ライフサイエンスコーパス: infancy

1 Nonetheless, the field is still in its infancy.

2 with reduced risk of clinical malaria during infancy.

3 d with acute respiratory tract infections in infancy.

4 ses of generalized arterial calcification of infancy.

5 However, the field is still in its infancy.

6 tion of peanut-containing foods beginning in infancy.

7 e initiation and development is still in its infancy.

8 iolitis and pneumonia, particularly in early infancy.

9 ir chemo-physical properties is still in its infancy.

10 for pediatric cardiomyopathies are in their infancy.

11 risk of Plasmodium falciparum malaria during infancy.

12 gy and a product development pipeline in its infancy.

13 -making between these two pathways is in its infancy.

14 lking, and who had been exposed to cattle in infancy.

15 he severity of respiratory diseases in early infancy.

16 enterocolitis (NEC)-associated deaths during infancy.

17 tect against malaria and other infections in infancy.

18 atory tract illness, on lung function during infancy.

19 d via gesture and vocalization is present in infancy.

20 s transfer may reduce the risk of malaria in infancy.

21 explained by differential adiposity gains in infancy.

22 the development of targeted agents is in its infancy.

23 and near emmetropia in their left eyes from infancy.

24 rpesvirus (HHV) infections are common during infancy.

25 d adiposity and lower fat-free mass in early infancy.

26 let autoimmunity in patients is still in its infancy.

27 ism is a developmental disorder evident from infancy.

28 y consumption in weight regulation begins in infancy.

29 f Sphagnum-microbiome associations is in its infancy.

30 ation of primary human cells is still in its infancy.

31 ons of target fisheries species are in their infancy.

32 ts, root research has long been stuck in its infancy.

33 The fourth had viral encephalitis during infancy.

34 understanding the origins of this ability in infancy.

35 a functional social cognitive skill in later infancy.

36 cases being severe and resulting in death in infancy.

37 osed to maternal smoking during pregnancy or infancy.

38 vet syndrome or severe myoclonic epilepsy in infancy.

39 e, the focus of this review, is still in its infancy.

40 s made some progress, but it is still in its infancy.

41 ntegrate widely into the frontal lobe during infancy.

42 y of age, with 23% of deaths occurring after infancy.

43 h protection against clinical malaria during infancy.

44 d in the immunopathogenesis of dengue during infancy.

45 ion was relentless and many children died in infancy.

46 astroesophageal reflux disease (GERD) during infancy.

47 diposity, and arterial wall thickness during infancy.

48 with nanometer precision are still in their infancy.

49 se diverse microbes function is still in its infancy.

50 between human and viruses are still in their infancy.

51 e associated with an increased risk of AD in infancy.

52 reased transepidermal water loss), and AD in infancy.

53 hemangioma (IH) is the most common tumor of infancy.

54 ions of synthetic biology are still in their infancy.

55 rinary species is a field of research in its infancy.

56 children, particularly those infected during infancy.

57 t and is the most common and deadly tumor of infancy.

58 ective C-allylation reaction is still in its infancy.

59 their role in brain function is still in its infancy.

60 rstanding of how they are silenced is in its infancy.

61 hose who will and will not develop eczema in infancy.

62 e captured by repeated questionnaires during infancy.

63 UCB is associated with eczema development in infancy.

64 th and predicts the development of eczema in infancy.

65 n 2008 to promote peanut introduction during infancy.

66 cause monogenic disease, often presenting in infancy.

67 omes challenging for IOL implantation during infancy.

68 lopment of spatial-numerical associations in infancy.

69 ed with abnormalities among offspring during infancy.

70 dial-up specific properties is still in its infancy.

71 were hospitalized for bronchiolitis in early infancy.

72 nent of the nasopharyngeal microbiome during infancy.

73 on [lateral occipital cortex (LOC)] early in infancy.

74 ses of generalized arterial calcification of infancy.

