitochondrial dysfunction and induction of apoptosis are promising strategies in cance
140 e main function ascribed so far has been the induction of apoptosis as a crucial mediator of KRAS-dri
141  the cytotoxic effect may be associated with induction of apoptosis as evidenced by increased percent
142  an intrinsic innate immune response and the induction of apoptosis as mechanisms to restrict viral r
143      This effect is related to a synergistic induction of apoptosis as seen in the combination of 4j
144           miR-205 overexpression resulted in induction of apoptosis, as evidenced by increased cleave
145   An opposite pattern was observed regarding induction of apoptosis, as studied by Western blotting f
146 osphocholine, inhibition of cell growth, and induction of apoptosis at low micromolar concentrations.
147 e mediated by the block of autophagy and the induction of apoptosis; (b) decreased metastatic potenti
148 r fate by regulating both cytoprotection and induction of apoptosis based on the metabolic state.
149                                              Induction of apoptosis because of aggresomal compartment
150 v toxin is linked to V. vulnificus dependent induction of apoptosis, but the region of the large mult
151 e inhibition of tumor cell proliferation and induction of apoptosis, but their effect on tumor cell i
152 x and decreased Bcl-2 expression, indicating induction of apoptosis by 48 hours after SP treatment.
153 t significantly affect AcMNPV replication or induction of apoptosis by a mutant of AcMNPV lacking the
154  signaling to AMPK, making them resistant to induction of apoptosis by adenosine 3',5'-cyclic monopho
155  cells transfected with miR-K10a showed less induction of apoptosis by annexin V staining and termina
156 n of Caspase3 at 200muM at 48h confirmed the induction of apoptosis by apigenin and quercetagetin.
157                                  Synergistic induction of apoptosis by AZD8055 and ABT-737 is confirm
158 as responsible for the activation of JNK and induction of apoptosis by both agents.
159 ance of significant caspase3/7 activity upon induction of apoptosis by doxorubicin.
160 we examined and quantitatively evaluated the induction of apoptosis by drug combination therapies.
161                                          The induction of apoptosis by ErPC3 requires TSPO, a mitocho
162     Early L3 animals are highly resistant to induction of apoptosis by expression of IAP-antagonists,
163       In the investigated cancer cell lines, induction of apoptosis by FasL or DR5 agonist antibody c
164                                        After induction of apoptosis by H. pylori or camptothecin, the
165 ruption of Bcl-2-ERK complex, as well as the induction of apoptosis by H2O2.
166 92 or a D-box destruction motif inhibits the induction of apoptosis by microtubule poisons.
167     The aim of this study was to explore the induction of apoptosis by MTX.
168                           Here, we show that induction of apoptosis by Myc in breast epithelial cells
169                                              Induction of apoptosis by OSI-027 appears to negatively
170                         Here, we describe an induction of apoptosis by p21 in sarcoma cell lines that
171 NA attenuated the downregulation of EGFR and induction of apoptosis by p73 in HNSCC cells.
172                       On the other hand, the induction of apoptosis by PIK-75 did not require the exp
173 wnregulation that contributes to synergistic induction of apoptosis by PP242 plus TRAIL.
174 vity of elephant cells to DNA damage and the induction of apoptosis by regulating activity of the TP5
175                                          The induction of apoptosis by RNAi-mediated depletion or pha
176                                              Induction of apoptosis by SHP activation inhibits perito
177 small cell lung cancer cell lines toward the induction of apoptosis by the DNA-damaging agent etoposi
178 us, mutation of cleavage sites inhibited the induction of apoptosis by treatment with tumor necrosis
179 ate that Exo1 plays an important role in the induction of apoptosis by unrepaired O(6)MeGs.
180                           It is thought that induction of apoptosis can lead to enhanced antigen pres
181  AKT2-amplified PDAC, was able to bypass the induction of apoptosis caused by KRAS* ablation, highlig
182 is demonstrates that BIM is critical for the induction of apoptosis caused by potentially oncogenic D
183  time-dependent inhibition of proliferation, induction of apoptosis, cell-cycle arrest, and terminal
184  showed the largest inhibition of growth and induction of apoptosis compared with the double combinat
185                                              Induction of apoptosis correlated with increased express
186 ed to significant inhibition of cell growth, induction of apoptosis, decreased invasiveness and decre
187 on (that resulted in loss of nuclear CK2 and induction of apoptosis) demonstrated dynamic modulation
188                                     However, induction of apoptosis does not explain how scrib(-) cel
189  as a scaffold for pro-caspase-8 binding and induction of apoptosis downstream of induced-proximity a
190                   Here we show the efficient induction of apoptosis even by a non-toxic nsEP exposure
191 crease in LC3 levels that was accompanied by induction of apoptosis, evidenced by increases in cleave
192 Usp9x in myeloma cells resulted in transient induction of apoptosis, followed by a sustained reductio
193                   TRAIL receptors facilitate induction of apoptosis for selective elimination of mali
194                                   The direct induction of apoptosis has emerged as a powerful antican
195                                          The induction of apoptosis has long been proposed as a targe
196 ough mostly cytosolic in healthy cells, upon induction of apoptosis, IBRDC2 accumulates in mitochondr
197  MV-4-11 human leukemia cells, showed potent induction of apoptosis (IC(50)'s approximately 20 muM) w
198                      The best binders showed induction of apoptosis in a human cancer cell line with
199 lence functions for P. aeruginosa, including induction of apoptosis in a variety of target host cells
200 immunoregulatory activity via B7-H1-mediated induction of apoptosis in activated T cells.
