ライフサイエンスコーパス: infect

1 populations of NIKS (uninfected) and NIKS16 (infected).

2 As of August 11, 2016, the virus has infected 1,791 patients, with a mortality rate of 35.6%.

3 ommon intracellular infection on the planet, infects 40% of insects as well as nematodes, isopods and

4 ion of B cells.IMPORTANCE Gammaherpesviruses infect a majority of the human population and are associ

5 alating study of BMS-936559, including HIV-1-infected adults aged >18 to <70 years on suppressive ant

6 warranted to screen all sexually active HIV-infected adults for syphilis, chlamydia, and gonorrhea.

7 As a result, most HIV-infected adults in the United States are >50 years of ag

8 than non-host individuals that do not become infected, although high tumour loads lead to high mortal

9 LP avian H7 influenza A viruses are able to infect and cause disease in mammals without prior adapta

10 udy of matrix turnover in HIV type 1 (HIV-1)-infected and -uninfected TB patients and controls, and a

11 MMP activity differed between HIV-1-infected and -uninfected TB patients and corresponded wi

12 Efficient removal of infected and dying neutrophils is required to protect th

13 weakness in the bowel wall, which can become infected and inflamed causing diverticulitis, with poten

14 Similarly, hSSR2-containing OV vaccinia infected and lysed cancer cells.

15 lones segregated based on responses to HIV-1-infected and peptide-loaded target cells.

16 e classified into two groups: those who were infected and those who were not, according to circulatin

17 rotein were found to be internalized in both infected and transfected cells after the addition of eit

18 Apoptosis was observed in both infected and uninfected bystander cortical neurons, sugg

19 family cytokines was performed in H. pylori-infected and uninfected gastric biopsy specimens.

20 azinamide resistance both in vitro and in an infected animal model.

21 AVV and treated with NP siRNA-LNP, with MARV-infected animals beginning treatment four or five days a

22 espiratory status of mitochondria from prion-infected animals is unknown.

23 t four or five days after infection and RAVV-infected animals starting treatment three or six days af

24 y from MRV infection and led to lethality in infected animals, whereas B cell-deficient mice showed C

25 not previously been applied in mycobacteria-infected animals.

26 med in HEV gt1, but not in Hepatitis B Virus infected animals.

27 peripheral blood mononuclear cells from HIV-infected ART-treated individuals in response to M. tuber

28 ulosis occurred in six (<1%) of 795 contacts infected at baseline versus four (<1%) of 518 contacts u

29 the isolation facility, 3 (3%) of whom were infected at baseline, before treatment initiation.

30 ls have been reported to respond against EBV-infected B cells in the lytic cycle and to control the v

31 characteristics of women giving birth to an infected baby after primary and nonprimary infection are

32 of AnxA1 was investigated in L. braziliensis-infected BALB/c mice.

33 Virus-infected bees had greater expression of genes involved i

34 We infected boa constrictors and ball pythons with purified

35 Mice were infected by means of oral gavage with 200 stage 3 H poly

36 anation for the behavioural changes in hosts infected by rabies virus.

37 L functionalities, yet they are unable to be infected by SIV.

38 ich is suspected of extracellular release by infected CD4+ T cells on protein quality control and aut

39 remain transcriptionally silent in latently infected CD4+ T cells.

40 ly upregulated in human papillomavirus (HPV)-infected cell lines and tissues.

41 To counter premature death of a virus-infected cell, poxviruses use a range of different molec

42 ells and restoring filament formation at the infected-cell surface.

43 (TSC2) inhibits lipophagy induction in DENV-infected cells and decreases viral replication.

44 ed with mycobacteria efficiently killing the infected cells and decreasing survival of mycobacteria.

45 virus expresses more VP26 fusion protein in infected cells and incorporates more VP26 fusion protein

46 cell survival mechanisms is unique to virus-infected cells and relies on regulation of MCL-1 mitocho

47 ficiency with which virus particles bud from infected cells and restoring filament formation at the i

48 om uninfected cells activate latent HIV-1 in infected cells and that true transcriptional latency may

49 HIV-1 reverse transcription products within infected cells are not well understood.

50 herapeutic strategy for eliminating latently infected cells before haematopoietic stem cell transplan

51 Superinfection of latently infected cells by productive virus could similarly remov

52 s to reactivate the latent reservoir so that infected cells can be recognized and targeted, with the