75 Liver cancer occurrence in infancy, 1.5 for million children per year, falls far be

76 associations between vitamin B-12 status in infancy (2-12 mo) and the development and cognitive func

77 ain and behavior mechanisms in autism during infancy, a period which precedes the emergence of the de

78 jects who received 3 doses of HBV vaccine in infancy and 251 nonvaccinated subjects.

79 as change in absolute tissue volume, between infancy and 7 years was calculated for cortical gray mat

80 p with generalized arterial calcification of infancy and arterial calcification due to CD73 deficienc

81 nclude generalized arterial calcification of infancy and arterial calcification due to CD73 deficienc

82 vestigate the association between VDI during infancy and challenge-proven food allergy at 1 year.

83 range of human diseases, and environments in infancy and childhood are important determinants of infl

84 in early life can cause disease and death in infancy and childhood as well as chronic, non-communicab

85 Epileptic encephalopathies of infancy and childhood comprise a large, heterogeneous gr

86 iated with increased risk of rapid growth in infancy and childhood obesity.

87 ngiomas are the most common benign tumors of infancy and childhood with a reported incidence of 4% to

88 In infancy and childhood, the brain may be affected by risk

89 to estimate exposure and body burden during infancy and childhood.

90 follow-up provided data on feeding modes in infancy and doctor's diagnosed eczema/skin allergy in th

91 LOC specialization from early childhood into infancy and earlier than developmental trajectories of h

92 itivity after hepatitis A vaccination during infancy and early childhood is unclear.

93 se association between having a furry pet in infancy and EoE (aOR, 0.58; 95% CI, 0.34-0.97).

94 ate the feasibility of introducing peanut in infancy and explore effects on growth and nutritional in

95 Neuroblastoma is the most common cancer in infancy and extremely heterogeneous in clinical presenta

96 all association was indicated between SHS in infancy and food sensitization up to age 16 years (OR 1.

97 edicine in ophthalmology is currently in its infancy and has yet to gain wide acceptance.

98 500 g) is associated with iron deficiency in infancy and high blood pressure (BP) later in life.We in

99 ffer from intense pain that can start during infancy and increase in severity throughout life, leadin

100 of this metabolic interface is still in its infancy and its therapeutic targeting is largely untappe

101 group 14 elements is already well beyond its infancy and may be an instrumental factor in awakening t

102 Secondary HLH presents after infancy and may be associated with heterozygous mutation

103 tive and undermining coparental behaviors in infancy and preschool, assayed oxytocin (OT) and vasopre

104 hypotonia and difficulties in feeding during infancy and reduced energy expenditure, hyperphagia, and

105 We measured skin barrier function in early infancy and related it to the later development of FS/FA

106 care in HF is promising, it is still in its infancy and requires additional high-quality, methodolog

107 ssible association between antibiotic use in infancy and risk of childhood obesity, with implications

108 nsition of the skin microbiome between early infancy and the dysbiosis of established AD is unknown.

109 food epitope-specific IgA develops in early infancy and the relationship of IgA epitope recognition

110 sfunctional feeding patterns carry over into infancy and toddler age groups.

111 gility, trauma-induced skin blistering since infancy, and development of remarkable diffuse mottled p

112 d nitrogen dioxide exposure in utero, during infancy, and in childhood were negatively associated wit

113 s ground breaking technology is still in its infancy, and its applicability for routine disease diagn

114 ral palsy are more difficult to ascertain in infancy, and magnetic resonance imaging and the Hammersm

115 s that inhibit these enzymes is still in its infancy, and the identification of suitable inhibitors c

116 the channel structure, although still in its infancy, and the role of the TRPM8 protein signalplex to

117 del of generalized arterial calcification of infancy, and we have now explored the potential efficacy

118 elivery: aOR, 1.77; 95% CI, 1.01, 3.09), and infancy (antibiotic use: aOR, 2.30; 95% CI, 1.21-4.38; u

119 SHS exposure in infancy appears to increase the risk of sensitization to

120 rmine whether microbial alterations in early infancy are associated with the development of atopic wh

121 arietal and sub-cortical neurogenesis during infancy are critical to the pathophysiology of ID.

