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1 es in the host immune response to Leishmania infantum.
2 immunity mediated by prior infection with L. infantum.
3 n against visceral infection with Leishmania infantum.
4 irst linked to the clinical syndrome roseola infantum.
5 tment murine model for acute infection by L. infantum.
6 14alpha-demethylase (CYP51) from Leishmania infantum.
7 eral leishmaniasis (VL) caused by Leishmania infantum.
8 tabase of the parasitic protozoan Leishmania infantum.
9 murine infection model employing Leishmania infantum.
10 positive patients coinfected with Leishmania infantum.
11 pesvirus 6 is the causative agent of roseola infantum, a generally benign rash illness of infants.
12 iasis is caused by infection with Leishmania infantum, a Protist parasite transmitted by blood-feedin
14 ed tandem repeat (TR) proteins of Leishmania infantum and an evaluation of VL patient antibody respon
18 ies of Leishmania, L. tarentolae, Leishmania infantum, and L. major, produced hypersensitivity to bot
19 ful development of an SPR sensor for anti-L. infantum antibodies detection in short time, showing a g
22 gly indicate that hamsters infected with Le. infantum become significantly more attractive to a great
23 ntly, this analysis suggests that Leishmania infantum chagasi alters the expression profile of certai
25 si-containing dermal leukocytes and total L. infantum chagasi parasites in draining lymph nodes were
26 noculation site, we introduced metacyclic L. infantum chagasi promastigotes intradermally into BALB/c
28 n abundance during development of Leishmania infantum chagasi to a virulent metacyclic stage, as did
29 e dermis even late after inoculation, and L. infantum chagasi trafficked through neutrophils in both
30 role of HO-1 in the infection by Leishmania infantum chagasi, the causative agent of VL cases in Bra
36 lpis sand flies, known vectors of Leishmania infantum/chagasi parasites, in a Brazilian city endemic
37 oplasma gondii, Neospora caninum, Leishmania infantum, Cryptosporidium parvum, or canine DNA under an
40 ination experiments employing the Leishmania infantum D-13 (p80) antigen, significantly higher levels
41 L. major and a visceral strain of Leishmania infantum, each bearing a different drug-resistant marker
42 roM semipermeable membrane suggested that L. infantum-exposed keratinocytes release soluble factors t
45 a protein of unknown function in Leishmania infantum (hypothetical C1 protein) and specific antibodi
48 agent of zoonotic leishmaniasis, Leishmania infantum, in France and Iberia, and provides a rare case
54 SE induced significant protection against L. infantum infection, with reductions in parasite loads of
59 es datasets from Leishmania braziliensis, L. infantum, L. major, L. tarentolae, Trypanosoma brucei an
60 15 were screened in vitro against Leishmania infantum , Leishmania braziliensis , Leishmania guyanens
61 e vector-borne protozoan parasite Leishmania infantum, mitochondrial peroxiredoxin (Prx) exerts intri
62 s sorted on contact with visceral Leishmania infantum on a susceptible mice model evaluating the subs
64 39 in HIV-negative patients infected with L. infantum or L. chagasi declined during treatment with me
66 rtalized human keratinocytes with Leishmania infantum or Leishmania major, which cause visceral or cu
68 ts suggest that TR regions from the novel L. infantum proteins identified in this study are immunodom
71 rypanosoma brucei, Trypanosoma cruzi, and L. infantum) suggests that substrate preferences of plant-
73 ondrial chaperone reservoir, which allows L. infantum to deal successfully with protein unfolding con
74 if the odour of hamsters, infected with Le. infantum, was more attractive than the odour of the same
77 y gut populations of both L. mexicana and L. infantum were significantly reduced in caspar-depleted f
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