ライフサイエンスコーパス: infant death

1 ence of the effects of MMN on fetal loss and infant death.

2 trophy (SMA) is the leading genetic cause of infant death.

3 n 50 and is the most common genetic cause of infant death.

4 ences in newborn babies and result in sudden infant death.

5 iated with an increased risk of neonatal and infant death.

6 e the burden of fetal death, stillbirth, and infant death.

7 th, stillbirth, and neonatal, perinatal, and infant death.

8 ate neonatal death, and 50.2 (42.8-59.0) for infant death.

9 th, stillbirth, and neonatal, perinatal, and infant death.

10 gly associated with the risk of neonatal and infant death.

11 a new, potentially treatable cause of sudden infant death.

12 have serious consequences, including sudden infant death.

13 s a significant risk factor for neonatal and infant death.

14 e to thrive, behavioral deficits, and sudden infant death.

15 revent the majority of needless maternal and infant deaths.

16 fetal deaths exceeded that of crash-related infant deaths.

17 nt 107000 (UR, 20000-198000) stillbirths and infant deaths.

18 54% of estimated cases and 65% of all fetal/infant deaths.

19 7-1.23; I2 = 78.5%; n = 12 studies); and for infant death, 1.18 (95% CI, 1.09-1.28; I2 = 79%; n = 4 s

20 een collected for 369 consecutive unexpected infant deaths (300 SIDS and 69 explained deaths) in Avon

21 sociated with 9208 (95% CI, 8601-9814) fewer infant deaths; 3195 (95% CI, 3017-3372) infant deaths co

22 CI, 19-23), and 24 (95% CI, 22-27); and for infant death, 33, 37 (95% CI, 34-39), and 43 (95% CI, 40

23 e 34 weeks (6.0% vs. 2.6%, P=0.04) and early infant death (4.4% vs. 0.6%, P=0.001), but there were no

24 The only high sensitivity was for past infant death (85.4%).

25 The sudden infant death/ALTE families had a greater frequency of tw

26 ntrol) included members with OSA, and all 10 infant death/ALTE families were among these (versus zero

27 Thus, OSA in adults and sudden unexpected infant death/ALTE in their biologic relatives appear to

28 Information on sudden unexpected infant death/ALTE was obtained by questionnaire and was

29 rom families with OSA plus sudden unexpected infant death/ALTE.

30 milies had 10 infants with sudden unexpected infant death/ALTE; two control families had three infant

31 Congenital anomalies are a leading cause of infant death and disability and their incidence varies b

32 ontroversies in the evaluation of unexpected infant death and inflicted traumatic brain injury are on

33 had an unexpected and apparently unexplained infant death and is currently available in over 90% of h

34 y associated with the risk of stillbirth and infant death and neonatal morbidity.

35 hyxia at term remains a significant cause of infant death and neurodevelopmental impairment, probably

36 m birth is the leading cause of neonatal and infant death and of disability among survivors.

37 e past, these vaccines were thought to cause infant deaths and brain damage.

38 Data on residual infant deaths and maternal seroprevalence would be valua

39 in the 40 years since 1970, particularly for infant deaths and mortality from non-communicable diseas

40 There were no maternal or infant deaths and no significant between-group differenc

41 11 mo, lost future earnings due to premature infant death, and the costs of purchasing infant formula

42 ity rates for SIDS, other sudden, unexpected infant deaths, and cause unknown/unspecified, and they e

43 nd 147000 (UR, 47000-273000) stillbirths and infant deaths annually.

44 tudied the relationship of sudden unexpected infant death/apparent life-threatening events (ALTE) to

45 nization was at least 1 million to prevent 1 infant death, approximately 100 000 for ICU admission, a

46 Rates of infant death are highest in children of Pakistani origin

47 Repeat unexpected infant deaths are most probably natural.

48 gnancy is a risk factor for sudden fetal and infant death as well as obstructive airway disease in ch

49 to identify those at risk of stillbirth and infant death at term.

50 The number of infant deaths at age 0-6 months was similar in each grou

51 rly 10 000 laboratory-confirmed cases and 14 infant deaths attributed to pertussis.