122 wth and reduce risk for overweight status in infancy are lacking.

123 and progression of the GIT microbiota during infancy are necessary to promote robust adaptive antivir

124 nt of gut microbial communities during early infancy are relatively rare, particularly for infants fr

125 giomas are the most common benign tumours of infancy, arising in the first few weeks of life and exhi

126 The field is in its infancy as compared to their class I counterparts.

127 with nonbreastfed individuals differ during infancy as well as into adulthood.

128 fic gastrointestinal bacterial taxa in early infancy associated with less asthma risk, possibly by pr

129 affected patients usually do not live beyond infancy because of infections.

130 nt in childhood, exposure to animal feces in infancy, birth in the dry season, or duration of exclusi

131 ugar intakes are associated with unfavorable infancy BMI peak characteristics and higher early childh

132 ft actuators is an area that is still in its infancy but has the potential to provide quantitative in

133 nce of increased endogenous DHA synthesis in infancy but not at school-age.

134 ccines) are immunogenic and can be used from infancy but there are no efficacy data for the leading c

135 endently of continuous magnitudes already in infancy, but is nonetheless more deeply connected to oth

136 W) is associated with increased mortality in infancy, but its association with mortality in later chi

137 ans and other social primates; it emerges in infancy, but the development of the neural mechanisms su

138 ns on disease susceptibility is still in its infancy, but the integration of all the above-mentioned

139 exclusive for 4 months) of breastfeeding in infancy by postal questionnaires.

140 al disorder that, if not treated promptly in infancy, can cause mental retardation, as the TH decreas

141 undergo numerous medical interventions from infancy, can suffer from lifelong debilitation caused by

142 ion in generalized arterial calcification of infancy caused by ENPP1 mutations.

143 icated early-life factors (antibiotic use in infancy, cesarean delivery, breast-feeding, neonatal int

144 Congenital hyperinsulinism of infancy (CHI) can be caused by inactivating mutations in

145 eficiency virus (HIV) infection during early infancy (commonly known as "early infant HIV diagnosis"

146 triction to darkness, benign tonic upgaze of infancy, congenital fibrosis syndrome, and the constella

147 nd maternal education, all tissue volumes in infancy correlated with IQ (r >/= 0.35, P < .05) and lan

148 d we hypothesized that reduced protection in infancy could be due to impaired establishment of lung T

149 such, the microbiome trajectory during early infancy could be predictors of healthy development.

150 autoantibodies during fetal development and infancy could contribute to brain dysfunction such as th

151 Research in this area, now in its infancy, could potentially provide a novel therapeutic s

152 Priming with 1, 2, or 3 PCV7 doses during infancy did not affect serotype-specific immunity or car

153 ed CD4(+) T cell imunosensors remains in its infancy due to the poor immobilization efficiency, lack

154 LA excitatory and inhibitory neurons in late infancy, events that coincide with a massive increase in

155 Our data suggest that in utero and infancy exposures to air toxics generated by industrial

156 reds died of severe pulmonary disease during infancy following viral pneumonia with evidence of combi

157 Exposures to air toxics during pregnancy/infancy for 43 PNET, 34 medulloblastoma, and 106 astrocy

158 the efficacy of oral tolerance induction in infancy for the prevention of FA.

159 disentangle the effect of antibiotic use in infancy from that of underlying infection on the risk of

160 Although the field is in its infancy, future investigations of human BNST function ha

161 s with generalized arterial calcification of infancy (GACI) due to loss-of-function mutations in the

162 However, infection during infancy generated markedly fewer lung TRMs, and heterosu

163 spontaneously resolved cytopenic episodes in infancy, harbored hematopoietic revertant mosaicism by u

164 to establish whether general anaesthesia in infancy has any effect on neurodevelopmental outcome.