53 oplasmic virus assembly compartment (vAC) of infected cells co-localizing with virus tegument protein

54 Thus the increase of miRNP stability in Ld-infected cells curtails production of proinflammatory cy

55 ing and monitoring of HIV genomic DNA within infected cells during cytoplasmic transit, nuclear impor

56 nt cellular cytotoxicity (ADCC) to eliminate infected cells following reactivation from HIV-1 latency

57 We tested this approach in HIV-infected cells grown in the lab and in animal infections

58 entiviral and gammaretroviral integration in infected cells have been discovered, but the factors tha

59 recapitulate the characteristics of latently infected cells in vivo is crucial to identifying and dev

60 (CRISPR)-enzymatically inactive Cas9 in MVM-infected cells increased both cyclin B1 protein and RNA

61 Thus, the proliferation of HIV-1-infected cells may play a role in viral persistence, but

62 d phosphoantigens (pAgs) that are present in infected cells or accumulate intracellularly in certain

63 y ET at 5-days post-infection, whereas HIV-1-infected cells surrounded by pools of free virions were

64 Short exposure of HCV-infected cells to daclatasvir reduced viral assembly and

65 uses have evolved multiple ways to adapt the infected cells to their needs, but knowledge about these

66 High titers of ADCC-Abs against H7N9 virus-infected cells were detected in sera from adults and chi

67 Free virions and infected cells were not readily detectable by ET at 5-da

68 ing immunotherapeutics to clear persistently infected cells will soon allow measurable clinical advan

69 sed on the gene expression levels in the non-infected cells, and demonstrated reasonable performance

70 umulate in a long-lasting manner in Shigella-infected cells, causing subsequent formation of covalent

71 fragment with the highest copy number in the infected cells, is derived from Salmonella 5'-leader of

72 n to trigger HIV transcription from latently infected cells, via a CDK9/HMBA inducible protein-1 depe

73 tion, replication, and genome maintenance in infected cells.

74 , LMP1 blocks IRF5-mediated apoptosis in EBV-infected cells.

75 nctional antibodies that can eliminate HIV-1-infected cells.

76 s and induced KSHV reactivation in naturally infected cells.

77 does not eliminate the reservoir of latently infected cells.

78 estored CD4 expression to the surface of HIV-infected cells.

79 thering, also shows reduced expression in Ld-infected cells.

80 y important to efforts to eradicate latently infected cells.

81 ted and dramatically reduced in level in the infected cells.

82 ent from transmission electron microscopy of infected cells.

83 ed the fates of multiple viral components in infected cells.

84 ment for detecting exceedingly rare latently infected cells.

85 lipophagy, but not basal autophagy, in DENV-infected cells.

86 s triggers both apoptosis and necroptosis in infected cells; however, encoded inhibitors of caspase-8

87 Eimeria species that infect chickens are most significant, with Eimeria tenel

88 Infected children and adults present with a range of med

89 Among perinatally HIV-infected children born in 2002-2013, 836 (63.0%) of the

90 l, we enrolled virologically suppressed, HIV-infected children from five hospital clinics in Uganda,

91 ions, but its role on CD4 T cells and in HIV-infected children is poorly understood.

92 y single-tablet regimen is available for HIV-infected children under 12 years.

93 Infected children with a better long-term outcome had hi

94 y B cell mobilization and recruitment to the infected CNS, while delayed accumulation of virus-specif

95 144 with severe influenza in three influenza-infected cohorts characterized by different levels of in

96 sporozoites isolated from salivary glands of infected Colombian mosquitoes.

97 lless viruses of the family Tectiviridae can infect commensal and pathogenic Gram-positive and Gram-n

98 Naturally diseased and laboratory infected coral systematically exhibited fragmented fluor

99 cells exposed to exosomes derived from HIV-1 infected DCs.

100 nonuclear cells in six samples from four HIV-infected donors (one with viremia and not on ART and thr

101 ly of macrophages and neutrophils infiltrate infected DRGs and account for the development of herpeti

102 Although T4 was more effective than PEf1 in infecting E. coli K-12 in pure cultures, PEf1 was 20-fol

103 ntrast, if female and male partners are both infected, embryos are viable.