52 Numbers of early infant deaths (between 24 hours and 6 months of age) cod

53 Low birth weight (LBW) increases the risk of infant death, but little is known about its causes among

54 SV is the second leading infectious cause of infant death, but no vaccine is available.

55 y outcome was the composite of stillbirth or infant death by 1 year of corrected age or moderate or s

56 R) estimates for fetal death, stillbirth, or infant death by at least 3 categories of maternal BMI we

57 Recurrent late-term fetal loss or sudden infant death can result from unsuspected parental mosaic

58 Birth certificate data linked to infant death certificates and to infant discharge abstra

59 ding interhospital transfers), and fetal and infant death certificates to assess neonatal mortality r

60 Among the categories of causes of infant death, congenital anomalies (APC = -7.87%), asphy

61 ewer infant deaths; 3195 (95% CI, 3017-3372) infant deaths could have been avoided had there been no

62 We used national birth cohort linked birth-infant death data (2000-2010) to evaluate the risk of in

63 r for Health Statistics' period linked birth/infant death data set files for 2007-2013 for 26546503 U

64 Infant death due to NEC in preterm babies was identified

65 Infant deaths due to external causes were most likely to

66 Homicide is the leading cause of infant deaths due to injury.

67 in the United States, equal to the number of infant deaths each year.

68 rd (1980-98) and the Scottish Stillbirth and Infant Death Enquiry (1985-98).

69 d its determinants by using the US birth and infant death files for 1989-1997.

70 Data were derived from linked US birth/infant death files for 1995 and 1996, comprising 7,465,8

71 ed by lack of T and natural killer cells and infant death from infection.

72 ublic between 1986 and 1993, including 3,254 infant deaths from 350,978 first births to married and s

73 Early infant deaths from critical congenital heart disease thr

74 Early infant deaths from other/unspecified cardiac causes decl

75 d a lethal phenotype characterized by sudden infant death (from cardiac and respiratory arrest) with

76 nown to reduce the risk of sudden unexpected infant death has contributed to a slowing in the decline

77 ses the risk of fetal death, stillbirth, and infant death; however, the optimal body mass index (BMI)

78 Secretory diarrhea is the leading cause of infant death in developing countries and a major cause o

79 gyo virus had a live birth with maternal and infant death in Isiro, the Democratic Republic of the Co

80 th MMN, compared with IFA, on fetal loss and infant death in the setting of routine prenatal care ser

81 s to be the most common cause of unexplained infant death in Western countries.

82 infant mortality, accounting for 35% of all infant deaths in 2008.

83 er 5 years worldwide, and constituted 65% of infant deaths in India.

84 The five infant deaths in the group of potential survivors result

85 tation) are approximately equal in number to infant deaths in the United States and are twice as like

86 e on Sudden Infant Death, Sudden Unexplained Infant Death, infant and child death due to maltreatment

87 ical effects on the parents following sudden infant death is discussed and reveals maternal anxiety a

88 nvestigations in cases of sudden unexplained infant death is emphasized.

89 ed to estimate the probability that a second infant death is natural versus unnatural.

90 ostneonatal deaths (first 28 days), and five infant deaths (<1 year).

91 After an event of sudden infant death, many parents moved without a forwarding ad

92 Because most infant deaths occur in the first few month of life, mate

93 hat when two or three unexpected unexplained infant deaths occur within a family they are more likely

94 Overall, for every 1,000 births, 3.36 more infant deaths occurred among non-Hispanic black women re

95 ted the relative risks (RRs) of neonatal and infant death of neonates with low (0-3) and intermediate

96 The primary outcomes were infant death or CP and severe maternal outcome potential

97 ts CP and reduces the combined risk of fetal/infant death or CP.

98 dicate that whole cell vaccines do not cause infant deaths or neurologic disease.

99 the risk of fetal death (P = 0.99) or early infant death (P = 0.19).