165 Exposure to air pollution during infancy has been related to lung function decrements in

166 Eyes treated for monocular cataract in infancy have axial growth similar to that of fellow eyes

167 iversity and low mucosal total IgA levels in infancy have been associated with allergy development, I

168 on supplementation (1 or 2 mg Fe . d(-1)) in infancy have lower SBP at 7 y.

169 tal compounds of this ligand is still in its infancy; however, it has received growing attention in r

170 From infancy, humans develop dedicated neural circuits [1] to

171 rine growth retardation, febrile seizures in infancy, impaired speech, stereotypic behavior, hyperton

172 oderate burden of pertussis disease in early infancy in Pakistan.

173 ldren with milk allergy who were enrolled at infancy in the Consortium of Food Allergy observational

174 mages and losses, but its practice is in its infancy in the region.

175 32% lower risk of symptomatic malaria during infancy (incidence rate ratio [IRR] = 0.68 [95% CI 0.51,

176 gene for a group of paroxysmal syndromes of infancy, including epilepsy, paroxysmal movement disorde

177 Exposure to mold or dampness during infancy increased the risk of asthma and rhinitis up to

178 less than 1 h of sevoflurane anaesthesia in infancy increases the risk of adverse neurodevelopmental

179 whether exposure to mold or dampness during infancy increases the risk of asthma, rhinitis, or IgE s

180 ials have shown that avoiding peanuts during infancy increases the risk of peanut allergy; however, t

181 RSV-induced wheezing illnesses during infancy influence respiratory health for years.

182 The gut microbiome in infancy influences immune system maturation, and may hav

183 iming of allergenic food introduction during infancy influences risk of allergic or autoimmune diseas

184 natal period is approximately 10% and beyond infancy is <5%, with 90% to 95% of patients surviving wi

185 idence that VDI during the first 6 months of infancy is a risk factor for food allergy at 1 year of a

186 Early infancy is a window during which gut microbiota may shap

187 ry microbiota composition and development in infancy is affecting microbiota stability and thereby re

188 Generalized arterial calcification of infancy is an intractable ectopic mineralization disorde

189 scores, respectively.Vitamin B-12 status in infancy is associated with development and performance o

190 Low brain volume in infancy is associated with long-term functional outcomes

191 Infection, but not antibiotic use, during infancy is associated with risk of childhood obesity.

192 N: Infection, but not antibiotic use, during infancy is associated with risk of childhood obesity.

193 Optimal iodine intake during infancy is critical for brain development, but no estima

194 RSV immunoprophylaxis during infancy is efficacious in preventing RSV-related hospita

195 Treatment during infancy is highly efficacious and well tolerated, but cu

196 Development of the gut microbiota in infancy is important in maturation of the immune system.

197 Exposure to traffic-related air pollution in infancy is negatively associated with FEV1 at age 16 yea

198 The importance of dietary lipids during infancy is paramount for rapid growth and development.

199 Because sustained attention in late infancy is predictive of future development, and because

200 signaling is required to prevent colitis in infancy is unknown.

201 ent of common infections with antibiotics in infancy is unlikely to be a main contributor to childhoo

202 mechanisms and physical aggression onset in infancy, it appears clear that preventive interventions

203 on homeostasis is absent or limited early in infancy largely because of a lack of regulation of the i

204 arly effective, long-term ART initiated from infancy leads to decay of HIV-1-infected cells to exceed

205 Infancy literature suggests an extension of the GANE (gl

206 the most common pulmonary infection in early infancy, making it important to determine whether it dam

207 lated functional connectivity changes during infancy, marking critical points in the development of t

208 High protein intake during infancy may contribute to obesity later in life in infan

209 tomegalovirus transmission prenatally and in infancy might further decrease HIV transmission and lead

210 Using a sign language since infancy might shape the representation of words and othe

211 CHD cases were diagnosed at birth and in infancy (n=72 591).

212 -term effects of poor vitamin B-12 status in infancy need further investigation in randomized control

213 diseases neuroendocrine cell hyperplasia of infancy (NEHI) and pulmonary interstitial glycogenosis.