104 Each Acanthamoeba isolate was used to infect EpiCorneal cells, a 3-dimensional human corneal t

105 ctor mechanism against Plasmodium falciparum-infected erythrocytes (IE); however, current phagocytosi

106 hrocyte membranes, preventing the rupture of infected erythrocytes but not parasitophorous vacuoles,

107 stimated that nearly 1 million people become infected every day with any of four curable sexually tra

108 ndothelial cells (a target of MAV-1 in vivo) infected ex vivo with MAV-1 had no difference in activit

109 at ZIKV, when produced from mammalian cells, infects fetal endothelial cells much more efficiently th

110 superfamily homolog in Drosophila, Coxiella-infected flies exhibit reduced mortality from infection.

111 ntaining vsRNAs, purified from haemolymph of infected flies, confer passive protection against virus

112 HIV-infected frail men (n = 155) were matched to nonfrail, H

113 that facilitate direct analysis of a single infecting genome in a sterile blood specimen are availab

114 is of trigeminal ganglia from latently HSV-1-infected, glutamine-treated WT mice showed upregulation

115 On post operative day 6, an infected haematoma and an area of proximal skin necrosis

116 preferred target cells for this virus in the infected host.

117 selective advantage in the immune systems of infected hosts to recall of memory B cells that recogniz

118 e chemicals identified from the headspace of infected hosts, 3-Methyl-2-buten-1-ol (prenol) and 3-Hyd

119 reases vector attraction and reproduction on infected hosts.

120 s induces drastic behaviour modifications in infected hosts.

121 th the behavioral changes of IJs towards the infected hosts.

122 ESRD than those who were nonchronically HCV-infected (HR, 2.11, 95% CI 1.16-3.86, and HR, 3.06, 95%

123 ir expression in cell culture models and HCV-infected human livers.

124 reased chemotaxis of both uninfected and HIV-infected human monocytes, suggesting a role for sPrP(c)

125 gic and rheologic alterations in P cynomolgi-infected human reticulocytes that are strikingly similar

126 tomic and quantitative proteomic analyses of infected human, primary, neuronal stem and monocytic cel

127 a novel subtype of HCV genotype 6, and a co-infecting human pegivirus.

128 WNV were produced in insect cells, they also infected HUVECs in an AXL-dependent manner.

129 M. tuberculosis-infected IL-21R KO mice had enhanced bacterial burden an

130 d a very different inflammatory profile from infected Il1r1(-/-) and Pycard(-/-) mice.

131 99.6%, NAAT sensitivity of 100% for infants infected in pregnancy or at least 4 weeks prior to testi

132 ld-living bonobos are endemically Plasmodium infected in the eastern-most part of their range.

133 of E. histolytica is the cyst, with a single infected individual passing up to 45 million cysts per d

134 terferon for treatment of acute HCV in HIV-1 infected individuals (SWIFT-C) is an open-label, 2-cohor

135 py (ART) suppresses viral replication in HIV-infected individuals but does not eliminate the reservoi

136 s collected from 70 virally suppressed HIV-1-infected individuals from Rakai District, Uganda, who ha

137 y in which 15 virologically suppressed HIV-1-infected individuals on antiretroviral therapy received

138 nt phagocytosis assays use IE collected from infected individuals or from in vitro cultures of P. fal

139 Lymph node cells from untreated HIV-infected individuals revealed that TFR cells harbored th

140 and potency of NAb responses in 98 CRF07_BC-infected individuals using a large, multi-subtype panel

141 al blood mononuclear cells isolated from HIV-infected individuals with suppressed infection.

142 tes to loss of immune control of LTBI in HIV-infected individuals, although the precise mechanisms wh

143 fic CD4 T cells was markedly impaired in HIV-infected individuals, compared with HIV-uninfected indiv

144 be large benefits to vaccinating previously infected individuals.

145 -restricted cellular immune responses in HIV-infected individuals.

146 at Cx43 is dysregulated in the hearts of HIV-infected individuals.

147 firmatory testing, LE was 26.2 years for HIV-infected infants and 61.4 years for all HIV-exposed infa

148 exposed infants; clinical outcomes for truly infected infants did not differ by strategy.

149 se leading to neurodevelopmental deficits in infected infants remain undefined, but postulated pathwa

150 spiratory distress were reported in 11 (29%) infected infants.

151 Some studies detected infected intestinal immune cells (8-12) , other studies

152 usually requires surgical replacement of the infected joint and weeks of antibiotic therapy, due to t

153 ever, we recently established that HIV-1 can infect kidney transplant epithelial cells in the absence

154 or alpha (TNF-alpha)/interleukin-6 (IL-6) in infected kidneys.