100 5 (95% confidence interval: 2.20, 2.71) more infant deaths per 1,000 births among non-Hispanic black

101 paid maternity was associated with 7.9 fewer infant deaths per 1,000 live births (95% CI 3.7, 12.0),

102 tudied, SIDS rates ranged from a high of 3.0 infant deaths per 1,000 live births for American Indians

103 ported cases of disease and from 933 to 5796 infant deaths per year for the diseases under study are

104 en food is shared only within kin groups, an infant death permits reallocation of its unneeded food t

105 Additionally, among selective causes of infant death, pneumonia, congenital heart disease, neura

106 tality among American Indians, a group whose infant death rate is consistently above the US national

107 screening policies has been associated with infant death rates is unknown.

108 sible or if it reduces sudden and unexpected infant death rates remains to be studied.

109 es for critical congenital heart disease and infant death rates.

110 obtained from the 2000-2002 US Linked Birth/Infant Death records and included 677,777 black infants

111 in infant mortality when national birth and infant death records began to be linked.

112 identified from the US Linked Livebirth and Infant Death records between 2000 and 2004.

113 onal Center for Health Statistics' birth and infant death records for all twin births occurring in th

114 Therefore, it seems likely that infant deaths result from sepsis secondary to intestinal

115 Sixty percent of infant deaths resulted from delay in recognition of feta

116 A), one of the most common genetic causes of infant death, results from the selective loss of motor n

117 iking changes in rates of sudden unexplained infant death (SIDS) around 1990, four large case-control

118 ces the resources recaptured by kin after an infant death, so evolved infant mortality is lower.

119 The primary outcomes were infant death, stillbirth, overall mortality (infant and

120 own/unspecified and other sudden, unexpected infant deaths, such as accidental suffocation and strang

121 e most recent published literature on Sudden Infant Death, Sudden Unexplained Infant Death, infant an

122 ased cohort of 221 anonymous cases of sudden infant death syndrome (84 females, 137 males; average ag

123 certificates/autopsy reports included sudden infant death syndrome (n = 544 [44%]), asphyxia (n = 74

124 th of an infant that had succumbed to sudden infant death syndrome (SIDS) (and no other cause of deat

125 The association between sudden infant death syndrome (SIDS) and maternal smoking was co

126 lities in HMs have been implicated in sudden infant death syndrome (SIDS) and obstructive sleep apnoe

127 moking is the highest risk factor for sudden infant death syndrome (SIDS) and prenatal nicotine expos

128 is growth in our understanding of how sudden infant death syndrome (SIDS) and the symptom complex see

129 Many sudden infant death syndrome (SIDS) cases exhibit a partial ( a

130 An estimated 10% to 15% of sudden infant death syndrome (SIDS) cases may stem from channel

131 Sudden infant death syndrome (SIDS) cases often have abnormalit

132 evalence during the past two decades, sudden infant death syndrome (SIDS) continues to be the leading

133 uction campaigns have been conducted, sudden infant death syndrome (SIDS) has become increasingly con

134 ears suggest that the epidemiology of sudden infant death syndrome (SIDS) has changed since the 1991

135 infant sleeping position and risk of sudden infant death syndrome (SIDS) in an ethnically diverse US

136 Sudden infant death syndrome (SIDS) is a leading cause of postn

137 The likelihood of recurrence of sudden infant death syndrome (SIDS) is an issue of biological,

138 Sudden infant death syndrome (SIDS) is postulated to result fro

139 The sudden infant death syndrome (SIDS) is the sudden death of an i

140 The recent US decline in sudden infant death syndrome (SIDS) rates may be explained by a

141 d here support a recent proposal that sudden infant death syndrome (SIDS) results from a developmenta

142 Unexplained stillbirth and the sudden infant death syndrome (SIDS) share some features.

143 o examine recent research relevant to sudden infant death syndrome (SIDS) to determine whether there

144 in the medullae of infants dying from sudden infant death syndrome (SIDS) were identified, suggesting

145 associated with an increased risk of sudden infant death syndrome (SIDS), but few studies have asses

146 used in infants at increased risk for sudden infant death syndrome (SIDS), but the efficacy of such d

147 to understanding the aetiology of the sudden infant death syndrome (SIDS), in which there is medullar

148 This has special implications for sudden infant death syndrome (SIDS), insofar as seemingly norma

149 us state in two infants who died from sudden infant death syndrome (SIDS), one with documented prolon

150 edisposes an infant to a high risk of sudden infant death syndrome (SIDS), the authors conducted a po

151 Sudden infant death syndrome (SIDS), the leading cause of postn

152 associated with an increased risk of sudden infant death syndrome (SIDS).

