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1 ram per day for 6 months in the treatment of infantile hemangioma.
2 clinical evaluation and treatment of CMN and infantile hemangioma.
3 guide future standard-of-care treatment for infantile hemangioma.
4 ng from systemic use of propranolol to treat infantile hemangioma.
5 lockers have been used to successfully treat infantile hemangioma.
6 c potential of stem cells derived from human infantile hemangioma.
7 -1 and merosin, immunodiagnostic markers for infantile hemangioma.
8 ar malformations, juvenile angiofibromas and infantile hemangiomas.
9 treatment of benign vascular tumors such as infantile hemangiomas.
10 romise for the treatment of certain types of infantile hemangiomas.
11 ine therapy for the treatment of complicated infantile hemangiomas.
12 fantile hemangiomas and treating complicated infantile hemangiomas.
13 sm of action of propranolol on regression of infantile hemangiomas.
14 nsible for the formation and rapid growth of infantile hemangiomas.
15 Objectives: To describe the sequelae left by infantile hemangiomas after natural involution and to id
16 opranolol has been used to treat complicated infantile hemangiomas, although data from randomized, co
17 tifies a stem cell as the cellular origin of infantile hemangioma and describes for what we believe i
18 s in a previously described in vivo model of infantile hemangioma and in cultured hemangioma-derived
19 es of these lesions are shared with those of infantile hemangioma and tufted angioma of children, but
20 is and psoriasis, use of topical timolol for infantile hemangiomas and bone marrow transplantation fo
21 e treatment of atopic dermatitis, psoriasis, infantile hemangiomas and dystrophic epidermolysis bullo
22 treatments for atopic dermatitis, psoriasis, infantile hemangiomas and dystrophic epidermolysis bullo
23 escribing clinical syndromes associated with infantile hemangiomas and treating complicated infantile
24 as wet age-related macular degeneration and infantile hemangioma are more common in light-skinned in
25 hat the transcriptomes of human placenta and infantile hemangioma are sufficiently similar to suggest
34 Corticosteroids are commonly used to treat infantile hemangioma, but the mechanism of action of thi
35 ccurred in redefining the clinical course of infantile hemangiomas, describing clinical syndromes ass
36 Little is known about the pathogenesis of infantile hemangiomas despite the fact that they are rel
38 (3 mg/kg/day) in the treatment of periocular infantile hemangioma (IH) based on clinical and radiolog
42 (HemSCs) are multipotent cells isolated from infantile hemangioma (IH), which form hemangioma-like le
46 nign vascular tumors that differ from common infantile hemangiomas in that they grow in utero and are
53 evolutionized the treatment of proliferating infantile hemangiomas, laser and/or surgical excision ar
54 common, predominantly benign complication in infantile hemangioma, little is known about the prognosi
55 gests that the unique self-limited growth of infantile hemangioma may, in fact, mirror the lifetime o
56 e pathogenic C482R VEGFR-2 mutant, linked to infantile hemangioma, promotes ligand-independent signal
57 220 parents/caregivers completed the 35-item Infantile Hemangioma Quality-of-Life (IH-QoL) instrument
58 hat EndMT induced by M1 macrophages promotes infantile hemangioma regression and may lead to novel th
61 etrospective cohort study of images from 187 infantile hemangiomas that had not received systemic tre
63 he ECs of mature human placental vessels and infantile hemangiomas, the most common tumor of infancy
64 cells recapitulated the unique evolution of infantile hemangioma--the formation of blood vessels fol
65 udies have shown a promising new therapy for infantile hemangioma using nonselective beta-blockers, i
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