155 elerated metamorphosis increased survival in infected larvae.

156 Mice with high levels of viremia had HBV-infected liver progenitor cells.

157 We find the HCV-infected liver to have a pattern of acquisition of DNA m

158 pigenetic and transcriptional changes in HCV-infected livers in comparison with control, uninfected l

159 ng this tool for Yersinia pseudotuberculosis-infected lymphatic tissues, we revealed numerous alterat

160 eplication was also found in EBV/KSHV dually infected lymphoproliferative disorders in humans.

161 nriched leukocyte populations from SIVmac251-infected macaques with or without CD8(+) lymphocyte depl

162 cation in cell culture and in experimentally infected macaques.

163 Finally, selective killing of Mtb-infected macrophages and subsequent bacterial release en

164 rial growth only, or both intracellularly on infected macrophages as well as extracellularly on bacte

165 ntal for survival of the parasite within the infected macrophages.

166 To investigate these differences, we infected male and female mice of different age groups wi

167 Lifetime CVD risk was 64.8% for HIV-infected males compared to 54.8% for males in the US gen

168 gocytosis, and destruction of microorganisms infecting mammals, their implication in plant virus reco

169 a pipientis is an intracellular endosymbiont infecting many arthropods and filarial nematodes.

170 the extent of proviral integration in HIV-1-infected MDDCs was unaffected by the absence of Vpr, the

171 a use is a risk factor for CV disease in HIV-infected men ages 40-60, independent of tobacco smoking

172 ar year, and antiretroviral therapy use (HIV-infected men only).

173 an increased risk to female partners of HIV-infected men resuming sex early after male circumcision.

174 cytes, and demonstrated decreased ability to infect mice.

175 was up-regulated in the brains of influenza-infected mice and was elevated in cerebrospinal fluid of

176 d in enhanced L. pneumophila in the lungs of infected mice but not within cultured host cells, which

177 red with wild-type infected mice, CD151-null infected mice exhibited a significant reduction in virus

178 In infected mice that overexpress plasminogen activator inh

179 CD8(+) T cells from infected mice were polyfunctional and mediated cytotoxic

180 As compared with wild-type infected mice, CD151-null infected mice exhibited a sign

181 al slowing of parasite maturation in acutely infected mice, extending the life cycle from 24 h to 40

182 rmalized neurodevelopmental abnormalities in infected mice, providing evidence that virus-induced inf

183 ssion profiling data from hearts of T. cruzi infected mice.

184 DAT and rescued antibiotic-treated influenza-infected mice.

185 Moreover, infecting mice with the LRV1-cured Leishmania guyanensis

186 related to exposing a human subject's arm to infected mosquitoes in the standard arm-in-cage assay.

187 hether repellents are effective against ZIKV-infected mosquitoes, in part because of the ethical conc

188 rearing infrastructure specific to Wolbachia-infected mosquitoes.

189 The attenuated 10-del ZIKV was incapable of infecting mosquitoes after oral feeding of spiked-blood

190 ESAT-6, IL-6 and phosphorylated-STAT3 in Mtb-infected mouse lungs.

191 trong binding to platelets within thrombi in infected mouse lungs.

192 Mycobacteriophages are viruses that infect mycobacterial hosts including Mycobacterium tuber

193 cy, and one viral gene expressed by latently infected myeloid cells is US28.

194 men (n = 155) were matched to nonfrail, HIV-infected (n = 141) and HIV-uninfected (n = 150) men by a

195 Host-generalist pathogens sporadically infect naive hosts, potentially triggering epizootics.

196 Human astroviruses (HAstVs) infect nearly every person in the world during childhood

197 POCT detected all 30 HIV-infected neonates (sensitivity 100%; 95% CI 88.4-100) wi

198 In productively infected neuronal cultures, epinephrine treatment signif

199 id not inactivate EBOV in blood samples from infected NHPs.

200 tantly, exogenous superoxide addition to CB3-infected NOD.Ncf1(m1J) bone marrow-derived macrophages r

201 We infected nonhuman primate cell cultures and then crab-ea

202 I. scapularis nymphal ticks, B. burgdorferi-infected nymphal ticks and B. mayonii-infected nymphal t

203 orferi-infected nymphal ticks and B. mayonii-infected nymphal ticks by measuring metabolism every 24

204 ipants from a TB-endemic setting, either HIV-infected or uninfected and with latent or active TB (aTB

205 We recruited 77 HIV-infected participants (37 PPI+ and 40 PPI-) and 20 HIV-u

206 HIV-infected participants enrolled in a study of cerebrospin

207 nscription from pre-formed RNPs deposited by infecting particles is unaffected.