214 Sudden unexpected death in infancy occurs in apparently healthy infants and remains

215 ren living with HIV who are not diagnosed in infancy often remain undiagnosed until they present with

216 s) provided during pregnancy, lactation, and infancy on attained size by 18 mo of age.

217 ct of paternal exposure during pregnancy and infancy on infectious disease incidence was evaluated us

218 cause of a novel syndrome including neonatal/infancy-onset diabetes, congenital cataracts, and sensor

219 n TANGO2 in three unrelated individuals with infancy-onset episodic metabolic crises characterized by

220 ion, but HbF is epigenetically silenced from infancy onward by DNA methyltransferase 1 (DNMT1).

221 le magnetic resonance imaging data at either infancy or 7 years were collected for 214 VPT children (

222 Affected individuals presented in infancy or childhood with slowly progressive proximal an

223 ten allergic in origin, frequently begins in infancy or childhood with variable airflow obstruction a

224 with subacute neurological deterioration in infancy or childhood, preceded by a febrile infection, a

225 ypertension of indeterminate etiology during infancy or childhood.

226 viorally defined condition that manifests in infancy or early childhood as deficits in communication

227 of infection in the first or second half of infancy or the likelihood of stunting at 12 mo postpartu

228 he incidence of CMV infection is high during infancy, our data provide a mechanistic framework to exp

229 outinely observed in chronic lung disease of infancy patients.

230 In infancy, perinatal factors, particularly respiratory iss

231 linical events associated with pregnancy and infancy period) was reported for 310 (38%) of 808 partic

232 obial colonization of mucosal tissues during infancy plays an instrumental role in the development an

233 responsiveness to the gut microbiota during infancy precedes asthma and allergy development, possibl

234 t the parental caudate-vmPFC connectivity in infancy predicted lower child externalizing symptoms at

235 tamin D supplementation during pregnancy and infancy prevents aeroallergen sensitization and primary

236 evidence demonstrates that protein intake in infancy programs linear growth.

237 Persistent tachypnea of infancy (PTI) is a specific clinical entity of undefined

238 gression showed that iron supplementation in infancy reduced the odds of having an SBP within the hyp

239 cyanosis requiring surgical repair in early infancy reflects poor anatomy and is associated with mor

240 n and the host-microorganism relationship in infancy remain poorly understood.

241 ion against systemic viral infections during infancy remains undefined.

242 are advanced from human and nonhuman primate infancy research for the exaptation of ingestive mouth m

243 s of iron homeostasis to pregnancy and early infancy, respectively, and their implications, 2) improv

244 agocytic lymphohistiocytosis (HLH) occurs in infancy resulting from homozygous mutations in NK and CD

245 se is a severe disorder that presents during infancy, resulting in failure to thrive, hepatomegaly, a

246 Sepsis in early infancy results in one million annual deaths worldwide,

247 STING)-associated vasculopathy with onset in infancy (SAVI) develop systemic inflammation characteriz

248 STING-associated vasculopathy with onset in infancy (SAVI).

249 tients with STING-associated vasculopathy in infancy (SAVI).

250 yndrome and STING-associated vasculopathy of infancy (SAVI).