201                                Moreover, the induction of apoptosis in airway epithelial cells activa
202 genes, decreased dendritic spine density and induction of apoptosis in astrocytes may contribute to t
203 function of WT p53 is directly linked to the induction of apoptosis in both cancer cells and tumor xe
204 domain protein Miz-1 restricts p53-dependent induction of apoptosis in both pro-B and DN3a pre-T cell
205 imary myoblasts and C2C12 cultures, chemical induction of apoptosis in C2C12 cells results in indiscr
206                                          The induction of apoptosis in CACs by microparticles derived
207      Inhibition of phagocytosis prevents the induction of apoptosis in CACs by microparticles.
208                                              Induction of apoptosis in cells infected by Sendai virus
209 tion protects stalled forks from collapse or induction of apoptosis in CHK1 inhibited cells during re
210 us in response to altered growth stimuli and induction of apoptosis in cultured prostate cells and in
211 , irreversible DNA damage, and ATM-dependent induction of apoptosis in cytokine-treated beta cells.
212             These in vitro studies show that induction of apoptosis in DC by MVA vectors is detriment
213  synergistic effect in growth inhibition and induction of apoptosis in DLBCL cells.
214 e importance of hnRNP A1 was demonstrated by induction of apoptosis in eIF2alpha phosphorylation-defi
215                                    The rapid induction of apoptosis in FAM129B knockdown cells was re
216 ailable for clinical studies potentiates the induction of apoptosis in glioma CSCs.
217  and evaluate their inhibition of growth and induction of apoptosis in human cancer cells.
218 ection gonorrhea, protects against exogenous induction of apoptosis in human cervical epithelial cell
219  kinase and PI3K pathways is involved in the induction of apoptosis in LPS-treated TBBEC.
220 lpain, CAPN1, which were requisite steps for induction of apoptosis in MDS cells and in acute myeloid
221                                     Although induction of apoptosis in melanoma cells by melphalan in
222                    Here, we studied in vitro induction of apoptosis in mouse bone marrow-derived macr
223 ensory epithelium (OSE) in hamsters and that induction of apoptosis in olfactory receptor neurons (OR
224                                       Strong induction of apoptosis in one compartment of the Drosoph
225 ment of multiple oncogenic processes and the induction of apoptosis in pancreatic and breast cancer c
226 on causes reduction in adipogenesis, through induction of apoptosis in pre-adipocytes, by inhibition
227  for cell cycle arrest and caspase-dependent induction of apoptosis in proliferating U937 cells.
228 cept in cancer therapy, coiled-coil mediated induction of apoptosis in Raji B cells, for treatment of
229 the expression of IFN-beta and the efficient induction of apoptosis in reovirus-infected cells.
230 ied the molecular mechanisms involved in the induction of apoptosis in response to cisplatin, and fou
231 tical role in cell-cycle progression and the induction of apoptosis in response to DNA damage.
232  activation plays a major regulatory role in induction of apoptosis in response to ER stress and indi
233 of E2F regulation is critical for the normal induction of apoptosis in response to irradiation.
234 these two microRNAs in mediating p16-related induction of apoptosis in response to this genotoxic str
235 sclerosis (RRMS) is not well understood, but induction of apoptosis in specific leukocyte subsets is
236 s revealed that Dicer1 deficiency led to the induction of apoptosis in T-ALL cells, whereas cell cycl
237                            Here we show that induction of apoptosis in the Drosophila wing imaginal d
238 ERK phosphorylation and the late response of induction of apoptosis in the human prostatic carcinoma
239 , attenuation of cell survival pathways, and induction of apoptosis in the ischemic territory, which
240  depletion causes impaired proliferation and induction of apoptosis in the OP9 cell line and in prima
241 ly demonstrated that Heme is involved in the induction of apoptosis in vascular endothelial cells.