208 tor performs the best in identifying viruses infecting particular hosts.

209 type analyses in a cohort of HIV-1 subtype C-infected patients (n = 168), together with site-directed

210 ized trial of behavioral weight loss for HIV-infected patients (n = 40).

211 ripheral blood B cells of 30 MC-negative HCV-infected patients and 15 healthy controls revealed that

212 (representative of Italian hepatitis C virus-infected patients in care).

213 ls accumulate in the blood of aviremic HIV-1-infected patients on long-term antiretroviral therapy, a

214 le and SVR12 rates of 96.7% among HIV/HCV co-infected patients participating in an Italian compassion

215 HIV-infected patients were significantly less accurate on th

216 e considered in genotype 1 hepatitis C virus-infected patients who are treatment-naive, do not have c

217 Among HIV-infected patients with advanced immunosuppression, enhan

218 xacerbated neurocognitive dysfunction in HIV-infected patients.

219 ents, particularly among HIV and hepatitis C infected people among whom cardiovascular disease risk i

220 protein was detectable only in Sendai virus-infected PHHs from individuals with the dG allele, where

221 ilitates WNV neuroinvasion by recruiting WNV-infected PMNs into the brain.

222 s in a real-life cohort of hepatitis C virus-infected policy 1, "universal," treat all patients, rega

223 phoblast within the colonies, quickly become infected, produce infectious virus and undergo lysis wit

224 TDR among human immunodeficiency virus (HIV)-infected PWUD, and assessed its impacts on first-line AR

225 om heterozygous (ARQ/VRQ or ARH/ARQ) scrapie-infected Rasa Aragonesa sheep was analyzed using this MR

226 enine pathway in brains of newborn and adult infected rats and cultured astroglioma cells, shunting t

227 The median frequency of latently infected rCD4 cells in this Ugandan cohort was 0.36 infe

228 nt increased fluorescence signal at the MRSA infected site.

229 eumonia cases were due to pneumococci in HIV-infected South African adults.

230 re permissive, suggesting that enteroviruses infect specific cell populations in the human intestine.

231 providing faster detection of drug-resistant infecting strains and to help inform therapeutic managem

232 1.83-7.07) compared with nonchronically HCV-infected subjects.

233 ly, we also demonstrate that a norovirus can infect T cells, a previously unrecognized target, in vit

234 A long-lived reservoir of latently infected T cells prevents antiretroviral therapy from el

235 ols, and a prospective cohort study of HIV-1-infected TB patients at risk of TB immune reconstitution

236 Different influenza A viruses (IAVs) infect the same cell in a host, and can subsequently pro

237 Here we show that ZIKV infects the subventricular zone in human fetal brain tis

238 inants or the evaluation of therapeutics, we infected them with a green fluorescent protein-expressin

239 (80%) were HCV treatment-naive, and 84% were infected through perinatal transmission.

240 y the age of three years and then repeatedly infects throughout life; this it does despite relatively

241 xodes ticks, mice fed upon by the DeltacheY2-infected ticks did not develop a persistent infection in

242 was gradually decreased with the increase of infected time in CD8(+) T cells, but not with that in NK

243 he importance of multiscale imaging of HIV-1-infected tissues and are adaptable to other animal model

244 we demonstrate the subpassage of prions from infected to naive astrocyte cultures, indicating the gen

245 the exchange of viral or prion proteins from infected to naive cells, it is not clear whether the vir

246 virus, inhibition of HIV-1 transmission from infected to uninfected CD4(+) T cells through virologica

247 iler's murine encephalomyelitis virus (TMEV)-infected transgenic FVB mice that express the D(b) class

248 Medical records of the donor and infected transplant recipients were reviewed for clinica

249 Based on the brain cytokine levels, MAV-1-infected Unc93b1(-/-) mice had a very different inflamma

250 ntly more IL-17 than gammadelta T cells from infected unprimed mice.

251 ditional binary classification of apparently infected versus uninfected to a probability-based interp

252 ANCE Poxviruses comprise a large family that infect vertebrates and invertebrates, cause disease in b

253 It infects virtually all children by the age of three years

254 t with a targeted processing of persistently infecting virus genomes.