251 l-motor and visual-tactile linkages in early infancy since infants' muscular strength and control can

252 mechanoregulation in cancer is still in its infancy, some agents against key mechanoregulators have

253 ent and schistosomiasis can occur from early infancy, such that the high disease burden found in adol

254 onal studies of the language system in early infancy suggest that the basic neural mechanisms are in

255 t and skills relative to males, appearing in infancy, suggesting biological roots; however, male and

256 ic motor neuron disease with an onset during infancy that results in failure to achieve motor milesto

257 ed little evidence of adaptations present in infancy that support sophisticated reasoning about perce

258 sponses on the role of social interaction in infancy, the methodological issues about imitation exper

259 After IOL implantation during infancy, the rate of myopic shift occurs most rapidly du

260 ings; the manifestation of severe disease in infancy; the presence of comorbid developmental abnormal

261 During infancy, there is a sexually dimorphic growth response t

262 Although still in its infancy, this approach is being applied to malignant per

263 he emergence of a language-cognition link in infancy, this article reveals a dynamic developmental ca

264 fat mass (FM) or fat-free mass (FFM) during infancy.This study aimed to examine associations of 3 de

265 the temporal-parietal and sub-cortex during infancy through adulthood.

266 d growth in both structure and function from infancy through early childhood, and this significantly

267 acterized childhood wheezing phenotypes from infancy to adolescence and their associations with asthm

268 Our results provide striking evidence from infancy to adulthood that bilinguals monitor their langu

269 ostructural changes in HD monkey brains from infancy to adulthood, suggesting differentiated degenera

270 n infants and a change in learning mode from infancy to adulthood.

271 natal human brain, ranging in age from early infancy to adulthood.

272 he progression from atopic dermatitis during infancy to asthma and allergic rhinitis in later childho

273 1 in 2000 newborns, is treated surgically in infancy to prevent adverse neurologic outcomes.

274 relating directly measured VDI during early infancy to subsequent challenge-proven food allergy.

275 heightened risk of serious influenza during infancy, vaccination is not recommended in infants young

276 the animal's age (isolation from the dam in infancy, versus nociceptive stimulation in adults).

277 ren who had received iron supplementation in infancy was 2.2 mm Hg (95% CI: 0.3, 4.2 mm Hg) lower tha

278 tionnaire data on soy formula feeding during infancy was ascertained for 1,553 participants (89% base

279 egnancy, infection without antibiotic use in infancy was associated with an increased risk of childho

280 current symptoms of allergic disease, SHS in infancy was associated with an overall elevated risk of

281 Weight gain in infancy was categorized into four groups based on weight

282 938 girls, PM10 exposure in utero and during infancy was negatively associated with pubertal stage an

283 h untreated infection, antibiotic use during infancy was not associated with risk of childhood obesit

284 lthough modern phage therapy is still in its infancy, we conclude that phages, such as OMKO1, represe

285 f polyunsaturated fatty acids were higher in infancy (week 1 to week 3) compared with later ages, and

286 er indoor levels of pet or pest allergens in infancy were associated with lower risk of asthma.

287 CMA and the use of hypoallergenic formula in infancy were associated with the later development of JI

288 y, and antibiotic or acid suppressant use in infancy, were associated with risk of pediatric EoE; hav

289 d an important cause of mortality throughout infancy, whereas the contribution of injuries to mortali

290 Here we highlight an area in its infancy which we believe will increasingly engage the co

291 FPIES) is a non-IgE-mediated food allergy of infancy whose pathophysiology is poorly understood.

292 itic skin disease that often occurs in early infancy with a chronic course.

293 types include Ohtahara syndrome, epilepsy of infancy with migrating focal seizures, and intellectual

294 afflicted individuals had an onset in early infancy with optic atrophy, and in four patients anemia

295 lepsies are frequently severe, presenting in infancy with pharmaco-resistant seizures; are often acco

296 Presentation was delayed beyond infancy with proximal muscle weakness and most patients

297 ected individuals (n = 9) presented in early infancy with severe irritability, followed by dystonia a

298 y later.Vitamin B-12 status was assessed in infancy with the use of plasma cobalamin, total homocyst

299 ter introduction of conjugate Hib vaccine in infancy without a booster dose in Kenya.

300 Exposure to SHS in infancy without prior exposure in utero was associated w