153 Thousands die each year from sudden infant death syndrome (SIDS).

154 is (PGCL), in many infants who die of sudden infant death syndrome (SIDS).

155 sure are the highest risk factors for sudden infant death syndrome (SIDS).

156 ly associated with cardiomyopathy and sudden infant death syndrome (SIDS).

157 may be responsible for some cases of sudden infant death syndrome (SIDS).

158 e sleep position (on the stomach) and sudden infant death syndrome (SIDS).

159 ic exposure (PNE), is responsible for sudden infant death syndrome (SIDS).

160 necrotizing enterocolitis (NEC), and sudden infant death syndrome (SIDS).

161 ties are present in many cases of the sudden infant death syndrome (SIDS).Mice with a targeted disrup

162 for disorders such as sleep apnea or sudden infant death syndrome and for regulating uterine contrac

163 is often a fine line differentiating sudden infant death syndrome and infanticide.

164 erized SCN5A variants associated with sudden infant death syndrome and provide further biophysical co

165 tionately affect males, including the sudden infant death syndrome and sleep apnea.

166 mend against it to reduce the risk of sudden infant death syndrome and suffocation deaths.

167 genital abnormalities, accidents, and sudden infant death syndrome are predicted to continue increasi

168 We classified sudden infant death syndrome as a separate cause of death consi

169 12 months or younger; most (52%) had sudden infant death syndrome as the reported cause of death.

170 evaluation and prevention of possible Sudden Infant Death Syndrome cases were published this year, wi

171 and an apparent relationship to some sudden infant death syndrome cases.

172 rowning, respiratory arrest, and near sudden infant death syndrome cause significant mortality and mo

173 riants were identified in a Norwegian sudden infant death syndrome cohort (n=201).

174 Sudden infant death syndrome contributed to 5% of deaths in chi

175 Pediatrics in 1992, the incidence of Sudden infant death syndrome has decreased by almost 50%.

176 in a large population-based cohort of sudden infant death syndrome has elucidated mutations in 5-10%

177 t with the observed 50% male bias for Sudden Infant Death Syndrome in humans.

178 here has been a dramatic reduction in sudden infant death syndrome in this country.

179 es were used to locate 230 parents of sudden infant death syndrome infants who died in Southern Calif

180 andardized and systematic approach to sudden infant death syndrome is also reviewed.

181 ns: While its exact cause is unknown, sudden infant death syndrome is believed to be multifactorial,

182 The current literature on sudden infant death syndrome is reviewed in this section by Ste

183 strongly supports the hypothesis that sudden infant death syndrome is the result of dysregulation of

184 rd ratios for total, respiratory, and sudden infant death syndrome mortality per-interquartile-range

185 covariates to total, respiratory, and sudden infant death syndrome mortality.

186 iation between infant bed sharing and sudden infant death syndrome or unintentional sleep-related dea

187 Controversy regarding the etiology of sudden infant death syndrome persists, and risk factors are rev

188 of Pediatrics new recommendations on sudden infant death syndrome prevention.

189 al "Back to Sleep" campaign to reduce sudden infant death syndrome provides an opportunity to study w

190 Given the risk of sudden infant death syndrome related to bedsharing, multipronge

191 Sudden infant death syndrome remains the leading cause of death

192 Despite a putative diagnostic shift, sudden infant death syndrome remains the most common cause of d

193 apparent life-threatening events and sudden infant death syndrome remains to be explored further, bu

194 Sudden infant death syndrome reports after IPV were consistent

195 athogenic cause for a small subset of sudden infant death syndrome via a secondary loss-of-function m

196 between meteorologic temperature and sudden infant death syndrome was investigated in the 1982-1983

197 Apgar score at 5 min and the risk of sudden infant death syndrome was noted at any gestational age (

198 identified 18 families with two SIDS(sudden infant death syndrome) deaths and two families with prob

199 ion continues to be a risk factor for sudden infant death syndrome, although immunizations may not be

200 ty, apparent life-threatening events, sudden infant death syndrome, and central hypoventilation are r

201 sudden unexpected death in epilepsy, sudden infant death syndrome, and sleep apnea.