255 mber of one of the largest families of plant-infecting viruses, increases vector attraction and repro

256 Infected WBCs can also function as 'Trojan horses' and c

257 s 1 (HBoV1) is an autonomous parvovirus that infects well-differentiated primary human airway epithel

258 Studies were performed using C57BL/6 mice infected with a moderately virulent strain of Cryptococc

259 KO), and C57BL/6 wild-type mice were orally infected with A. actinomycetemcomitans and analyzed for

260 e in IL-17C mRNA and protein levels in cells infected with cagA-positive infections compared to cells

261 Not all women infected with chlamydiae develop upper genital tract dis

262 SE disease in offspring born to muntjac dams infected with chronic wasting disease (CWD) (1).

263 ing adaptive immune responses in individuals infected with clade C, which is responsible for the majo

264 Mice infected with covR-deficient S. agalactiae produced less

265 Protein-deficient mice were infected with Cryptosporidium parvum oocysts for 6-13 da

266 h cagA-positive infections compared to cells infected with either cagA-negative or cag pathogenicity

267 lis were depleted of mucus, and in vivo mice infected with G. duodenalis had a thinner mucous layer a

268 tion includes those who either currently are infected with HBV or have had past exposure to HBV.

269 Thirty-two chronic HCV patients infected with HCV genotypes 1, 3, and 4 received a singl

270 duals from 15 countries who were chronically infected with HCV genotypes 1-6 (HCV RNA >/=10 000 IU/mL

271 Patients co-infected with hepatitis C virus (HCV) and human immunode

272 Patients who are chronically infected with hepatitis C virus (HCV) and who do not hav

273 e found 80% of 29382 young persons currently infected with hepatitis C virus lived >10 miles from a s

274 ed to enrol four controls for every case not infected with HIV and six controls for every case with H

275 ry human cord blood-derived MCs (CBMCs) were infected with HRV or UV-irradiated HRV at increasing mul

276 ansformed lymphocytic cell lines chronically infected with HTLV-1, particularly the MT-2 cell line, w

277 intrapulmonary KGF before isolation and then infected with IAV ex vivo exhibited the same temporal pa

278 pheral blood mononuclear cells from patients infected with L. braziliensis and reduced IL-1beta level

279 Primary NVRMs were infected with lentivirus containing ChR2 or YFP gene and

280 Sixteen rhesus monkeys were lethally infected with MARV or RAVV and treated with NP siRNA-LNP

281 cterial burdens and kidney pathology in mice infected with MRSA.

282 roaches to classify susceptibility to TB, we infected with MTB dendritic cells (DCs) from putatively

283 vitro, CD44TA-SMP accumulated in macrophages infected with mycobacteria efficiently killing the infec

284 on of European badgers Meles meles naturally infected with Mycobacterium bovis.

285 ronin-60 (cpn60) showed that the plants were infected with phytoplasma subgroup16SrXIII-(A/I)I (SbGP/

286 CB17 SCID mice infected with R. typhi(GFPuv) succumb to the infection w

287 D4(+) T cell responses in healthy volunteers infected with rDEN2Delta30.

288 2,588 (65.5%) infants with severe LRTI were infected with RSV.

289 +) splenic T cells compared to those of mice infected with SL3261/cyto.

290 and specific tetramers, we showed that mice infected with the SL3261/surf or SL3261/sec strain gener

291 cell cycle progression are arrested in cells infected with vaccinia virus, but mass fluctuations cont

292 ed less proinflammatory cytokines than those infected with wild-type 874391.

293 n with kinetics similar to those for animals infected with wild-type R. typhi and develop comparable

294 was investigated by comparing melanoma cells infected with wild-type-like VZV or hyperfusogenic mutan

295 Comparing the transcriptomes of tomato infected with X. gardneri vs. XgDeltaavrHah1 revealed th

296 of universal AC screening offered to all HIV-infected women.

297 the threshold temperature associated with an infected wound.

298 5% confidence interval (CI): 1.27, 2.71; for infected wounds, IRR = 3.04, 95% CI: 1.54, 5.98); exposu

299 fection, IRR = 3.28, 95% CI: 1.95, 5.51; for infected wounds, IRR = 4.96, 95% CI: 2.18, 11.29).

300 cells in lungs compared with M. tuberculosis-infected WT mice.