242 nducing ligand (TRAIL) for markedly enhanced induction of apoptosis in VHL-null 786-O cells but not i
243 ulting in viral RNA replication and a potent induction of apoptosis in vitro and antitumor effects in
244 inical Akt and CDK inhibitors, demonstrating induction of apoptosis in vitro and providing a preclini
245        PE induced a rapid and dose-dependent induction of apoptosis in wild-type (WT) and Bax knockou
246   siParp1 inhibits cell growth, primarily by induction of apoptosis, in Brca1-deficient cells both in
247 c protein that forms a complex with Bax upon induction of apoptosis, in the presence of gamma-secreta
248 ents that require BID activation for maximal induction of apoptosis, including topoisomerase inhibito
249                                          The induction of apoptosis increased osteocytic Rankl expres
250            To investigate if EBOV blocks the induction of apoptosis, infected cells were treated with
251 tinoblastoma and current models propose that induction of apoptosis is a key p53 tumor suppressive fu
252             We further demonstrated that the induction of apoptosis is associated with the GNMT nucle
253     The regulation of neutrophil lifespan by induction of apoptosis is critical for maintaining an ef
254 in the activation of Bid, and the consequent induction of apoptosis is inhibited by Noxa silencing.
255             Killing cancer cells through the induction of apoptosis is one of the main mechanisms of
256                                              Induction of apoptosis is the primary mechanism through
257  impaired growth and proliferation in vitro, induction of apoptosis, loss of colony formation in meth
258       Furthermore, we demonstrate that, upon induction of apoptosis, lymphoma cells not only activate
259 ificant inhibition of cell proliferation and induction of apoptosis, making it an attractive target f
260  R (PKR) was blocked in EBOV-infected cells, induction of apoptosis mediated by dsRNA was not suppres
261 in orchestrating various pathologies via the induction of apoptosis, necroptosis, and nuclear factor-
262  blood-brain barrier integrity during ECM by induction of apoptosis of brain vascular endothelial cel
263 e capacity to help GC-B cells because of the induction of apoptosis of GC-B cells through the FAS/FAS
264  B-ALL development, which was accompanied by induction of apoptosis of leukemic B-lymphoid progenitor
265 : (i) inhibition of cell proliferation, (ii) induction of apoptosis of tumor xenograft cells, (iii) d
266 receptor, death receptor 5 (DR5), leading to induction of apoptosis, offers a promising anticancer st
267 lthough p53 limits tumorigenesis through the induction of apoptosis or cell cycle arrest, its molecul
268 on state of the Bnip3 LIR signals either the induction of apoptosis or pro-survival mitophagy.
269 f c-Jun N terminal kinases (JNK)1/2, but not induction of apoptosis or the JAK/STAT pathway that is n
270 lts in suppression of cell proliferation and induction of apoptosis process.
271                                 Furthermore, induction of apoptosis proved to be dependent on the sub
272 70alpha is AR-dependent and mediated through induction of apoptosis rather than cell cycle arrest.
273 f breast cancer cells, which correlated with induction of apoptosis, reduced expression of SOX2 but e
274 ation of oxidant and antioxidant levels, and induction of apoptosis-related proteins in the lung tiss
275 , including cell activation, protection from/induction of apoptosis, release of inflammatory cytokine
276 ppression of TWIST in stroma interfered with induction of apoptosis sensitivity in cocultured CD34(+)
277    Degradation of FAT10 is accelerated after induction of apoptosis, suggesting that it plays a role
278 lts in loss of MCL-1 expression prior to the induction of apoptosis, suggesting that MCL-1 may be an
279      Combination treatment resulted in rapid induction of apoptosis that coincided with increased DNA
280            M-phase arrest and the consequent induction of apoptosis that follows could possibly be at
281                 These findings indicate that induction of apoptosis through Fas is dependent on recep
282  strong anticancer activity exhibited as the induction of apoptosis through p53 activation.
283 um by down-regulation of XIAP expression and induction of apoptosis through specific targeting of a S
284                   Although the time from the induction of apoptosis to the change of mitochondrial pe
285                   We unexpectedly found that induction of apoptosis triggered high-level viral replic
286  Independent cell cycle arrest and increased induction of apoptosis underlies the synergistic effect
287 is inducing factor (AIFM1) have roles in the induction of apoptosis upon association with PA.
288  of Lmo4 in nitroxidative conditions and the induction of apoptosis upon repression of Lmo4.
289 previously unidentified role of ISG54 in the induction of apoptosis via a mitochondrial pathway and s
290 BID and BIM have nonoverlapping roles in the induction of apoptosis via BAK and BAX, affecting chemot
291 t that cellular levels of PPARgamma regulate induction of apoptosis via caspase-8 activation, whereas
292                                              Induction of apoptosis was caspase dependent, as it was
293 rimary epidermal keratinocytes, photodynamic induction of apoptosis was elicited by the combined acti
294                                              Induction of apoptosis was increased using a combination
295                                              Induction of apoptosis was observed in Cucurbitacin D tr
296 served that, in clade B infected astrocytes, induction of apoptosis was significantly higher than in
297 ynthesis inhibition, cell cycle effects, and induction of apoptosis were determined for two character
298 or, significant reduction in cell viability, induction of apoptosis were observed when treated with a
299           These effects were due to striking induction of apoptosis, which was mediated by Bim upregu
300    5q inhibits tumor cell growth through the induction of apoptosis, with detailed mechanism of its s

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