202 e sudden unexplained death, including sudden infant death syndrome, can be caused by cardiac channelo

203 ent in a variety of disorders such as sudden infant death syndrome, depression, and anxiety.

204 hy, and now encompasses more cases of sudden infant death syndrome, fulminant hepatic failure, and se

205 thought to underlie the aetiology of sudden infant death syndrome, including: (1) a vulnerable neona

206 ventable-cause mortality in children (sudden infant death syndrome, unintentional injury, and homicid

207 k factor for late fetal death and the sudden infant death syndrome, we investigated cardiorespiratory

208 rovide insight into the mechanisms of sudden infant death syndrome, which has been associated with ab

209 phenotype in mice, with relevance to Sudden Infant Death Syndrome.

210 ve AR capacity has been implicated in Sudden Infant Death Syndrome.

211 nifestations include fetal hydrops or sudden infant death syndrome.

212 have been identified in some cases of sudden infant death syndrome.

213 ome cases of sudden unexplained death/sudden infant death syndrome.

214 me, familial atrial fibrillation, and sudden infant death syndrome.

215 pathophysiology and risk factors for sudden infant death syndrome.

216 s one of the biggest risk factors for sudden infant death syndrome.

217 rs that may contribute to the risk of Sudden Infant Death Syndrome.

218 c 5-HT brainstem disorders, including sudden infant death syndrome.

219 KCNH2 gain of function mutations and sudden infant death syndrome.

220 associated with an increased risk of sudden infant death syndrome.

221 sturbances culminating in events like Sudden Infant Death Syndrome.

222 mptoms in children and be a factor in sudden infant death syndrome.

223 veness of these campaigns in reducing sudden infant death syndrome.

224 ged QT electrocardiogram interval and sudden infant death syndrome.

225 reatest influence on the reduction of sudden infant death syndrome.

226 find other modifiable risk factors of sudden infant death syndrome.

227 the relationship between smoking and sudden infant death syndrome.

228 ged QT electrocardiogram interval and sudden infant death syndrome.

229 rt rates and cardiac function such as sudden infant death syndrome.

230 nts who are labeled as having died of sudden infant death syndrome.

231 associated with an increased risk of sudden infant death syndrome.

232 normalities thought to be involved in sudden infant death syndrome.

233 sto-pathological findings reported in sudden infant death syndrome.

234 as of prematurity, Rett syndrome, and sudden infant death syndrome.

235 recently long-QT syndrome (LQTS) and sudden infant death syndrome.

236 accine would reduce the maternal anaemia and infant deaths that are associated with malaria in pregna

237 the physiologic mechanisms that cause sudden infant death, the mainstay of risk reduction continues t

238 enetic diseases are a leading cause of early infant death, to our knowledge, the contribution of sing

239 Vaccine effectiveness against infant deaths was estimated at 95% (95% confidence inter

240 and social risk factors, and cause-specific infant death were ascertained via linkage between nation

241 Adjusted hazard ratios for infant deaths were 145 (95% CI 141, 149) and 9.8 (95% CI

242 tal deaths, 11,294 neonatal deaths, and 4983 infant deaths were included.

243 m 1997 to 2014, 970,583 live births and 6510 infant deaths were registered.

244 data, developmental outcomes, including four infant deaths, were documented for 888 of (88%) 1010 sin

245 e identified two cases of sudden unexplained infant death where no lung LCAD antigen was detectable.

246 ty loci for a rare monogenic disease (sudden infant death with dysgenesis of the testes syndrome), a

247 ght (m)(2)) of women who had stillbirths and infant deaths with those of their sisters or of populati

248 The possibility of a second sudden infant death within a family is discussed.

249 atrophy (SMA) is a major inherited cause of infant death worldwide.